ライフサイエンスコーパス: cell interaction

1 rises from the head-tail symmetry of cell-to-cell interaction).

2 can directly activate basophils via cell-to-cell interaction.

3 tin and dramatically inhibits exosome-target cell interaction.

4 ntial homo- and heterotypic energies of cell-cell interaction.

5 during reperfusion, suggesting a direct cell-cell interaction.

6 regulated and involves trophoblast-decidual cell interaction.

7 le still capable of initiating cognate APC/T cell interaction.

8 nutrients can affect growth but also a cell-cell interaction.

9 eable, allowing the possibility of PMP-tumor cell interaction.

10 ting the cellular phenotype to alter hepatic cell interactions.

11 st cells is a model system for studying cell-cell interactions.

12 croenvironmental patterns, including cell-to-cell interactions.

13 l responses to exogenous cues depend on cell-cell interactions.

14 tes to Myosin II polarization via local cell-cell interactions.

15 egulator of AJ mechanics and long-range cell-cell interactions.

16 h in studying early virus-mammalian receptor/cell interactions.

17 e ECM as cords, involving heterotypical cell-cell interactions.

18 er the cell cycle phases influence bacterium-cell interactions.

19 s and to identify strategies for robust cell-cell interactions.

20 ma patients, significantly inhibited exosome-cell interactions.

21 mechanism to sharpen the specificity of cell-cell interactions.

22 ent on adjacent cells, thus creating cell to cell interactions.

23 titers, suggesting insulin-specific T- and B-cell interactions.

24 n was higher than that exerted by stromal-MM cell interactions.

25 ronment and in the establishment of T cell-B cell interactions.

26 lfate-binding ligand and mediator of exosome-cell interactions.

27 cellular activities, processes, and cell-to-cell interactions.

28 vel and very efficient insight to individual cell interactions.

29 ility of DLX4 to stimulate tumor-mesothelial cell interactions.

30 the immunological synapse, and other immune cell interactions.

31 d the molecules involved in eosinophil-tumor cell interactions.

32 retention in the lung by reducing MAM-cancer cell interactions.

33 collectively make decisions through cell-to-cell interactions.

34 nscripts, some of which are involved in cell-cell interactions.

35 reased efficiency of immune surveillance and cell interactions.

36 r cells, distinct from any responses to cell-cell interactions.

37 l size is an emergent property of local cell-cell interactions.

38 various biological processes including cell/cell interactions.

39 izosaccharomyces pombe, Zfs1, regulates cell-cell interactions.

40 hyl cycle--and is socially mediated via cell-cell interactions.

41 opment of nonspecific CD22-mediated T cell-B cell interactions.

42 tion, proliferation, tubulogenesis, and cell-cell interactions.

43 on structure and closer interspecies cell-to-cell interactions.

44 irulence factor that functions in early host-cell interactions.

45 ns of tumor cells through stromal cell-tumor cell interactions.

46 ates rearrangement and promotes higher-order cell interactions.

47 pact on understanding other enterovirus-host cell interactions.

48 rom stromal, lymphoid, or antigen presenting cell interactions.

49 nce of single cell analysis when studying NP-cell interactions.

50 er and the fetus, and is thus the epitome of cell interactions.

51 properties are known to affect nanoparticle-cell interactions.

52 nvolving both homotypic and heterotypic cell-cell interactions.

53 ved as signaling molecules that mediate cell-cell interactions.

54 t role in redox signalling and pathogen-host cell interactions.

55 e theoretically calculated limit and cell-to-cell interactions.

56 tabolism as crucial for cancer - endothelial cells interaction.

57 s work highlights the complex nature of cell-cell interactions after injury and introduces perineuria

58 lex network of soluble mediators and cell-to-cell interactions allowing human classically activated (

59 Several molecules implicated in cell interactions also control collective migration, but

60 These cytokines regulate hepatic cell interaction and crosstalk of the various inflammato

61 romotes tumor growth and metastasis via cell-cell interaction and extracellular vesicles.

62 he associated changes in the physics of cell-cell interaction and its impact on the architecture of b

63 vide help to B cells both by direct B cell-T cell interaction and production of IL-21.

64 nd (Dll) pathway, a master regulator of cell-cell interaction and vascular patterning.

65 ignaling, Polycomb repression, innate immune cell interactions and a cluster of zinc finger-encoding

66 ramework allows us to directly quantify cell-cell interactions and biofilm dynamics.

67 Cognate T-B cell interactions and CD40-CD154 costimulation are essen

68 changes in relation to cell attachment, cell-cell interactions and cell viability respectively.

69 engraftment in bone marrow and enhances cell-cell interactions and cytokine-mediated cell migration.

70 ace molecules required for NKT and apoptotic cell interactions and developed suppressive immune funct

71 ssue hierarchies, account for spatiotemporal cell interactions and discover rare cells that drive met

72 are essential for long-lasting cognate Tfh-B cell interactions and efficient selection of low-affinit

73 we investigated in vitro macrophage-myogenic cell interactions and found that Dysf-deficient muscle i

74 , and type IV pili] that likely mediate cell-cell interactions and gut colonization.

75 ding is important in understanding EV71-host cell interactions and has potential impact on understand

76 iprocal, phenotype-dependent melanoma-immune cell interactions and highlights a critical role for mas

77 n, thereby compromising pericyte-endothelial cell interactions and inter-endothelial cell junctions.

78 lation and function of epidermal cell-immune cell interactions and into how components that are class

79 phic factor that may regulate glial-neuronal cell interactions and is a potential therapeutic molecul

80 These glycoconjugates are involved in host-cell interactions and may be associated with the virulen

81 features of IFM myogenesis, this sequence of cell interactions and membrane events and the mechanisti

82 impact of ECM composition, heterotypic cell-cell interactions and patterns of signaling heterogeneit

83 possible linkage between place cell to grid cell interactions and PCA.

84 t of tumours is characterized by heterotypic cell interactions and physiological gradients of nutrien

85 use SEGGA to analyze changes in cell shape, cell interactions and planar polarity during convergent

86 mplex engaged to CD6 during dendritic cell-T cell interactions and provide novel information on the m

87 nections between proteins that regulate cell-cell interactions and provides evidence for a previously

88 nt role in regulating neutrophil-endothelial cell interactions and recruitment to the inflamed lung.

89 umor microenvironment is mediated by cell-to-cell interactions and soluble factors.

90 Both altered pDC-CD24(+)CD38(hi) Breg cell interactions and STAT1-STAT3 activation were normal

91 owing understanding of dynamic tumour-immune cell interactions and the mechanisms by which tumour cel

92 tool for investigation of epithelial-stromal cell interactions and to optimize HLE culture conditions

93 mely immune semaphorins, facilitating immune cell interactions and trafficking.

94 EphA2, a guidance molecule involved in cell-cell interactions and tumorigenesis.

95 of processes, including cell signaling, cell-cell interaction, and cell migration.

96 umor-initiating potential, tumor-endothelial cell interaction, and lung metastasis.

97 ce, but also aberrant T-cell spreading, cell-cell interaction, and migration.

98 new parallel between exosome and virus host cell interaction, and suggest unanticipated routes of su

99 uantitative basis for understanding nanowire-cell interactions, and a means for rapidly assessing mem

100 fferentiation state heterogeneity in pigment cell interactions, and an unanticipated morphogenetic be

101 crucial for tissue structure and mechanics, cell interactions, and other functions in vivo.

102 n cell adhesion molecules to coordinate cell-cell interactions, and to provide navigational cues duri

103 reduced (Fgf8) and factors involved in cell-cell interactions are affected (Jag1, Hes5).

104 Transient cell-cell interactions are formed between T cells and both MF

105 r mechanisms underlying Cryptosporidium-host cell interactions are poorly understood.

106 s; however, the mechanisms mediating exosome-cell interactions are poorly understood.

107 coordinated cell movements and physical cell-cell interactions are required for the formation of thre

108 that a greater number of Ag-dependent CD4 T cell interactions are required to generate Th2 than Th1

109 DC-NK cell interactions are thought to influence the developme

110 the early signaling events initiated by cell-cell interactions are transduced into diverse functional

111 molecular determinants of sporozoite-Kupffer cell interactions are unknown.

112 lling networks, we prepared heterotypic cell-cell interaction arrays using DNA-programmed adhesion.

113 ides a microenvironment that ensures 3D cell-cell interactions as a unique tool to investigate new in

114 r transcriptome database for changes in cell-cell interactions as the characteristic of malignancy.

115 l to recapitulate spatial aspects of cell-to-cell interactions as well as tissue gradients present in

116 was, in part, caused by uncoupling of Tfh-B cell interactions, as evidenced by reduced expression of

117 Here, we present a microfluidics-based cell-cell interaction assay that enables longitudinal investi

118 lasia, and redifferentiation, driven by cell-cell interactions between acinar cells, leukocytes, and

119 ion system (T6SS) mediates antagonistic cell-cell interactions between competing Gram-negative bacter

120 that bidirectional short- and long-distance cell interactions between epithelial and mesenchyme-like

121 Cell-cell interactions between normal cells and RasV12 cells

122 ian constructs that recapitulate native cell-cell interactions between ovarian granulosa and theca ce

123 esult from the disruption of inhibitory cell-cell interactions between the HVEM and BTLA (B and T lym

124 of signalling processes influencing cell-to-cell interactions between the vascular endothelial cells

125 This mechanism relies on cell interactions between two functionally distinct cell

126 stances that prevent the first step of virus-cell interaction by mimicking heparan sulfate proteoglyc

127 m Asn-25 reinforces dynamic endothelial cell-cell interactions by stabilizing the PECAM-1 homophilic

128 ngstanding questions about dynamics of virus-cell interactions can be answered by combining fluoresce

129 utum in which changes in rare cells and cell-cell interactions can be masked.

130 ling as one mechanism by which CDH2-mediated cell interactions can control NP cell phenotype and bios

131 he organization of cells, emerging from cell-cell interactions, can give rise to collective propertie

132 r to synergize their respective strength and cell interaction capabilities.

133 ified general Lotka-Volterra model with cell-cell interaction coefficients to predict the overall met

134 w play a dominant role in NP margination and cell interaction, compared to Brownian motion, gravity,

135 dings suggest that airway smooth muscle/mast cell interactions contribute to asthma severity by trans

136 that alteration in pDC-CD24(+)CD38(hi) Breg cell interaction contributes to the pathogenesis of SLE.

137 e explore the case for stromal-hematopoietic cell interactions contributing to neoplastic myeloid dis

138 Craniofacial development depends on cell-cell interactions, coordinated cellular movement and dif

139 protein Apelin and hematopoietic-endothelial cell interactions create this arrangement, providing gen

140 s II in regulating antigen-presenting cell-T cell interactions critical to the initiation and control

141 acrophages as gatekeepers of the cancer-host cell interaction: depriving transplants from macrophages

142 In order to track virus-target cell interactions during acute infection in vivo, we dev

143 fector molecules that are necessary for cell-cell interactions during late developmental processes an

144 ecular determinants of neutrophil-epithelial cell interactions during ongoing inflammation.

145 Here we explore FCSC and endothelial cell interactions during vascularized bone formation.

146 lates non-TRA coding genes that enhance cell-cell interactions (e.g., claudins, integrins, and select

147 f TCR-mediated signals, as opposed to single-cell interaction events, is needed to gain a full view o

148 These findings provide a new aspect of virus-cell interaction for rendering efficient HBV replication

149 affected the outcome of Chlamydia-dendritic cell interactions for both the bacterium and the host ce

150 Myelination is one of the most pivotal cell-cell interactions for normal brain development, involvin

151 During inflammation, leukocyte-endothelial cell interactions generate molecular signals that regula

152 enduring nature of the infection, this cell-cell interaction has become highly intimate, and the bac

153 heparan sulfate plays a dual role in exosome-cell interaction; heparan sulfate on exosomes captures f

154 that uterine decidual cells represent a cell-cell interaction hub with a large number of potential in

155 ure in-depth studies of VF dynamics and host cell interactions.IMPORTANCE MRV has historically been u

156 o develop new models to study niche-leukemic cell interaction in human myeloid malignancies; and fina

157 show the importance of an antibody/effector cell interaction in mediating humoral immunity.

158 d HtrA3 are crucial for trophoblast-decidual cell interaction in the control of trophoblast invasion.

159 omplexity of cell fate specification by cell-cell interactions in a rapidly dividing embryo.

160 lar junctions, because the weakening of cell-cell interactions in an alpha-catenin knockdown monolaye

161 This system will be useful to monitor cell-cell interactions in animals, and can be used to genetic

162 sputum can further our understanding of cell-cell interactions in asthmatic patients with the potenti

163 ton intravital microscopy as a tool to study cell interactions in different areas of the immune syste

164 sult of T follicular helper cell (TFH) and B cell interactions in germinal centers.

165 ll responses and follicular T helper (TFH)-B cell interactions in germinal centers.

166 ated the role of the NKG2D axis in T cell/NK cell interactions in hepatitis B.

167 e of the importance of gut-microbiota/immune-cell interactions in immune homeostasis and responsivene

168 m the inside out, and all the way up to cell-cell interactions in microbial communities.

169 lysis of CXCL1-induced leukocyte-endothelial cell interactions in postcapillary venules revealed that

170 suggesting a functional role for local place cell interactions in shaping firing fields.

171 ltiple human liver cell types can mimic cell-cell interactions in specific types of DILI.

172 er mortality, enhanced leukocyte-endothelial cell interactions in the brain microvasculature, and inc

173 EV biogenesis, secretion, cargo, and target cell interactions in the context of select liver disease

174 fine a nonredundant function of neutrophil-T cell interactions in the regulation of vascularization a

175 sh it as a novel, important mediator of DC-T cell interactions in type-2 immunity.

176 at offer new opportunities for studying cell-cell interactions in ultralow-volume environments.

177 Recreating heterotypic cell-cell interactions in vitro is key to dissecting the role

178 Precise reconstruction of heterotypic cell-cell interactions in vitro requires the coculture of dif

179 n, and indeed such a model fits the bare-tip/cell interaction, in agreement with earlier work.

180 ng suggests that faster dopamine-induced T-B-cell interactions increase total germinal centre output

181 lay between this antiviral response and cell-cell interactions, indicating that low cell densities in

182 Without this DC-T cell interaction, insufficient effector CD8 T cells are

183 monstrate how the transfer of molecular wood cell interactions into hemicellulose-based materials may

184 e, suggesting that a stable Ag-dependent T-B cell interaction is required.

185 ect examination of viral particles and virus-cell interactions is now possible via advanced microscop

186 A better understanding of virus-host cell interactions is relevant for developing improved th

187 ls are governed by a complex network of cell-cell interactions, knowing the specific immune cell comp

188 nse peptide GKY25, based on combined LPS and cell interactions leading to inhibition of TLR4 dimeriza

189 se catalyses reciprocal melanoma-endothelial cell interactions leading to perivascular invasion, a ph

190 d IL-6 in myofibroblasts (MFs) - lung cancer cell interactions, lung cancer cells (Lewis and CTM-167

191 Their natural composition and selective cell interactions make them promising drug carriers.

192 Accordingly, we propose that cell-cell interaction may be a factor responsible for the gra

193 Deciphering the mechanisms of EV-cell interactions may facilitate the design of EVs that

194 We propose an updated cell-cell interaction model for Myosin II polarization that w

195 ith a prisoner's dilemma game-theoretic cell-cell interaction model to design chemotherapeutic strate

196 mhtt in the corticostriatal projection cell-cell interaction model, we used BACHD/Emx1-Cre (BE) mice

197 enhanced resolution on cell-surface and cell-cell interactions modulated by membrane related protein

198 nce by a balance of cell-substratum and cell-cell interactions, modulated by cell phenotype-specific

199 at plays a pivotal role in a variety of cell-cell interactions, mostly studied during development.

200 Instead, evidence of NP-cell interactions must be preserved in derivative (usual

201 To investigate the cell-cell interactions necessary for the formation of retinal

202 trates that CCL2 enables the prolonged MSC-T cell interactions needed for sufficient suppression of a

203 ablish the PP SED as a niche supporting DC-B cell interactions needed for TGFbeta activation and indu

204 To investigate the early stages of cell-cell interactions occurring between living biological sa

205 tion from pluripotency fails to recapitulate cell interactions occurring during organogenesis.

206 In a TH2-promoting environment, T-cell/B-cell interactions occurring in regional lymph nodes lead

207 tistep process that involves sequential cell-cell interactions of circulating leukocytes with IL-1- o

208 gs showed important insights into virus-host cell interactions of NYVAC vectors expressing HIV antige

209 s cancer-immune system interactions, stromal cell interactions, oncoviruses, and sensitivity to thera

210 the basis for studying complex parasite-host cell interactions or drug effects with spatio-temporal r

211 serve as a binding site for a potential host cell interaction partner.

212 e summarize the biochemical activities, host cell interaction partners, and physiological functions o

213 To study cap sugar-T-cell interactions, pathogen mimics (namely glycodendrime

214 Cell-cell interactions play crucial roles in the maintenance

215 results suggest that direct or indirect cell-cell interactions prevent most coinfected cells from bei

216 ive albumin, thus showing the most favorable cell interaction profiles (low uptake by J774A.1 macroph

217 Cell-cell interactions promote juxtacrine signals in specific

218 function for IL-21 in tuning NK and CD4(+) T cell interactions promoting a specific expansion of cent

219 rogram in which secreted morphogens and cell-cell interactions prompt the adoption of unique cell fat

220 n tolerance in mice, identification of FRC-T cell interactions provides a new research target for tol

221 ur findings highlight the critical ECM-tumor cell interactions regulated by miR-200/Zeb1-dependent EM

222 enable the noninvasive visualization of cell-cell interactions relevant to organismal biology.

223 the precise mechanisms modulating MSC-immune cells interactions remain largely elusive.

224 macroscopic patterns resulting from cell-to-cell interactions remains largely qualitative.

225 receptors, that the greater number of CD4 T cell interactions, required to generate Th2 over Th1 cel

226 binding to heparan sulfate mediates exosome-cell interactions, revealing a fundamental mechanism imp

227 Cell-cell interactions, soluble factors, and extracellular ma

228 that are involved in a diverse array of host cell interactions, some of which directly activate cell

229 Microbial and host cell interactions stimulate rabbit B cells to diversify

230 Furthermore, IL-8 induced by stromal-MM cell interactions strongly contributed to osteoclast for

231 Using a 3D model of mitral and granule cell interactions supported by experimental findings, co

232 Cell competition is a form of cell interaction that causes the elimination of less fit

233 We aimed to investigate the ILC3-B-cell interaction that probably takes place in human tons

234 cells and platelets contributes to the cell-cell interactions that are involved in the pathogenesis

235 es innervate peripheral tissues through cell-cell interactions that are poorly understood.

236 cells ignores physiologically relevant cell-cell interactions that may be critical for circuit level

237 The rapid T-B-cell interactions that occur during this process are rem

238 nd products and their receptors mediate cell-cell interactions that regulate several biological funct

239 sizes the complex immunoglobulin light chain-cell interactions that result in fibril internalization,

240 To gain insight into the microbial and host cell interactions that stimulate B cells to diversify th

241 allotolerance by interacting with APCs and T cells, interactions that require their proper homing to

242 MS-derived BWMs require two distinct cell-cell interactions, the first inhibitory and the second,

243 Despite the importance of these virus-immune cell interactions, the specific mechanisms of HPV16 entr

244 Chlamydia-host cell interaction therefore constitutes a unique system t

245 proliferation, death, migration, and cell-to-cell interaction through contact inhibition.

246 ng that targeting osteocyte-multiple myeloma cell interactions through specific Notch receptor blocka

247 ontaining vacuole." The bacteria govern host cell interactions through the Icm/Dot type IV secretion

248 their receptors and signaling networks, cell-cell interactions, tissue microenvironment, and the host

249 t humans, we examined the adipocyte-leukemia cell interactions to determine if they are essential for

250 tanding of gene regulatory networks and cell-cell interactions to enable the reliable and robust engi

251 on cell membranes and the modulation of cell-cell interactions to generate three-dimensional (3D) tis

252 erference with PDGF-BB or PDGF receptor-beta cell interactions to implicate PDGF-BB as a primary effe

253 ly transitions from one that is rich in cell-cell interactions to one that is dominated by cell-ECM (

254 ight on how global cues integrate with local cell interactions to organize cellular polarity at the t

255 Cell-cell interactions underlie fundamental biological proces

256 within the IVD, specifically, mast cell-IVD cell interactions using immunohistochemistry and 3D in-v

257 The ability to control CAR-T cell and cancer cell interactions using intermediate switch molecules ma

258 tigated the molecular basis of the CagL-host cell interactions using structural, biophysical, and fun

259 pithelial cell pathobiology and related cell-cell interactions, using ischemic AKI as a model.

260 reproduced in this system via physical cell-cell interaction, vascular mechanical cues and cell migr

261 impact of HIV antigenemia on B cells and Tfh cell interactions warrants further exploration.

262 r biosynthesis during neutrophil-endothelial cell interactions was initiated by endothelial cyclooxyg

263 insight into the dynamics of place and grid cell interaction, we built a computational model with th

264 To study cancer cell-vascular cell interactions, we engineered a 2-layer hydrogel with

265 of DLX4 on CD44 levels and tumor-mesothelial cell interactions were abrogated when IL-1beta or NF-kap

266 Heterotypic cell interactions were also studied.

267 g a successful immune response requires cell-cell interactions, where the nature of antigen presentat

268 ology in CD19-hBtk mice was dependent on B-T cell interaction, whereas IL-10 production and IgM autoa

269 se and human cells, we identified a T cell-T cell interaction whereby antigen-experienced subsets dir

270 atelets and neutrophils is required for cell-cell interactions, which contribute to the pathology of

271 To characterize these cell-cell interactions, which include contact inhibition of l

272 cyte-associated antigen 1 is required for NK cell interaction with and elimination of iRBCs.

273 Our results show a unique pattern of NK cell interaction with C. albicans, which involves direct

274 Their activation requires cell-cell interaction with different myeloid cell populations

275 ver, the physical mechanism that governs the cell interaction with fibrous 3D ECM is still not known.

276 ll shape recognition can also be achieved by cell interaction with imprints that can be made into pol

277 al distinct roles in T cell activation and T cell interaction with other immune cells.

278 egenerative factors) and membrane-based cell-cell interaction with the injured cells.

279 Tumor cell interaction with various cellular components of the

280 role and suggest that Mac-1 may mediate cell-cell interactions with a previously unrecognized counter

281 our molecular understanding of those social cell interactions with a relevant function in tumor init

282 to dissect fundamental mechanisms of immune-cell interactions with bacteria and fungi.

283 nity by using intravital imaging of CD4(+) T cell interactions with dendritic cells (DCs) exposed to

284 microscopy, we captured 2,330 hours of tumor cell interactions with functional microvessels and provi

285 umors, pathogens and other tools, to study T-cell interactions with many different cell types, to mod

286 r morphology is sculpted by specific cell to cell interactions with neurons and each other.

287 automated methods for profiling dynamic cell-cell interactions with single-cell resolution from high-

288 n of desired cell types, or to interrogate T-cell interactions with specific populations.

289 that migratory DCs execute targeted cell-to-cell interactions with stationary MCs before leaving the

290 mimicry and vascular co-option involve tumor cell interactions with the abluminal vascular surface.

291 tin receptors play important roles in immune cell interactions with the environment.

292 Cell interactions with the extracellular matrix (ECM) ca

293 nced breast cancer patients, depend on tumor cell interactions with the mineralized bone extracellula

294 form integrated systems that mediate dynamic cell interactions with their environment or other cells

295 Endothelial cell interactions with transitional matrix proteins, suc

296 espect to normal ones (In the first 6h after cells interaction with surface) are the key mechanism in

297 nation of cancer cells via tumor-endothelial cell interactions, with possible implications for microR

298 PEL is a cationic polymer that promotes cell-cell interactions within the biofilm matrix through elec

299 coculture model that mimics bone cell/cancer cell interactions within the bone microenvironment.

300 t features to cancer cells and affects tumor cell interactions within the tumor microenvironment.