コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 tin) at the leading edge generates force for cell locomotion.
2 rmation of lamellipodia and filopodia during cell locomotion.
3 etal remodeling, intracellular transport and cell locomotion.
4 ate in modulating actin dynamics to optimize cell locomotion.
5 they fail to release their back ends during cell locomotion.
6 required to maintain cell shape and promote cell locomotion.
7 adhesion turnover, leading to inhibition of cell locomotion.
8 ular processes, including cell spreading and cell locomotion.
9 s of strong cell adhesion, but yet allow for cell locomotion.
10 ating a direct involvement of this enzyme in cell locomotion.
11 ht-directed perturbation techniques to study cell locomotion.
12 and force of membrane extension required for cell locomotion.
13 able of activating pro-MMP-2 and stimulating cell locomotion.
14 ction tail move forward, in the direction of cell locomotion.
15 normal and pathological processes involving cell locomotion.
16 t this aspect of transport is independent of cell locomotion.
17 induced mitogenic signals, cell survival and cell locomotion.
18 eta-gal, whereas AA-Akt blocked VEGF-induced cell locomotion.
19 a gradient of CSF-1 but is not essential for cell locomotion.
20 he contractility of both ends is involved in cell locomotion.
21 ain key aspects of cytokinesis and polarized cell locomotion.
22 ot elicit identical responses with regard to cell locomotion.
23 frame from what could be expected of single cell locomotion.
24 xplore four different minimal mechanisms for cell locomotion: 1), a biophysical model for myosin cont
25 This finding also suggests that directional cell locomotion against resistive forces requires a turg
26 te that the flagellar motors responsible for cell locomotion also play a role, adding or subtracting
27 By recording the position of beads during cell locomotion and after cell removal, we discovered th
28 during coordinated cell movements, including cell locomotion and cell division, we generated a phosph
29 alysis revealed an elevation in the speed of cell locomotion and cell proliferation rate in the conne
30 Cdc42 activity, we show changes in speed of cell locomotion and cell roundness also result from exog
31 ucial for many cellular processes, including cell locomotion and cytokinesis, but are poorly understo
37 rich structures, likely play a major role in cell locomotion and in mechanical signaling with the sur
40 nizing the cilium, a structure important for cell locomotion and sensing of the surrounding environme
41 lts suggest that the increased plasticity of cell locomotion and the invasiveness of MTLn3 cells resu
42 hput wound closure assay in which individual cell locomotion and wound closure kinetics were quantifi
43 translocation of proteins across membranes, cell locomotion, and catalyzed protein and nucleic acid
45 -1 upregulation and TGF-beta1+EGF-stimulated cell locomotion, as pharmacologic disruption of MEK/p38
47 motors in living cells are involved in whole-cell locomotion, contractility, developmental shape chan
50 roperties of biological cells play a role in cell locomotion, embryonic tissue formation, and tumor m
58 gh the N-WASp/Arp2/3 pathway is important in cell locomotion, membrane trafficking, and pathogen infe
59 much the same way the cytoskeleton mediates cell locomotion, mitosis and intracellular vesicular tra
60 but not against alpha1 and alpha2, inhibited cell locomotion on a reconstituted basement membrane in
61 l cell invasive behavior, but not epithelial cell locomotion over the gel surface, is partially regul
64 within a three-dimensional collagen matrix, cell locomotion results in translocation of the flexible
65 biochemical signals and forces necessary for cell locomotion, suggesting that P2Y(2)Rs may be linked
66 gh microtubules have long been implicated in cell locomotion, the mechanism of their involvement rema
67 mical processes are integrated to accomplish cell locomotion, the same issues, along with certain new
69 latory role for cell traction generation and cell locomotion under the physical confinement of the th
70 a that microtubules normally act to restrain cell locomotion was confirmed by treating cells with hig
71 e role of these macromolecular assemblies in cell locomotion, we have determined the 2.6 A resolution
72 ously associated metabolic oscillations with cell locomotion, we hypothesized that patients with abno
73 nt controls invadopodium formation and tumor cell locomotion, we systematically analyzed components o
74 ta1 integrin binding activates RhoA, slowing cell locomotion, whereas laminin-5-alpha3beta1 integrin
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。