ライフサイエンスコーパス: cell marker

1 9 and CD138 immune cells (plasmablast/plasma cell markers).

2 ary acidic acid (GFAP)-expressing supporting cell marker.

3 anglioside GD2 as a novel breast cancer stem cell marker.

4 is regarded as a multipotent and neural stem cell marker.

5 FR/ERBB tyrosine kinase receptors and a stem cell marker.

6 germ cells turning on an early-stage somatic cell marker.

7 19 as a segment-committed nephron progenitor cell marker.

8 as might be expected for a tumor-initiating cell marker.

9 ositive for CD45, considered a hematopoietic cell marker.

10 ntified keratin 23 (KRT23) as a new ductular cell marker.

11 ession of the master EMT regulators and stem cell markers.

12 , and, at the single-cell level, cancer stem cell markers.

13 cell profiling of any tissues that lack live-cell markers.

14 (ii) activated RICTOR; and (iii) progenitor cell markers.

15 These tumor-initiating cells express stem cell markers.

16 reduced the expression levels of neural stem cell markers.

17 es cell proliferation and expression of stem cell markers.

18 ndothelial, podocyte, or parietal epithelial cell markers.

19 nd maintained expression of mesenchymal stem cell markers.

20 d fewer cells expressing mammary cancer stem cell markers.

21 This group included 34 NK cell markers.

22 els of Wnt activity also express cancer stem cell markers.

23 al cells expressing multiple progenitor/stem cell markers.

24 els of either Trp63 or of standard secretory cell markers.

25 traits of juvenile astroglia and neural stem cell markers.

26 activates tissue-specific expression of stem cell markers.

27 on and were negative for regulatory CD8(+) T cell markers.

28 imately 72%) but did not coexpress CD4 or NK cell markers.

29 ells express both mesenchymal and epithelial cell markers.

30 ocesses and continuously express neural stem cell markers.

31 trated by the up-regulation of classical MZB cell markers.

32 me or regulation of conventional cancer stem cell markers.

33 ivascular tumor cells expressed hepatic stem cell markers.

34 hemoresistance and expression of cancer stem-cell markers.

35 ut allergy with B-cell, T-cell, and effector cell markers.

36 ways and expressing relevant stem/progenitor cell markers.

37 e islets it co-localizes with alpha and beta cell markers.

38 potential and downregulation of glioma stem cell markers.

39 rray of novel genes as putative corneal stem cell markers.

40 n of key epithelial, stromal and endothelial cell markers.

41 yte markers, oval cell markers, and stellate cell markers.

42 f EZC progeny that continued to express stem cell markers after SCI, increased the proportion of EZC

43 s resulted in a significant decrease in stem cell markers, Aldefluor expression, proliferation, and i

44 t for the previously established cancer stem cell marker ALDH (aldehyde dehydrogenase) in the mainten

45 y of mammospheres and downregulated the stem cell markers ALDH and CD44, while upregulating CD24.

46 or metastasis and the expression of the stem cell marker ALDH1A1.

47 -expressing cells colocalized with mesangial cell markers alpha8-integrin and PDGF receptor-beta but

48 rsistence of T cells targeting CD19, a pan-B cell marker, also depletes normal B cells and causes sev

49 CD25 is a leukemic stem cell marker and a conspicuous prognostic marker for over

50 mining region Y)-box 2 (Sox2) as cancer stem-cell marker and driver of cancer stemness.

51 atic activity has been used as a cancer stem cell marker and seems to correlate with tumour aggressiv

52 influenced gene expression of TH17 and Treg cell markers and cytokine secretion.

53 e infiltration and expression of cytotoxic T-cell markers and effector cytokines, similar in extent t

54 e identified using immunohistochemical Golgi cell markers and electron microscopic profiles of granul

55 ated with downregulation of mature dendritic cell markers and expansion of regulatory T cells resulti

56 Resultant iPSC clones expressed pluripotent cell markers and generated teratomas.

57 ent show increased expression of mature beta cell markers and improved glucose stimulated insulin sec

58 on promotes expression of hair follicle stem cell markers and induces elements of the core hair morph

59 n CD4 T cells expressing T follicular helper cell markers and inhibitory co-receptors.

60 nd circulating lymphocytes were extracted; T-cell markers and inhibitory receptors were quantified fo

61 ult beta cells, including expression of beta cell markers and insulin secretion following glucose sti

62 ifferential activation of hepatic progenitor cell markers and metabolic pathways: high-risk tumors we

63 ancer that were even more enriched with stem cell markers and more tumorigenic than the freshly isola

64 eveloped a mathematical model using T- and B-cell markers and mycophenolate mofetil (MMF) dosage.

65 mation and for the expression of neural stem cell markers and Notch target genes in primary neural pr

66 ween food diversity and gene expression of T-cell markers and of Cepsilon germline transcript, reflec

67 asured the gene expression of various immune cell markers and performed multivariate analysis of the

68 mesenchymal stem cell and hepatic progenitor cell markers and produce growth factor and cytokine mRNA

69 The cells also expressed known mesenchymal cell markers and promoted new bone formation to heal cri

70 he SASP exhibit increased expression of stem cell markers and regenerative capacity in vivo.

71 hese highly proliferative cells express stem cell markers and retain the ability to differentiate dow

72 ABCG2 can actively drive expression of stem cell markers and self-renewal in glioma cells.

73 Stem cell markers and self-renewal was enriched in cells with

74 We observed high expression of epithelial cell markers and similarity to the transcriptome for int

75 be given to use of multiple cancer stem-like cell markers and suitable procedures for cell isolation

76 rcoma cells decreases the expression of stem cell markers and suppresses sarcosphere formation, as we

77 st that expression of several canonical Treg cell markers and suppressive function could be Foxp3 ind

78 ed a computational screen for myeloma plasma cell markers and systematically evaluated 7 candidates.

79 ted and secreted from muscle to induce beige cell markers and the browning of WAT in Mstn(-/-) mice.

80 at tissue did not express myoblast or dermal cell markers and uniformly differentiated into brown adi

81 ntricular zone cells that express progenitor cell markers and upregulation of EZH2 expression within

82 leal epithelial enteroids had reduced Paneth cell markers and were highly sensitive to inflammatory c

83 pair signaling and expression of cancer stem cells markers and sensitized chemoresistant OC cells to

84 omato) (tracing marker), 2.3 Col1(GFP) (bone cell marker), and aggrecan Cre(ERT2) (onetime tamoxifen

85 y used as a pluripotent human embryonic stem cell marker, and its expression is correlated with the m

86 in the expression of Sox2, a GBM cancer stem cell marker, and was obligatory for tumor formation.

87 atant IL-4, serum IL-2 and IL-6, endothelial cell markers, and anti-ADAMTS13 antibody compared with c

88 symmetric division, express mesenchymal stem cell markers, and generate chondrocytes via both asymmet

89 NA damage response, pro-survival genes, stem cell markers, and self-renewal ability for survival and

90 y low expression of hepatocyte markers, oval cell markers, and stellate cell markers.

91 handling, increased mRNA expression of stem cell markers, and striking induction of many genes assoc

92 ed on a phenotype composed of conventional B cell markers, and that IL-10 production can be elicited

93 ates with decreased expression of the EC tip cell markers apelin and Dll4 and is associated with a re

94 The cells expressing five of stem/progenitor cell markers are localized in basal layer of entire muri

95 These studies identify this stem cell marker as a novel BRD4-NUT target that supports the

96 We identified ALDH1A1, a cancer stem cell marker as being overexpressed in OC spheroids and d

97 dentification of Lgr5 as the intestinal stem cell marker as well as the growth factors necessary to r

98 e expression of mesoderm and primordial germ cell markers asymmetrically on the embryonic and extraem

99 they expressed neuronal and retinal ganglion cell markers (ATOH7, POU4F2, beta-III tubulin, MAP2, TAU

100 RPMI, DMEM both expressed the putative stem cell marker Bmi-1 and yielded cell colonies.

101 4 (OLFM4) has emerged as an intestinal stem-cell marker, but its biological function in the intestin

102 ated the expression of lymphatic endothelial cell markers by the SC in murine and zebrafish models as

103 ein gene product 9.5 (PGP 9.5), and the mast cell marker c-kit.

104 suppressed the expression of the cancer stem cell markers c-Myc, CD133, and nestin, which could contr

105 lastoma multiforme which express neural stem cell markers, can differentiate into neurons and glia, a

106 ased percentage of cells expressing the stem-cell marker CD133 and increased apoptotic fraction.

107 nating hair placode cells expresses the stem cell marker CD133 during early morphogenesis.

108 osphere formation and the expression of stem cell markers CD133, Nestin, and Nanog.

109 akers (BrdU, cyclin D1, p53) and cancer stem cell markers (CD133 and nanog) are significantly up-regu

110 ed by HSCs augmented B-cell survival, plasma cell marker CD138 expression, and immunoglobulin G produ

111 that was positive for the known tendon stem cell marker CD146 and exhibited clonogenic capacity, as

112 mono express higher levels of the dendritic cell marker CD1c.

113 and immunohistochemistry and expressed the T-cell markers CD2, CD3, CD4, and terminal deoxynucleotidy

114 Depleting mAbs against the pan B cell marker CD20 are remarkably effective in the treatme

115 antibody specifically targeting the human B cell marker CD20, for its ability to image B cells in a

116 ed with type 2 immune response and dendritic cell markers (CD209 and CD207).

117 upernatants enhanced the expression of the B cell markers CD23 and CD40, which are important for B ce

118 aining factors (c-Myc, Nanog and Oct4), stem cell markers (CD24 and CD133), components of Shh pathway

119 lly, cell surface expression of the CML stem cell markers CD25, CD26, and IL1RAP is high in all subpo

120 relevant genes, as for instance the memory-B cell marker CD27 and PTPRC genes, providing us with biol

121 ) and immunostaining of vascular endothelial cell marker CD31 and VEGFR2.

122 ng alphaSMA, SM22, and CNN1, and endothelial cell marker CD31 were significantly downregulated in Dro

123 ment in liver-resident cells expressing stem cell marker CD34.

124 nted epithelium and the vascular endothelial cell marker CD34.

125 e exhibit increased expression of Osm, the T cell markers Cd4 and Cd8, and the macrophage markers F4/

126 ifying neuropilin-1 (NRP1); and the helper T cell marker, CD4.

127 Here, we show that the stem cell marker CD44 promotes cancer stem cell phenotypes an

128 egulates alternative splicing of cancer stem cell marker CD44 through a phosphorylated T179 of SMAD3-

129 astoma in a pattern correlated with the stem cell marker CD44.

130 ype with increased expression of cancer stem cell markers CD44 and CD133 and become more resistant to

131 e cells gain expression of de-differentiated cell markers CD44 and cytokeratin 5 (CK5), lose luminal

132 ting organoids expressed high levels of stem cell markers CD44 and Lgr5.

133 acterized by elevated surface levels of stem cell markers CD44 and Sca1 and by rapid metastasis.

134 requency of cells expressing the cancer stem cell markers CD44, CD133, and c-Met and the immunologic

135 l expression of other putative prostate stem cell markers (CD44 and integrins alpha2/beta1), when com

136 e characteristics of CSCs by expressing stem cell markers (CD44, CD133, LGR5 and DCLK1) and transcrip

137 a and progesterone receptors, expressed stem cell marker, CD44, and displayed increased clonogenicity

138 with the expression of putative cancer stem cell markers, CD44 and ALDH1, increased resistance to ch

139 Allelic variants of the pan-haematopoietic cell marker CD45, identified as CD45.1 and CD45.2, have

140 or memory T cells that reexpress the naive T cell marker CD45RA have many characteristics of cellular

141 bsets of PROM1pos cells coexpress progenitor cell marker CD49f, epithelial marker E-CADHERIN, biliary

142 -treated humanized mice that express both NK cell marker CD56 and myeloid markers such as CD36 and CD

143 level (p < 0.05), and immunostaining for NK cell marker CD56 revealed a striking reduction of uNK in

144 ate tumor tissues, elevated cancer stem-like cell markers coincide with those cells exhibiting high S

145 immunolabeling experiments by using various cell markers combined with synaptic markers.

146 that AML cells express lymphatic endothelial cell markers consistent with lymphatic endothelial cell

147 021 reduced the growth rate, stem/progenitor cell marker content and clonogenic capacity in the expla

148 shi1 (MSI1) has been characterized as a stem cell marker, controlling the balance between self-renewa

149 lving a panel of macrophage and other immune-cell markers could verify and validate these findings an

150 terleukin (IL)-4 and, unexpectedly, the mast cell marker CPA3 predicted UCP1 gene expression.

151 sin-negative and express pRb, the epithelial cell marker cytokeratin that is expressed in the retinal

152 tic lesions positive for the stem/progenitor cell marker cytokeratin-19, indicating that several HCC-

153 in D1 and the putative epithelial progenitor cell marker cytokeratin-5.

154 oplastic lesions positive for the progenitor cell marker, cytokeratin-19 (Krt-19) and characterized b

155 in greatly reduced expression levels of stem cell markers (DCLK1/CD44/ALDHA1/Lgr5/Nanog) in three-dim

156 by a live-cell sorting method using the germ cell marker DDX4, which has previously been assumed to b

157 mRNA expression of Th1/Th2/Th17-associated cell markers decreased between 4.5 and 6 years (P < 0.00

158 ount (CBC) and inflammation-associated blood cell markers derived from them have been reported to cor

159 Satellite cell markers did not correlate with Ki-67-affected myofi

160 -ErbB negative regulator and intestinal stem cell marker down-regulated in many malignancies.

161 erentiated epithelia reexpress apparent stem-cell markers during injury-induced dedifferentiation and

162 tes, with its cognate ligand, the epithelial cell marker E-cadherin on tumor cells, plays a major rol

163 enlargement, ii) altered expression of mural cell markers (eg, down-regulation of NG2 and up-regulati

164 These T cell markers enabled a new understanding of immune respo

165 cell lines increased the expression of stem cell markers EpCAM, Claudin7, and Oct4, as well as decre

166 stem cell (Sca-1, CD133, Dlk) and liver stem cell markers (EpCAM, CD14, CD24, CD49f); and negative fo

167 Stem cell markers ESRRB and CK5, myoepithelial marker CK14, a

168 milar phenotypic profile of mesenchymal stem cell markers, except a relatively higher level of CD146

169 ression across cells, (3) the variability of cell marker expression across samples that (4) may vary

170 cs include increased self-renewal, high stem cell marker expression and high tumorigenic capacity in

171 tion of Akt-EphA2 cross-talk attenuated stem cell marker expression and neurosphere formation while h

172 Polymorphisms in IL10 influenced Treg cell marker expression and TH1/TH2 and proinflammatory c

173 CD8(-) T cells, regulatory T cells, CD4(+) T cell marker expression, lifespan, and Th/regulatory T ce

174 on, and flow cytometry to assess airway stem cell marker expression.

175 Induced GH is a readily measurable cell marker for p53-mediated cellular senescence.

176 is one of the best-characterized tumor stem cell markers for many intra- and extracranial tumor enti

177 We examined the time course of effector cell markers (for basophils, dendritic cells and T cells

178 tor [IL23R], IL17, IL17F, and IL22) and Treg cell markers (forkhead box protein 3 [FOXP3], lymphocyte

179 y individuals did not coexpress regulatory T cell markers (Foxp3(+) CD25(+)) and cultured T cell clon

180 itical tools, including highly specific stem cell marker genes along with more rigorous experimental

181 rgely driven by identification of novel stem cell marker genes, revealing the existence of quiescent,

182 We focused on the reactive cell marker glial fibrillary acidic protein (GFAP) to st

183 ortin-3, or appeared to co-express the alpha-cell marker, glucagon.

184 xpress several pluripotent and myogenic stem cell markers; have the capability to form embryoid bodie

185 patterns of EpCAM, a hepatic stem/progenitor cell marker highly expressed in a subset of HCC with ste

186 ng MARCM (mosaic analysis with a repressible cell marker; i.e., GFP-labeled, homozygous mutant) on al

187 Instead, they expressed high levels of stem cell markers, IL-7R and RORgammat, consistent with the n

188 MUSASHI-1 (MSI-1) is a well-established stem cell marker in both normal and malignant colon cells and

189 r6, but not Lgr5, acts as an epithelial stem cell marker in SCCs in vivo.

190 ies have suggested that PHLDA1 may be a stem cell marker in the human intestine that contributes to t

191 LGR5 is known to be a stem cell marker in the murine small intestine and colon, how

192 ng female reproductive organs, and is a stem cell marker in the stomach and intestinal epithelium, ha

193 the expression of p63, a known basal/reserve cell marker in this tissue.

194 highly positive staining for epithelial stem cell markers in all areas of normal TM tissue.

195 ocalized to cells positive for smooth muscle cell markers in human coronary artery disease lesions, a

196 Staining for other cell markers in rats treated with GLP-1(1-37)-secreting

197 activated Wnt signalling, and distinct stem-cell markers in senescence.

198 -driven target genes and alveolar epithelial cell markers in the elastase, as well as in CS-induced m

199 hology and the presence of transcripts for T cell markers in the sorted CD4-1(+)CD3epsilon(+) cells w

200 ivity and higher expression of CD105, a stem cell marker, in hMSCs in random versus regular patterns

201 here formation and expression of cancer stem cell markers, including BMI1.

202 city of BTIC and decreased numerous GBM stem cell markers, including CD133, CD90, CD49f and A2B5.

203 ts with T1D expressed elevated levels of Tfh cell markers, including CXCR5, ICOS, PDCD1, BCL6, and IL

204 undly decreased expression of canonical Treg cell markers, including FOXP3, CD25, Helios, and cytotox

205 el of NSPC and lineage-restricted progenitor cell markers, including MSI-1, SOX9, and TUJ1.

206 and failure to sustain expression of repair cell markers, including Shh, GDNF, and BDNF.

207 LGA microspheres improves expression of stem cell markers, increases stem cell numbers and decreases

208 that Lgr5, a recently discovered adult stem cell marker, is exclusively expressed in GBCs in neonata

209 oupled receptor 5 (LGR5), an intestinal stem cell marker, is known to exhibit tumor suppressor activi

210 s) expressing the biliary/hepatic progenitor cell marker keratin 19 (K19) have been linked with a poo

211 ssion drove ectopic expression of the Merkel cell marker keratin 8 (K8) throughout the epidermis.

212 cadherin and the undifferentiated epithelial cell markers keratin 5 and 14 but not the differentiatio

213 These phmSG cells exhibited progenitor cell markers (keratin 5 and nanog) as well as acinar-spe

214 These nail LRCs express the hair stem cell marker, keratin 15 (K15), and lineage tracing show

215 er, they did not colocalize with the Schwann cell marker Krox20, spiral ganglion marker NF200, nor gl

216 characterized by high expression of the stem cell marker Lgr5, by the expression of liver progenitor

217 vel EE gene expression but co-expressed tuft cell markers, Lgr5 and Ascl2, reminiscent of label-retai

218 Here we report that the lung stem cell marker Lgr6 becomes enriched in non-small cell lung

219 cell patches expressing the undifferentiated cell marker LHX9, and a loss of differentiated cells in

220 CD3, CD19, CD4, CD8 and Foxp3) and dendritic cell markers (Lineage negative [CD3, CD14, CD16, CD19, C

221 BMSCs when induced to express neuronal stem cell markers lose immunoregulatory function when transfe

222 combining mosaic analysis with a repressible cell marker (MARCM) analysis with gene expression studie

223 s showed upregulation of T follicular helper cell markers, most notably CXCR5 and its ligand CXCL13,

224 al of hDPSCs and other stem cells, selective cell markers must be identified in the progenitor cells.

225 wi for more than 6 weeks along with the germ cell markers nanos3, dnd, dazl and vasa.

226 ellite glial cells expressing the progenitor cell marker nestin was increased at 1 and 3 days followi

227 ation largely negative for the skeletal stem cell markers Nestin-GFP, Leptin receptor and Gremlin1.

228 , a positive correlation between CCL5 and NK cell marker NKp46 expression was found in melanoma patie

229 tivation of expression of the embryonic stem cell markers OCT4, NANOG, SOX2 and SSEA1 and lacked expr

230 overlapping populations: one containing host cell markers of exosomes (CD9, CD63) and the other conta

231 which also had increased expression of stem cell markers (OLFM4, BMI1, and MSI1) compared with colon

232 e chain reaction to examine Sertoli and germ cell markers on rat testes and human fetal testis xenogr

233 AFSC derived EVs presented exosomal and stem cell markers on their surface membrane, including VEGFR1

234 Runx2 and Col1a1, which are early osteogenic cell markers, on day 10 after the subperiosteal injectio

235 tify genes representing common putative stem cell markers or determinants, which included Sox9, Fzd7,

236 hen become HCC cells that express progenitor cell markers), or to transdifferentiate into biliary-lik

237 However, IWP2 increased stem/progenitor cell marker (p63alpha and ABCG2) content and clonogenic

238 predominantly express the venous endothelial cell marker PAL-E, whereas CD144(+)PAL-E(-) vessels comp

239 sharply elevated the expression of the beta cell markers pancreatic and duodenal homeobox 1 (Pdx1) a

240 factors, while down-regulating the satellite cell marker Pax7.

241 2 silencing inhibited the expression of stem cell markers, pluripotency maintaining transcription fac

242 and expression of the lymphatic endothelial cell markers podoplanin and lymphatic vessel endothelial

243 cultures of DiO-labeled TMSCs expressed stem cell markers preferentially in DiO positive cells, demon

244 Early PVR vitreous showed upregulation of T-cell markers, profibrotic cytokines, and cytokines downs

245 tion expressing the putative stem/progenitor cell marker, PROMININ-1 (PROM1), adjacent to ductular re

246 found that during ee cell formation, the ee cell marker Prospero localizes to the basal side of divi

247 Double labeling with the ganglion cell marker RBPMS demonstrated that the large majority (

248 t contests the observations that cancer stem cell markers reliably identify the subset of tumor cells

249 of gut barrier dysfunction and phenotypic T-cell markers remains unclear.

250 d contained established granule and Purkinje cell markers, respectively.

251 These T1D SC-beta cells express beta-cell markers, respond to glucose both in vitro and in vi

252 Moreover, flow cytometric analyses for B-cell markers revealed an MDM2-ALT1-associated decrease i

253 ls expressing the myeloid-derived suppressor cell marker S100A9 only in a TN breast cancer environmen

254 Adventitial progenitors express the stem cell markers, Sca1 and CD34 (adventitial sca1-positive p

255 aled significantly higher expression of stem cell markers, self-renewal properties, thyrosphere forma

256 ession of the epithelial progenitor and stem cell marker sex-determining region Y-related box 2 (sox2

257 aracteristics, such as p63(+) cells (a basal-cell marker) showing luminal-like morphology, or cells d

258 not form neurospheres but expressed the stem cell markers Slc1a3-CreER(T), GFAP-CreER(T2), Sox2(CreER

259 2 as well as the supporting cell and Schwann cell marker Sox10; however, they did not colocalize with

260 factors led to the expression of the Schwann cell markers SOX10, KROX20 (EGR2), p75NTR (NGFR), MBP an

261 cells involved an up-regulation of the stem cell marker Sox2 and proliferation genes and decreased e

262 d with the stem cell and cochlear-supporting-cell marker Sox2 as well as the supporting cell and Schw

263 Additonally, the epithelial stem cell marker SOX2 was significantly upregulated in KCOT w

264 murine cerebellar cortex, expresses the stem cell markers Sox2 and Nestin, and lacks markers of glial

265 ress an exceptionally high level of the stem cell marker, SOX2.

266 Expression of the stem cell marker SOX9 was markedly higher in esophageal adeno

267 etween CDC42 and type II alveolar epithelial cells marker SP-A, indicating the potential importance o

268 The alpha-cell markers, Sst and Hhex, are upregulated in Snord116p

269 sphere growth and increased activity of stem cell markers such as Aldehyde Dehydrogenase 1 (ALDH1).

270 T-cell markers such as CD3-by examining active recruitment

271 beta cells, including the expression of beta cell markers such as insulin, PDX1, and NKX6.1.

272 n alpha-6, and that these cells express germ cell markers such as vasa, pumilio and piwi, as well as

273 ion, they expressed typical glioma stem-like cell markers, such as CD133, CD15, and CD90.

274 In contrast to other stem cell markers, such as CD38, EPCR expression is maintaine

275 mmunolabeled for an RGC marker, not amacrine cell markers, suggesting that they are dopaminergic ipRG

276 s expressed stem, endothelial, and pericytic cell markers, suggesting these cells represent an undiff

277 C3 expressed higher levels of the effector T cell markers than TC3, suggesting that PD-L1 expression

278 1 protein (DCLK1) is a gastrointestinal tuft cell marker that has been proposed to identify quiescent

279 d identify Lmx1a as a novel enterochromaffin cell marker that is also essential for the production of

280 ronan (HA) induces the up-regulation of stem cell markers that display the hallmark CSC properties.

281 ls stimulates a durable upregulation of stem cell markers that epigenetically reprogram these cells.

282 Owing to the absence of exclusive stem cell markers, the location, multiplicity, dynamics and f

283 They express normal and cancer stem cell markers, they divide asymmetrically and they cycle

284 ncluding a later increase in expression of B-cell markers, transcription factors, and signaling molec

285 ne, and for expression of the surface mucous cell marker Ulex europaeus.

286 A expression of Treg/Th1/Th2/Th17-associated cell markers was measured ex vivo.

287 h pure DCIS, a similar loss in myoepithelial cell markers was observed.

288 Notably, stem cell markers were also misregulated.

289 h serum IL-2, IL-4, and IL-6 and endothelial cell markers were analyzed by ELISA.

290 Cell markers were analyzed by using immunohistochemistry

291 Viral infection and expression of lung cell markers were analyzed using flow cytometry.

292 Cell markers were assessed by in situ staining and by qu

293 solated steatotic primary hepatocytes, and T-cell markers were assessed in hepatic lymphocytes after

294 Putative stem cell markers were detected in cultures grown in DMEM and

295 Yet TH17 and Treg cell markers were positively correlated and influenced b

296 , and glial fibrillary acidic protein (glial cells markers) were quantified in the lumbar region of t

297 ed expression of oncofetal proteins and stem-cell markers, while low-risk tumors had low lin-28 homol

298 ntification of an LITR subset as a cytotoxic cell marker will allow for more effective monitoring of

299 e the association of CPS-induced cytotoxic T-cell markers with protection.

300 We also examine five rhombic lip cell markers (Wls, Math1, Pax6, Lmx1a, and Tbr2) to iden