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1 ly control different aspects of the in vitro cell microenvironment.
2 ibits tumor invasion by inducing suppressive cell microenvironment.
3 tion of both the cytokine and cellular tumor cell microenvironment.
4 reprogrammed by inductive cues in the tumor cell microenvironment.
5 y can be suppressed by factors in the tumour-cell microenvironment.
6 chain and, thus, the remodeling of the tumor cell microenvironment.
7 cosphingolipids that are shed into the tumor cell microenvironment.
8 upon continuous guidance from thymus stromal cell microenvironments.
9 ess damaged than 2D culture due to different cell microenvironments.
10 erentiation within complex and heterogeneous cell microenvironments.
11 e ratio of protease and antiproteases in the cells' microenvironment.
12 on of PTN through stimulation of the stromal cell microenvironment alone may be sufficient to account
13 of ExoS ADPRT activity within the eukaryotic cell microenvironment and into possible modulatory roles
14 rtant for a better understanding of lymphoid cell microenvironment and migrating capacities and their
15 With precise control over small volumes, the cell microenvironment and other biological agents can be
16 y linked to its ability to modulate the host cell microenvironment and to facilitate efficient produc
17 ell adhesion cues that distinguish each stem cell microenvironment, and that are critically important
18 M molecules expressed in many different stem cell microenvironments, and their corresponding receptor
20 sh triggers the formation of an inflammatory cell microenvironment at the implant site through comple
21 egulator of EMT and preserves thymic stromal cell microenvironment by controlling age-related adipocy
22 els to emulate the complexity of the natural cell microenvironment during embryogenesis, particularly
24 concentrations of asparagine in the leukemic cell microenvironment - for the protective effects we ob
25 ility to re-programme the local inflammatory cell microenvironment from a 'hostile' to an 'instructiv
27 ches for controlling the embryonic stem (ES) cell microenvironment have been developed for regulating
28 and promote protease activation in the tumor cell microenvironment; however, uPAR also regulates cell
29 contribute to wound healing, the role of the cell microenvironment in tissue repair remains elusive.
30 l receptor (BCR) signaling and on a specific cell microenvironment, including T cells, macrophages, a
31 ological processes, such as 1) cell-cell and cell-microenvironment interaction; 2) transdifferentiati
33 ng developed that target these complex tumor cell-microenvironment interactions and target the signal
34 , the data highlight the importance of tumor cell-microenvironment interactions and the necessity to
35 multi-cellular in nature where cell-cell and cell-microenvironment interactions determine the emergen
36 deposition and explain how Bves facilitates cell-microenvironment interactions in the regulation of
37 can control precisely individual aspects of cell-microenvironment interactions is presented and they
41 ymph nodes (LNs) and formation of LN stromal cell microenvironments is dependent on lymphotoxin-beta
42 xtensive remodeling of the MCF-7-Ptn/NIH 3T3 cell microenvironment; it up-regulated expression of mar
44 croscale principal strains for a majority of cell microenvironments located across diverse microstruc
45 This work provides new insight into how the cell microenvironment may influence Raf-1 expression to
46 ese attributes include colocalization with B cell microenvironments, MHC class II expression, dendrit
50 pproach to therapy is to manipulate the stem cell microenvironment, or niche, to facilitate repair by
52 gens; however, physical constrainment by the cell microenvironment represents an additional mechanism
53 novel components of the haematopoietic stem cell microenvironment, revealing that haematopoietic ste
55 cations in basic research such as studies of cell microenvironments, stem cell niches, metaplasia, or
56 rmation and organ regeneration to engineered cell microenvironments, synthetic biomaterials and artif
57 the extracellular biochemical milieu of stem cell microenvironments that regulate pluripotent cell fa
58 activities are controlled by the local stem cell microenvironment, the "niche." Wnt signals emanatin
60 eature of hypoxia, as well as tumor and stem cell microenvironments, we hypothesized that pH may regu
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