ライフサイエンスコーパス: cell morphology

1 molecule inhibitors and disruption of normal cell morphology.

2 ine kinase, KSHV-TK modulates signalling and cell morphology.

3 assify leukocytes and characterize red blood cell morphology.

4 ductase, abrogates pilus assembly and alters cell morphology.

5 has no effect on focal adhesion integrity or cell morphology.

6 tion of hippocampal LTP and on CA1 pyramidal cell morphology.

7 oximately 15%, with only marginal changes to cell morphology.

8 ely on qualitative observation of changes in cell morphology.

9 he Spag6-deficient MEFs rescued the abnormal cell morphology.

10 l bursting discharge, Ih currents, and islet cell morphology.

11 nd, thus, regulation of axonal stability and cell morphology.

12 can of peripheral blood smears for red blood cell morphology.

13 lecules in multiple cells by normalizing for cell morphology.

14 e-associated beta-galactosidase activity and cell morphology.

15 arepsilon is sufficient to block mesenchymal cell morphology.

16 Piezo1 mechanotransduction to regulation of cell morphology.

17 dema within the submucosa and altered mucous cell morphology.

18 hrough changes in both cell biochemistry and cell morphology.

19 , loss of astrocyte coupling, and changes in cell morphology.

20 status of RhoA but also RhoA activation and cell morphology.

21 skeleton of a neuron, reconstructing the 3D cell morphology.

22 Lon, intracellular localization of Lon, and cell morphology.

23 city in addition to their role in regulating cell morphology.

24 uding cytokinesis, migration, and control of cell morphology.

25 ering profiles that correlate with perturbed cell morphology.

26 r both virus assembly and modulation of host cell morphology.

27 on for plant cell pigmentation and epidermal cell morphology.

28 ormation on the three-dimensional epithelial cell morphology.

29 ll growth and proliferation, cell death, and cell morphology.

30 revealing a link between filament length and cell morphology.

31 on growth rates, mode of PG incorporation or cell morphology.

32 which involve substantial changes in overall cell morphology.

33 tivation state within tissue based solely on cell morphology.

34 matin, affecting transcription, meiosis, and cell morphology.

35 ate RhoA signaling and downstream effects on cell morphology.

36 sponses including a characteristic change in cell morphology.

37 d ectopic septin fibers, as well as aberrant cell morphology.

38 method for unbiased, automated comparison of cell morphology.

39 ith UM advancement, but has little effect on cell morphology.

40 sites and for the establishment of migratory cell morphology.

41 eletal structures that can dynamically alter cell morphology.

42 chondria, a landmark signature of eukaryotic cell morphology.

43 c1-dependent cytoskeletal dynamics, and thus cell morphology.

44 orescence staining was conducted to evaluate cell morphology.

45 ociated beta-galactosidase activity and flat cell morphology.

46 g methods that match molecular expression to cell morphology.

47 um currents, and central, radial or vertical cell morphologies.

48 ng in adhesive asymmetries and non-hexagonal cell morphologies.

49 ear if or how myelodysplasia (abnormal blood cell morphology), a key MDS feature in humans, presents

50 Purbeta in cultured MEFs promoted changes in cell morphology, actin isoform expression, and cell migr

51 odulus that produced striking differences in cell morphology, actin organization, and membrane dynami

52 l effector proteins implicated in regulating cell morphology, adhesion, and migration in various cell

53 of the dioxin receptor (AhR) in maintaining cell morphology, adhesion, and migration.

54 fibroblasts induces changes in breast cancer cell morphology, adhesion, and motility that promote inv

55 not observed under these conditions, nor is cell morphology affected by alphavbeta6 expression.

56 d not affect cell viability; neither was the cells morphology affected as demonstrated by live cell i

57 Alterations in cell morphology after treatment of primary visceral SMCs

58 The sample fixation methodology preserves cell morphology, allows analysis in the ultrahigh vacuum

59 -1alpha-induced adhesion using shear stress, cell morphology alterations, and crawling on intercellul

60 Knockdown of TbeIF5A led to abnormal cell morphologies and detached flagella, suggesting that

61 aglpQ and DeltaphoD mutants revealed altered cell morphologies and effects on autolytic activity and

62 geneous adhesive behavior due to the diverse cell morphologies and membrane rigidities.

63 H3R17 methylation results in defective glial cell morphology and a sensory defect in a subpopulation.

64 Nicotine induced changes in cell morphology and ablate tight junctions consistent wi

65 cue of auditory/vestibular behavior and hair cell morphology and activity.

66 ment, thereby failing to support the Sertoli cell morphology and adhesion protein complexes (e.g., oc

67 Removal of blocking cargo also restores cell morphology and attenuates the ER-stress response.

68 ated particular longevity-related changes in cell morphology and characteristics, including critical

69 oaches permit the linking of connectivity to cell morphology and circuit function for particular cell

70 learing method for enhanced visualization of cell morphology and connections in neuronal and non-neur

71 tamine (24-48 hrs) produces gross changes in cell morphology and cytoskeletal architecture towards a

72 rotein that is involved in the regulation of cell morphology and cytoskeletal organization.

73 TEPA, a copper chelator, inhibited EMT-like cell morphology and cytoskeleton arrangement triggered b

74 Moreover, Sj7170 changed cell morphology and cytoskeleton of U87 cells by the GTP

75 en species, mitochondrial health, as well as cell morphology and determine that the hMSCs are minimal

76 We observed altered cell morphology and disrupted organization of F-actin in

77 -mesenchymal transition (EMT) as examined by cell morphology and EMT markers; knockdown or inhibiting

78 ) type 1 receptor, Ang II induced changes in cell morphology and expression of epithelial-to-mesenchy

79 ls from frame-to-frame, and characterize the cell morphology and fluorescence.

80 often involving complex alterations in host cell morphology and function.

81 d according to induced phenotypic changes of cell morphology and functionality measured by fluorescen

82 ensible, we use high-dimensional image-based cell morphology and gene expression profiles.

83 SLIP yields rich data sets on cell morphology and gene expression that illustrate the

84 lternatively, the documentation of a typical cell morphology and immunophenotype on blood cells coupl

85 ontrast, AR deletion in luminal cells alters cell morphology and induces transient over-proliferation

86 s are cytoskeletal filaments responsible for cell morphology and intracellular organization.

87 of developmental pathways specifying neural cell morphology and ion channel expression.

88 f cavities around MHs correlates with Muller cell morphology and is consistent with the hypothesis of

89 sly allows tracking the resulting changes in cell morphology and mechanics as well as measuring the f

90 her approaches for cancer diagnosis based on cell morphology and microscopy (biopsies) are too not co

91 The effects of ACM on tumor cell morphology and migration also depended on astrocyte

92 oteins play key roles not only in regulating cell morphology and migration but also in proliferation.

93 dition of exogenous TSP2 to WT cells induced cell morphology and migration rates that were similar to

94 tes the ability of PAK1 to induce changes in cell morphology and motility and to promote malignant tr

95 se (ROCK) has an essential role in governing cell morphology and motility, and increased ROCK activit

96 ys in generating the characteristic hook-end cell morphology and motility, have not been elucidated.

97 trix to the actin cytoskeleton and regulates cell morphology and motility.

98 ulators of cancer invasion via regulation of cell morphology and motility.

99 heir roles in epithelial phenotypes, such as cell morphology and movement.

100 d no effect on development of the embryo, or cell morphology and organization of auditory brainstem n

101 where it is responsible for proper epidermal cell morphology and overall plant growth.

102 constraints can influence three-dimensional cell morphology and packing within epithelial tissues.

103 where they function as master regulators of cell morphology and pathogenesis.

104 yze fluorescence intensity and localization, cell morphology and proliferation as well as other descr

105 ring which period they retained their normal cell morphology and proliferation rates.

106 ere reversible and accompanied by changes in cell morphology and pronounced reduction in both cell/ce

107 d atomic force microscopy were used to study cell morphology and propagation.

108 It contributes to the cell morphology and provides cell wall integrity against

109 uit analysis as well as analyzed the role of cell morphology and receptor internalization.

110 ters (cell membrane capacitance referring to cell morphology and seal resistance referring to adhesio

111 est, Rb dephosphorylation, flat and enlarged cell morphology and senescence-associated beta-galactosi

112 luding cell type distributions, variation in cell morphology and stomatal depth, differentiation of h

113 ents (IFs) are key players in the control of cell morphology and structure as well as in active proce

114 ard optical microscopy to examine changes in cell morphology and subcellular organization concomitant

115 nstitutes a biophysical stimulus that alters cell morphology and suppresses mesenchymal motility in h

116 Wor1/Mit1/Ryp1 protein controlled aspects of cell morphology and that movement of genes in and out of

117 affects gonococcal colony morphology but not cell morphology and that overexpression of ng1686 does n

118 al spindle position is determined largely by cell morphology and that spindles consistently center th

119 Furthermore, cell morphology and the arrangement of epidermal cell la

120 rescence (IF) microscopy adds information on cell morphology and the microenvironment that are not ob

121 Changes in cell morphology and the physical forces that occur durin

122 alladin, is essential for the maintenance of cell morphology and the regulation of cell movement.

123 It also rapidly affected CPE cell morphology and tight junction protein levels.

124 Cell death introduces alterations in cell morphology and tissue micro-structures that cause m

125 critical component in regulating endothelial cell morphology and vascular network formation.

126 Despite the central importance of MreB for cell morphology and viability, very little is known abou

127 2-dependent secretion is required for normal cell morphology and virulence in L. monocytogenes; howev

128 tematically analyzed cell cycle progression, cell morphology, and bud site selection after repression

129 ns in illumination, staining, imaging noise, cell morphology, and cell clustering.

130 arameters, such as gene expression profiles, cell morphology, and cytoskeleton arrangement, we demons

131 8 inhibited transferrin endocytosis, altered cell morphology, and decreased cell viability.

132 ular regulation influencing gene expression, cell morphology, and function.

133 ability were vacuolization, other changes in cell morphology, and increased macropinocytosis.

134 itical ERK effector that regulates motility, cell morphology, and invasiveness.

135 cell wall is one major determinant of plant cell morphology, and is an attractive bioresource.

136 TMIGD1 controls cell migration, cell morphology, and protects renal epithelial cells fro

137 ffector of EphB2/ephrinB signaling, controls cell morphology, and thereby cell repulsion.

138 The mutation in divK additionally affected cell morphology, and this effect was complementable by a

139 of later biosynthetic steps contributing to cell morphology, antibiotic resistance, and pathogenesis

140 zation and CT694-induced alterations in host cell morphology are dependent on an N-terminal domain.

141 inside the cell, the cell velocity, and the cell morphology are determined by the integration of act

142 w that neither channel noise nor a realistic cell morphology are responsible for the rate dependent s

143 nonbulky proteins, ER stress, and defective cell morphology are secondary consequences of bulky carg

144 (SEM) and in depth (IBA) information on the cell morphology as well as on the exact localization of

145 SICM can be used to analyze cell morphology at nanoscale, follow membrane dynamics,

146 d cells (RBCs) to represent different sickle cell morphologies based on a simulated annealing procedu

147 senescence is characterized by a large flat cell morphology, beta-gal staining and irreversible loss

148 on caused reduced cell growth and defects in cell morphology, both of which were suppressed by overex

149 not impact microtubule network integrity or cell morphology but contributed to microtubule stabiliza

150 e the matrilin-1 knockdown had no effects on cell morphology, but increased cell death was observed.

151 The cortex regulates cell morphology by controlling cellular mechanical prope

152 the marginal band, that flattens the overall cell morphology by pushing on the cell cortex.

153 Moreover, BrdU(+) cells with plasma cell morphology can be detected for 10 years after vacci

154 ed with collagen matrices including stellate cell morphologies, cell-mediated realignment of fibres,

155 other aspects of cellular behavior, such as cell morphology, cell mechanics, cell motility, cell sig

156 eas beta-lactams were responsible for strong cell morphology changes (spheroplast with imipenem, fila

157 including membrane traffic, yet its role in cell morphology changes, such as the budding to filament

158 ) induces constitutive activation of FAK and cell morphology changes, which are independent of SRC fa

159 ed to the plasma membrane to orchestrate the cell morphology changes.

160 s a powerful tool for automated detection of cell morphology changes.

161 muscle diseases present with aberrant muscle cell morphologies characterized by smaller myofibers wit

162 r data reveal a regulatory role of Fbxl10 in cell morphology, chemokine expression, and the metabolic

163 Our cell morphologies compare favorably with experimental ti

164 Cell morphology complexity assayed by fractal dimension

165 t in vitro or in ovo induces changes in hair cell morphologies consistent with a loss of tonotopic or

166 led to a more rounded, less mesenchymal-like cell morphology, consistent with decreased metastatic pr

167 We propose that these changes in ganglion cell morphology could impact the function of individual

168 nstrated the ability for inducing changes in cell morphology, cytoskeletal fiber orientation and chan

169 for neuronal differentiation and for normal cell morphology, cytoskeletal organization, proliferatio

170 al axis, which correlate with alterations in cell morphology, cytoskeleton and cell-cell contacts in

171 lls overexpressing Cpc2p display substantial cell morphology defects, disorientation of septum format

172 These changes in cell morphology deform the developing epithelial tube to

173 e screening data sets on nuclear and mitotic cell morphologies demonstrates that CellCognition Explor

174 These dramatic changes in cell morphology depend on the auto-phosphorylation of ty

175 res that distinguish this model are variable cell morphology described by a collection of particles c

176 Loss of capzb affects cell morphology, differentiation and neural crest migrat

177 seven bacterial strains with a wide range of cell morphology, dimension, and surface characteristics.

178 d myosin-IIA can suitably couple nuclear and cell morphology downstream of matrix mechanics.

179 nt11 ligand and regulated cell migration and cell morphology during gastrulation.

180 aracterized by abnormal cortical lamination, cell morphology (e.g., cytomegaly), and cellular polarit

181 ular protrusions are a ubiquitous feature of cell morphology, e.g., filopodia and microvilli, serving

182 ed by progressive alterations in endothelial cell morphology, excrescences (guttae) and thickening of

183 le (EpCAM) independent fluid biopsy based on cell morphology for CTC detection and enumeration (defin

184 e describe a method for extracting realistic cell morphologies from fluorescence microscopy images to

185 Thus, accurate determination of cell morphology from rapidly moving molecules requires t

186 a readout (e.g., fluorescence, luminescence, cell morphology) from each cell in the population.

187 t also controls a dramatic transformation in cell morphology, from cuboidal to the eponymous stellate

188 expression controls with dynamic changes in cell morphology, function, and control.

189 mellipodia formation, and membrane ruffling, cell morphologies generated by active Rac1.

190 Our work suggests that cell morphology has a strong impact within microbial com

191 These data indicate that cell morphology has different requirements on cytoskelet

192 positive cells did not display the typical B cell morphology, having in general a more dendritic cell

193 tron microscopy to investigate variations in cell morphology, immunostaining, and the distribution of

194 d keratinocytes (p.Ile482Lys) showed altered cell morphology, impaired tight junctions, adhesion defe

195 in cells has profound effects on MDA-MB-231 cell morphology, implying the presence of a pool of unme

196 r bacterial motility in Escherichia coli and cell morphology in Bacillus subtilis.

197 Changes in cell morphology in budding yeast, mediated by polarizati

198 nt to stabilize focal adhesions and maintain cell morphology in infected cells and cells infected wit

199 es, including proper development of regional cell morphology in Kupffer's vesicle and the establishme

200 ing protein 4, and assumption of a dendritic cell morphology in response to anti-CD40 plus IL-4 were

201 liferation, TGF-beta expression, and altered cell morphology in SSCT cells.

202 single neurons, the role of detailed single cell morphology in the population has not been studied q

203 reprogramming of acinar cells and regulates cell morphology in vivo and in vitro.

204 on neuronal proteostasis and maintenance of cell morphology in vivo.

205 skeletal organelles involved in establishing cell morphology, including the flagella connector, flage

206 y of mammalian cells and profound changes in cell morphology, including the loss of a single leading

207 ls specific alterations in cell motility and cell morphology indicating that the MG200-MG491 interact

208 as accelerated virus-induced alterations to cell morphology, indicating that TRAF2 influences early

209 e loss of MreB function, severely perturbing cell morphology, inhibiting growth and inducing cell lys

210 f cell biological effects, including altered cell morphology, inhibition of cell growth and, in some

211 of FAK and Nanog cross-regulation on cancer cell morphology, invasion, and growth that plays a signi

212 In addition, bacterial cell morphology is adaptable to changes in environmental

213 c images is challenging, especially when the cell morphology is complex.

214 In rod-shaped bacteria, cell morphology is correlated with the replication rate.

215 he cellular and molecular control of a lobed cell morphology is currently thought to involve PIN-FORM

216 y, high-throughput microscopy indicated that cell morphology is relatively insensitive to mild knockd

217 One of the most fundamental changes in cell morphology is the ingression of a plasma membrane f

218 ocyte injury results in a dramatic change in cell morphology known as foot process effacement.

219 rocess where a drastic change of endothelial cell morphology leads to the formation of blood stem and

220 an essential role of TbKIN-C in maintaining cell morphology, likely through regulating microtubule d

221 HMDMs were investigated by analyzing the cell morphology, LPS-induced cytokine profile, surface m

222 by microgrooves, suggesting that changes in cell morphology may be responsible for modulation of the

223 ovative approach to preserve human red-blood-cell morphology, mechanics, and function following vitri

224 which results in irreversible disruption of cell morphology, mechanics, and function.

225 Furthermore, the role of Hfq in the cell morphology, metabolism, cell wall integrity, resist

226 hesion size and distribution, thus affecting cell morphology, migration and ultimately localization.

227 nformation about individual cells, including cell morphology, molecular content and local cell densit

228 tion is frequently accompanied by changes in cell morphology (morphodynamics) on a range of spatial a

229 levels in bladder cancer cell lines affects cell morphology, motility and proliferation.

230 Cell morphology, neurotransmission, and cellular process

231 hing and quantitatively characterizing brain cell morphologies noninvasively.

232 explore the possibility of quantifying brain cell morphology noninvasively.

233 Our results demonstrate changes in cell morphology, observed as cytoskeleton protrusions-i.

234 nts to grow in nude mice with characteristic cell morphology of anaplastic thyroid cancer (ATC).

235 Fluorescent microscopy was used to assess cell morphology of different cell types which were stain

236 eening to identify molecules that affect the cell morphology of tobacco BY-2 cells.

237 cells in confluent samples, handles various cell morphologies, offers algorithms for quantitative an

238 es focused on developmental organization and cell morphology often use this layered stratification to

239 To systematically interrogate the impact of cell morphology on bacterial physiology, we used fluores

240 er, these results highlight the influence of cell morphology on fate determination processes.

241 mediated cellular traction, independently of cell morphology or matrix mechanics.

242 hroughput, they produce no information about cell morphology or spatial resolution offered by microsc

243 acrophages or dendritic cells (DCs) based on cell morphology, phenotype, or select functional propert

244 n motility (myosin light chain 1, myosin A), cell morphology (PhIL1), and host cell invasion (apical

245 At least ten distinct subregions differ in cell morphology, physiology and the expression of hundre

246 neurons have distinguishable differences in cell morphology, physiology, and synaptic circuit connec

247 , nor SSV2 induced any detrimental effect on cell morphology, plasma membrane and mitochondrial funct

248 features modulate cell behaviour, including cell morphology, proliferation and differentiation.

249 ombined mechano-topographical stimuli on PDL cell morphology, proliferation, and osteogenic and ligam

250 reveal new roles for SPAG6 in modulation of cell morphology, proliferation, migration, and ciliogene

251 ective concentrations, UNC0638 did not alter cell morphology, proliferation, or erythroid differentia

252 e cell state characterized by differences in cell morphology, proliferative kinetics, and tumor-initi

253 Further, ADAR1 knockdown altered cell morphology, promoted in vitro proliferation, and ma

254 Here we reported that MSI1 alters cell morphology, promotes cell migration, and increases

255 es of their microenvironment that can affect cell morphology, protein levels and localization, gene e

256 odology to quantify the relationship between cell morphology, pulse frequency, and electroporation re

257 Revealing VIP+ cell morphologies, receptive fields and synaptic connect

258 of integrin beta1 led to alterations in beta-cell morphology, reduced insulin gene expression, and en

259 ionally, betaIII-tubulin suppression altered cell morphology, reduced tumor spheroid outgrowth, and i

260 adosome under anaerobic conditions regulates cell morphology, resulting in Ecoli MG1655 cell filament

261 orphology as axons fragment, and how Schwann cell morphology reverses once regenerating growth cones

262 l myelopoiesis (TAM) disorder based on blood cell morphology review and screening for GATA1 mutations

263 anced segmentation algorithms, variations in cell morphology, sample preparation, and acquisition set

264 DM96 with the rates for a routine red blood cell morphology scan.

265 alyzed by CellaVision and microscopy for red cell morphology scans.

266 e, including root branching, root epithelial cell morphology, seed germination, and leaf conductance.

267 ative injury status (50 mum Fe(2+)) in which cell morphology showed changes typical of neuronal damag

268 ochastic, multicolor labeling for individual cell morphology studies.

269 ell division of C. trachomatis, although the cell morphology suggested differentiation into a metabol

270 relates with membrane defects and changes in cell morphology, suggesting a localized alteration in th

271 amically controlled independently of overall cell morphology, suggesting that protrusion formation is

272 ession leads to defective cortical pyramidal cell morphology, synaptic plasticity deficits, and alter

273 bandgap copolymers calls for optimized solar cell morphologies that are fundamentally different from

274 ls persisting for over 700 days demonstrated cell morphologies that were very small in size, with a h

275 This generates a distinctive repair cell morphology that is favorable for the formation of t

276 We showed light-inducible control of cell morphology that resulted in a substantial increase

277 ation is involved in invasion, motility, and cell morphology, the proteins that utilize this PTM rema

278 of the pyrazolobenzothiazines tested altered cell morphology, this undesired aspect was further inves

279 Despite similarities in neuromast hair cell morphology, three classes of these cells can be dis

280 e, showed a limited response of the Neuro-2a cell morphology to EGF stimulation.

281 trate stiffness, focal adhesion density, and cell morphology to show that the total amount of work a

282 features, such as tumor size, location, and cell morphology, to the slightly more sophisticated coun

283 ,25-(OH)2D3 at a time-dependent manner alter cell morphology towards osteoblast-associated characteri

284 energetically costly changes in P. mirabilis cell morphology translate into an advantage for adapting

285 We compete cells with different cell morphologies under a range of conditions and ask ho

286 ntration of free endotoxin released, and the cell morphology under light microscope.

287 and aggregation of ZnO NPs compromised algal cell morphology, viability, and membrane integrity, resu

288 Cell morphology was assessed by fluorescent staining.

289 Altered cell morphology was assigned to the viral M25 gene.

290 Endothelial cell morphology was evaluated by subjective scoring (ran

291 The cell morphology was imaged by confocal microscopy and an

292 nscription of pluripotency markers, the stem cell morphology was not significantly affected.

293 A striking change of cell morphology was observed with a rounded phenotype ar

294 YAP activation with smaller and more rounded cell morphologies were induced in hMSCs.

295 al effects on HEK proliferation kinetics and cell morphology were also assessed by using the trypan b

296 PIP5K6 expression on pollen tube growth and cell morphology were attenuated by coexpression of MPK6

297 Early, specific changes in endothelial cell morphology were found to "announce" an upcoming all

298 roduce three complementary models of rounded cell morphologies with a prescribed excess surface area.

299 ctive motility but a fundamentally different cell morphology with highly flexible snake-like swarming

300 unique among bacteria based on their helical cell morphology with hook-shaped ends and the presence o