ライフサイエンスコーパス: cell movement

1 ilamentous actin staining, indicating active cell movement.

2 vision, but that the number increases due to cell movement.

3 y promoting cell adherence and by inhibiting cell movement.

4 teins needed for virion assembly and cell-to-cell movement.

5 extracellular matrix, processes critical for cell movement.

6 ridge, abolished virion assembly and cell-to-cell movement.

7 which we now show involves concerted active cell movement.

8 in ratio that drives membrane protrusion and cell movement.

9 surface beta1 integrin required for forward cell movement.

10 vascular development, cell proliferation and cell movement.

11 but not cell velocity or the persistence of cell movement.

12 cluding cell division) with the mechanics of cell movement.

13 dissociation accompanied by FA turnover and cell movement.

14 nce of cell morphology and the regulation of cell movement.

15 ion number, cell-cell adhesion and polarized cell movement.

16 led similarities to cancer, development, and cell movement.

17 ional tension and inhibiting intraepithelial cell movement.

18 on, or enhanced virion stability and cell-to-cell movement.

19 e lineage boundary at the MHB by restricting cell movement.

20 ection of cell adhesion, cell signaling, and cell movement.

21 tion site, which is required for Plk4-driven cell movement.

22 to changes in ClC-3 conductance required for cell movement.

23 nt5b-induced Ca(2+) activity and directional cell movement.

24 Periodontal remodeling requires coordinated cell movement.

25 oEr2 ligands altered the effect of ApoEr2 on cell movement.

26 e cell motility and control the direction of cell movement.

27 bout the physics and biochemistry underlying cell movement.

28 ion for efficient leading edge formation and cell movement.

29 ing coordinated cytoskeletal dynamics during cell movement.

30 ma, achieve remarkable speeds of directional cell movement.

31 exclusion limits and promoting virus cell-to-cell movement.

32 namics, thereby determining the direction of cell movement.

33 ntrol of cytoskeleton remodeling involved in cell movement.

34 proliferation, in contrast to its effects on cell movement.

35 lk4 and Arp2 in mediating Plk4-driven cancer cell movement.

36 HDAC9 in GT1-7 cells increased apoptosis and cell movement.

37 r-789 phosphorylation to positively regulate cell movement.

38 C cells, AMPK blockade resulted in increased cell movement.

39 gulators of PIP3-sensitive mRNAs involved in cell movement.

40 tes to alter plasmodesmata for virus cell-to-cell movement.

41 nduced apoptosis and impaired basal neuronal cell movement.

42 (ECM) proteins serve as cues for directional cell movement.

43 egulate the fundamental processes that drive cell movement.

44 y that enables proper neuronal signaling and cell movement.

45 ses within the tissue to regulate collective cell movements.

46 ipital tissues participate in well-conserved cell movements.

47 s as a direct and essential mediator for C&E cell movements.

48 organization underlying PCP and gastrulation cell movements.

49 ich it regulates cell polarity and polarized cell movements.

50 irectionality as well as the coordination of cell movements.

51 ut not XTRPM7 disrupted radial intercalation cell movements.

52 n levels, suggesting a role in morphogenetic cell movements.

53 actin filaments is a major driving force for cell movements.

54 ombination of changes in gene expression and cell movements.

55 ophila pupal eye is characterized by precise cell movements.

56 f patterning signals and major morphogenetic cell movements.

57 erved role for Git2 in controlling embryonic cell movements.

58 ly reported with impaired integrin-dependent cell movements.

59 pecial emphasis on individual and collective cell movements.

60 skeleton and power the membrane machinery of cell movements.

61 for organised organ growth in the absence of cell movements.

62 WNT5A ligand, leading to JNK activation and cell movements.

63 es place via a series of dramatic collective cell movements.

64 in individual cell behaviors and collective cell movements.

65 -cell signaling, which consequently prevents cell movements across the MHB.

66 necessity of MAPK activation for coordinated cell movement after damage.

67 The mechanisms facilitating cell movement against the gradient have not been defined

68 that coordinate cytoskeletal events to bias cell movement along a chemoattractant gradient.

69 recent observations of extensive endothelial cell movement along growing blood vessels.

70 hs in the matrix and the temporal control of cell movements along these paths.

71 receptor for CXCL12, exhibit defective GnRH cell movement and a significant reduction in their numbe

72 renewed interest in the relationship between cell movement and affinity maturation.

73 in controlling FAK activation during planar cell movement and amoeboid motility during extracellular

74 ism and also cytoskeletal functions, such as cell movement and attachment.

75 during different stages of enamel formation; cell movement and attachment; regulation of ion and prot

76 , beta-catenin-independent pathway modulates cell movement and behavior.

77 studies indicate that DeltaNp63alpha affects cell movement and can reverse the increase of cell motil

78 he central role of cAMP in the regulation of cell movement and cell differentiation.

79 a stable demarcation line can form when both cell movement and cell growth cease at low nutrient leve

80 in the immune system have been implicated in cell movement and cell-cell interaction during the cours

81 a crucial control point for both collective cell movement and ciliogenesis in Xenopus embryos.

82 in these syndromes also influence collective cell movement and ciliogenesis.

83 Cell movement and cytokinesis are facilitated by contrac

84 plete, implicating a potential separation of cell movement and differentiation during this process.

85 We quantified defects in cell lifetime, cell movement and division axis in end-3(-) embryos, whi

86 ong to group I of the PAK family and control cell movement and division.

87 GF)-beta family ligand Nodal, regulates both cell movement and EMT during embryonic development.

88 ficient Tregs lost the ability to modulate T cell movement and failed to inhibit the T cell-dendritic

89 sion of about 1300 genes, mostly involved in cell movement and growth, and specifically affected meta

90 f the cytosolic protein actin is critical to cell movement and host cell invasion by the malaria para

91 ommonly increased in both GSC lines involved cell movement and included a number of genes that have b

92 infiltration, we analyzed the dynamics of T cell movement and interactions within individual islets

93 ally different behaviour, one showing robust cell movement and intercalation (in which the AVE migrat

94 ctin cytoskeleton, which is also involved in cell movement and invasion, were affected.

95 that coordinate Dictyostelium morphogenetic cell movement and is highly expressed at the organizing

96 poptosis rates were observed based solely on cell movement and local conditions.

97 the underlying mechanisms that regulate PDAC cell movement and metastasis remain little understood.

98 characteristics, including overexpression of cell movement and migration-associated genes in the Spin

99 d one that is static, with relatively little cell movement and mixing.

100 ngrailed 1 (En1) as a necessary regulator of cell movement and NeuC/Mes lineage boundary positioning

101 The ratio between the distance of red blood cell movement and plasma separation is the criterion for

102 owed the role of Rho-GDI2 in regulating both cell movement and proliferation.

103 Its modular structure, the lack of cell movement and relative accessibility to microscopic

104 Thus, SHEP1 orchestrates marginal zone B-cell movement and retention as a key downstream effector

105 ievable with soluble EGF, we examined single-cell movement and signaling in human immortalized HaCaT

106 s IIa HDAC9 with Class IIb HDAC6 to modulate cell movement and survival in GnRH neurons.

107 often lacks real-time monitoring of vertical cell movement and systematically controlled chemotactic

108 CV) was necessary for efficient TVCV cell-to-cell movement and systemic infection in Nicotiana bentha

109 ts that occur during gastrulation, including cell movement and the activation of some endodermal targ

110 quired for TGB1 self-interaction and cell-to-cell movement and the amino-terminal domain required for

111 steering explains the local coordination of cell movement and the maintenance of monolayer integrity

112 Furthermore, subtle relationships between cell movement and the positive and negative interactions

113 lls, such as alterations in the direction of cell movement and the regulation of gene transcription,

114 clude that microtubules act both to restrain cell movement and to establish directionality.

115 ut dysregulates a subset of genes related to cell movement and transport.

116 d used time lapse microscopy to characterize cell movements and behavior in wild type and mutant tail

117 K is necessary for the proper orientation of cell movements and cell division.

118 signaling plays a pivotal role in regulating cell movements and lineage induction during gastrulation

119 Moreover, coordinated cell movements and physical cell-cell interactions are r

120 ial cues that induce systematically oriented cell movements and promote tissue elongation.

121 Thus, both Ret-dependent cell movements and Ret-independent changes in the Wolffi

122 ulation, disrupting both the directedness of cell movements and the coherence of movements among neig

123 e first to demonstrate that ApoEr2 regulates cell movement, and both X11alpha and Reelin enhance this

124 Actin filaments play an essential role in cell movement, and many posttranslational modifications

125 arized protrusive cell activity, directional cell movement, and oriented cell division and is crucial

126 promotes cell-cell interaction, limits lone cell movement, and slows swarm expansion.

127 rotation create a permissive environment for cell movement, and that uniform levels in these two popu

128 egulates the formation of tissue boundaries, cell movements, and signaling.

129 -mediated vesicle induction to virus cell-to-cell movement are discussed.

130 rate gastrulation, convergence and extension cell movements are coordinated with the anteroposterior

131 Coordinated cell movements are crucial for vertebrate gastrulation a

132 Moreover, cell movements are highly correlated and in phase with E

133 Collective cell movements are integral to biological processes such

134 These cell movements are spearheaded by the occipital lateral

135 quiring human intervention to compensate for cell movement as a patch pipette approaches a targeted n

136 t TAZ, is strictly required for WSS-enhanced cell movement, as blockade of YAP1, TEAD1-4 or the YAP1-

137 in transcript splicing were also detected in cell movement associated genes including Cd44.

138 irectionality, indicating that the posterior cell movements associated with axis elongation in the PS

139 and a resultant failure of hearts to undergo cell movements associated with cardiac formation.

140 e metabolite concentration caused changes in cell movements at gastrulation that also altered the tis

141 chemokine receptors (cCKRs) directly control cell movement; atypical chemokine receptors (ACKRs) regu

142 tERF53 also has a function to regulate guard-cell movement because the stomatal aperture of AtERF53 o

143 Cell movement begins with a leading edge protrusion, whi

144 pathogens, and they define requirements for cell movement between parenchyma and SCS in what we spec

145 In the spleen, B cell movement between the blood-rich marginal zone and f

146 n zones that permit axon extension but limit cell movement between the CNS and PNS.

147 Cell movement biased by a chemical gradient, or chemotax

148 r resulted in an additive effect to increase cell movement but did not alter the acetylation of alpha

149 es that AprA affects the directional bias of cell movement, but not cell velocity or the persistence

150 raspanins CD9 and CD151, which also regulate cell movement, but not for the association between KAI1/

151 er Pyr or Ths was sufficient to redirect CVM cell movement, but only when the endogenous source of th

152 to regulate both ciliogenesis and collective cell movement, but the underlying mechanism is unknown.

153 rphogenesis depends on a series of concerted cell movements; but the roles of cell adhesion signaling

154 ate that the dmim product regulates directed cell movement by inhibiting endocytosis and antagonizing

155 e T4P is the S motility motor, and it powers cell movement by retraction.

156 strategy that automatically compensates for cell movement by tracking cell position and adjusting pi

157 cally, we show that Celsr1 regulates dynamic cell movements by inhibiting stabilization of VE-cadheri

158 We show that glioblastoma cell-cell movement can be described as Brownian motion biased

159 of the models showed that cytokine induced T cell movement can explain the very slow decline of CD4+

160 During morphogenesis, cell movements, cell divisions and cell death work toget

161 his arrangement is the result of coordinated cell movements, cell shape changes, and the organisation

162 grams in MCCs, regulating genes that control cell movement, ciliogenesis, and cilia function.

163 OCM depends instead on a complex set of cell movements coordinated between the prospective neura

164 ll transplantation experiments show that the cell movement defect is cell autonomous.

165 e inhibitor for myosin II activity-exhibited cell movement defects similar to git2a knockdown embryos

166 minus of CP functions as a dedicated cell-to-cell movement determinant.

167 dentified, but are thought to control either cell movement directly or the patterning of their axonal

168 Fundamental cellular processes such as cell movement, division or food uptake critically depend

169 ll polarity (PCP) pathway regulates directed cell movement during development and was recently found

170 ransition (EMT) is a core process underlying cell movement during embryonic development and morphogen

171 lates cell alignment required for collective cell movement during embryonic development.

172 0b, is required for convergent and extension cell movement during gastrulation.

173 n algorithms to track and analyze individual cell movements during expansion of P. aeruginosa biofilm

174 led an essential role for Git2a in zebrafish cell movements during gastrulation.

175 are able to track cell lineages and dynamic cell movements during germ cell differentiation.

176 Directional cell movements during morphogenesis require the coordina

177 that loss of either pcdh 19 or ncad impairs cell movements during neurulation, disrupting both the d

178 adhesion, cell proliferation, cell death and cell movements during otic development.

179 he primary regulator of convergent extension cell movements during vertebrate development, but the ro

180 y (PCP) signaling pathway governs collective cell movements during vertebrate embryogenesis, and cert

181 TRPM7 and Mg(2+) in Rac-dependent polarized cell movements during vertebrate gastrulation.

182 usly un-recognized role for an Rfx factor in cell movement, finding that Rfx2 cell-autonomously contr

183 scopy to monitor PI3K signaling dynamics and cell movements for extended periods.

184 T and B cell movement from blood into lymph nodes is reduced in

185 restricted to the DZ, with a net vector of B cell movement from the DZ to the LZ.

186 ases of precursor and migratory neural crest cell movements from the neural keel stage to times of ac

187 rs, and cell-cell cohesion during collective cell movements, further highlight that tension-dependent

188 TGB1 self-interaction is needed for cell-to-cell movement, importin-alpha-mediated nucleolar targeti

189 Chemokines like CXCL12 regulate cell movement in a biphasic pattern, with peak migration

190 ultured endothelial cells abrogated directed cell movement in a wound healing assay.

191 tested whether ApoEr2 played a novel role in cell movement in a wound-healing assay.

192 n and the basis of all cell shape change and cell movement in development.

193 ary approach to further our understanding of cell movement in epithelia.

194 nctional significance of this interaction on cell movement in MCF 10A epithelial cells.

195 tina in vivo and in correlation with dynamic cell movement in mouse embryonic stem cell-derived sprou

196 een responses to differentiation signals and cell movement in patterning based on 'salt and pepper' d

197 slocation to the rice cytoplasm, and cell-to-cell movement in rice.

198 that Dab1 activation is sufficient to orient cell movement in the absence of other signals.

199 dure to study marginal zone and follicular B-cell movement in the live mouse spleen.

200 l determinant of actin assembly and directed cell movement in the vascular endothelium.

201 igration, we develop a mathematical model of cell movement in the VE.

202 LGI1 protein acts as a suppressive agent for cell movement in this assay.

203 Matrix metalloproteinases (MMPs) facilitate cell movement in various tissues during development, and

204 anisms by which actomyosin drives collective cell movement in vertebrate embryos.

205 To identify new genes that regulate cell movement in vivo, we screened lethal mutations on c

206 ell adhesion, which contributes to defective cell movements in the gastrula.

207 ow that Ret/Etv4 signaling promotes directed cell movements in the ureteric bud tips, and suggest a m

208 by the Ret receptor tyrosine kinase promotes cell movements in the Wolffian duct that give rise to th

209 the whole animal level, they regulate early cell movements in zebrafish development.

210 a more than twofold increase in the rate of cell movement, in part due to a significant increase in

211 ll cortex and disrupted events essential for cell movement, including actin dynamics, lamellipodia pr

212 rrepresented biological functional group was cell movement, including E-selectin, which was coordinat

213 be responsible for the variety of nonmuscle cell movements, including the "saltatory cytoplasmic par

214 Furthermore, embryos treated with cell movement inhibitors (blebbistatin or RhoK inhibitor

215 We also find that directed cell movement initiated by Rac1b is dependent upon p120.

216 ndicates a mechanism for the coordination of cell movements initiated by receptor signaling.

217 ction mice, we demonstrated that mesothelial cell movement into the lung requires the direct action o

218 Instead, T cell movement is better described as a correlated random

219 ed analysis has definitively shown whether T cell movement is consistent with Brownian motion.

220 Collective cell movement is critical to the emergent properties of

221 B1 cell movement is largely governed by Cxc ligand 13 (Cxcl

222 , the interaction between ClC-3 activity and cell movement is poorly understood.

223 echniques, we demonstrate that the vector of cell movement is regulated by localised epidermal growth

224 olve an invasive phenotype if the consequent cell movement is rewarded by proliferation.

225 Cell movement is thought to involve motor complexes comp

226 Directional cell movement is universally required for tissue morphog

227 isting knowledge on the molecular control of cell movements, it is unclear how the different observed

228 which generates actomyosin-based tension and cell movement, JNK signaling is required to establish mi

229 cs has significant influence on the speed of cell movement, kinetics of mutation propagation, and sen

230 n fixing the body plan: it controls epiblast cell movements leading to primitive streak formation, ge

231 bud tips, and suggest a model in which these cell movements mediate branching morphogenesis.

232 and are widely studied for their effects on cell movement, navigation, angiogenesis, immunology and

233 mplexes present on ameloblasts to foster the cell movement necessary for formation of the decussating

234 zing mitotic profiles, we found that neither cell movement nor oriented cell division could explain t

235 derm) from a single sheet, while large scale cell movements occur across the entire embryo.

236 Extensive cell movements occur during zebrafish optic vesicle morp

237 Zebrafish gastrulation cell movements occur in the context of dynamic changes i

238 course of animal morphogenesis, large-scale cell movements occur, which involve the rearrangement, m

239 light cues are processed to regulate cell-to-cell movement of auxin to allow establishment of a trans

240 tory effect of RFA expression on the cell-to-cell movement of Bean dwarf mosaic virus, a single-stran

241 V failed to complement the defective cell-to-cell movement of BMV.

242 P kinase signalling is important for cell-to-cell movement of invasive hyphae in M. oryzae.

243 Demonstrating cell-to-cell movement of mitochondria reconstructs the evolution

244 We report cell-to-cell movement of mitochondria through a graft junction.

245 Here, we present evidence for cell-to-cell movement of the entire 161-kb plastid genome in the

246 Directed cell-to-cell movement of the plant growth hormone auxin is often

247 matic morphogenetic processes, including the cell movements of gastrulation, epiboly and dorsal conve

248 In addition to cell divisions, coordinated cell movements of the progeny allow the rapid expansion

249 gulation of alpha5beta1-mediated endothelial cell movement on fibronectin.

250 s not due to defects in replication, cell-to-cell movement, or virion assembly.

251 Also, by considering the kinetics of the cell movement, our model predicts a biphasic invasivenes

252 d3 depletion inhibited collective and border cell movement out from spheroids in a ROCK1/2-dependent

253 tastasis through down-regulation of multiple cell movement pathways by regulating transcript levels a

254 caffold protein is an essential regulator of cell movement processes required for normal eye developm

255 Tight regulation of Schwann cell movement, proliferation and differentiation is esse

256 rce are present, increased directionality of cell movement promotes chemotaxis.

257 r of the plant cell wall by encoding cell-to-cell movement proteins (MPs), which direct newly replica

258 LCuV) and Tobacco mosaic virus (TMV) cell-to-cell movement proteins.

259 ic expression domains as a passive effect of cell movements represents an alternative strategy for br

260 nuclear localization, whereas the effects on cell movement required a cytoplasmic site of action.

261 Cell movement requires the coordinated reception, integr

262 Efficient cell movement requires the dynamic regulation of focal a

263 gration because perturbations to these early cell movements result in the alteration of specific fate

264 ogenesis, and it is comprised of directional cell movement resulting from the polarization and reorga

265 icate that lamellipodia are not required for cell movement, suggesting an alternate mechanism.

266 virion assembly but are required for cell-to-cell movement, suggesting that the C terminus of CP func

267 and ubiquitous production of Toddler promote cell movement, suggesting that Toddler is neither an att

268 of Xenopus Amer2 blocks convergent extension cell movements, suggesting that the Amer2-EB1-APC comple

269 ding motility, a substrate-dependent form of cell movement that underpins the protozoan parasite's ab

270 ermal-myocardial communication can guide the cell movements that initiate cardiac morphogenesis.

271 of the cytoskeleton, accounting for precise cell movements that organize the functional retinal fiel

272 talloproteinase/PTK7 axis are detrimental to cell movements that shape the body plan and that chz rep

273 w that the nucleus dictates the direction of cell movement through mechanical guidance by its environ

274 for export to the cell periphery and cell-to-cell movement through plasmodesmata.

275 Directional cell movement through tissues is critical for multiple b

276 cover previously unappreciated long-distance cell movements throughout the life cycle of the hair fol

277 is requisite for TSWV infection and cell-to-cell movement; thus, this behavior is most likely to est

278 ssed, but, in normal conditions, facilitates cell movement to locally prepatterned sources of FGF.

279 c cells, subcapsular sinus macrophages, or B cell movement to the subcapsular sinus.

280 Medulla cortex cells follow two patterns of cell movements to acquire their final position: first, n

281 Integrating individual cell movements to create tissue-level shape change is es

282 ange of defects, from aberrant morphogenetic cell movements to failure to correctly orient structures

283 es, from fast subcellular rearrangements and cell movements to slow structural changes at the whole-o

284 electron shuttle and an attractant to direct cell movement toward local sources of insoluble electron

285 During vertebrate neurulation, extensive cell movements transform the flat neural plate into the

286 ients correlated positively with the rate of cell movement under a variety of pharmacological treatme

287 ngiogenesis, and incorporation of individual cell movement using a hybrid continuum-discrete approach

288 ved to act as permissive cues for vertebrate cell movement via Frizzled (Fz).

289 and function for the WRAMP structure during cell movement were not determined.

290 of PIK3CA may also stimulate intraepithelial cell movement, which could contribute to spread of cells

291 ed for efficient virion assembly and cell-to-cell movement, while the C-terminal 65 amino acids are d

292 In brief, cell movement will lead to a significant technical (samp

293 on and branching of their leading processes, cell movement with axon specification and extension, swi

294 Coordination of cell movement with cell differentiation is a major feat

295 Inhibiting basal-type cell movement with clinically relevant drugs blocked inv

296 We show that the direction of cell movement with respect to the cell-cell contact is c

297 tes is driven by a proximodistal gradient of cell movement, with WNT and FGF activities controlling d

298 and may modulate intercellular adhesion and cell movement within in epithelia during development and

299 t previously unknown, stimulus for directing cell movement within porous extracellular matrix.

300 pplied to more complex systems of collective cell movement without prior knowledge of the cellular ma