ライフサイエンスコーパス: cell phagocytosis

1 G) and N297D/S298A-IYG optimally drove tumor cell phagocytosis.

2 or I, thus eliciting highly selective cancer cell phagocytosis.

3 atelet and endothelial activation and immune cell phagocytosis.

4 demonstrated their ability to promote target cell phagocytosis.

5 O mice displayed enhanced rates of apoptotic-cell phagocytosis.

6 the efficiency of serum-dependent apoptotic cell phagocytosis.

7 ma-hemorrhage-induced suppression of Kupffer cell phagocytosis.

8 hich sublethal oxidative stress inhibits RPE cell phagocytosis.

9 gulation of the cytoskeleton during stellate cell phagocytosis.

10 tissues and a macrophage defect in apoptotic cell phagocytosis.

11 e are capable of infection and inducing host cell phagocytosis.

12 lls are deleted through apoptosis or Sertoli cell phagocytosis.

13 ot appear related to inactivation of Kupffer cell phagocytosis.

14 blocking it with antibodies leads to cancer cell phagocytosis.

15 with GdCl3, which is known to block Kupffer cell phagocytosis and antigen processing, the spontaneou

16 and blockade of its function leads to tumor cell phagocytosis and elimination.

17 e will outline the consequences of apoptotic cell phagocytosis and illustrate how apoptotic cells cou

18 receptor tyrosine kinase mediates apoptotic cell phagocytosis and modulates macrophage cytokine prod

19 pha2 but not alpha1 AMPK was involved in RPE cell phagocytosis and that activation of alpha2 AMPK con

20 ta5 receptor is required for lens epithelial cell phagocytosis and that UV light treatment of lens ep

21 s a downregulatory signal that inhibits host cell phagocytosis, and CD47 therefore functions as a "do

22 or cells and induction of antibody-dependent cell phagocytosis as one of the primary mechanisms of ac

23 Inhibition of RPE cell phagocytosis by activation of alpha2 AMPK was assoc

24 Inhibition of RPE cell phagocytosis by AICAR was fully reversed by blockad

25 We find that androgen regulates Sertoli cell phagocytosis by controlling expression of miR-471-5

26 Trauma-hemorrhage suppressed Kupffer cell phagocytosis by decreasing Fc receptor expression a

27 recruitment of C1q, which enhanced apoptotic cell phagocytosis by immature dendritic cells.

28 lls in vitro with specificity, trigger tumor cell phagocytosis by macrophages, and efficiently clear

29 aired in their abilities to induce apoptotic cell phagocytosis by murine peritoneal macrophages.

30 a2 AMPK contributed to the inhibition of RPE cell phagocytosis by oxidative stress.

31 s dose-dependently impaired apoptotic Jurkat cell phagocytosis by primary rat or human AM, irrespecti

32 and new phagocyte functions after apoptotic cell phagocytosis demonstrates the enormity of ways to m

33 Apoptotic cell phagocytosis (efferocytosis) is mediated by specifi

34 nation nearly abolished macrophage apoptotic cell phagocytosis; elimination of Axl, Tyro3, or both, r

35 The increase in Kupffer cell phagocytosis following hypoxemia was also prevented

36 ngs indicate that the suppression of Kupffer cell phagocytosis following trauma-hemorrhage is indepen

37 expression, and augmented mAb-mediated tumor cell phagocytosis in vitro However, only STINGa reversed

38 ntaining viral DNA also showed evidence of T cell phagocytosis in vivo, suggesting that their viral D

39 We investigated whether Kupffer cell phagocytosis is differentially regulated following

40 Cell phagocytosis is impaired in type 2 diabetes and req

41 cytoskeletal involvement, although apoptotic cell phagocytosis is not involved.

42 hagocytic receptor(s) responsible for tumour cell phagocytosis is(are) largely unknown.

43 The uptake of large particles by cells (phagocytosis) is an important factor in cell biol

44 mbospondin 1 (TSP1) indicated that mesangial cell phagocytosis of apoptotic cells involved an alpha(v

45 Hence, KIM-1-mediated epithelial cell phagocytosis of apoptotic cells protects the kidney

46 g the injury-limiting potential of mesangial cell phagocytosis of apoptotic cells.

47 Mesangial cell phagocytosis of apoptotic neutrophils in vitro was

48 We conclude that mesangial cell phagocytosis of apoptotic neutrophils involves a no

49 etermined that IFN-gamma reduced endothelial cell phagocytosis of C. albicans by 41.3% compared with

50 Since endothelial cell phagocytosis of C. albicans is required for damage

51 Since endothelial cell phagocytosis of C. albicans is required for endothe

52 Thus, inhibiting endothelial cell phagocytosis of C. albicans may be a mechanism by w

53 cause inactivation or activation of Kupffer cell phagocytosis of colloidal carbon.

54 dative stress dose-dependently inhibited RPE cell phagocytosis of photoreceptor outer segments (POS)

55 macological activator of AMPK, inhibited RPE cell phagocytosis of POS in a dose-dependent manner.

56 to C. albicans, suggesting that endothelial cell phagocytosis of the organism is required to induce

57 lm bacteria was mediated through mononuclear cell phagocytosis since treatment with cytochalasin B, w

58 before transplantation, or LEW donor Kupffer cell phagocytosis was blocked with GdCl3 (7 mg/kg) on da

59 ation by GdCl3 inhibition of hepatic Kupffer cell phagocytosis was pivotal in preventing the developm

60 are revealing universal principles of immune-cell phagocytosis, while also dispelling misconceptions