ライフサイエンスコーパス: cell proliferation

1 galectin-10 by itself was able to suppress T cell proliferation.

2 y by increasing the rates of respiration and cell proliferation.

3 be a function for autophagy in germline stem cell proliferation.

4 g components responsible for epithelial stem cell proliferation.

5 rols, likely attributable to diminished beta-cell proliferation.

6 find cell geometry affects TNFalpha-induced cell proliferation.

7 hages is one pathway that suppresses local T cell proliferation.

8 V, enhance maturation of uninfected DC and T cell proliferation.

9 utes to increased G1-S phase transitions and cell proliferation.

10 n of irisin improved GSIS and increased beta-cell proliferation.

11 calcium influx, aldosterone production, and cell proliferation.

12 nobiotic metabolism, energy homeostasis, and cell proliferation.

13 mosaicism, and couple somatic mosaicism with cell proliferation.

14 modulate MEK/ERK activity in favor of tumor cell proliferation.

15 e vascular branching density and endothelial cell proliferation.

16 ortical cell activation without uncontrolled cell proliferation.

17 ngiomatous lesions that features endothelial cell proliferation.

18 n positive and negative signals that control cell proliferation.

19 pathway genes and concomitant inhibition of cell proliferation.

20 bundance of CDKN1A/P21 and TP53, and reduced cell proliferation.

21 and antagonizing endogenous let-7 triggered cell proliferation.

22 t, very low-carbohydrate diet did not affect cell proliferation.

23 s including elevated cytokine production and cell proliferation.

24 or inhibition to hyperglucagonemia and alpha-cell proliferation.

25 y, stress responses and altered apoptosis or cell proliferation.

26 ot its ubiquitin-binding domain, supported T cell proliferation.

27 ion for genes involved in translation during cell proliferation.

28 velopment, reduced tumor size, and inhibited cell proliferation.

29 results from altered cell cycle and reduced cell proliferation.

30 l cycle progression, signal transduction and cell proliferation.

31 tabolism to favor mitochondrial activity and cell proliferation.

32 e that was caused by a 39% reduction in beta-cell proliferation.

33 tic drugs, which are mainly directed against cell proliferation.

34 ll differentiation and taste stem/progenitor cell proliferation.

35 control diverse biological processes such as cell proliferation.

36 xerted a significant suppressive effect on T cell proliferation.

37 inhibit mediator secretion, or modulate mast cell proliferation.

38 s mTORC1/4E-BP2-eIF4E pathway regulates beta-cell proliferation.

39 and MDA-MB-231 resulted in a reduced rate of cell proliferation.

40 we demonstrated that HMGA2 knockdown reduces cell proliferation.

41 nk between organellar genome maintenance and cell proliferation.

42 xM1 deficiency also blocks compensatory beta-cell proliferation.

43 with the consequent activation of c-Myc and cell proliferation.

44 gnalling promotes competition-induced winner cell proliferation.

45 ) signaling pathway to promote germline stem cell proliferation.

46 cle constriction, neuronal excitability, and cell proliferation.

47 ibly in humans, exendin-4 can stimulate beta-cell proliferation.

48 phorylation, c-Myc up-regulation and reduced cell proliferation.

49 sential enzyme for nucleotide metabolism and cell proliferation.

50 and prolonged survival through inhibition of cell proliferation.

51 s cell differentiation, coupled with reduced cell proliferation.

52 larization, with greater asymmetry promoting cell proliferation.

53 he mutant p53, significantly inhibited tumor cell proliferation.

54 fficiently reversed E1S- and E1-induced T47D cell proliferation.

55 adout for effects of CDK4/CDK6 inhibitors on cell proliferation.

56 beta together drove their suppression of TH2 cell proliferation.

57 /peptide, further enhancing stimulation of T-cell proliferation.

58 cell proliferation, but did stimulate VDR KO cell proliferation.

59 o facilitate cell-cycle progression and stem cell proliferation.

60 YAP target genes and promotes YAP-dependent cell proliferation.

61 elomerase is further up-regulated to sustain cell proliferation.

62 hynyl-2'-deoxyuridine to determine satellite cell proliferation, activation and fusion.

63 peptide-exposed eosinophils induced CD8(+) T cell proliferation, activation, and effector functions.

64 ion, increasing LIGHT expression increased T-cell proliferation, activation, and infiltration, result

65 However, the mechanisms mediating cell proliferation after hypoxia or HIF-2alpha activatio

66 e loss of expression led to a decrease in NB cell proliferation and 16 also induced differentiation.

67 s T-cell responses as evidenced by reduced T-cell proliferation and a switch from a Th1 to a Th2 phen

68 in-draining lymph nodes, where they induce T-cell proliferation and an antigen presentation gene expr

69 n in gastric cancer cells leads to increased cell proliferation and angiogenesis, which may, in turn,

70 volved in melanoma cell migration as well as cell proliferation and anoikis-independent growth, which

71 ized role for IL-27 in regulating epithelial cell proliferation and antiviral host defense during the

72 broblast growth factor receptor 1 (FGFR1) to cell proliferation and apoptosis via the PIM and PI3K ki

73 naling is active, and NG2 inhibition targets cell proliferation and apoptosis.

74 The genes identified converge on aspects of cell proliferation and cell cycle regulation, including

75 signalling via IL-6/8, which is amplified by cell proliferation and cell density, to directly promote

76 vironment by controlling the balance between cell proliferation and cell motility, but the regulators

77 Whereas activation of ERalpha stimulates cell proliferation and cell survival, ERbeta promotes ap

78 r BEC-1/BECN1/Beclin1 in the control of stem cell proliferation and cell-cycle progression, which may

79 transporter gene, thus contributing to tumor cell proliferation and chemoresistance.

80 lines, HepG2 and Huh7, profoundly suppressed cell proliferation and colony formation, and induced cel

81 Cell proliferation and cycle were evaluated by flow cyto

82 HDV-specific T-cell proliferation and cytokine production were weak and

83 Wnt proteins modulate cell proliferation and differentiation and the self-rene

84 requires a coordinated program of epithelial cell proliferation and differentiation as well as resist

85 ide hormones are critical regulators of many cell proliferation and differentiation mechanisms in pla

86 ases (MAPKs) and thereby critically modulate cell proliferation and differentiation.

87 signaling, a key regulator of germline stem cell proliferation and differentiation.

88 ption factor that plays an important role in cell proliferation and differentiation.

89 With roles in development, cell proliferation and disease, micro-RNA (miRNA) biolog

90 helial cells, adhesion to stromal cells, and cell proliferation and display an increased resistance t

91 homeobox 5 (Nkx2.5), resulting in decreased cell proliferation and enhanced cardiomyocyte specificat

92 t into the role of FLCN in regulating kidney cell proliferation and facilitate the development of nov

93 Modulation of T cell proliferation and function by immunoregulatory myel

94 shown to be involved in the regulation of T cell proliferation and function.

95 uced skeletal muscle regeneration, satellite cell proliferation and fusion.

96 xic to J774.2 macrophage cells, and supports cell proliferation and growth.

97 invasion front indicated an association with cell proliferation and higher expression of growth diffe

98 ion, and delineated the relationship between cell proliferation and iCM induction.

99 in BRAF mutant melanoma cell lines, melanoma cell proliferation and in vivo tumor growth were signifi

100 oparticles could efficiently suppress glioma cell proliferation and induce cell apoptosis in vitro.

101 inhibitor cobimetinib (GDC-0973) suppresses cell proliferation and induces cell death better than ei

102 PC1 was previously shown to slow cell proliferation and inhibit apoptosis but the underly

103 ding A4 (S100A4), a protein linked to cancer cell proliferation and invasiveness.

104 respond to insulin stimulation by increasing cell proliferation and invasivity, and that such a respo

105 Organ growth and pancreatic islet cell proliferation and mass were examined in sheep fetus

106 hances the ability of ECs to stimulate tumor cell proliferation and metastasis through stimulation of

107 cisely assign therapies counteracting cancer cell proliferation and metastatic spread.

108 Cell proliferation and migration are known to be regulat

109 ped tailored mathematical models to quantify cell proliferation and migration under normal conditions

110 has been shown to play a role in regulating cell proliferation and migration, and to interact with R

111 re deletion of trkB.T1 in astrocytes reduced cell proliferation and migration.

112 RCC cell lines in vitro led to a decrease in cell proliferation and migration.

113 glycolysis, leading to impaired endothelial cell proliferation and migration.

114 ducing Group I Pak activity diminished MPNST cell proliferation and motility, and that these effects

115 markers and also exhibited the capacity for cell proliferation and neovascularization.

116 Cell proliferation and neuroinflammation in the adult hy

117 palatal shelves, as marked by an increase in cell proliferation and osteogenesis in utero, while othe

118 s, with or without antibody against CTLA4; T-cell proliferation and protein expression were quantifie

119 AM depletion in chemoresistant cells reduced cell proliferation and reduced the IC50 inhibitory conce

120 ecovery; however, mechanisms underlying ATII cell proliferation and spreading are not well understood

121 of the Hippo signaling pathway that regulate cell proliferation and stem cell functions.

122 c energy homeostasis to influencing hepatoma cell proliferation and suggest a potential role of SLC13

123 in or SHH pathway inhibitors decreased tumor cell proliferation and suppressed metastatic tumor growt

124 not Th1/Th17-derived) EVs inhibited CD4(+) T cell proliferation and suppressed two relevant targets o

125 ays both regulate tissue growth by affecting cell proliferation and survival, but interactions betwee

126 yclin D1 is associated with normal and tumor cell proliferation and survival.

127 lated in stem cells due to a deficit in stem cell proliferation and survival.

128 is of thymidylate (TMP) and is essential for cell proliferation and survival.

129 uces the levels of c-Jun, a key regulator of cell proliferation and survival.

130 tion at Ser727, a modification that promotes cell proliferation and the DDR.

131 intricately associated with the reduction in cell proliferation and the enhancement of cancer cell ch

132 ct tumor suppression, the former by blocking cell proliferation and the latter by recruiting immune c

133 development to promote growth by regulating cell proliferation and tissue size.

134 the capacity of ILT3.Fc to inhibit CD4(+) Th cell proliferation and to induce the generation of CD8(+

135 YAP and TAZ are required both to maintain TA cell proliferation and to inhibit differentiation.

136 ractions within the ERK pathway can regulate cell proliferation and transformation, and suggest oncog

137 well as OP9 preadipocytes were assessed for cell proliferation and triglyceride accumulation followi

138 Vgll4 inhibits cell proliferation and tumor growth by competing with YA

139 ranscription of downstream genes and promote cell proliferation and tumor growth in vivo Taken togeth

140 mgGDS (RAP1GDS1) is a key promoter of cancer cell proliferation and tumorigenesis.

141 littermate controls, and analyzed epithelial cell proliferation and ultrastructure from their intesti

142 ression of tumor-related genes and result in cell proliferation and uncontrolled growth, eventually i

143 atrigel substrate coatings greatly increased cell proliferation and uniquely affected beta-crystallin

144 me K-Ras effectors, leading to reduced tumor cell proliferation and viability.

145 WISP-1 induced epithelial cell proliferation and wound closure by activating epith

146 ction of wild-type FLCN in regulating kidney cell proliferation and, therefore, act as an oncoprotein

147 ess than 10% naive T cells, reduced/absent T-cell proliferation, and at least 1 significant clinical

148 , TGR5 stimulation enhanced cAMP production, cell proliferation, and cyst growth by approximately 40%

149 Levels of cAMP, cell proliferation, and cyst growth in vitro were decrea

150 elial stem cell activity, transit-amplifying cell proliferation, and enamel formation in the mouse in

151 ng genes involved in leukocyte infiltration, cell proliferation, and extracellular matrix accumulatio

152 amide production is important for apoptosis, cell proliferation, and immune modulation, highlighting

153 pathways controlling metabolic homeostasis, cell proliferation, and immunity.

154 d exhibited higher oxidative stress, reduced cell proliferation, and increased cell motility.

155 ld-type MLL degradation impedes MLL leukemia cell proliferation, and it downregulates a specific grou

156 ar lactate levels strongly correlated with T cell proliferation, and measuring lactate compared favor

157 there to control T-cell receptor signaling, cell proliferation, and survival.

158 the production of IFN-gamma and TNF-alpha, T cell proliferation, and the expression of CD25, PD-L1, a

159 nstrumental in histone succinylation, tumour cell proliferation, and tumour development.

160 The cell proliferation antigen Ki-67 is widely used in cance

161 n to be closely associated with glioblastoma cell proliferation, apoptosis and drug resistance.

162 n the gene body of DAPK3, a gene involved in cell proliferation, apoptosis, and autophagy.

163 owever, the GLI2 targets that promote cancer cell proliferation are unknown.

164 The live/dead staining, cell proliferation assay and immunohistochemistry study

165 Cell proliferation assays revealed lower proliferation r

166 Here, we performed marker analysis, cell proliferation assays, and genetic lineage tracing t

167 niquely effective source of Ag in standard T cell proliferation assays.

168 ave reduced adult intestinal epithelial stem cell proliferation at the end of metamorphosis (for the

169 ASD-derived NPCs display increased cell proliferation because of dysregulation of a beta-ca

170 from IL233-treated mice suppressed CD4(+) T cell proliferation better than Tregs from saline-treated

171 sCD83 inhibited T cell proliferation, blocked IL-2 secretion, and rendered

172 related drugs identified samples with higher cell proliferation both in vitro and in vivo as leukemia

173 Knockdown had minimal effects on cell proliferation but blocked tumor cell invasion.

174 In turn, this suppressed tumor cell proliferation but increased tumor cell invasion via

175 he masked ANG does not stimulate endothelial cell proliferation but protects astrocytes from oxidativ

176 ons were not sufficient to initiate leukemic cell proliferation but rather only augmented signals fro

177 1,25(OH)2D3 did not affect WT cell proliferation, but did stimulate VDR KO cell prolif

178 locking IDO1 activity completely restored Th cell proliferation, but not IFN-gamma production.

179 Blocking HFD-induced cell proliferation by central delivery of the antimitoti

180 ntrols murine intestinal stem and progenitor cell proliferation by modulating the Wnt pathway via c-M

181 The tumor suppressor PTEN controls cell proliferation by regulating phosphatidylinositol-3-

182 risingly, however, only LECs showed enhanced cell proliferation by regulating the vascular endothelia

183 ve feedback loop in which EZH2 controls GC B cell proliferation by suppressing CDKN1A, enabling cell

184 Arabidopsis RETINOBLASTOMA RELATED (RBR) in cell proliferation can be separated from a novel functio

185 cid (triac) and 3-iodothyronamine (T1AM), on cell proliferation, cell death and DNA damage was studie

186 in RNA (shRNA) significantly inhibited tumor cell proliferation, colony formation and anchorage-indep

187 In contrary, depletion of SATB2 inhibited cell proliferation, colony formation, cell motility and

188 significant decrease in bone-residing tumor cell proliferation compared with free docetaxel.

189 umor cell conditioned media increased glioma cell proliferation compared with media from macrophages

190 that the inhibitory effects of DANCR loss on cell proliferation could be partially rescued by p21 sil

191 ough activation of NFAT1, as well as reduced cell proliferation, cytokine production, and the antitum

192 eover, CDK20-depleted cells display impaired cell proliferation, defective G2/M arrest and increased

193 This stimulation induces antigen-specific B cell proliferation, differentiation of B cells into plas

194 s with the G4 motif in the 5' UTR to promote cell proliferation during liver development and carcinog

195 velopmental phase transitions and to control cell proliferation during organ growth and development,

196 idase called DA1 that limits the duration of cell proliferation during organ growth in Arabidopsis th

197 s involved in regulating uterine endothelial cell proliferation during pregnancy.

198 ATII cell proliferation during repair was attenuated in ATII

199 modulate vascular branching and endothelial cell proliferation during retinal angiogenesis.

200 [(3)H]thymidine incorporation and the use of cell proliferation dyes).

201 state cancer model systems, was required for cell proliferation, enhanced AR's transcriptional activi

202 ted by ELISA, rat basophil leukemia assay, T-cell proliferation experiments using recombinant wildtyp

203 cells with high CD44 expression, and inhibit cell proliferation in a dose-dependent manner.

204 While calcitriol treatment did not decrease cell proliferation in BVE(Cyp24a1-null) cells, it streng

205 of nutrients, growth factors and drugs; and cell proliferation in complex 3D geometries.

206 Selective inhibitory effects of zinc on cell proliferation in esophageal squamous cell carcinoma

207 ation of the midface associated with reduced cell proliferation in forebrain neuroectoderm and fronto

208 y, we show that USP36 down-regulation alters cell proliferation in human cancer cells by inducing bot

209 activates a second ARK, Ssp2/AMPKalpha, for cell proliferation in low environmental glucose.

210 ent cell adhesion is critical for supporting cell proliferation in mesenchymal cells both in vivo and

211 Chronic hyperglycemia also decreased brain cell proliferation in most neurogenic niches throughout

212 granulocytic MDSC suppressed CD4+ and CD8+ T cell proliferation in response to polyclonal or SIV-spec

213 eased by bronchial fibroblasts on epithelial cell proliferation in severe asthma.

214 n chromosome segregation, p53 regulation and cell proliferation in somatic cells, but its role in emb

215 T cell proliferation in the pituitary was confirmed in pat

216 However, the factors that trigger epithelial cell proliferation in this inflammatory process are inco

217 immunosuppression and show that regulatory T-cell proliferation in tissues distal to site of lymphati

218 We found no significant change in prostate cell proliferation in treated mice when compared to cont

219 N3 was sufficient to effectively reduce CPRC cell proliferation in vitro and to abolish xenograft tum

220 ignificantly abolished platelet-induced EOMA cell proliferation in vitro and tumor development in viv

221 specific inhibitors significantly inhibited cell proliferation in vitro and tumor growth in vivo of

222 rget of rapamycin (mTOR) signaling and ADPKD cell proliferation in vitro Homozygous deletion of ILK i

223 rmore, EPO directly inhibited conventional T cell proliferation in vitro via tyrosine phosphatase SHP

224 a consistent critical role in retinoblastoma cell proliferation in vitro, as well as in orthotopic xe

225 sCD83 inhibits T cell proliferation in vitro, supports allograft survival

226 tient suffering from recurrent EBV-induced B cell proliferations including Hodgkin's lymphoma because

227 a 2-fold overexpression of genes linked with cell proliferation, including most targets of the anapha

228 pecies, inhibition of neocortical progenitor cell proliferation, induction of premature neuronal diff

229 Taking cell proliferation into account may explain the paradoxi

230 s, in turn leading to the elevation of NSCLC cell proliferation, invasion and migration.

231 egetative tissues, providing evidence of how cell proliferation is controlled in an identity-specific

232 Cell proliferation is essential to rapid tissue growth a

233 Regulation of cell proliferation is necessary for immune responses, ti

234 chanical force production is compromised and cell proliferation is reduced in Yap mutant lungs.

235 vertebrate ribonuclease, is known to promote cell proliferation, leading to neovascularization as wel

236 genes involved in the inflammatory response, cell proliferation, leukocyte extravasation and choleste

237 rent survival times) were processed for DCX, cell proliferation markers (Ki-67, BrdU), pallial/subpal

238 CD8(+) T cells, and increased expression of cell proliferation markers.

239 logical capacity of the ligand, by measuring cell proliferation, maturation markers and cytokine prod

240 adiol- and catecholamine-induced endothelial cell proliferation may be indicative of unappreciated ev

241 ypoxia mediated signaling pathways including cell proliferation, metabolism and cell survival.

242 tate intraepithelial neoplasia, by promoting cell proliferation, micro-invasion, tissue inflammation,

243 neck and lung) significantly promote cancer cell proliferation, migration and invasion.

244 sfer the signals inside the cells regulating cell proliferation, migration and survival.

245 ore, Gab2 knockout in HepG2 cells restrained cell proliferation, migration and tumor growth in nude m

246 MR and which have no deleterious effects on cell proliferation, migration or differentiation.

247 and metastasis, largely by stimulating tumor cell proliferation, migration, adhesion, and transendoth

248 les plays diverse roles in the regulation of cell proliferation, migration, and differentiation, thes

249 It mediates effects such as cell proliferation, migration, and differentiation.

250 vitro angiogenesis and also for endothelial cell proliferation, migration, and invasion.

251 )2D3 and 24R,25(OH)2D3 on corneal epithelial cell proliferation, migration, and on the vitamin D acti

252 stress-regulated mitochondrial switch of the cell proliferation-motility balance in cancer, and its p

253 horylation of Akt, collagen-I synthesis, and cell proliferation of ASMCs and attenuated airway smooth

254 2 and significantly increased the epithelial cell proliferation of both healthy and severe asthmatic

255 These data suggest that zinc may inhibit cell proliferation of esophageal cancer cells through Or

256 for proper leaf patterning, development and cell proliferation of leaf support tissues, and for rest

257 of the Forkhead family that is required for cell proliferation of normal cells.

258 lls suppressed c-Met expression and enhanced cell proliferation, perhaps by modulating other targets.

259 perated with oncogenic RAS to induce thyroid cell proliferation, pointing to ATXN7 as a previously un

260 g to maximal group persistence and show that cell proliferation prevents the buildup of intercellular

261 Rapid cancer cell proliferation promotes the production of reducing e

262 le-mediated processes, including neural stem cell proliferation, radial migration, and growth cone dy

263 racellular Ca(2+) resulted in an increase in cell proliferation rate, store-operated calcium entry (S

264 othyronine (T3 ), insulin and leptin on beta cell proliferation rates were determined in isolated fet

265 from 12 to 18 mg/L, significantly inhibited cell proliferation, reduced cell viability, and was dire

266 of the 2 identified SAMD9L mutants decreased cell proliferation relative to wild-type protein.

267 zinc and its association with Orai1-mediated cell proliferation remains unknown.

268 Conversely, CD4 T cell proliferation requires an invariant minimal intensi

269 ne H3 (PH3) staining to assess apoptosis and cell proliferation, respectively, showed a significant i

270 imized this discrimination by combining stem cell proliferation responses with a phenotypic screening

271 s a variety of cellular processes, including cell proliferation, RNA processing, protein translation,

272 ISPR-Cas9 gene editing resulted in a halt of cell proliferation, severely impaired EGFR signaling and

273 activation involving the ternary complex in cell proliferation signaling by oncogenic K-Ras.

274 expression, glycolytic flux, and endothelial cell proliferation, sprouting and tubule formation.

275 pate in the control of hormone secretion and cell proliferation, suggesting a potential endocrine/par

276 CH1 target genes include key regulators of B-cell proliferation, survival, and signal transduction.

277 linositol 3-kinase (PI3-kinase) signaling in cell proliferation that is supported by protein macropin

278 TOR-dependent energy signals alone stimulate cell proliferation, the development of a normal leaf lam

279 -Rb1 and E2F-downstream targets, diminishing cell proliferation; these effects are recovered by CDK6

280 l and PACAP/VIP synergistically promote beta-cell proliferation through a FoxM1-dependent mechanism.

281 unfolded protein response and inducing tumor cell proliferation through a TF-dependent mechanism, a s

282 ter ES treatment significantly decreased PCa cell proliferation through down-regulation of GR and up-

283 lar superoxide and inhibited cyst epithelial cell proliferation through extracellular signal-related

284 LDHA also promoted GH3 cell proliferation through induction of cell cycle progr

285 umber by increasing apoptosis and decreasing cell proliferation through initiation of cell cycle arre

286 trans-signaling directly induces endothelial cell proliferation to promote tumor angiogenesis.

287 lioma samples and positively correlated with cell proliferation, tumor grade and malignancy.

288 e, we found that a PKA inhibitor impaired ES cell proliferation, tumor growth and metastasis, which w

289 in vitro and in vivo by inhibiting leukemia cell proliferation/viability and by promoting cell-cycle

290 Bronchial epithelial cell proliferation was evaluated by BrdU incorporation t

291 Cell proliferation was examined in ITPR2-knockout HepG2

292 Whereas the initial step of parenchymal cell proliferation was not affected by acute hyperglycem

293 T cell proliferation was restored when MDSC were treated w

294 Even stiffness-mediated cell proliferation was unaffected by deletion of CD44.

295 ales decreased expression of CRABP2 leads to cell proliferation, whereas in old females it leads to c

296 alveoli cells prevents Kras (G12D) -induced cell proliferation, which leads to reduced tumor formati

297 expression level is critical for lung cancer cell proliferation, which may serve as a prognostic mark

298 , during early infection EBV induces rapid B cell proliferation with low levels of LMP1 and little ap

299 Cell proliferation within small intestinal crypts is the

300 cause concentration-dependent inhibition of cell proliferation, yielding an IC50 value of 356 +/- 21