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1  major glycoprotein 2 (GP2) as a PP-specific cell surface marker.
2  cells has been difficult without a defining cell surface marker.
3 nto Th17 cells in vitro and in vivo via CD25 cell surface marker.
4 e ISCs from mouse and human tissues based on cell surface markers.
5 ytometry together with various hematopoietic cell surface markers.
6 al nerve, and immunofluorescence staining of cell surface markers.
7 4, KDR) and osteoblastic (osteocalcin [OCN]) cell surface markers.
8 her coagulation, inflammatory, or lymphocyte cell surface markers.
9 subpopulation characterized previously using cell surface markers.
10 he result of context-dependent expression of cell surface markers.
11 arly-outgrowth colony-forming unit assay and cell surface markers.
12 ke cysts that selectively incorporate apical cell surface markers.
13 ined challenging due to the lack of specific cell surface markers.
14 eased up-regulation of activation-associated cell surface markers.
15 xpression of both DC and monocyte/macrophage cell surface markers.
16 iling cytokines, intracellular molecules and cell surface markers.
17 an be identified by the presence of specific cell surface markers.
18 oskeleton regulators and the localization of cell surface markers.
19 et been achieved due to the lack of specific cell surface markers.
20 ls that previously identified based on other cell surface markers.
21 y Ab responses due to an absence of specific cell surface markers.
22 using flow cytometry to determine lymphocyte cell surface markers.
23  protein assays for cytokine, chemokine, and cell surface markers.
24 eukemias expressing early B lineage and stem cell surface markers.
25  effect on iDC survival or the expression of cell surface markers.
26 ytokine secretion and expression of specific cell surface markers.
27 essing erythroid, myeloid, lymphoid and stem cell surface markers.
28 cell cycle, and failed to express multiple B-cell surface markers.
29 emokine messenger RNA (mRNA) expression, and cell surface markers.
30  be phenotypically distinguished by very few cell surface markers.
31 pol mRNA, intracellular p24 Gag protein, and cell surface markers.
32 lliliter of blood and the fold expression of cell surface markers.
33 human fetal pancreatic differentiation using cell surface markers.
34 ypically purified from the bone marrow using cell surface markers.
35 re limited by the lack of available specific cell surface markers.
36 ession profiles and identification of unique cell-surface markers.
37 roid progenitor cells that express analogous cell-surface markers.
38 expression of APC (macrophages and dendritic cells) surface markers.
39 unbiased manner a panel of all commonly used cell surface markers (280 genes) from individual cells.
40 ogenase-bright cells expressed CD34 or CD133 cell surface markers (57.0% and 27.1%, respectively), co
41                      We show herein that the cell surface marker 6-sulfo LacNAc (slan) can define sla
42 for viral haemagglutinin (HA) expression and cell surface markers 8-16 hours post infection.
43                         From the panel of 21 cell surface markers, 9 were overexpressed in fetal prog
44 nfection of DC with live 35000HP caused less cell surface marker activation than infection with heat-
45  also expressed higher levels of more mature cell surface markers, additionally linking inflammasome
46 ses were assessed by measuring expression of cell surface markers (adhesion molecules, fibrinogen-lik
47                        Additional cytokines, cell surface markers, adhesion molecules, and accessory
48 ated with respect to endocytosis properties, cell surface markers, allostimulatory activity, and cyto
49                                        These cell surface markers allowed direct isolation of rare ce
50    Superparamagnetic antibodies specific for cell surface markers allowed imaging of CD4+ T cells, CD
51  the basis of Hoechst exclusion and a single cell-surface marker allows enrichment levels similar to
52                                              Cell surface marker analysis reveals that PP T cells con
53 ary, we identified GD2 as a new CSC-specific cell surface marker and GD3S as a potential therapeutic
54     These cells continue to express the CD34 cell surface marker and have ongoing telomerase activity
55 thelial cells expressed specific endothelial cell surface markers and also exhibited the capacity for
56 bpopulations of CSCs, characterized by their cell surface markers and colony morphology, which can se
57 to different sub-populations on the basis of cell surface markers and examine their function in an in
58 comprised of distinct subsets with different cell surface markers and functional characteristics and
59 ntional dendritic cells (cDCs) with distinct cell surface markers and functions exist in mouse and hu
60 ancer cell line, which we first confirmed by cell surface markers and gene profiling to be highly enr
61 otypes, express telomerase activity, express cell surface markers and genes that characterize human E
62                        Peripheral lymphocyte cell surface markers and HLA Ab levels (%PRA and titers)
63                                  Analysis of cell surface markers and immunoglobulin H gene rearrange
64 MAPK signaling pathways and up-regulation of cell surface markers and intracellular molecules associa
65 re and extent of abnormalities in lymphocyte cell surface markers and NK cell activity in patients wi
66 s include (1) the identification of distinct cell surface markers and other cellular properties in he
67                                   Changes in cell surface markers and patterns of gene expression are
68 d expression of CD80, CD86, CD40, and MHC-II cell surface markers and production of proinflammatory c
69 this likeness extended into the non-ISCT MSC cell surface markers and trilineage differentiation, whi
70 ubsets based on their expression of specific cell surface markers and used them in our adoptive trans
71 s and healthy control donors share a similar cell-surface marker and gene expression profile.
72 l death (AICD) and expressed a unique set of cell-surface markers and gene profiles.
73  display significantly altered expression of cell-surface markers and produce increased inflammatory
74 ly, neutrophil morphology (nucleus shape and cell-surface markers) and functions (phagocytosis, degra
75 nology for measuring cell entity, evaluating cell surface marker, and peculiarly in the field of stem
76 ion of apoptosis, acquisition of tumorigenic cell surface markers, and epithelial-mesenchymal transit
77 including those encoding effector cytokines, cell surface markers, and key transcription factors.
78 iptional program, expression of B lymphocyte cell surface markers, and reprogramming of cell fate.
79 ptor (uPAR), a uniquely overexpressed cancer cell-surface marker, and facilitating the immune-mediate
80                                 Because many cell surface markers are shared between AML blasts and h
81 wed AME downregulates the expression of such cell surface markers as CD80, CD86, and major histocompa
82  interferon-gamma production, without losing cell surface markers associated with memory.
83 al cells in part by increasing the levels of cell surface markers associated with mesenchymal stem ce
84                                We identified cell surface markers associated with repression of p16(I
85  effector T cells, and caused an increase in cell surface markers associated with T(Regs) such as Fox
86 eration of reagents that specifically target cell-surface markers, because transmembrane proteins are
87 in muscle tissue express memory and effector cell surface markers but have sharply attenuated effecto
88 n NMR biosensor that can identify a specific cell surface marker by targeted (129)Xe MRI.
89                          Analysis of several cell surface markers by flow cytometry showed that EPC,
90 nel of cytokines, chemokines, receptors, and cell surface markers by RNase protection assays.
91  and FACS analyses demonstrate that specific cell surface markers can be used to discriminate prostat
92 ete subset identified by coexpression of the cell-surface markers CD123 and CCR6.
93                                       As the cell surface marker CD133 identifies cancer-initiating c
94                                          The cell surface marker CD133 is frequently used to identify
95                               Currently, the cell surface marker CD138 (SDC1) is used for this enrich
96 ive L3 for 48 h showed no alterations in the cell surface markers CD14, CD86, CD83, CD207, E-cadherin
97 imeric monoclonal antibody against the pan B cell surface marker CD20 (rituximab).
98 ammatory T cells into tissues, or target the cell surface marker CD20 (rituximab; Rituxan for hematol
99 data in K562 leukemic cells, we identify the cell surface marker CD24 as co-varying with chromatin ac
100                     A new study shows that a cell-surface marker, CD27, identifies the first point of
101 s with great thermogenic potential using the cell surface marker CD29.
102  (MLN), spleen and thymus were labeled for T cell surface markers (CD3, CD4, CD8) and intracellular F
103                                          The cell surface marker CD34 marks mouse hair follicle bulge
104 including CTNND1 and the early hematopoietic cell surface marker CD34, resulted in reduced leukemic g
105                                        Using cell surface markers (CD34, CD133, kinase insert domain
106 nterferon-gamma with a unique combination of cell surface markers (CD4(+)CD25(-)CD44(hi)CD62L(lo)) an
107 ed dendritic cells, assessed by increases in cell-surface markers CD40, CD80, CD86, and MHC class I a
108 s of breast cancer stem cells (BCSCs) is the cell surface marker CD44.
109 d stem cell-like characteristics express the cell surface marker CD44.
110  a Matrigel-based differentiation assay, and cell surface markers CD44 and CD24.
111 ese pancreatic cancer stem cells express the cell surface markers CD44, CD24, and epithelial-specific
112 on of pancreatic cancer cells expressing the cell surface markers CD44, CD24, and epithelial-specific
113  stage transitions, marked by changes in the cell-surface markers CD44 and ICAM1, and a Nanog-enhance
114 Here, we explored this hypothesis by using 2 cell surface markers, CD44 and CD137, to isolate antitum
115 hat express IL-10, as well as Tr1-associated cell surface markers, CD49b and LAG-3, and transcription
116                Using the natural killer (NK) cell-surface marker CD56 to study NK T cells in peripher
117 )/CCR4(+) T cells that also lack the usual T cell surface markers CD7 and/or CD26.
118 -surface protein also known as the leukocyte cell-surface marker CD82.
119 n the low nanomolar range, we identified the cell surface marker CD86 as a sensitive surrogate biomar
120 ls based on a variety of phenotypes, such as cell surface markers, cell proliferation and drug respon
121 B in vitro, as measured by the expression of cell surface markers, cellular signaling events, and cyt
122 man MDDCs exposed to galectin-1 up-regulated cell surface markers characteristic of DC maturation (CD
123 ubsets of HPCs examined, including HPCs with cell surface markers consistent with immature hematopoie
124 ssion signatures, morphological changes, and cell surface markers consistent with myeloid maturation.
125 e cells (CSCs) from DCIS.com cell line using cell surface markers (CS24(-)CD44(+)ESA(+)) and found th
126  within the tumor preferentially express the cell surface markers CTLA-4 and OX40.
127              Given the limited repertoire of cell-surface markers currently available for neural prog
128 oadly characterized by the expression of the cell surface marker CXCR4.
129 etailed study has been hampered by a lack of cell surface markers defining tumor-specific dysfunction
130                                        Forty cell-surface markers, distinguishing all major leukocyte
131 is approach is that the presence of specific cell surface markers does not directly reflect the trans
132 umor cell (CTC) detection strategies rely on cell surface marker EpCAM and intracellular cytokeratins
133  We demonstrate that by the judicious use of cell surface markers, especially CD11b and CD11c, severa
134 XCL9, CXCL10, CXCL12, CXCL13 and CXCL16) and cell surface marker expression (CD3, CD4 and CXCR3) in p
135 eripheral blood, cutaneous mTregs had unique cell surface marker expression and cytokine production.
136                       Although not affecting cell surface marker expression and phagocytotic function
137 ted control iDCs to WT capsule did not alter cell surface marker expression but did elicit IL-8.
138                      Cytokine production and cell surface marker expression of murine PP mononuclear
139  normal, displaying no obvious compromise in cell surface marker expression or antibody production ei
140 n in vivo, and except for CD138, plasmablast cell surface marker expression was unaffected.
141 tro proliferation responses, alloreactivity, cell surface marker expression, and antibody production.
142  recipient T cells as assessed by frequency, cell surface marker expression, cytokine production, and
143 T cells showed convergence in the pattern of cell surface marker expression, cytokine profiles, and g
144 n of MEK1/2 did not reduce CT-B induction of cell surface marker expression, it did attenuate CT-B-me
145 nd, when isolated from skeletal muscle using cell surface marker expression, these cells showed compa
146 entiation for at least 25 d, as evidenced by cell surface marker expression.
147 rom the nontumorigenic cancer cells based on cell surface marker expression.
148 ains the decrease in cytokine production and cell surface marker expression.
149  isolation, cells were characterized through cell-surface marker expression and lineage-specific diff
150  and, regardless of methodology for harvest, cell-surface marker expression of CD73, CD90, CD105, and
151                                      We used cell-surface marker expression to purify from the satell
152 lectively increased polyploidization, mature cell-surface marker expression, and apoptosis of maligna
153                                              Cell surface markers' expression and chemotaxis were det
154  transmembrane glycoprotein, is an important cell surface marker for both stem cells and cancer stem
155                            CCR6 was the best cell surface marker for IL-17A+ cells when compared with
156                 Moreover, huEGFRt provides a cell surface marker for in vivo tracking of adoptively t
157                    Dual staining for CD68 (a cell surface marker for macrophages) and PCNA was also p
158 xpression of CD83 (previously described as a cell surface marker for mature dendritic cells) on CD4 T
159                      We further identified a cell surface marker for prospective isolation of iNCs, w
160                    MUC16 is a well-validated cell surface marker for serous adenocarcinomas of the ov
161 matic stem cells, and it is widely used as a cell surface marker for the isolation and characterizati
162                    Our identification of new cell surface markers for enriching mammosphere-initiatin
163                                     Existing cell surface markers for GSC are developed from embryoni
164     Our analysis identified 24 new/potential cell surface markers for murine fetal hepatic stem cells
165 ss I molecules offers unique cancer-specific cell surface markers for the identification and targetin
166 ceptor (MC1R), which has been evaluated as a cell-surface marker for melanoma.
167 timulation during cancer immunotherapy and a cell-surface marker for pancreatic cancer.
168      In this study, we attempted to identify cell-surface markers for leukemia-initiating cells in FA
169                                              Cell-surface markers for prospective isolation of stem c
170 of commercial antibodies, we have identified cell-surface markers for the separation of pancreatic ce
171 cently, a simple phenotype for HSCs based on cell surface markers from the signaling lymphocyte activ
172 ood, we acquired an immunological profile of cell-surface markers from healthy control and untreated
173 ssessment, nitro-blue tetrazolium reduction, cell-surface markers, genome-wide patterns of gene expre
174 h all the hallmarks of stem cells, including cell surface markers, global gene expression profiles, a
175 ent, both targeting the erythrocyte-specific cell surface marker glycophorin A.
176      However, lack of known unique mesangial cell surface markers has hampered this process.
177  allowing detection of increasing numbers of cell surface markers, has challenged the traditional tec
178 ietic, neural, and skeletal muscle, and stem cell surface markers have been characterized.
179 rious combinations of antibodies directed to cell surface markers have been used to isolate human and
180                                      Several cell-surface markers have been reported to identify cand
181  the basis of the differential expression of cell-surface markers, here we identify a mesenchymal str
182 ific membrane antigen (PSMA), a prototypical cell surface marker highly overexpressed in prostate can
183 utrophils, fibroblasts, and lymphocytes; and cell surface markers, ie, F4/80, CD11b, CD11c, and Ly-6C
184 ify and purify anergic T cells by a distinct cell surface marker in an autoimmune disease and paves t
185 he up-regulation of macrophage/hematopoietic cell surface markers in a large proportion of NIH 3T3 ce
186                                        Novel cell surface markers in adult progenitor cells included
187 e due to the impaired expression of relevant cell surface markers in Eklf(-/-) erythroid cells.
188 o studied expression of the identified novel cell surface markers in fetal rat liver progenitor cells
189 S), we examined mRNA expression of dendritic cell surface markers in individual sporadic ALS (sALS),
190     In this work, we studied progenitor/oval cell surface markers in the liver of rats subjected to 2
191                    Persistence of respective cell surface markers in vitro is confirmed both by flow
192 responses, alloreactivity, and expression of cell surface markers in vitro.
193  selection using an optimized combination of cell surface markers including CD30.
194 n be separated into two populations based on cell surface markers including CD34.
195 ells that exhibit differential expression of cell surface markers, including CD105 (or endoglin), Thy
196 ethod was reported, using the SLAM family of cell-surface markers, including CD150 (SlamF1), to offer
197                          Although exhibiting cell surface markers indicative of activation, the IL-10
198 roteins and were monitored for expression of cell surface markers indicative of maturation.
199     The transcriptional profile identified 2 cell-surface markers, ITGA6 and NGFR, which can be used
200 n inflammatory cell numbers and cytokine and cell-surface marker levels on monocytes and macrophages.
201 gland stem cells (MaSCs) using combinatorial cell surface markers (Lin(-)CD24(+)CD29(h)CD49f(h)) has
202 lation based on the expression profiles of 2 cell-surface markers LNGFR (CD271) and THY-1 (CD90).
203 re the identification of TIM3 as an AML stem cell surface marker more highly expressed on multiple sp
204                             PSMA is a unique cell surface marker, negatively regulated by androgen an
205   We have isolated rare cells bearing the NK cell surface marker NK1.1, as well as the dendritic cell
206  rapidly upregulate the expression of the NK cell-surface marker NK1.1 in response to MSU crystals bu
207 ty in vitro but lacked expression of myeloid cell surface markers normally seen with MLL-CBP.
208 e attenuates acute loss of the developing OL cell surface marker O1 and the mature OL marker MBP (mye
209 ir expression of CD4, CD8, naive, and memory cell-surface markers, occupy distinct homeostatic compar
210                   CD44 is commonly used as a cell surface marker of cancer stem-like cells in epithel
211 xpression of CD146, a hypoxia down-regulated cell surface marker of human BM-MSCs.
212 e DLL1(+) cluster, revealed it to be a novel cell surface marker of human epidermal stem cells.
213 11 receptor alpha subunit (IL-11Ralpha) as a cell surface marker of tumor progression that correlates
214           Doubly deficient T cells displayed cell surface markers of activation yet were significantl
215 ukocytes (VLC), due to sharing functions and cell surface markers of both dendritic cells and endothe
216  protein resulted in increased expression of cell surface markers of DC maturation and an increase in
217  that a group of immature myeloid cells with cell surface markers of Gr-1(+) CD11b(+) are highly enri
218 ion, cytokine production, proliferation, and cell surface markers of immune cells between GA-treated
219 ry CD8 T cells from naive old mice expressed cell surface markers of memory in addition to receptors
220 MSCs) were established and characterized for cell surface markers of mesenchymal stem cell origin in
221  also known as CD143), a recently identified cell-surface marker of adult human hematopoietic stem ce
222             CD30, originally identified as a cell-surface marker of Reed-Sternberg and Hodgkin cells
223  associated with GVHD leads to expression of cell surface markers on both effector and regulatory T c
224                   The expression of lymphoid cell surface markers on PBMC and splenocytes of mice hom
225 S, and microglial activation was assessed by cell surface marker or phospho-MAPK immunofluorescence.
226 eomic or flow cytometric characterization of cell surface markers or adoptive transfer.
227 ls (CSCs) can be identified by expression of cell surface markers or enzymatic activity, but these me
228 intercellular adhesion, without knowledge of cell-surface markers or intracellular proteins.
229                 Flow cytometry utilizing the cell surface markers p75 and HNK-1 was used to isolate l
230 haracteristics such as cell cycle status and cell surface marker phenotype, they respond to different
231     These results demonstrate that CD96 is a cell surface marker present on many AML-LSC and may serv
232 ese results demonstrate that IL-3Ralpha is a cell-surface marker present on FA-AML leukemia-initiatin
233 vity was confirmed by examining cytokine and cell surface marker production in bone-marrow-derived de
234 n cytokine-supplemented medium changed their cell surface marker profile and gene expression pattern
235 sms are cells characterized by a CD44+/CD24- cell surface marker profile.
236  over 24 h, as indicated by up-regulation of cell surface marker proteins CD40, CD80, and CD86.
237 OV-1 antigen, a6 integrin, and connexin 43), cell surface markers recently identified by us (CD44, CD
238  in rat tissue, they distinctively express a cell surface marker recognized by the SE-1 antibody.
239 cycle, lowered levels of macrophage-specific cell surface markers, resistance to Legionella pneumophi
240 ation; SP) in conjunction with canonical HSC cell-surface markers (Sca-1, c-Kit, and lineage markers)
241           When combined with high-throughput cell surface marker screening, this approach facilitates
242         This study identifies FolR1 as a new cell surface marker selectively expressed in mesDA proge
243                           T1/ST2 is a stable cell surface marker selectively expressed on type 2 T he
244     The antennal lobe neuropil expressed the cell surface marker semaphorin 1a.
245                                  Analysis of cell surface markers showed an age-dependent increase in
246                        These MDSCs express a cell surface marker signature (CD11b(+) Gr-1(+) Ly6C(+))
247 n is reliant on the presence of well-defined cell surface markers specific for diverse progenitor pop
248                          We identified novel cell surface markers specific for hepatic progenitor/ova
249  cells as demonstrated by expression of stem cell surface markers such as aldehyde dehydrogenase 1, s
250 m is complicated by the shared expression of cell surface markers such as CD11c.
251 ) are commonly purified by the expression of cell surface markers such as CD34.
252                            Identification of cell surface markers sufficient to purify Treg cells exp
253 ed by a remarkable up-regulation of specific cell surface markers, suggesting that LPS stimulation le
254 n of MSC with both endothelial and pericytic cell surface markers suppresses the homing of cancer cel
255 olobus purpureas agglutinin (TPA) as a novel cell surface marker that allows for such delineation.
256           These data suggest that AA4.1 is a cell surface marker that can identify the earliest lymph
257                                  CD20 is a B cell surface marker that is expressed in various stages
258        We identified 33 transcripts encoding cell surface markers that are differentially expressed b
259 e and chemokine responses, and expression of cell surface markers that are related to T cell activati
260        Cluster designation (CD) antigens are cell surface markers that can be used to identify consti
261 nitive endoderm with the goal of identifying cell surface markers that can be used to track the devel
262 t LSC, one potential strategy is to identify cell surface markers that can distinguish LSC from norma
263                       We sought to delineate cell surface markers that could distinguish NK cells tha
264 acteristics, as well as the precise panel of cell surface markers that uniquely define this newly des
265 ha,and IL-2, and up-regulation of numerous T cell surface markers that would promote T-T Ag presentat
266                       These results reveal a cell-surface marker that delineates functional activity
267 , zymosan, heat-killed or live bacteria, and cell-surface markers that coexpress with FR were identif
268                                  We identify cell-surface markers that delineate a series of stress e
269 ptor type I and II (CD121a/CD121b) as unique cell-surface markers that distinguish activated Tregs fr
270 lls have been hampered by a lack of suitable cell-surface markers that specifically enable their puri
271  cell subsets that display either CD4 or CD8 cell surface markers, the leukemic cell is exclusively o
272 ltiple defects in the expression of effector cell surface markers, the synthesis of cytokines, and in
273                Upon recognizing the targeted cell-surface marker, the APH enters the host cell via en
274 e cell lines were negative for hematopoietic cell surface markers, they gave rise to hematopoietic co
275 D(2) receptor, CRTH2, the best selective Th2-cell surface marker to date.
276 op an assay based on loss of expression of a cell surface marker to monitor epigenetic instability at
277 ins on fibroblast cells, which are potential cell surface markers to differentiate endothelial and fi
278 tential in part independent of commonly used cell surface markers to discriminate effector and memory
279                        We have used specific cell surface markers to examine the association of NG2 c
280 nhanced green fluorescent protein (eGFP) and cell surface markers to FACS-isolate DeltaSox2-eGFP(+) G
281  is due, in part, to the difficulty of using cell surface markers to identify CD4(+)CD25(+) T reg cel
282  we combine H2B-GFP-based pulse-chasing with cell-surface markers to distinguish quiescent from proli
283 ave used flow cytometry and a defined set of cell-surface markers to identify and subsequently isolat
284 ly carried out by quantification of multiple cell surface markers, transcription factors and cytokine
285 eries of heterogeneous subpopulations in its cell surface markers, tumorigenicity, invasion and metas
286               The cells lacked expression of cell surface markers typically expressed by germinal cen
287 ver based on the expression of CD4 and other cell surface markers uniquely expressed by pDCs.
288                                   Sca-1 is a cell surface marker used to identify hematopoietic stem
289                   They discovered a panel of cell surface markers useful for isolating living hair fo
290           The specificity of progenitor/oval cell surface markers was confirmed by ISH and double IF
291                                   Additional cell surface markers were also used to quantify differen
292 he differential expression patterns of these cell surface markers were dependent on Ly49H recognition
293                                              Cell surface markers were screened for their ability to
294             Antibodies against the following cell surface markers were used: CD45 (leukocytes), CD3 (
295 cently identified myeloid lineage restricted cell surface marker, which is overexpressed in over 90%
296 Cs and a germline seminoma that share a CD38 cell-surface marker, which collectively defines likely p
297                                    CX3CR1, a cell surface marker whose expression is associated with
298 lyzed for the expression of CD105 and CD166, cell surface markers whose coexpression defines mesenchy
299          Initially, we determined that the 3 cell surface markers widely used to identify naive T cel
300 r-, costimulation-, or activation-associated cell surface markers yet were associated with lower Ly6C

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