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1 major glycoprotein 2 (GP2) as a PP-specific cell surface marker.
2 cells has been difficult without a defining cell surface marker.
3 nto Th17 cells in vitro and in vivo via CD25 cell surface marker.
4 e ISCs from mouse and human tissues based on cell surface markers.
5 ytometry together with various hematopoietic cell surface markers.
6 al nerve, and immunofluorescence staining of cell surface markers.
7 4, KDR) and osteoblastic (osteocalcin [OCN]) cell surface markers.
8 her coagulation, inflammatory, or lymphocyte cell surface markers.
9 subpopulation characterized previously using cell surface markers.
10 he result of context-dependent expression of cell surface markers.
11 arly-outgrowth colony-forming unit assay and cell surface markers.
12 ke cysts that selectively incorporate apical cell surface markers.
13 ined challenging due to the lack of specific cell surface markers.
14 eased up-regulation of activation-associated cell surface markers.
15 xpression of both DC and monocyte/macrophage cell surface markers.
16 iling cytokines, intracellular molecules and cell surface markers.
17 an be identified by the presence of specific cell surface markers.
18 oskeleton regulators and the localization of cell surface markers.
19 et been achieved due to the lack of specific cell surface markers.
20 ls that previously identified based on other cell surface markers.
21 y Ab responses due to an absence of specific cell surface markers.
22 using flow cytometry to determine lymphocyte cell surface markers.
23 protein assays for cytokine, chemokine, and cell surface markers.
24 eukemias expressing early B lineage and stem cell surface markers.
25 effect on iDC survival or the expression of cell surface markers.
26 ytokine secretion and expression of specific cell surface markers.
27 essing erythroid, myeloid, lymphoid and stem cell surface markers.
28 cell cycle, and failed to express multiple B-cell surface markers.
29 emokine messenger RNA (mRNA) expression, and cell surface markers.
30 be phenotypically distinguished by very few cell surface markers.
31 pol mRNA, intracellular p24 Gag protein, and cell surface markers.
32 lliliter of blood and the fold expression of cell surface markers.
33 human fetal pancreatic differentiation using cell surface markers.
34 ypically purified from the bone marrow using cell surface markers.
35 re limited by the lack of available specific cell surface markers.
36 ession profiles and identification of unique cell-surface markers.
37 roid progenitor cells that express analogous cell-surface markers.
38 expression of APC (macrophages and dendritic cells) surface markers.
39 unbiased manner a panel of all commonly used cell surface markers (280 genes) from individual cells.
40 ogenase-bright cells expressed CD34 or CD133 cell surface markers (57.0% and 27.1%, respectively), co
44 nfection of DC with live 35000HP caused less cell surface marker activation than infection with heat-
45 also expressed higher levels of more mature cell surface markers, additionally linking inflammasome
46 ses were assessed by measuring expression of cell surface markers (adhesion molecules, fibrinogen-lik
48 ated with respect to endocytosis properties, cell surface markers, allostimulatory activity, and cyto
50 Superparamagnetic antibodies specific for cell surface markers allowed imaging of CD4+ T cells, CD
51 the basis of Hoechst exclusion and a single cell-surface marker allows enrichment levels similar to
53 ary, we identified GD2 as a new CSC-specific cell surface marker and GD3S as a potential therapeutic
54 These cells continue to express the CD34 cell surface marker and have ongoing telomerase activity
55 thelial cells expressed specific endothelial cell surface markers and also exhibited the capacity for
56 bpopulations of CSCs, characterized by their cell surface markers and colony morphology, which can se
57 to different sub-populations on the basis of cell surface markers and examine their function in an in
58 comprised of distinct subsets with different cell surface markers and functional characteristics and
59 ntional dendritic cells (cDCs) with distinct cell surface markers and functions exist in mouse and hu
60 ancer cell line, which we first confirmed by cell surface markers and gene profiling to be highly enr
61 otypes, express telomerase activity, express cell surface markers and genes that characterize human E
64 MAPK signaling pathways and up-regulation of cell surface markers and intracellular molecules associa
65 re and extent of abnormalities in lymphocyte cell surface markers and NK cell activity in patients wi
66 s include (1) the identification of distinct cell surface markers and other cellular properties in he
68 d expression of CD80, CD86, CD40, and MHC-II cell surface markers and production of proinflammatory c
69 this likeness extended into the non-ISCT MSC cell surface markers and trilineage differentiation, whi
70 ubsets based on their expression of specific cell surface markers and used them in our adoptive trans
73 display significantly altered expression of cell-surface markers and produce increased inflammatory
74 ly, neutrophil morphology (nucleus shape and cell-surface markers) and functions (phagocytosis, degra
75 nology for measuring cell entity, evaluating cell surface marker, and peculiarly in the field of stem
76 ion of apoptosis, acquisition of tumorigenic cell surface markers, and epithelial-mesenchymal transit
77 including those encoding effector cytokines, cell surface markers, and key transcription factors.
78 iptional program, expression of B lymphocyte cell surface markers, and reprogramming of cell fate.
79 ptor (uPAR), a uniquely overexpressed cancer cell-surface marker, and facilitating the immune-mediate
81 wed AME downregulates the expression of such cell surface markers as CD80, CD86, and major histocompa
83 al cells in part by increasing the levels of cell surface markers associated with mesenchymal stem ce
85 effector T cells, and caused an increase in cell surface markers associated with T(Regs) such as Fox
86 eration of reagents that specifically target cell-surface markers, because transmembrane proteins are
87 in muscle tissue express memory and effector cell surface markers but have sharply attenuated effecto
91 and FACS analyses demonstrate that specific cell surface markers can be used to discriminate prostat
96 ive L3 for 48 h showed no alterations in the cell surface markers CD14, CD86, CD83, CD207, E-cadherin
98 ammatory T cells into tissues, or target the cell surface marker CD20 (rituximab; Rituxan for hematol
99 data in K562 leukemic cells, we identify the cell surface marker CD24 as co-varying with chromatin ac
102 (MLN), spleen and thymus were labeled for T cell surface markers (CD3, CD4, CD8) and intracellular F
104 including CTNND1 and the early hematopoietic cell surface marker CD34, resulted in reduced leukemic g
106 nterferon-gamma with a unique combination of cell surface markers (CD4(+)CD25(-)CD44(hi)CD62L(lo)) an
107 ed dendritic cells, assessed by increases in cell-surface markers CD40, CD80, CD86, and MHC class I a
111 ese pancreatic cancer stem cells express the cell surface markers CD44, CD24, and epithelial-specific
112 on of pancreatic cancer cells expressing the cell surface markers CD44, CD24, and epithelial-specific
113 stage transitions, marked by changes in the cell-surface markers CD44 and ICAM1, and a Nanog-enhance
114 Here, we explored this hypothesis by using 2 cell surface markers, CD44 and CD137, to isolate antitum
115 hat express IL-10, as well as Tr1-associated cell surface markers, CD49b and LAG-3, and transcription
119 n the low nanomolar range, we identified the cell surface marker CD86 as a sensitive surrogate biomar
120 ls based on a variety of phenotypes, such as cell surface markers, cell proliferation and drug respon
121 B in vitro, as measured by the expression of cell surface markers, cellular signaling events, and cyt
122 man MDDCs exposed to galectin-1 up-regulated cell surface markers characteristic of DC maturation (CD
123 ubsets of HPCs examined, including HPCs with cell surface markers consistent with immature hematopoie
124 ssion signatures, morphological changes, and cell surface markers consistent with myeloid maturation.
125 e cells (CSCs) from DCIS.com cell line using cell surface markers (CS24(-)CD44(+)ESA(+)) and found th
129 etailed study has been hampered by a lack of cell surface markers defining tumor-specific dysfunction
131 is approach is that the presence of specific cell surface markers does not directly reflect the trans
132 umor cell (CTC) detection strategies rely on cell surface marker EpCAM and intracellular cytokeratins
133 We demonstrate that by the judicious use of cell surface markers, especially CD11b and CD11c, severa
134 XCL9, CXCL10, CXCL12, CXCL13 and CXCL16) and cell surface marker expression (CD3, CD4 and CXCR3) in p
135 eripheral blood, cutaneous mTregs had unique cell surface marker expression and cytokine production.
137 ted control iDCs to WT capsule did not alter cell surface marker expression but did elicit IL-8.
139 normal, displaying no obvious compromise in cell surface marker expression or antibody production ei
141 tro proliferation responses, alloreactivity, cell surface marker expression, and antibody production.
142 recipient T cells as assessed by frequency, cell surface marker expression, cytokine production, and
143 T cells showed convergence in the pattern of cell surface marker expression, cytokine profiles, and g
144 n of MEK1/2 did not reduce CT-B induction of cell surface marker expression, it did attenuate CT-B-me
145 nd, when isolated from skeletal muscle using cell surface marker expression, these cells showed compa
149 isolation, cells were characterized through cell-surface marker expression and lineage-specific diff
150 and, regardless of methodology for harvest, cell-surface marker expression of CD73, CD90, CD105, and
152 lectively increased polyploidization, mature cell-surface marker expression, and apoptosis of maligna
154 transmembrane glycoprotein, is an important cell surface marker for both stem cells and cancer stem
158 xpression of CD83 (previously described as a cell surface marker for mature dendritic cells) on CD4 T
161 matic stem cells, and it is widely used as a cell surface marker for the isolation and characterizati
164 Our analysis identified 24 new/potential cell surface markers for murine fetal hepatic stem cells
165 ss I molecules offers unique cancer-specific cell surface markers for the identification and targetin
168 In this study, we attempted to identify cell-surface markers for leukemia-initiating cells in FA
170 of commercial antibodies, we have identified cell-surface markers for the separation of pancreatic ce
171 cently, a simple phenotype for HSCs based on cell surface markers from the signaling lymphocyte activ
172 ood, we acquired an immunological profile of cell-surface markers from healthy control and untreated
173 ssessment, nitro-blue tetrazolium reduction, cell-surface markers, genome-wide patterns of gene expre
174 h all the hallmarks of stem cells, including cell surface markers, global gene expression profiles, a
177 allowing detection of increasing numbers of cell surface markers, has challenged the traditional tec
179 rious combinations of antibodies directed to cell surface markers have been used to isolate human and
181 the basis of the differential expression of cell-surface markers, here we identify a mesenchymal str
182 ific membrane antigen (PSMA), a prototypical cell surface marker highly overexpressed in prostate can
183 utrophils, fibroblasts, and lymphocytes; and cell surface markers, ie, F4/80, CD11b, CD11c, and Ly-6C
184 ify and purify anergic T cells by a distinct cell surface marker in an autoimmune disease and paves t
185 he up-regulation of macrophage/hematopoietic cell surface markers in a large proportion of NIH 3T3 ce
188 o studied expression of the identified novel cell surface markers in fetal rat liver progenitor cells
189 S), we examined mRNA expression of dendritic cell surface markers in individual sporadic ALS (sALS),
190 In this work, we studied progenitor/oval cell surface markers in the liver of rats subjected to 2
195 ells that exhibit differential expression of cell surface markers, including CD105 (or endoglin), Thy
196 ethod was reported, using the SLAM family of cell-surface markers, including CD150 (SlamF1), to offer
199 The transcriptional profile identified 2 cell-surface markers, ITGA6 and NGFR, which can be used
200 n inflammatory cell numbers and cytokine and cell-surface marker levels on monocytes and macrophages.
201 gland stem cells (MaSCs) using combinatorial cell surface markers (Lin(-)CD24(+)CD29(h)CD49f(h)) has
202 lation based on the expression profiles of 2 cell-surface markers LNGFR (CD271) and THY-1 (CD90).
203 re the identification of TIM3 as an AML stem cell surface marker more highly expressed on multiple sp
205 We have isolated rare cells bearing the NK cell surface marker NK1.1, as well as the dendritic cell
206 rapidly upregulate the expression of the NK cell-surface marker NK1.1 in response to MSU crystals bu
208 e attenuates acute loss of the developing OL cell surface marker O1 and the mature OL marker MBP (mye
209 ir expression of CD4, CD8, naive, and memory cell-surface markers, occupy distinct homeostatic compar
213 11 receptor alpha subunit (IL-11Ralpha) as a cell surface marker of tumor progression that correlates
215 ukocytes (VLC), due to sharing functions and cell surface markers of both dendritic cells and endothe
216 protein resulted in increased expression of cell surface markers of DC maturation and an increase in
217 that a group of immature myeloid cells with cell surface markers of Gr-1(+) CD11b(+) are highly enri
218 ion, cytokine production, proliferation, and cell surface markers of immune cells between GA-treated
219 ry CD8 T cells from naive old mice expressed cell surface markers of memory in addition to receptors
220 MSCs) were established and characterized for cell surface markers of mesenchymal stem cell origin in
221 also known as CD143), a recently identified cell-surface marker of adult human hematopoietic stem ce
223 associated with GVHD leads to expression of cell surface markers on both effector and regulatory T c
225 S, and microglial activation was assessed by cell surface marker or phospho-MAPK immunofluorescence.
227 ls (CSCs) can be identified by expression of cell surface markers or enzymatic activity, but these me
230 haracteristics such as cell cycle status and cell surface marker phenotype, they respond to different
231 These results demonstrate that CD96 is a cell surface marker present on many AML-LSC and may serv
232 ese results demonstrate that IL-3Ralpha is a cell-surface marker present on FA-AML leukemia-initiatin
233 vity was confirmed by examining cytokine and cell surface marker production in bone-marrow-derived de
234 n cytokine-supplemented medium changed their cell surface marker profile and gene expression pattern
237 OV-1 antigen, a6 integrin, and connexin 43), cell surface markers recently identified by us (CD44, CD
238 in rat tissue, they distinctively express a cell surface marker recognized by the SE-1 antibody.
239 cycle, lowered levels of macrophage-specific cell surface markers, resistance to Legionella pneumophi
240 ation; SP) in conjunction with canonical HSC cell-surface markers (Sca-1, c-Kit, and lineage markers)
247 n is reliant on the presence of well-defined cell surface markers specific for diverse progenitor pop
249 cells as demonstrated by expression of stem cell surface markers such as aldehyde dehydrogenase 1, s
253 ed by a remarkable up-regulation of specific cell surface markers, suggesting that LPS stimulation le
254 n of MSC with both endothelial and pericytic cell surface markers suppresses the homing of cancer cel
255 olobus purpureas agglutinin (TPA) as a novel cell surface marker that allows for such delineation.
259 e and chemokine responses, and expression of cell surface markers that are related to T cell activati
261 nitive endoderm with the goal of identifying cell surface markers that can be used to track the devel
262 t LSC, one potential strategy is to identify cell surface markers that can distinguish LSC from norma
264 acteristics, as well as the precise panel of cell surface markers that uniquely define this newly des
265 ha,and IL-2, and up-regulation of numerous T cell surface markers that would promote T-T Ag presentat
267 , zymosan, heat-killed or live bacteria, and cell-surface markers that coexpress with FR were identif
269 ptor type I and II (CD121a/CD121b) as unique cell-surface markers that distinguish activated Tregs fr
270 lls have been hampered by a lack of suitable cell-surface markers that specifically enable their puri
271 cell subsets that display either CD4 or CD8 cell surface markers, the leukemic cell is exclusively o
272 ltiple defects in the expression of effector cell surface markers, the synthesis of cytokines, and in
274 e cell lines were negative for hematopoietic cell surface markers, they gave rise to hematopoietic co
276 op an assay based on loss of expression of a cell surface marker to monitor epigenetic instability at
277 ins on fibroblast cells, which are potential cell surface markers to differentiate endothelial and fi
278 tential in part independent of commonly used cell surface markers to discriminate effector and memory
280 nhanced green fluorescent protein (eGFP) and cell surface markers to FACS-isolate DeltaSox2-eGFP(+) G
281 is due, in part, to the difficulty of using cell surface markers to identify CD4(+)CD25(+) T reg cel
282 we combine H2B-GFP-based pulse-chasing with cell-surface markers to distinguish quiescent from proli
283 ave used flow cytometry and a defined set of cell-surface markers to identify and subsequently isolat
284 ly carried out by quantification of multiple cell surface markers, transcription factors and cytokine
285 eries of heterogeneous subpopulations in its cell surface markers, tumorigenicity, invasion and metas
292 he differential expression patterns of these cell surface markers were dependent on Ly49H recognition
295 cently identified myeloid lineage restricted cell surface marker, which is overexpressed in over 90%
296 Cs and a germline seminoma that share a CD38 cell-surface marker, which collectively defines likely p
298 lyzed for the expression of CD105 and CD166, cell surface markers whose coexpression defines mesenchy
300 r-, costimulation-, or activation-associated cell surface markers yet were associated with lower Ly6C
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