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1 engineered T cells 2 d after autologous stem cell transplant.
2 ion after unrelated-donor hematopoietic stem cell transplant.
3 in lymphoma received chemotherapy and a stem cell transplant.
4 e treatment or autologous or allogeneic stem-cell transplant.
5 to identify an etiology, 6 weeks after stem cell transplant.
6 free without a consolidative allogeneic stem cell transplant.
7 ib treatment and high-dose melphalan in stem cell transplant.
8 ortality after allogeneic hematopoietic stem cell transplant.
9 ortality after allogeneic hematopoietic stem cell transplant.
10 tion in first remission with autologous stem cell transplant.
11 ion is a significant complication after stem cell transplant.
12 3%) to lenalidomide; 39 (59%) had prior stem cell transplant.
13 hal complication of allogeneic hematopoietic cell transplant.
14 ne of these viruses after hematopoietic stem cell transplant.
15 rior methotrexate, and prior autologous stem-cell transplant.
16 ts (range 1-11); 18 (38%) had undergone stem-cell transplant.
17 ponses among the 18 patients with prior stem-cell transplant.
18 re not planned for immediate autologous stem-cell transplant.
19 reatment that included an HIV-resistant stem cell transplant.
20 um-based chemotherapy before autologous stem cell transplant.
21 secutive patients undergoing allogeneic stem cell transplant.
22 tic progenitor cells in preparation for stem cell transplant.
23 ical malignancies and the recipients of stem-cell transplant.
24 development of allogeneic hematopoietic stem cell transplant.
25 s, particularly those who have received stem cell transplants.
26 omide), and 76% had received autologous stem-cell transplants.
27 :CLEC2D) interactions in human hematopoietic cell transplants.
28 on in recipients of allogeneic hematopoietic-cell transplants.
29 ressive therapies and/or solid organ or stem cell transplants.
30 of CMV events in recipients of hematopoietic-cell transplants.
31 to both solid organ and haematopoietic stem cell transplants.
32 ronic administration of immunosuppression in cell transplants.
33 id formation in type 2 diabetes and in islet cell transplants.
34 reatment of blood cancers with hematopoietic cell transplants.
35 itution in recipients of haploidentical stem-cell transplants.
36 s host disease in matched or mismatched stem cell transplants.
37 educe the rate of relapse in allogeneic stem cell transplants.
38 aftment rates, with similar numbers of CD34+ cells transplanted.
39 h patients should receive an allogeneic stem cell transplant?
40 ncrystalline LCPT, of whom 11 underwent stem cell transplant, 16 received chemotherapy only, and nine
41 in adults receiving allogeneic hematopoietic cell transplants (1999-2005) from HLA-identical sibling
47 lity to distinguish allogeneic hematopoietic cell transplant (allo-HCT) recipients at risk for cytome
48 on a cohort of 237 allogeneic hematopoietic cell transplant (allo-HCT) recipients, that can predict
50 arting treatment achieved hematopoietic stem cell transplant (alloHSCT) compared with 46% in the imat
51 omplication of allogeneic hematopoietic stem cell transplant (alloHSCT), underscoring the need to fur
52 do not appear to require an allogeneic stem cell transplant (alloSCT) if they achieve a good respons
55 nts in need of allogeneic hematopoietic stem cell transplant and has recruited millions of volunteers
57 , persons with autologous hematopoietic stem cell transplant and those without graft-versus-host dise
58 n additional 300 patients who underwent stem cell transplant and were enrolled on multicentre clinica
59 of the children received hematopoietic stem cell transplants and all showed poor graft function with
61 ibody is associated with early graft loss of cell transplants and reduced long-term survival of solid
62 consolidative hematopoietic allogeneic stem cell transplant, and 9 (9% of all enrolled patients) rem
65 Studies of critically ill, oncologic or stem cell transplant, and solid organ transplant patients sho
66 mmunosuppressive drugs, 13 had received stem cell transplants, and 7 were infected with human immunod
67 nsfusions, bone marrow or hematopoietic stem cell transplants, and experimental pharmacologic chapero
68 Recipients of allogeneic hematopoietic stem cell transplant appear to have the highest risk of morta
70 e field of hematology, as hematopoietic stem cell transplants are already commonplace in clinics arou
72 an adequate blood supply for the survival of cell transplants are major hurdles that need to be overc
74 on emission tomography or hematopoietic stem cell transplant as independent prognostic factors for ou
75 e-ferumoxytol-positive macrophages into stem cell transplants, as visualized with IVM and histopathol
78 dose chemotherapy (HDC) with autologous stem-cell transplant (ASCT) may provide an alternative to add
83 halan with low-dose TBI after haplocord stem cell transplant assures good engraftment and leads to ac
86 mes of patients who underwent haplocord stem cell transplant between May 2013 and March 2015 and who
87 or the use of RIC regimens in all adult stem cell transplant candidates with acute leukemia in remiss
88 ent admissions with a non-hematopoietic stem cell transplant cohort and excluded solid-organ transpla
89 ortality was 32.9% in non-hematopoietic stem cell transplant cohort, which was similar to autologous
91 imaging of donor-matched and mismatched stem cell transplants demonstrated decreased signal intensity
92 esis that selective GABA agonists as well as cell transplant-derived GABA are antipruritic against ac
95 ority of patients in need of a hematopoietic-cell transplant do not have a matched related donor.
97 blative chemotherapy with hematopoietic stem-cell transplant followed by adjuvant retinoid differenti
98 n a patient who received a heterologous stem cell transplant for acquired immunodeficiency syndrome-r
99 230 subjects receiving a first hematopoietic cell transplant from a human leukocyte antigen-matched s
100 icient SCID (ADA-SCID) is hematopoietic stem cell transplant from an HLA-matched sibling donor, altho
101 received a first myeloablative hematopoietic-cell transplant from an unrelated cord-blood donor (140
103 two patients who received hematopoietic stem cell transplants from 2004 through 2008 at the Universit
106 itive recipients of allogeneic hematopoietic-cell transplants from matched related or unrelated donor
107 with AML who had received hematopoietic stem-cell transplants from unrelated donors matched for HLA-A
110 ents with crystalline LCPT treated with stem cell transplant had stable or improved kidney function,
111 undergoing haploidentical hematopoietic stem cell transplant (haplo-HSCT) without the risk for uncont
114 yndrome (BOS) after allogeneic hematopoietic cell transplant (HCT) conferred nearly universal mortali
115 man herpesvirus 6 (ciHHV-6) in hematopoietic cell transplant (HCT) donors or recipients confounds mol
116 who receive an unrelated donor hematopoietic cell transplant (HCT) for the treatment of blood disorde
117 al virus (RSV) pneumonia after hematopoietic cell transplant (HCT) is associated with severe morbidit
119 therapy (HDIT) with autologous hematopoietic cell transplant (HCT) may, in contrast, induce sustained
120 omosome (HY-Abs) develop after hematopoietic cell transplant (HCT) of male recipients with female don
121 CD4(+) </=200; autologous hematopoietic stem-cell transplant (HCT) or allogeneic-HCT recipients.
122 lasia (MDS), and in allogeneic hematopoietic cell transplant (HCT) recipients (early, until day 40; l
123 ults of MVA in allogeneic hematopoietic stem cell transplant (HCT) recipients and Triplex in healthy
124 lid organ transplant (SOT) and hematopoietic cell transplant (HCT) recipients at a single center over
125 disseminated viral disease in hematopoietic cell transplant (HCT) recipients but does not lead to re
126 5-specific T cells can protect hematopoietic cell transplant (HCT) recipients from CMV complications.
134 apsed CLL following allogeneic hematopoietic cell transplant (HCT) who subsequently received ibrutini
139 eloablative (NMA) conditioning hematopoietic cell transplants (HCTs) have changed the therapeutic str
140 atients who receive allogeneic hematopoietic cell transplants (HCTs), and the skin is the most common
141 after high-dose therapy and autologous stem-cell transplant (HDT/ASCT) for patients with relapsed or
142 livery of potent immune suppressor cells for cell transplants holds great clinical application potent
143 etes and contributes to the failure of islet cell transplants, however the mechanisms of IAPP-induced
145 missions for induction or hematopoietic stem-cell transplant (HSCT) from 2006 to 2014 was selected.
149 Autologous and allogeneic hematopoietic stem cell transplant (HSCT) patients are susceptible to pulmo
152 V-seronegative allogeneic hematopoietic stem cell transplant (HSCT) recipient is generally accepted.
153 irus (CMV) viral loads in hematopoietic stem cell transplant (HSCT) recipients are typically monitore
154 he risk of skin cancer in hematopoietic stem-cell transplant (HSCT) recipients has not been extensive
155 2 study in 50 allogeneic hematopoietic stem cell transplant (HSCT) recipients who received donorderi
156 ed in >/=9% of allogeneic hematopoietic stem cell transplant (HSCT) recipients, in whom it can cause
177 other than myeloablative hematopoietic stem cell transplant (HSCT); however, relapse remains a major
178 ections often occur after hematopoietic stem cell transplant (HSCT); vaccination is important for pre
181 2 treatments (range, 1-6), including a stem cell transplant in 105 patients (75%), were administered
182 cured following a delta32/delta32 CCR5 stem cell transplant in 2007 revealed no antibodies against p
183 sible, then high-dose chemotherapy with stem cell transplant in an experienced center is a reasonable
185 nt; 25 patients underwent an allogeneic stem cell transplant in first CR and were excluded, leaving 7
187 suggests that the rejection of similar stem cell transplants in humans will be dependent upon the pr
189 ons could reduce the number of hematopoietic cell transplants in patients with AML by 20-25% while ma
190 n contrast, Sharpin(cpdm) mammary epithelial cells transplanted in vivo into wild-type stroma, fully
191 he Lancet of embryonic-stem-cell-derived RPE cell transplants indicate no serious adverse outcomes an
193 igration of nmMYLK(-/-) microglia (2 x 10(5) cells transplanted into corpus callosum) compared with W
197 into old recipients; conversely, young beta-cells transplanted into old mice decrease their replicat
198 ave increased replication rate compared with cells transplanted into old recipients; conversely, youn
201 w that for encapsulated rat pancreatic islet cells transplanted into streptozotocin-treated diabetic
203 In addition, we demonstrate that old beta-cells transplanted into young recipients have increased
205 The role of allogeneic hematopoietic stem cell transplant is less clear but may be useful in selec
207 ansfer of human peripheral blood mononuclear cells, transplanted LEA-tg ICCs were completely protecte
208 ia-like" regimen followed by allogeneic stem-cell transplant may be advisable; addition of a tyrosine
209 ad received an allogeneic hematopoietic stem cell transplant (median, 61 [interquartile range {IQR},
213 own postnatal mice-plus syngeneic fibroblast cell-transplant models-we demonstrate that the regenerat
214 y Hodgkin lymphoma following autologous stem cell transplant (N = 102) after a median observation per
215 66; HR = 0.66; P = .001) and autologous stem cell transplant (n = 273; HR = 0.55; P = .004) were inde
216 2.39-6.07) and autologous hematopoietic stem cell transplant (odds ratio, 1.28; 95% CI, 1.06-1.53) re
217 l mortality in allogeneic hematopoietic stem cell transplant (odds ratio, 3.81; 95% CI, 2.39-6.07) an
218 de effects of lifetime immunosuppression for cell transplants often outweigh the benefits; therefore,
221 a who previously received an autologous stem cell transplant or two previous multiagent chemotherapy
225 conditioning regimen for hematopoietic stem cell transplant (P = .002), and the cumulative dose of c
226 splant recipients and one hematopoietic stem-cell transplant patient with positive A. calidoustus cul
227 entified all hematologic malignancy and stem cell transplant patients diagnosed with proven mucormyco
228 infused 12 haploidentical hematopoietic stem cell transplant patients with increasing numbers of allo
233 ng patients who received blood-building stem cell transplants, patients with chronic granulomatous di
234 3 unrelated marrow and peripheral blood stem cell transplants performed from 1995 to 2007 for treatme
236 idomide/pomalidomide, double autologous stem cell transplant plus bortezomib, or combination of immun
237 dysplastic syndromes, and hematopoietic stem cell transplant populations has been evaluated in recent
240 egionnaires' disease in a hematopoietic stem cell transplant recipient caused by this organism is des
241 ection was diagnosed in a hematopoietic stem cell transplant recipient during conditioning regimen.
243 was similar to autologous hematopoietic stem cell transplant recipients (30.1%) and those who did not
244 ant recipients (n = 197), 0% to 5.8% in stem-cell transplant recipients (n = 103), and 11.2 to 15.2%
248 a cluster of fatal toxoplasmosis among stem cell transplant recipients at 2 hospitals, surveillance
249 unit knockout mice, we demonstrate that stem cell transplant recipients lacking the ability to genera
250 reventing RSV LRTIs in 50 hematopoietic stem cell transplant recipients or patients with hematologic
251 t cytomegalovirus infection in hematopoietic cell transplant recipients provided initial data on the
252 fects of GvHD itself, all serve to make stem cell transplant recipients vulnerable to disease from en
253 uency of severe sepsis in hematopoietic stem cell transplant recipients was five times higher when co
255 ion therapy or allogeneic hematopoietic stem cell transplant recipients were randomized (1:1 ratio) t
256 in subsequent admissions, hematopoietic stem cell transplant recipients with graft-versus-host diseas
257 as detected in 9 of 79 (11.4%) hematopoietic cell transplant recipients with influenza, and was less
258 es and mortality in allogeneic hematopoietic cell transplant recipients with invasive pulmonary asper
259 We compared outcomes of hematopoietic stem cell transplant recipients with severe sepsis during eng
261 patients (renal transplant recipients, stem cell transplant recipients, and congenitally infected ch
262 omen, neutropenic hosts, solid-organ or stem cell transplant recipients, and patients receiving tumor
264 taneous malignant neoplasms in hematopoietic cell transplant recipients, this population should be ed
275 (cGVHD) after allogeneic hematopoietic stem cell transplant reflects a complex immune response resul
276 JMML relies on allogeneic hematopoietic stem cell transplant, relapse is the most frequent cause of t
277 een applied for decades, outcomes of somatic cell transplants remain disappointing, presumably due to
280 linical factors, induction therapy, and stem cell transplant (SCT) on the outcomes of 311 patients wi
283 tients who relapse following autologous stem cell transplant (SCT), multiple treatment options are av
284 < 0.0001) and not undergoing allogeneic stem cell transplant (SCT, p = 0.0005) predicted poor overall
285 g risk in patients receiving allogeneic stem cell transplants (SCTs) or chemotherapy but not in those
288 nts in 3 medical centers using hematopoietic cell transplants to establish mixed or complete chimeris
291 Salvage high-dose chemotherapy plus a stem-cell transplant was required in six (6%) patients in the
294 recipients of allogeneic hematopoietic stem cell transplants who underwent GBCA-enhanced MR imaging
295 ents who require an allogeneic hematopoietic cell transplant will have an HLA-matched sibling donor.
296 biopsy in patients who have undergone a stem cell transplant with eruptions on the head and neck.
297 detection of innate immune responses to stem cell transplants with magnetic resonance (MR) imaging.
300 recipients of allogeneic hematopoietic stem cell transplant, with their infections caused by five Le
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