ライフサイエンスコーパス: cell tumor

1 rmal tumor, and one desmoplastic small round cell tumor).

2 inal stromal tumor, leiomyosarcoma, granular cell tumor).

3 ally named PEComas (perivascular epithelioid cell tumors).

4 d in the aggressive desmoplastic small round cell tumor.

5 fication of the specific Ig genes from the B-cell tumor.

6 ) and in Kaposi sarcoma (KS), an endothelial cell tumor.

7 pathological features of tenosynovial giant-cell tumor.

8 tral nervous system tumors, or renal or germ-cell tumors.

9 rge B cell lymphoma (DLBCL) and additional B cell tumors.

10 the development of adenocarinoma and spindle cell tumors.

11 either seminomas or nonseminomas/mixed germ cell tumors.

12 the survival of mice with implanted human B-cell tumors.

13 ightly stronger for B-cell tumors than for T-cell tumors.

14 are at increased risk of developing Sertoli cell tumors.

15 ovarian serous adenocarcinomas and granulosa cell tumors.

16 the poor outcome reported in all MHC II(-) B-cell tumors.

17 ficacy should be examined in patients with B-cell tumors.

18 PIK3CA were previously described in squamous cell tumors.

19 ent of certain categories of testicular germ cell tumors.

20 omatography to membrane preparations of beta-cell tumors.

21 29% of clear cell samples and 8% of nonclear cell tumors.

22 rtility and are associated with ovarian germ cell tumors.

23 a key determinant of oncogenic fate in germ-cell tumors.

24 ent of certain categories of testicular germ cell tumors.

25 or misexpression of Chinmo results in blood cell tumors.

26 ly 35% of patients with newly diagnosed germ-cell tumors.

27 ent of certain categories of testicular germ cell tumors.

28 ant mice were infertile but lacked granulosa cell tumors.

29 men are at increased risk of testicular germ-cell tumors.

30 ed using the terms testicular cancer or germ cell tumors.

31 es of initial treatments for testicular germ cell tumors.

32 is a tumor suppressor for testicular stromal cell tumors.

33 ng frame and is expressed in human male germ cell tumors.

34 oach from the Brazilian GCT-99 study on germ cell tumors.

35 ma skin cancer (NMSC), particularly squamous cell tumors.

36 a potential target for the therapy of Leydig cell tumors.

37 e most prevalent molecular features of clear cell tumors.

38 rs, lung and mammary carcinomas, and spindle cell tumors.

39 s, systemic mastocytosis, and localized mast cell tumors.

40 echanism that controls cell growth in Leydig cell tumors.

41 with newly diagnosed non-germinomatous germ cell tumors.

42 with newly diagnosed non-germinomatous germ cell tumors.

43 RAS/LKB1-mutant adenocarcinomas and squamous cell tumors.

44 gene is elevated in most tenosynovial giant-cell tumors.

45 e inactivated in a majority of lung squamous cell tumors.

46 typical prolonged washout observed in Leydig cell tumors (12 of 21 patients, P < .001 when compared w

47 y higher in adenocarcinomas than in squamous cell tumors (17.73 vs 11.46; P = .043).

48 umors (29%), predominantly in Sertoli-Leydig cell tumors (26 of 43, or 60%), including 4 tumors with

49 primitive neuroectodermal tumors, 57.8; germ cell tumors, 63.5; ependymoma or high-grade glioma, 69.8

50 h hepatoblastoma or desmoplastic small round cell tumor achieved a partial response; eight patients h

51 cells (Tregs) and myeloid-derived suppressor cells, tumor Ag-specific Tregs interfere with the detect

52 nce of FOXL2 and KRAS mutations in granulosa cell tumors and in mucinous tumors, respectively.

53 e of cisplatin resistance in testicular germ cell tumors and may also be implicated in ovarian and pr

54 cal margins, and indolent tumor types (islet cell tumors and mucinous cystadenocarcinoma).

55 thyroid, thymic and lymph node lesions; germ cell tumors and vascular lesions.

56 a target of chromosomal translocations in T-cell tumors and was activated by retroviral vector inser

57 ion can be abrogated by exposing susceptible cells (tumor and mycobacteria-infected cells, or aminobi

58 nd paraganglioma, pericardial cyst, Purkinje cell tumor, and papillary fibroelastoma (n = 1, each).

59 lution in relation to retinoblastoma or germ cell tumors, and both cancers are rare, these findings r

60 lps explain oncogene mutations observed in B-cell tumors, and further, that many oncogenes are vulner

61 sion was validated in Ewing sarcoma cells, B-cell tumors, and human keratinocytes where NOTCH dysregu

62 expression is deregulated in testicular germ cell tumors, and loss of imprinting occurs frequently in

63 ype-specific variations, chromophobes, clear-cell tumors, and oncocytomas were composed exclusively o

64 nocarcinomas, squamous cell carcinomas, mast cell tumors, and soft tissue sarcomas.

65 vel marker for this lymphoma across mature B-cell tumors, and support the distinction of NMZL as an i

66 d-glucose (6-[(18)F]FDG) was studied in EMT6 cells, tumors, and muscle and correlated to GLUT1 and GL

67 and is highly expressed in a variety of stem cells, tumors, and tumor cell lines.

68 ic antigen receptors (CAR) specific to the B cell tumor antigen CD19 can successfully eradicate syste

69 Germ cell tumors are a heterogeneous group of neoplasms deriv

70 Endothelial cell tumors are the most common soft tissue tumors in in

71 Endothelial cell tumors are the most common soft tissue tumors in in

72 Leydig cell tumors are the most frequent interstitial neoplasms

73 Insulinomas (pancreatic islet beta cell tumors) are the most common type of functioning pan

74 ic astrocytoma (PA) is the most common glial cell tumor arising in children.

75 Follicular lymphoma (FL) is a B-cell tumor arising in germinal centers and retaining fea

76 te Phf7 expression, is also observed in germ cell tumors arising from the loss of bag of marbles (bam

77 OCT4 is commonly expressed in germ-cell tumors as well as putative cancer stem cells in sev

78 d-MyD88 infection of primary human dendritic cells, tumor-associated fibroblasts, and colorectal carc

79 Among tumor-infiltrating immune cells, tumor-associated macrophages (TAMs) take a center

80 ular immune modulators, such as regulatory T cells, tumor-associated macrophages, and myeloid-derived

81 PD-L1 is expressed on tumor cells, tumor-associated macrophages, and other cells in

82 iates multidirectional crosstalk among tumor cells, tumor-associated myeloid cells, and ECs that cont

83 ose proximity to colonies of prostate cancer cells, tumor-associated stromal cells and newly formed w

84 noculated with 2 x 10(7) of Walker 256 tumor cells [tumor bearing (TB) rats].

85 her the humoral immune response affects germ cell tumor biology.

86 the diagnosis of adrenomedullary chromaffin cell tumors can be jeopardized by physiologic uptake by

87 nal translocations, nearly all analyzed CP B-cell tumors carried clonal translocations.

88 antly increases CD20 levels in established B-cell tumor cell lines and primary malignant cells.

89 We analyzed three pairs of testicular germ cell tumor cell lines using Affymetrix expression microa

90 ubpopulations of tumor cells through stromal cell-tumor cell interactions.

91 For nonfixed, viable cells, tumor cell recovery ranged from 72.5% to 93.9% wi

92 be killed by CD8(+)alphabetaT or gammadeltaT cells; tumor cell killing was partially restored by trea

93 of PD-L1 expression in both tumor and immune cells (tumor cells: 22c3, 2.96; 28-8, 3.26; SP142, 1.99;

94 lon cancer (GI(50) value of 565 nM in SW-620 cells) tumor cells.

95 their proximity to potential immune effector cells, tumor cells grow aggressively on these immune agg

96 Unlike normal differentiated cells, tumor cells metabolize glucose via glycolysis und

97 The interplay between stromal cells, tumor cells, and migratory cells such as lymphocy

98 CD8(+) T cells (CTLs) kill virally infected cells, tumor cells, or other potentially autoreactive T

99 Analysis of testicular germ cell tumor clinical samples by quantitative reverse tran

100 g NKG2D ligands had increased incidence of B cell tumors, confirming that the inability to clear NKG2

101 for the different stages of testicular germ cell tumors continue to be defined and refined, as resea

102 ffector T-cell activation and natural killer cell tumor cytotoxicity.

103 Among B-cell tumors, deletion of 7q and NOTCH2 mutations are alm

104 We also profiled the microRNA of B-cell tumors derived from diffuse large B-cell lymphoma,

105 nografted with human EGFRvIII-expressing GBM cells, tumor-derived suPAR was detected in the plasma, a

106 w that let-7 functionally inhibits non-small cell tumor development.

107 Desmoplastic small round cell tumor (DSRCT) is a rare disease of children, adoles

108 Desmoplastic small round cell tumor (DSRCT) is a rare sarcoma in adolescents and

109 al translocation in desmoplastic small round-cell tumors (DSRCT), a rare but aggressive soft tissue s

110 ily tumors (EFT) or desmoplastic small round cell tumors (DSRCT).

111 One patient (desmoplastic round cell tumor [DSRCT]) achieved a confirmed partial respons

112 c niche consisting of transformed epithelial cells, tumor-educated fibroblasts, endothelial cells, an

113 The extragonadal germ cell tumors (EGCTs) represent a unique entity, and as su

114 endocrine tumors (pNET), also known as islet cell tumors, exhibit a wide range of biologic behaviors

115 A subset of B-cell tumors exhibits recurrent translocations of Bcl-3,

116 veral human B cell cancer lines, and human B cell tumors expressing AID at high levels have genomic u

117 n cohort of patients with tenosynovial giant-cell tumors (extension study).

118 a critical role in the progression of Hut78 cell tumor formation in skin, thus providing a new thera

119 wn to be critically required for endothelial cell tumor formation.

120 Squamous cell tumors from patients treated with an RAF inhibitor

121 tivated by retroviral vector insertions in T-cell tumors from X-SCID patients in gene therapy trials.

122 rowth, were enriched in NMZL across mature B-cell tumors, functionally caused the loss of the phospha

123 rpose Patients with relapsed metastatic germ cell tumor (GCT) can be cured with second-line and even

124 Germ cell tumor (GCT) is the most common malignancy in young

125 Giant cell tumor (GCT) of bone is a histologically benign oste

126 toposide [E] with stem-cell support) in germ cell tumor (GCT) patients predicted to have a poor progn

127 re conducted on 488 North American male germ cell tumor (GCT) survivors in relation to cumulative cis

128 nicopathologic heterogeneity, malignant germ cell tumors (GCT) share molecular abnormalities that are

129 sitivity, most patients with metastatic germ cell tumors (GCTs) are cured with cisplatin-based chemot

130 The anterior mediastinal germ cell tumors (GCTs) are the most common EGCT.

131 are in children, with pineoblastoma and germ cell tumors (GCTs) being the most common.

132 or children with extracranial malignant germ cell tumors (GCTs) have increased significantly.

133 granulosa cells causes metastatic granulosa cell tumors (GCTs) in female mice and phenocopies human

134 alignant lesions that develop into granulosa cell tumors (GCTs) spontaneously later in life or follow

135 l dietary intake patterns and pediatric germ cell tumors (GCTs) using principal components analysis a

136 in two thirds of patients with advanced germ cell tumors (GCTs) who relapsed after first-line chemoth

137 mainstay of treatment for disseminated germ cell tumors (GCTs), is associated with venous thromboemb

138 Germ-cell tumors (GCTs), which arise from pluripotent embryon

139 py (HDCT) in the management of advanced germ cell tumors (GCTs).

140 e-wide association study for testicular germ cell tumor, genotyping 298,782 SNPs in 979 affected indi

141 - and drug-resistant Emu-myc p53-/- lymphoma cell tumors grown in live mice.

142 In slices of LLC1 cell tumors grown in mice to approximately 5 mm diameter

143 Inhibiting Sod2 expression reduces OCCC ES-2 cell tumor growth and metastasis in a chorioallantoic me

144 y, chronic MAF overexpression enhanced MPNST cell tumor growth in vivo, correlating with elevated pS6

145 g L1CAM was sufficient to reduce glioma stem cell tumor growth in vivo, we targeted L1CAM in glioma c

146 dicer and microRNA in promoting endothelial cell tumor growth in vivo.

147 inhibitor imatinib after injection of cancer cells; tumor growth was measured based on bioluminescenc

148 n study, 12 patients with tenosynovial giant-cell tumors had a partial response and 7 patients had st

149 edical treatment of advanced testicular germ cell tumors has changed over the past 30 years, with lon

150 mphocytic leukemia (CLL) and other related B-cell tumors have been described in the literature and st

151 tients with clinical stage I testicular germ cell tumors have been managed with adjuvant radiotherapy

152 spectrum of human neoplasms, including germ cell tumors, high-grade and low-grade carcinomas and ben

153 cell clones were often observed in T- and NK-cell tumors in a percentage higher than observed in reac

154 BLV infection is strongly associated with B-cell tumors in cattle.

155 s), suggesting similar etiology between germ cell tumors in mouse and man.

156 In this study, we characterized the B-cell tumors in MSH6-deficient mice and describe their hi

157 lood vessels of spontaneous pancreatic islet-cell tumors in RIP-Tag2 transgenic mice.

158 Loss of nito activity results in stem cell tumors in the female germ line as well as female-to

159 al important developments in testicular germ cell tumors in the last year.

160 al important developments in testicular germ cell tumors in the past year.

161 al important developments in testicular germ cell tumors in the past year.

162 al important developments in testicular germ cell tumors in the past year.

163 d slows the growth of these "addicted" pre-B-cell tumors in vivo, suggesting a promising therapeutic

164 s in 1 of 14 nonseminomatous testicular germ-cell tumors, in 2 of 5 embryonal rhabdomyosarcomas, and

165 tients with intermediate- and poor-risk germ cell tumors, in whom 4 cycles of bleomycin, etoposide, a

166 rated lymphocytes, low level of regulatory T-cell tumor-infiltrated lymphocytes, and low PD-L1 expres

167 region from 320 sequences expressed by germ cell tumor-infiltrating B cells revealed clear evidence

168 DQB2, HLA-DMA, and HLA-DOA), increased CD8 T-cell tumor infiltration (P=7.6x10(-5)), and trans-regula

169 growth, increased antigen-specific CD8(+) T-cell tumor infiltration, and increased median survival,

170 type and in facilitating CD4(+) and CD8(+) T-cell tumor infiltration.

171 zation of cancer in which only a fraction of cells, tumor-initiating cells (TICs), can sustain tumor

172 ith menin knockdown, in MEN1-associated beta cell tumors (insulinomas), and also in human sporadic in

173 n oncogenic factor in insulin-secreting beta-cell tumors (insulinomas).

174 Our model provides a system to study T cell-tumor interactions in detail and to test the effica

175 have implications for bidirectional CD4(+) T-cell:tumor interactions within the TME.

176 idence interval (CI): 2.65, 3.50), with germ cell tumors (IRR = 5.19, 95% CI: 2.67, 9.41), retinoblas

177 transformed human cell grows into a billion-cell tumor is uncertain because serial observations are

178 etween chlordane isomers and testicular germ cell tumors, it is reasonable to assume that chlordanes

179 Secondly, a subset of testicular germ cell tumors, known as non-seminomas, often contain diffe

180 However, chemotherapy of germ-cell tumors led to the induction of anti-OCT4 immunity i

181 g peptide that specifically recognizes tumor cells, tumor lymphatics, and tumor-associated macrophage

182 tramer-positive CD8 T cells, increased CD8 T-cell tumor lytic activity, augmented antigen-specific IF

183 data indicate that TC-PTP is required for B-cell tumor maintenance.

184 biopsy-proven peripheral T1-T2N0M0 non-small cell tumors (measuring <5 cm in diameter) and medical co

185 Effects against mitochondria and general cell tumor metabolism were noted at higher concentration

186 s with intermediate-risk (IR) malignant germ cell tumors (MGCT) if the administration of cisplatin, e

187 ting of a genetic disease, the B-cell/plasma cell tumor microenvironment (TME) contributes significan

188 spontaneous translocations in other mouse B-cell tumor models, CP B-cell tumor translocations were n

189 ic characteristics of SED against H1650 stem cell tumor models.

190 ent studies demonstrate that testicular germ cell tumor mortalities can vary significantly in differe

191 vival outcomes in metastatic testicular germ cell tumor (MT-GCT), but how the initial risk changes ov

192 own as HHV8) is the causative agent of two B cell tumors, multicentric Castleman disease (MCD) and pr

193 tivation of genes expressed in retinal glial cells, tumor necrosis factor-alpha, and transforming gro

194 gement of patients with nonseminomatous germ cell tumor (NSGCT) who achieve a serologic and radiograp

195 osteosarcoma and, less frequently, in giant cell tumor of bone (GCT).

196 d villonodular synovitis (dtPVNS), and giant cell tumor of bone (GCTB).

197 in DSRCT, solitary fibrous tumor, and giant cell tumor of bone suggests that future evaluation of su

198 PURPOSE OF REVIEW: Giant cell tumor of tendon sheath and pigmented villonodular s

199 -year-old man with a history of a mixed germ cell tumor of the testis presented with acute-onset, rig

200 In contrast, in 92% (49/53) of giant cell tumors of bone, we found histone H3.3 alterations e

201 that has been implicated in testicular germ cell tumors of mouse and human.

202 The majority of small-cell tumors of the gynecologic tract will require system

203 Germ cell tumors of the ovary constitute less than one percen

204 Validation in a bigger cohort of clear-cell tumors of the ovary is warranted.

205 acteristics of large-cell calcifying Sertoli cell tumors of the testes (LCCSCTs).

206 ciplinary approach to the management of germ cell tumors of the testis has resulted in survival rates

207 e the surgical management of metastatic germ cell tumors of the testis, highlighting the indications

208 Like several other tumor types, germ cell tumors often harbor an immune cell infiltrate that

209 f health care resources between ovarian germ cell tumor (OGCT) survivors and age/race/education-match

210 ratio (OR) = 1.05; 95% CI: 1.01, 1.10]; germ cell tumors (OR = 1.16; 95% CI: 1.04, 1.29), particularl

211 3 concentrations and risk of testicular germ-cell tumors (p > 0.05).

212 only associated with endometrioid and clear cell tumors (P-het </= .01).

213 mutant p80HT, which develop predominantly B cell tumors, p52 transgenic mice are not prone to lympho

214 riplatin in the National Cancer Institute 60-cell tumor panel screen revealed a spectrum of activity

215 We extracted nascent plasma cell tumor (PCT) cells from within inflammatory oil gran

216 ecapitulating human perivascular epithelioid cell tumors (PEComas) from patients with TSC.

217 PURPOSE Perivascular epithelioid cell tumors (PEComas) represent a family of mesenchymal

218 f the resected tissue showed metastatic germ cell tumor predominantly consisting of a yolk sac elemen

219 Mice susceptible to plasma cell tumors provide a useful model for human multiple my

220 NK group 2D ligands in the impairment of NK cell tumor recognition and killing.

221 Upon direct recognition of cancer cells, tumor-recognizing CD4(+) T cells (TR-CD4) potentl

222 induction promoted drug resistance in MCF-7 cells, tumor regeneration in Du145 cells and, most impor

223 Significant challenges for poor-risk germ cell tumors remain.

224 postorchiectomy early-stage nonseminoma germ cell tumors remains a topic of debate.

225 s a partner in recurrent translocations in B cell tumors, resulting in deregulated expression.

226 Analysis of human germ cell tumors reveals similar gene expression changes corr

227 xamined associations between testicular germ-cell tumor risk and circulating concentrations of insuli

228 Human germ cell tumors show a strong sensitivity to genetic backgro

229 ve been reported with IFNgamma-producing Th1 cells, tumor-specific Th2 cells have been largely neglec

230 the immune cell infiltrate of all three germ cell tumor subtypes and defined the molecular characteri

231 y broad and high expression levels in plasma cell tumors such as multiple myeloma (MM).

232 on of human carcinomas, e.g. diffuse large B cell tumors, T cell lymphomas, etc.

233 hepatoblastomas (HBs), 1 transitional liver cell tumor (TCLT), 1 hepatocellular carcinoma, and 2 mal

234 lex genetic factors underlie testicular germ cell tumor (TGCT) development.

235 association (GWA) studies of testicular germ cell tumor (TGCT) have identified 18 susceptibility loci

236 Testicular germ cell tumor (TGCT) is the most common cancer in young men

237 aggressive fibromatosis, tenosynovial giant cell tumor (TGCT) or diffuse-type pigmented villonodular

238 molecular pathogenesis of tenosynovial giant cell tumor (TGCT) or pigmented villonodular synovitis (P

239 conventional inheritance for testicular germ cell tumor (TGCT) risk both in humans and mice implicate

240 it male infertility, a known testicular germ cell tumor (TGCT) risk factor.

241 yzed population-wide data on testicular germ-cell tumor (TGCT) status in 1,135,320 two-generational N

242 the most potent modifiers of testicular germ cell tumor (TGCT) susceptibility in mice and rats.

243 c modifier known to suppress testicular germ cell tumor (TGCT) susceptibility in mice or humans.

244 variants act as modifiers of testicular germ cell tumor (TGCT) susceptibility in the 129/Sv mouse mod

245 e-wide association study for testicular germ cell tumor (TGCT), genotyping 307,666 SNPs in 730 cases

246 new susceptibility loci for testicular germ cell tumor (TGCT).

247 inct susceptibility loci for testicular germ cell tumor (TGCT).

248 -wide association studies of testicular germ cell tumor (TGCT; 3,558 cases and 13,970 controls) to id

249 Testicular germ cell tumors (TGCT) are considered a paradigm of chemosen

250 Testicular germ cell tumors (TGCT) are sex limited, occurring only in ma

251 Testicular germ cell tumors (TGCT) are the most common solid tumors of 1

252 Testicular germ cell tumors (TGCT) are the most frequently diagnosed sol

253 Testicular germ cell tumors (TGCT) generally respond well to chemotherap

254 Susceptibility to testicular germ cell tumors (TGCT) has a significant heritable component

255 Testicular germ cell tumors (TGCT) have been expected to have a strong u

256 control of susceptibility to testicular germ cell tumors (TGCT) in humans or mice.

257 Testicular germ cell tumors (TGCT) originate from germ cells.

258 Testicular germ cell tumors (TGCT) represent the most common malignancy

259 actor for the development of testicular germ cell tumors (TGCT), but the initiating event linking the

260 influences susceptibility to testicular germ cell tumors (TGCT), the most common testicular cancer in

261 and thereby increase risk of testicular germ cell tumors (TGCT).

262 Testicular germ cell tumors (TGCTs) are highly responsive to and curable

263 e enhances susceptibility to testicular germ cell tumors (TGCTs) in mice, in part by interacting epig

264 , and risk stratification of testicular germ cell tumors (TGCTs) in the past year.

265 al important developments in testicular germ cell tumors (TGCTs) over the past year.

266 Testicular germ cell tumors (TGCTs) share germline ancestry but diverge

267 d and highly expressed in tenosynovial giant cell tumors (TGCTs), and this observation allowed us to

268 tanding of susceptibility to testicular germ cell tumors (TGCTs), but much of the heritability remain

269 so are misregulated in human testicular germ cell tumors (TGCTs), suggesting similar etiology between

270 Testicular germ cell tumors (TGCTs), the most common neoplasms of young

271 ed with an increased risk of testicular germ cell tumors (TGCTs).

272 er drug for the treatment of testicular germ cell tumors (TGCTs).

273 verage type and were slightly stronger for B-cell tumors than for T-cell tumors.

274 l zone lymphoma (NMZL) is a rare, indolent B-cell tumor that is distinguished from splenic marginal z

275 agnosis and therapy of malignant small round cell tumors that affect children, particularly in rhabdo

276 Insulinomas are beta-cell tumors that cause hypoglycemia through inappropriat

277 eloped aggressive ovarian surface epithelial cell tumors that did not occur in the Ptenfl/fl;KrasG12D

278 is, leading to the accumulation of granulosa cell tumors that reactivates the epithelial program from

279 results in an ovarian phenotype of granulosa cell tumors that renders the animals infertile.

280 rtoli cells also caused testicular granulosa cell tumors that showed gene expression patterns that pa

281 e for NF1 tumor suppressor function in glial cell tumors, the mechanisms underlying transformation re

282 protein in primary human colonic epithelial cells, tumor tissues, and colon cancer cell lines.

283 with the widespread expression in epithelial cells, tumor tissues, and macrophages detected using BTN

284 e to the hypersensitivity of testicular germ cell tumors to cisplatin, are discussed.

285 amined DNA methylation patterns in TCL1-tg B-cell tumors to discover tumor-associated epigenetic chan

286 ons in other mouse B-cell tumor models, CP B-cell tumor translocations were not recurrent and did not

287 ere indeed found in 7% of 43 testicular germ cell tumor trios; this percentage exceeds background CNV

288 s expression levels in human testicular germ cell tumors using patient tissues, model cell lines, and

289 glucopyranosyl lipid A (GLA), using a whole-cell tumor vaccine.

290 r the interaction between RRV and human host cells (tumor versus nontumor) in vitro.

291 importance of versican in this smooth muscle cell tumor, we used versican-directed siRNA to knock dow

292 SYK credentials as a therapeutic target in B-cell tumors, we explored the role of PDE4B in these resp

293 Lung epithelial TC1 cell tumors were 84% greater in mice subjected to IH for

294 Malignant germ cell tumors were identified in 2.8% (31 of 1097) of boys

295 treatment of patients with nonseminoma germ cell tumor, whereas radiotherapy, as a standard treatmen

296 skin developed significantly fewer squamous cell tumors with a delayed onset when compared with wild

297 The treatment of advanced testicular germ cell tumors with cisplatin combination chemotherapy is b

298 Treatment of tenosynovial giant-cell tumors with PLX3397 resulted in a prolonged regress

299 529 was highly active against subcutaneous B-cell tumor xenografts in severe combined immunodeficient

300 ur subsequent extensive analysis of cultured cells, tumor xenografts, and cancer patient biopsies sup