ライフサイエンスコーパス: cell-cell contact

1 invasion and arises as a result of intimate cell-cell contact.

2 HIV-1 structural protein Gag to the site of cell-cell contact.

3 increased by the TGF-beta/Smad3 pathway and cell-cell contact.

4 PRF was induced during cell proliferation by cell-cell contact.

5 ecules such as E-cadherin are present at the cell-cell contact.

6 kinase in response to apoptotic stimuli and cell-cell contact.

7 ization of the small GTPase RhoA at sites of cell-cell contact.

8 vers signaling proteins directly at sites of cell-cell contact.

9 bule organization and dynamics near sites of cell-cell contact.

10 ibit the growth of susceptible bacteria upon cell-cell contact.

11 mation of signaling clusters at the sites of cell-cell contact.

12 an be detected during the first minute after cell-cell contact.

13 P-B colocalize with p120-catenin at sites of cell-cell contact.

14 lular protein localization, and detection of cell-cell contact.

15 modulates tissue homeostasis in response to cell-cell contact.

16 with prostaglandin E2 (PGE2) production and cell-cell contact.

17 omotes tumor cell survival in the absence of cell-cell contact.

18 ll contact and transwell systems to separate cell-cell contact.

19 n restore functional autophagic flux through cell-cell contact.

20 ctomyosin contraction along the periphery of cell-cell contact.

21 junction-associated actin bundles soon after cell-cell contact.

22 expression, leading to a loss of epithelial cell-cell contact.

23 VCAM-1 expression, resulting in insufficient cell-cell contact.

24 sue structure and function following de novo cell-cell contact.

25 herens junction maturation following de novo cell-cell contact.

26 t migration to the tips of myofibers through cell-cell contact.

27 hrough Eph receptors is largely dependent on cell-cell contact.

28 cells that involved the gamma-delta TCR and cell-cell contact.

29 ract within the trabecular layer to form new cell-cell contacts.

30 ing through the formation of uropod-mediated cell-cell contacts.

31 nhibits cell proliferation upon establishing cell-cell contacts.

32 ing through the formation of uropod-mediated cell-cell contacts.

33 architecture through apicobasal polarity and cell-cell contacts.

34 cell (DC) functions via soluble mediators or cell-cell contacts.

35 inase Src and stimulates Src signaling along cell-cell contacts.

36 enchymal, lacked ruffles but formed abundant cell-cell contacts.

37 for KRIT1 depletion-dependent disruption of cell-cell contacts.

38 nin conformational epitope immunostaining at cell-cell contacts.

39 tion of the RASIP1 protein pool localizes to cell-cell contacts.

40 oA activity, and results in loss of p190B at cell-cell contacts.

41 s ameloblast movement by cleaving ameloblast cell-cell contacts.

42 al filopodia to discrete membrane domains at cell-cell contacts.

43 catenin and the Arp2/3 complex at developing cell-cell contacts.

44 rtance of Syx function in maintaining stable cell-cell contacts.

45 es the dynamic stability of E-cadherin-based cell-cell contacts.

46 ation and lumen morphogenesis by maintaining cell-cell contacts.

47 lated in the cytoplasm at the cell edges and cell-cell contacts.

48 helial barrier function that is regulated by cell-cell contacts.

49 tes the re-assembly of E-cadherin-containing cell-cell contacts.

50 is fragment localized to cadherin-containing cell-cell contacts.

51 t S285 to promote the maturation of immature cell-cell contacts.

52 protein CdGAP, a GAP for Rac1 and Cdc42, at cell-cell contacts.

53 hat APLP1 forms homotypic trans complexes at cell-cell contacts.

54 he virus and its ability to spread by way of cell-cell contacts.

55 ons without a corresponding reinforcement of cell-cell contacts.

56 ring, may contribute to the reinforcement of cell-cell contacts.

57 d in cardiomyocytes disrupts localization at cell-cell contacts.

58 basal epithelial polarity, as well as proper cell-cell contacts.

59 al features by perturbing cell-substrate and cell-cell contacts.

60 This HIV-1 transmission was mediated by cell-cell contacts.

61 of the existence of oligomeric cadherins at cell-cell contacts.

62 nterferes with the initiation of sustainable cell-cell contacts.

63 immunogenic stimulus, physical disruption of cell-cell contacts a tolerogenic stimulus.

64 Tbx3, Klf4 and Esrrb transcript repression, cell-cell contact abrogation, cell survival in suspensio

65 Here we show that in the absence of cell-cell contact, actomyosin contractility suppresses Y

66 bjected to serum starvation, allowed to form cell-cell contacts, after microtubule disruption, or inh

67 Cell-cell contacts also direct ingression of the cleavag

68 of whether or not E-cadherin is recruited to cell-cell contacts, although tension is further increase

69 ormation requires regulation at the level of cell-cell contacts among brain cells.

70 anges of EMT markers, suggesting that direct cell-cell contact and aberrant COX-2 expression synergis

71 ucing microtubule catastrophe at the site of cell-cell contact and abrogating CIL.

72 ation was found to be independent of loss of cell-cell contact and Activin/Nodal-dependent pluripoten

73 Tight cell-cell contact and an increase in E-cadherin but a co

74 nterestingly, trogocytosis of PD-L1 requires cell-cell contact and cannot be induced by uptake of sol

75 oach was employed to decouple the effects of cell-cell contact and cell-matrix adhesion in TGFbeta1-i

76 us maturation of Swap-70(-/-) DCs depends on cell-cell contact and does not involve beta-catenin sign

77 egulatory cytokines such as IL-10 and direct cell-cell contact and have been linked to experimental m

78 remodelling to facilitate cadherin-mediated cell-cell contact and promote beta-catenin signalling.

79 the Notch receptor pathway is responsive to cell-cell contact and regulates keratinocyte growth and

80 Ed/Sav interaction is promoted by cell-cell contact and requires dimerization of Ed cytopl

81 ns appear to be mediated through both direct cell-cell contact and secreted ligands.

82 reated cells did not require phagocytosis or cell-cell contact and thus occurs through a different me

83 in murine or human MCs by using both direct cell-cell contact and transwell systems to separate cell

84 s within 24 hours that were shown to exhibit cell-cell contact and uniform size (201 +/- 2 mum).

85 CCM2) are critical regulators of endothelial cell-cell contact and vascular homeostasis.

86 s induced Treg proliferation, which required cell-cell contact and was MHC class II-dependent.

87 teral clustering of adhesion receptors after cell-cell contact and, more generally, to the formation

88 liver Syndrome patients strongly destabilize cell-cell contacts and (ii) CdGAP mRNA levels are invers

89 east epithelium results in disruption of the cell-cell contacts and acquisition of a mesenchymal phen

90 sed the localization of the other protein to cell-cell contacts and altered AJ dynamics and stability

91 ell confinement in such geometries increased cell-cell contacts and altered colony organization.

92 esion, Elmo2 and Dock1 no longer localize to cell-cell contacts and are not required subsequently for

93 ced endocytosis of E-cadherin, disruption of cell-cell contacts and cell scatter.

94 -independent growth, reduced ability to form cell-cell contacts and chromosomal aberrations.

95 We also determined that cell-cell contacts and cortically associated EB1/beta-ca

96 s, resulting in delocalization of ROCK2 from cell-cell contacts and decreased junctional contractilit

97 y expressed adhesion molecule that regulates cell-cell contacts and facilitates leukocyte transendoth

98 imensional (3D) culture, whereas it promotes cell-cell contacts and induces various hallmarks of diff

99 ests that RhoA exists as an inactive pool at cell-cell contacts and is recruited to cell-ECM contacts

100 ering, epithelial cell islands rupture their cell-cell contacts and migrate away as single cells on t

101 is required for PIP5KIgamma accumulation at cell-cell contacts and phosphatidyl inositol 4,5-bisphos

102 ced by some chemotherapeutic agents required cell-cell contacts and proceeded through an endocytic pa

103 duces more complete EMT, including disrupted cell-cell contacts and reduced E-cadherin expression, an

104 lective guidance does not require persistent cell-cell contacts and strong short range adhesion.

105 iral envelope glycoprotein (Env) establishes cell-cell contacts and subsequently recruits Gag by a pr

106 clear whether they do so directly via stable cell-cell contacts and sustained TCR signals.

107 y disrupted and a tension difference between cell-cell contacts and the free cell surface at gaps of

108 protein-Myo10 localizes to lateral membrane cell-cell contacts and to filopodia-like structures imag

109 because they are well positioned to form the cell-cell contacts and to provide the intercellular comm

110 t positively affect GSIR and led to impaired cell-cell contacts and vesicle trafficking.

111 tion was blocked by anti-4-1BBL Ab, required cell-cell contact, and did not require the cytoplasmic s

112 This event was dependent on cell-cell contact, and experiments using blocking Abs in

113 L3 expression in primary leucocytes required cell-cell contact, and induction was suppressed by plasm

114 which cells move away from each other after cell-cell contact, and it contributes to malignant invas

115 ucts the assembly of the LGN/NuMA complex at cell-cell contacts, and define a mechanism that couples

116 pithelial cells lose apicobasal polarity and cell-cell contacts, and gain mesenchymal phenotypes with

117 et T cells, requires the formation of stable cell-cell contacts, and is an active, calcium-dependent

118 s ECM stiffness, adhesion ligand density, or cell-cell contacts, and thus strongly influences cell fa

119 (-/-)mEF is greater at higher densities when cell-cell contacts are abundant.

120 Cell-cell contacts are fundamental to multicellular orga

121 cts in distance and is independent of direct cell-cell contact.ARMMs are extracellular vesicles that

122 actin assembly and turnover at newly formed cell-cell contacts as well as for human epithelial lumen

123 ly acts by sterically interfering with close cell-cell contacts at the NK cell-target cell interface

124 Angiogenic sprouting requires that cell-cell contacts be maintained during migration of end

125 eration of specific T cells was dependent on cell-cell contact between B cells and moDCs, which was e

126 We hypothesized that cell-cell contact between plasmacytoid dendritic cells (

127 ted with progressive loss of Tregs, impaired cell-cell contact between Tregs and dendritic cells (DCs

128 l barrier is established by highly regulated cell-cell contacts between epithelial cells.

129 The establishment of cell-cell contacts between presynaptic GABAergic neurons

130 ial cytokinesis, the remodelling of adhesive cell-cell contacts between the dividing cell and its nei

131 At cell-cell contacts, both Elmo2 and Dock1 are essential f

132 he molecular role of E-cadherin not only for cell-cell contact but also for clonal propagation of hPS

133 ragment of Tat, (iii) did not require direct cell-cell contact but appeared rather to be mediated by

134 -binding domains restores cadherin-dependent cell-cell contacts but cannot strengthen intercellular a

135 ll-cycle arrest upon mitogen deprivation and cell-cell contact, but did contribute to RAS(V12)- and r

136 thelial barrier dysfunction by disruption of cell-cell contacts, but not CCL20 secretion.

137 through p190RhoGAP-B, which is localized to cell-cell contacts by association with p120-catenin that

138 erodimer is not only recruited to the apical cell-cell contacts by binding to Crb but depends on func

139 Rack1 stabilizes E-cadherin and catenins at cell-cell contacts by inhibiting the Src phosphorylation

140 ave demonstrated that complete disruption of cell-cell contact can promote transforming growth factor

141 ing important cellular processes involved in cell-cell contact, cell adhesion and motility.

142 lated by intrinsic cell machineries, such as cell-cell contact, cell polarity, and actin cytoskeleton

143 ellular and intracellular signals, including cell-cell contact, cell polarity, mechanical cues, ligan

144 tributions to heterogeneity from cell cycle, cell-cell contact, cell stochasticity and heritable morp

145 hat the formation of STK11-dependent lateral cell-cell contacts competent for tyrosine kinase signali

146 ecular level, little is known about how this cell-cell contact dependent feedback is transmitted at t

147 ated, but their role in juxtacrine (that is, cell-cell contact dependent) signaling in NSC niches has

148 omoted apoptosis of primary murine HSCs in a cell-cell contact-dependent manner, involving Fas-ligand

149 Notch regulates cell-cell contact-dependent signaling and is activated b

150 Cell-cell contact-dependent signaling involved beta2 int

151 g, with bulk cell stiffness and stiffness of cell-cell contacts dictating the invasion pattern.

152 hile, E-cadherin clusters throughout lateral cell-cell contacts display dynamic movements in the plan

153 the interplay between cell polarization and cell-cell contact drives the segregation of these lineag

154 Induction of PTPRF by cell-cell contact during cell proliferation quenched the

155 crotubule plus ends interact with regions of cell-cell contact during tissue development and morphoge

156 al morphogenetic movements while maintaining cell-cell contacts during embryogenesis and post-embryon

157 ension-dependent adaptation of AJs regulates cell-cell contact dynamics and coordinated collective ce

158 We showed that, without direct cell-cell contact, ECs secrete factors that promoted the

159 We surmise, however, that cell-cell contacts enabling HIV-1 fusion with the plasma

160 nhibiting ATE1 disrupted E-cadherin-mediated cell-cell contacts, enhanced formation of actin-rich pro

161 ading is controlled, the presence of partial cell-cell contacts enhances expression of alphaSMA.

162 CD73 associated with cell-cell contacts, filopodia, and membrane zippers, ind

163 trols the anchoring of cadherin to actin and cell-cell contact fluidity.

164 Cell-cell contact formation following cadherin engagemen

165 er in modulating myosin II activation during cell-cell contact formation in Madin-Darby canine kidney

166 Cell-cell contact formation is a dynamic process requiri

167 cell adhesion assay and live cell imaging of cell-cell contact formation revealed that inhibition of

168 duces cell-cell internalization during early cell-cell contact formation, which involves active invas

169 is robust to severe perturbations affecting cell-cell contact formation.

170 y localized by active Cdc42 at the external, cell-cell contact-free surfaces of apically constricting

171 ypothesis, we found that the expression of a cell-cell contact gene, Desmoplakin, is greatly reduced

172 ell types and formation of multiple sites of cell-cell contact, giving rise to nascent fusion pores w

173 cell proliferation is complete and adhesive cell-cell contacts have been refined, the vast majority

174 HLSECs internalized HCV, independent of cell-cell contacts; HCV RNA was translated but not repli

175 ought to examine how the presence of partial cell-cell contacts impacts EMT.

176 G and betaII-spectrin colocalize at sites of cell-cell contact in columnar epithelial cells and promo

177 and zebrafish that Par3 is localised to the cell-cell contact in neural crest cells and is essential

178 Whether HIV also spreads by cell-cell contact in vivo is a matter of debate.

179 We find that Rab35 accumulates at cell-cell contacts in a cadherin-dependent manner.

180 rations in cell morphology, cytoskeleton and cell-cell contacts in a gradient-like manner.

181 side binding cholera toxin B subunit Ctb, at cell-cell contacts in a p120-catenin-independent manner,

182 induced intramembrane proteolysis to monitor cell-cell contacts in animals.

183 CDH11 mediates cell-cell contacts in both valvular fibroblasts and myof

184 of cadherin trafficking and stabilization at cell-cell contacts in C2C12 myoblasts and HeLa cells.

185 Hakai, an E3 ubiquitin ligase, disrupts cell-cell contacts in epithelial cells and is up-regulat

186 we show that Elmo2 recruits Dock1 to initial cell-cell contacts in Madin-Darby canine kidney cells.

187 hold for nonpatterned cells allowed to form cell-cell contacts in monolayer culture.

188 nificant increase in Rho activity at nascent cell-cell contacts in myosin-IXA depleted cells compared

189 he only contributor to disrupted endothelial cell-cell contacts in the absence of KRIT1.

190 ells, and morphological changes with loss of cell-cell contacts in the epidermal tissue of zebrafish.

191 Cell-cell contacts in tissues are continuously subject t

192 ibits directional migration and destabilizes cell-cell contacts, in part by disturbing the localizati

193 ied E-cadherin as a major contributor to the cell-cell contact-induced osteoblast differentiation.

194 Additionally, we observed that cell-cell contact induces a mitochondrion-dependent incr

195 Here, we show that cell-cell contact induces rapid recruitment of mitochond

196 Cell-cell contacts inhibit cell growth and proliferation

197 e endogenous induction of PXR as a result of cell-cell contact inhibited proliferation and subsequent

198 A reduced responsiveness to cell-cell contact inhibition and an increase in E6/E7 ac

199 Cell-cell contact inhibition and the mechanical environm

200 hat may alter the diffusion dynamics through cell-cell contact interactions.

201 Disruption of endothelial cell-cell contact is a key event in many cardiovascular

202 rection of cell movement with respect to the cell-cell contact is correlated with changes in the aver

203 In contrast, viral spread by direct cell-cell contact is largely unaffected by most of these

204 How the stability of cell-cell contacts is modulated to effect such morpholog

205 ing domain promotes accumulation in areas of cell-cell contact, leading to enhanced adhesion and inhi

206 ligand (FASL)/FAS-mediated death pathway via cell-cell contact, leading to up-regulation of regulator

207 nd both relied on IL-10 secretion as well as cell-cell contact, likely mediated through CD80 and CD86

208 a collagen matrix unravelled the efficacy of cell-cell contact loosening and 3D emigration into an en

209 The process requires direct cell-cell contact made possible by a multiprotein comple

210 s and may depend on secreted factors, direct cell-cell contact, matrix interactions, or a combination

211 PKC phosphorylates JAM-A at S285 to regulate cell-cell contact maturation, TJ formation, and single l

212 ted by chNKG2D T cells through IFN-gamma and cell-cell contact mechanisms.

213 ceptually distinct routes to synaptogenesis: cell-cell contact mediated by adhesion proteins, cell-ce

214 ical inhibition of these pathways abates the cell-cell contact mediated expression of alphaSMA.

215 s early cytoskeletal remodelling and lateral cell-cell contacts, mediated through the RAC1 guanine nu

216 This cell-cell contact-mediated (CCCM) HCV transfer occurs re

217 ty microvascular endothelial cells that lack cell-cell contacts migrate in the flow direction.

218 Adherens junctions (AJs) are essential for cell-cell contacts, morphogenesis, and the development o

219 ether the MDSC induction mechanism relies on cell-cell contact of melanoma cells with CD14(+) cells.

220 of resting cells, at active protrusions and cell-cell contacts of randomly moving cells, and at the

221 SLOs), we characterized the localization and cell-cell contacts of splenic neutrophils at several sta

222 Notch signaling is mediated either by direct cell-cell contact or by Dll4-containing exosomes from a

223 control dynamic F-actin distribution at the cell-cell contact or in membrane protrusions.

224 in the establishment of tight junction (TJ) cell-cell contacts orchestrated by the protein occludin

225 long with PKCzeta-dependent stabilization of cell-cell contacts promotes directed and collective endo

226 However, how MA targets Gag to sites of cell-cell contact remains unknown.

227 In vitro coculture under direct cell-cell contact resulted in indirect suppression of Ig

228 o the cytoplasm of neighboring bacteria upon cell-cell contact, resulting in growth inhibition or dea

229 ial cells reduced the level of active Src at cell-cell contacts, resulting in delocalization of ROCK2

230 lacement and force redistribution in guiding cell-cell contact rupture during scattering.

231 To explore the role of cell motion and cell-cell contact rupture, we examine the biophysical ch

232 ar force as well as the initial direction of cell-cell contact rupture.

233 n-perturbed cells to mitogen deprivation and cell-cell contact seems a better predictor of tumor deve

234 e as a general platform for generating novel cell-cell contact signaling pathways.

235 for secretion of such large granules at the cell-cell contact site.

236 They accumulate at cell-cell contact sites and assemble into large macromol

237 FMNL3 patches also exist, which enrich near cell-cell contact sites and fuse with the plasma membran

238 s Daple/beta-catenin/E-cadherin complexes to cell-cell contact sites, enhances noncanonical Wnt signa

239 of defects in the molecular architecture of cell-cell contact sites, including the adherens and tigh

240 of receptor recruitment and distribution at cell-cell contact sites.

241 throughout the biofilm where it localizes to cell-cell contact sites.

242 cells determined RhoA/Rho-kinase activity at cell-cell contact sites.

243 However, whether cell-cell contact specifically induces dynamic T cell re

244 in alpha3 restores directional migration and cell-cell contact stability, suggesting that cells recog

245 omerization in cellulo and for its impact on cell-cell contact stability.

246 l division was not dependent on septins when cell-cell contacts, such as those with antigen-presentin

247 inhibition of locomotion (CIL), meaning that cell-cell contacts suppress force transduction to the su

248 gradients and repulsive effects arising from cell-cell contact, termed contact inhibition of locomoti

249 involve transmission through zones of tight cell-cell contact that are resistant to neutralizing ant

250 lumen morphogenesis require close epithelial cell-cell contacts that are maintained as a result of ad

251 scores the complexity of actin regulation at cell-cell contacts that involves actin activators, inhib

252 Upon ligand-receptor interaction in sites of cell-cell contact, the transmembrane domain of an engine

253 coated surfaces was employed to mimic native cell-cell contacts.The dynamic strength of the homomeric

254 35 impaired N- and M-cadherin recruitment to cell-cell contacts, their stabilization at the plasma me

255 As motile bacteria reach cell-cell contacts they form plasma membrane protrusions

256 hard surfaces, myxobacteria glide, and upon cell-cell contact, they can efficiently exchange their O

257 of desmoplakin (DP) accumulation at sites of cell-cell contact, they play distinct roles in later ste

258 This striking effect was mediated by direct cell-cell contact through cross-linking of MHC class II

259 t formation and functions by means of direct cell-cell contact through ligands and receptors, such as

260 tivation of the Src tyrosine kinase disrupts cell-cell contacts through an E-cadherin/catenin-depende

261 s demonstrate that RhoA is down-regulated at cell-cell contacts through p190RhoGAP-B, which is locali

262 We propose that Ed may link cell-cell contact to Hpo signaling through binding and s

263 on system that typically engages at sites of cell-cell contact to initiate bidirectional signaling.

264 the body without an apparent requirement for cell-cell contact to persist in vivo.

265 v-containing secretory apparatus to sites of cell-cell contact to support polarized viral assembly an

266 EMT, epithelial cell adhesion switches from cell-cell contacts to mainly cell-ECM interactions, rais

267 ansfer retroviruses in vitro across synaptic cell-cell contacts to uninfected cells, a process called

268 ably, this behavior was independent of local cell-cell contact topologies and of position within the

269 perties could be inferred by the analysis of cell-cell contact topologies, and the nonequilibrium phe

270 eline for its ability to detect vertices and cell-cell contacts, track cells, and identify mitosis an

271 Cell-cell contact triggers active polarization of organe

272 three hypotheses have been proposed: direct cell-cell contact, tunneling nanotubes, and exosomes.

273 Epithelial differentiation and augmented cell-cell contacts underlie the anti-migratory action ex

274 hich involves active invasion of the lateral cell-cell contact underneath the apical-junctional compl

275 beta-PIX, which is specifically recruited at cell-cell contacts upon CCM.

276 oA activity to be significantly decreased at cell-cell contacts versus cell-ECM adhesions, and, of im

277 2 h or 1-3 days, and in selected experiments cell-cell contact was blocked.

278 e of E-cadherin appeared to be enhanced when cell-cell contact was established.

279 When cell-cell contact was minimized either by decreasing the

280 nce axonal growth, demonstrating that direct cell-cell contact was not required.

281 Jun involved in proliferation, adhesion, and cell-cell contact, we found that AP-1 repressed the expr

282 junction reinforcement to stabilize adhesive cell-cell contacts, we propose an alternative mechanosen

283 lling cell migration and actin regulation at cell-cell contacts, we were interested to investigate th

284 ts, although tension is further increased at cell-cell contacts when adhering cells are stretched.

285 oteins are actively recruited to the site of cell-cell contact where the viral material is efficientl

286 endocytic itinerary of Cx43 is altered upon cell-cell contact, which causes Cx43 to traffic by EEA1-

287 rface, setting in motion E-cadherin-mediated cell-cell contact, which establishes apicobasolateral po

288 inate the specific shortening of mesenchymal cell-cell contacts, which in turn powers cell interdigit

289 Immunosuppression via cell-cell contact with apoptotic cells is a well studied

290 This required cell-cell contact with live neutrophils.

291 However, T cells do not require direct cell-cell contact with monocytes, suggesting the importa

292 oma cells required viral replication, direct cell-cell contact with pDCs, and receptor-mediated endoc

293 bserved to specifically establish long-lived cell-cell contact with uninfected cells.

294 mation of capillary-like networks induced by cell-cell contact with vascular smooth muscle cells (vSM

295 diate hepatoblast motility and long-distance cell-cell contacts with the LPM beyond immediate tissue

296 both at the cell-substratum interface and at cell-cell contacts, with the latter being 10-fold more s

297 cytoplasm and subsequent localization to the cell-cell contact zone, assembly of adherens junction co

298 Apical cell-cell contact zones and actomyosin only later moved

299 al tension, but by dynamic linking of apical cell-cell contact zones to an already contractile apical

300 ving cells rapidly drove FMNL2 to epithelial cell-cell contact zones.