ライフサイエンスコーパス: cell-to-cell interaction

1 e that arises from the head-tail symmetry of cell-to-cell interaction).

2 a process that facilitates immune evasion or cell to cell interaction.

3 that ECs can directly activate basophils via cell-to-cell interaction.

4 ors, while the CD8+ T cell response required cell-to-cell interaction.

5 ation during PMN vascular as well as mucosal cell to cell interactions.

6 des present on adjacent cells, thus creating cell to cell interactions.

7 numerous cellular activities, processes, and cell-to-cell interactions.

8 ems that collectively make decisions through cell-to-cell interactions.

9 population structure and closer interspecies cell-to-cell interactions.

10 ear cells (PBMCs) in order to study in vitro cell-to-cell interactions.

11 feration and differentiation, through direct cell-to-cell interactions.

12 ion via stimulating both cell-to-surface and cell-to-cell interactions.

13 These changes implicate extensive cell-to-cell interactions.

14 release and CD4+ T cell proliferation during cell-to-cell interactions.

15 se to the theoretically calculated limit and cell-to-cell interactions.

16 tumor microenvironmental patterns, including cell-to-cell interactions.

17 s a complex network of soluble mediators and cell-to-cell interactions allowing human classically act

18 on as a unique signaling system triggered by cell-to-cell interaction and have been shown to mediate

19 trafficking of cells that affect the dynamic cell-to-cell interactions and determine the outcome of i

20 lates endothelial cytoskeletal architecture, cell-to-cell interactions and lumen formation.

21 nd the tumor microenvironment is mediated by cell-to-cell interactions and soluble factors.

22 t regulate growth, transcription regulation, cell to cell interactions, and morphology.

23 chemokines, CCL5 and CCL6, was dependent on cell-to-cell interaction, and was observed only with fib

24 hich fail to recapitulate spatial aspects of cell-to-cell interactions as well as tissue gradients pr

25 mily of proteins that are thought to mediate cell to cell interactions between neurons.

26 bel-free, and noninvasive probing of dynamic cell-to-cell interactions between adherent and nonadhere

27 These findings show that cell-to-cell interactions between intraepithelial gammad

28 Cell-to-cell interactions between intraepithelial lympho

29 local non-MHC stimulatory factors or direct cell-to-cell interactions between the T cells themselves

30 plethora of signalling processes influencing cell-to-cell interactions between the vascular endotheli

31 istance, are required for citrate-stimulated cell-to-cell interactions, but the GraRS regulatory syst

32 propose that alpha-hemolysin is required for cell-to-cell interactions during biofilm formation.

33 f secreted signaling molecules that regulate cell-to-cell interactions during development.

34 odel predicts three possible benefits of the cell-to-cell interactions: First, the asymmetric interac

35 is important in establishing and maintaining cell to cell interactions in epithelial cells, thereby p

36 active eicosanoids that can be formed during cell to cell interactions in human tissues to self limit

37 , however, bypasses the normal mechanisms of cell-to-cell interactions in which Notch-1 participates.

38 function and that this may be largely due to cell-to-cell interactions mediated by P-selectin.

39 We propose that besides cell-to-cell interaction-mediated Fas triggering, stimul

40 Treatment with ZD1839 reduced the cell-to-cell interaction of HUVECs, resulting in disrupt

41 For the first time, the direct cell-to-cell interactions of tumor cells with bead-activ

42 ollowed by a relatively slower step in which cell-to-cell interactions predominate (the aggregation p

43 d of the macroscopic patterns resulting from cell-to-cell interactions remains largely qualitative.

44 n that the bystander phenomenon must involve cell-to-cell interactions, the relevance of such single-

45 rs cell proliferation, death, migration, and cell-to-cell interaction through contact inhibition.

46 This antiapoptotic effect required direct cell-to-cell interactions, was associated with phosphory

47 hat their morphology is sculpted by specific cell to cell interactions with neurons and each other.

48 OC formation requires cell-to-cell interactions with osteoblasts and can be ac

49 evidence that migratory DCs execute targeted cell-to-cell interactions with stationary MCs before lea