ライフサイエンスコーパス: cell-to-cell movement

1 expression, or enhanced virion stability and cell-to-cell movement.

2 odes proteins needed for virion assembly and cell-to-cell movement.

3 a salt bridge, abolished virion assembly and cell-to-cell movement.

4 ta size-exclusion limits and promoting virus cell-to-cell movement.

5 chanistic link between nuclear transport and cell-to-cell movement.

6 ion spanning these residues was required for cell-to-cell movement.

7 gly suggest that IH co-opt plasmodesmata for cell-to-cell movement.

8 uppression were also non-functional in viral cell-to-cell movement.

9 dependent of silencing but also required for cell-to-cell movement.

10 ship between calreticulin, TMV MP, and viral cell-to-cell movement.

11 ed 3aDeltaC33 MP) resulted in CP-independent cell-to-cell movement.

12 virion tail as a specialized device for BYV cell-to-cell movement.

13 the size of the viral genome at the level of cell-to-cell movement.

14 encoded by RNAbeta but is not essential for cell-to-cell movement.

15 s a tail represents a specialized device for cell-to-cell movement.

16 virus RNA amplification, virus invasion, and cell-to-cell movement.

17 B3), and betad (TGB2), are each required for cell-to-cell movement.

18 nding region resulted in inactivation of TMV cell-to-cell movement.

19 these sites to alter plasmodesmata for virus cell-to-cell movement.

20 leoprotein complexes to facilitate potyvirus cell-to-cell movement.

21 ons that are distinct from those involved in cell-to-cell movement.

22 lones that showed further increased rates of cell-to-cell movement and degrees of systemic invasion i

23 virus (BYV) functions in virion assembly and cell-to-cell movement and is autonomously targeted to pl

24 of MP(TVCV) was necessary for efficient TVCV cell-to-cell movement and systemic infection in Nicotian

25 in a coordinated manner to facilitate virus cell-to-cell movement and that one of these (BR1) is a n

26 omain required for TGB1 self-interaction and cell-to-cell movement and the amino-terminal domain requ

27 a-glucuronidase (GUS), genome amplification, cell-to-cell movement, and long-distance movement were m

28 on of CP-mediated vesicle induction to virus cell-to-cell movement are discussed.

29 viruses is thought to involve two processes: cell-to-cell movement between adjacent cells and long-di

30 f the wild-type TGB3 protein interfered with cell-to-cell movement but movement was not affected by t

31 from TGB2 and TGB3 could function in limited cell-to-cell movement but that the rates of movement dep

32 ficking of the 126-bodies and VRCs and virus cell-to-cell movement, but did not decrease virus accumu

33 ains are capable of genome amplification and cell-to-cell movement, but only TEV-Oxnard is capable of

34 -amino acid peptide was sufficient to impart cell-to-cell movement capacity to GST, a normally cell-a

35 ody and VRC intracellular movement and virus cell-to-cell movement correlates with the disruption of

36 he C terminus of CP functions as a dedicated cell-to-cell movement determinant.

37 component of the viroid infection process is cell-to-cell movement; however, there is virtually no in

38 at while TGB1 self-interaction is needed for cell-to-cell movement, importin-alpha-mediated nucleolar

39 were able to support genome replication and cell-to-cell movement in inoculated leaves.

40 that each of these proteins is required for cell-to-cell movement in monocot and dicot hosts.

41 Cs, translocation to the rice cytoplasm, and cell-to-cell movement in rice.

42 due to suppression of virus replication and cell-to-cell movement in the inoculated leaves of these

43 single cells, suggesting that essentially no cell-to-cell movement occurred in the latter host.

44 his work, we analyze the virion assembly and cell-to-cell movement of a plant closterovirus and revea

45 The rates of cell-to-cell movement of a reporter-tagged TEV strain (T

46 nses to light cues are processed to regulate cell-to-cell movement of auxin to allow establishment of

47 e inhibitory effect of RFA expression on the cell-to-cell movement of Bean dwarf mosaic virus, a sing

48 Cell-to-cell movement of beet yellows closterovirus requ

49 tion, CMV failed to complement the defective cell-to-cell movement of BMV.

50 ate that p20 is dispensable for assembly and cell-to-cell movement of BYV but is required for the lon

51 propose that the 35S RNA may be involved in cell-to-cell movement of CaMV as an intermediate that is

52 lts imply a role for P6 in intracellular and cell-to-cell movement of CaMV.

53 ification on these residues is essential for cell-to-cell movement of Cm-PP16-1.

54 Cell-to-cell movement of different-sized GFP reporters r

55 utant CP interferes with MP accumulation and cell-to-cell movement of infection.

56 , and MAP kinase signalling is important for cell-to-cell movement of invasive hyphae in M. oryzae.

57 s, the TMV-MP was still capable of mediating cell-to-cell movement of itself and the 9.4-kDa F-dextra

58 Demonstrating cell-to-cell movement of mitochondria reconstructs the e

59 We report cell-to-cell movement of mitochondria through a graft ju

60 Plant viral movement proteins mediate the cell-to-cell movement of nucleic acids.

61 sor and for the subcellular localization and cell-to-cell movement of plant viral movement protein.

62 The cell-to-cell movement of plant viruses involves transloc

63 vel, PSTVd cDNA also appears able to mediate cell-to-cell movement of plasmid DNA.

64 e are routinely determined by monitoring the cell-to-cell movement of protein tracers of different si

65 We previously proposed that the cell-to-cell movement of RYMV in xylem involves chelatio

66 The cell-to-cell movement of the Beet yellows virus (BYV) is

67 Here, we present evidence for cell-to-cell movement of the entire 161-kb plastid genom

68 ) along with unlabeled CMV 3a MP resulted in cell-to-cell movement of the F-dextran in control plants

69 Directed cell-to-cell movement of the plant growth hormone auxin

70 Cx43 but not Cx32/Cx26 channels allowed the cell-to-cell movement of the siRNA.

71 t the endoplasmic reticulum as a conduit for cell-to-cell movement of the viral genome.

72 o reach plasmodesmata substantially impaired cell-to-cell movement of the virus.

73 ular proteins known to be involved in local, cell-to-cell movement of tobacco mosaic virus (TMV), is

74 is potentially involved in the regulation of cell-to-cell movement of viral infectious material durin

75 The rates of cell-to-cell movement of virus in inoculated leaves were

76 ts that enhance or suppress TEV replication, cell-to-cell movement, or long-distance movement.

77 ically is not due to defects in replication, cell-to-cell movement, or virion assembly.

78 randed DNA (dsDNA) pararetrovirus capable of cell-to-cell movement presumably through intercellular c

79 This tagged virus was competent in cell-to-cell movement, producing multicellular infection

80 Tomato bushy stunt virus and its cell-to-cell movement protein (MP; P22) provide valuable

81 , and cooperative interaction with the viral cell-to-cell movement protein MP.

82 e barrier of the plant cell wall by encoding cell-to-cell movement proteins (MPs), which direct newly

83 irus (CaLCuV) and Tobacco mosaic virus (TMV) cell-to-cell movement proteins.

84 Examination of the role of L-Pro in BYV cell-to-cell movement revealed that none of the 20 exami

85 ble for virion assembly but are required for cell-to-cell movement, suggesting that the C terminus of

86 nucleus for export to the cell periphery and cell-to-cell movement through plasmodesmata.

87 pex symplastically via the phloem and/or via cell-to-cell movement through plasmodesmata.

88 Thus cell-to-cell movement throughout the epithelium of cytos

89 nal cell is requisite for TSWV infection and cell-to-cell movement; thus, this behavior is most likel

90 e required for efficient virion assembly and cell-to-cell movement, while the C-terminal 65 amino aci