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1 y produce IL-12, whereas freshly isolated decidual CD4(+) T cells expressed high levels of activation markers (CD25, HLA-
2 l studies confirmed that the cell bodies and endfeet of Sus cells expressed high levels of apoE.
3                   Following contraction, BCG-specific CD4 T cells expressed high levels of Bcl-2 and displayed a predomin
4                                             These unusual T cells expressed high levels of Bcl2, p16, and p53, and had li
5                                                       These cells expressed high levels of CC chemokine receptor 5 and we
6                Further analyses showed that these TREM-2(+) cells expressed high levels of CCR-7 and CD86 suggesting a po
7                      In diabetic mice, circulating CD11b(+) cells expressed high levels of CCR5 (P = 0.04), whereas splee
8 , in contrast to CD25(hi) natural Treg cells, CD103(+) Treg cells expressed high levels of CCR5 but low levels of CD62L a
9                                  In contrast, CD22(+) Daudi cells expressed high levels of CD22 mRNA and protein, and wer
10                         Most IFN-gamma- and IL-17-producing cells expressed high levels of CD226, but production of IL-13
11                                                       These cells expressed high levels of CD25 and Foxp3 and suppressed
12                                           AF DENV-labeled B cells expressed high levels of CD27 and CD38 during acute inf
13                                  The activated gammadelta T cells expressed high levels of CD39 and NKG2D on their cell s
14 pressing cells were detected among both CD4(+) and CD8(+) T cells, expressed high levels of CD44 and Tbet, and were able
15                                               The engrafted cells expressed high levels of CD45 and CCR2 and appeared in
16                         Mechanistic study revealed that CLL cells expressed high levels of CPT1 and CPT2.
17                             We found that 4T1 breast cancer cells expressed high levels of CXCR7, but not of CXCR4, both
18 ional profiles were also distinct among subsets--CD16(+) NK cells expressed high levels of cytolytic molecules, and CD56(
19 trast to wild-type cells, these differentiating Gpc4-mutant cells expressed high levels of DOPA decarboxylase and the dop
20                     We revealed that plasma cells but not B cells expressed high levels of DR3 and were direct targets of
21                                PDA-infiltrating gammadeltaT cells expressed high levels of exhaustion ligands and thereby
22                                                       These cells expressed high levels of Foxp3, GITR, and CTLA4 molecul
23              Freshly isolated naive peripheral blood CD8+ T cells expressed high levels of GATA-3 and failed to down-regu
24                                              Most surviving cells expressed high levels of GLUT1, and 4% of these survivo
25  of poly(I:C), polyclonally activated naive IRF3(-/-) CD8 T cells expressed high levels of IL-17 and IL-23R in comparison
26                                 IL-12/15/18-preactivated NK cells expressed high levels of IL-2Ralpha (CD25), and their r
27               We report that resting and activated CD4(+) T cells expressed high levels of IL-7 receptor a chain but very
28                                        JAG2-depleted HCC827 cells expressed high levels of inflammation-related genes, in
29                                                 Growing GCT cells expressed high levels of inhibin betaA and nuclear SMAD
30   Among the cell lines, the cisplatin-resistant IGROV-1/Pt1 cells expressed high levels of integrin alphaVbeta3, making t
31                                                        NG2+ cells expressed high levels of laminin and fibronectin, which
32  we showed that acquired cisplatin-resistant ovarian cancer cells expressed high levels of MKP-1 and PARP-1 proteins, and
33 activated T cells expressed FOXP3, these induced FOXP3(+) T cells expressed high levels of multiple effector cytokines an
34                                         Haematopoietic stem cells expressed high levels of oestrogen receptor-alpha (ERal
35                                                       These cells expressed high levels of osteopontin (OPN), a cytokine
36 st to E6 + E7-immortalized cells, the Myc + E7-immortalized cells expressed high levels of p53 protein as well as two p53
37                   Furthermore, the tumor-infiltrating CD8 T cells expressed high levels of programmed death-1 and were pa
38    Furthermore, osteocytes in contact with multiple myeloma cells expressed high levels of Sost/sclerostin, leading to a
39 proteins; 2) normal human and mouse large airway epithelial cells expressed high levels of SPLUNC1; and 3) although Mp in
40                                                        Mast cells expressed high levels of ST2 and responded directly to
41                                     ARPE-19 and primary RPE cells expressed high levels of the antiapoptotic proteins Bcl
42                        FcepsilonRII-bearing monocytes and B cells expressed high levels of the FcepsilonRII sheddase a di
43                                                CD56(bright) cells expressed high levels of the glucose uptake receptor, G
44                                  These IL-10 reporter(+) NK cells expressed high levels of the IL-12 target genes T-bet,
45      Mechanistic investigations showed that BDCA1(+)CD14(+) cells expressed high levels of the immune checkpoint molecule
46 e to enhance function in exhausted CD8 T cells, these CD4 T cells expressed high levels of the programmed cell death (PD)
47                                   We found that Twist1-null cells expressed high levels of the T cell chemoattractant CXC
48                                   ILC2s, in contrast to Th2 cells, expressed high levels of the IL-9R, and IL-9 signaling
49 7(KIP1) were extremely low; conversely, Wnt10b-null mammary cells expressed high levels of this protein, suggesting Wnt-d
50  Early-forming MVs and GMPs in which the lining endothelial cells expressed high levels of VEGFR-2 were highly susceptibl

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