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1 ernalization was inhibited with drugs or mutations, or when cells expressed a chimeric receptor likely to have impaired d
3 issues, had a mature phenotype and, distinct from other NKT cells, expressed almost no ThPOK or Tbet.
5 ugh both PCR and immunostaining that mouse lymphatic muscle cells expressed Ca(v)3.1 and Ca(v)3.2 and produced functional
8 toxin administration to DEREG donor mice whose Foxp3+ Treg cells expressed diphtheria toxin receptor, restored rejection
10 In parallel, MCL cells as compared with normal B cells expressed elevated levels of WNT16, a NF-kappaB target
15 activation markers, although the majority of total CD8(+) T cells expressed granzymes and/or perforin.
21 In healthy individuals, CCR9+ memory T cells expressed higher levels of CCR6, a CNS-homing chemokine
23 ice were treated with rmTNFalpha protein, the Col6alpha2-KO cells expressed higher levels of TNFalpha mRNA compared with
25 ntly more nitrites and SEAP into perfusion effluent, and SC cells expressed increased GFP near collector channel ostia co
26 In addition, unresponsive MLL-rearranged leukemia cells expressed increased levels of MEIS1, an important leuke
28 Both secretory epithelial and stromal cells expressed Ki67 that was detectable at day 3 and largely
32 or influenza virus infections, all virus-specific CD8(+) T cells expressed low PRF levels without restimulation.
33 latory CD4(+) T-cell phenotypes, whereas conventional CD4 T cells expressed lower levels of costimulation molecules and o
36 of CL patients compared with healthy subjects, and that NK cells expressed more interferon-gamma, tumor necrosis factor
37 Consistent with this, resting CD5(hi) T cells expressed more of the NF-kappaB p65 protein than CD5(lo
41 effector memory activated phenotype, whereas EBV-infected B cells expressed plasma cell differentiation markers.
42 es and expanded upon antigen re-encounter, whereas memory B cells expressed receptors capable of neutralizing virus when
43 Compared with control CD4+ T cells, PGGT1B-deficient CD4+ T cells expressed significantly higher levels of integrin alpha
44 ive units expressed multiple opsins, while UV photoreceptor cells expressed single opsins; 2) most of the long-wavelength
45 In vitro primary bronchial epithelial cells expressed ST2 and IL-33 stimulation led to an increase
49 reg)-like transcriptome, although only 21% of all apoB(+) T cells expressed the T(reg) transcription factor FoxP3 (Forkhe