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2 Moreover, residual CD28-deficient TFR cells showed a diminished suppressive capacity as assessed by
3 metastases generated in mice by IL-1beta-expressing cancer cells showed a dramatic increase in S100A4, COX-2 and the alt
4 er-like, dysplastic growth pattern, whereas GALNT6 knockout cells showed a more normal differentiation pattern, reduced p
5 ne with this, a highly HBV permissive cell clone of HepAD38 cells showed a prominent association of core-microtubule and
6 differentiation of patient-derived induced pluripotent stem cells showed a reduced expansion of lymphohematopoietic proge
7 alysis of miR-183 family cluster overexpression in prostate cells showed an enrichment for cancer-related pathways includ
9 The constructs seeded with porcine AF cells showed approximately 11-, approximately 5.1-, and appro
10 CCL7-deficient bone marrow-derived mast cells showed decreased degranulation following IgE and Ag tre
11 ice did not have obvious abnormality; however, VHR-knockout cells showed decreased expression of integrins and FAK but st
12 In response to injury, AQP3-depleted colonic epithelial cells showed defective lamellipodia, focal adhesions, and rep
14 alanine) mutant in either HEK293 cells or colon cancer HT29 cells showed dramatically reduced NF-kappaB-activating abilit
15 XLKO heart and skeletal muscle, as well as XLKO Ocy454 cells, showed elevated SNX9 protein levels, and siRNA-mediate
16 normal counterpart, tumor-derived Lineage-EpCAM-CD73+CD90+ cells showed enhanced expression of the immunosuppressive lig
17 r, chromatin immunoprecipitation analysis in embryonic stem cells showed extensive binding of Smad2/3/4 to GC-rich cis-re
20 antify interassay variability for PD-L1 expression in tumor cells showed high concordance between antibodies for tumor ce
27 gly, NOD/SCID mice, which have a deficiency in T, B, and NK cells, showed minimal disease at cutaneous sites but develope
28 In contrast to control cells, the probands' cells showed mitochondrial swelling, which was exacerbated up
29 Compared with G0-expressing cells, however, G1-expressing cells showed more dramatic phenotypes, resembling those obser
30 Further analysis revealed that p205-knockdown cells showed reduced expression of apoptosis-associated speck
34 Live imaging of meiotic divisions in condensin-depleted cells showed repeated nuclear stretching and contraction as t
35 Notably, naive Foxp3(Cre)xT-bet(fl/fl) mice, lacking Treg1 cells, showed spontaneous skewing toward Th1 immunity.
36 Moreover, monitoring lineages of individual colicinogenic cells showed stochastic switching between expressers and non-
39 nally, in vitro IH exposures of murine macrophages and LLC1 cells showed that both cell types increased PGE2 release in r
40 nd pharmacological studies in human megakaryoblastic MEG-01 cells showed that DREAM is important for A23187-induced Ca(2+
42 g nanobody-labeled nuclear pore complexes in Xenopus laevis cells showed that MINFIELD-STED microscopy resolved details s
43 agged with EGFP (EB3-GFP) in primary external granule layer cells showed that netrin-1 differentially increased MT dynami
44 tudies that have been performed in primary human epithelial cells showed that PPARgamma agonists are able to restore barr
45 reover, forced expression and knockdown of SPZ1 in hepatoma cells showed that SPZ1 was able to regulate the cellular prol
46 Transcriptome comparisons of SOX9+ cells with GLT1+ cells showed that the two populations of cells exhibit largel
47 rthermore, a transgenic mouse line specifically labeling SL cells showed that they send profuse axonal projections to olf
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