ライフサイエンスコーパス: cells were treated with

1 Rat lung epithelial cells were treated with 52 stimuli, and gene expression and p

2 When AtT-20 cells were treated with a cell-permeant derivative of the Kal

3 Human NP cells were treated with a range of pH, from that of a non-deg

4 mediated migration was also observed when human endothelial cells were treated with a small molecule inhibitor of PTP4A3.

5 in ChlR1-depleted cells, but not in FANCJ(-/-) cells, when cells were treated with a triplex stabilizing compound benzoq

6 of BST2 was substantially inhibited by MLN4924 only when T cells were treated with alpha interferon (IFN-alpha) to induc

7 KSHV-infected-cell growth, primary effusion lymphoma (PEL) cells were treated with an iASPP inhibitor in the presence or

8 xplore the effects of EMD and TGF-beta1 on CTGF expression, cells were treated with and without TGF-beta inhibitor, and C

9 fect of the drug on MF stem cells (MF-SCs), splenic CD34(+) cells were treated with AZD1480 and transplanted into immunod

10 miR-34a levels also increased when cells were treated with c-SRC inhibitors, suggesting a negati

11 When LS174T cells were treated with C12-HSL and TQ416 simultaneously, TQ4

12 When POTS cells were treated with choline, transporter was up-regulated

13 dditive or synergistic promoter activation when transfected cells were treated with combined stimuli.

14 DNA damage was also observed when cells were treated with commercially available Ginkgo biloba

15 To study the role of CTGF in PDL development, cells were treated with CTGF inhibitor.

16 In further experiments, liver cells were treated with curcumin after exposure to acrylamide

17 When these cells were treated with DNA methyltransferase inhibitor, TLR2

18 For AST, test cells were treated with drugs before ferricyanide addition; c

19 Mice infected with 10(6) PbN-parasitized red blood cells were treated with either ET-1 or saline from 2 to 8 day

20 lation and inducibility of major human P450 enzymes, HepaRG cells were treated with either the WNT/beta-catenin signaling

21 treatment, with two distinct vesicle fractions present when cells were treated with epibromohydrin (EBH), a model epoxide

22 A549 cells were treated with fullerene (C60), long or short multi-

23 yonic stem cell-derived cardiomyocytes (hESC-CMs) and HepG2 cells were treated with glucose, and expression levels of NPs

24 Rat aortae and cultured rat aortic endothelial cells were treated with Hcy, BT extract, and theaflavin-3,3'-

25 Primary rat mesangial cells were treated with high glucose (30 mm) or mannitol as o

26 Porcine aortic endothelial cells were treated with HMGB1, human (h)TNFalpha or lipopolys

27 BEAS2B (bronchial epithelial) cells were treated with IL-4 followed by ADMA and investigate

28 resting cells myoferlin was bound to EHD2 protein and when cells were treated with IL-6, myoferlin dissociated from EHD2

29 MDA-MB-468 breast and HCT8 colorectal cancer cells were treated with inhibitors including LY294002, MK2206

30 When these cells were treated with inhibitors of both FLT3 and MEK in co

31 more necrotic/apoptotic cells were found when these cancer cells were treated with IR-780 iodide with US irradiation.

32 However, when the infected TN cells were treated with latency-reversing agents, including a

33 Human hepatoma cells were treated with Lp(a) purified from human plasma and

34 METHODS AND Human hepatoma cells were treated with Lp(a) purified from human plasma and

35 In vitro, human CCA cells were treated with MC supernatant fluids before evaluati

36 key markers of pancreatic CSC was observed when MIA PaCa-2 cells were treated with MDB5 compared to GDC-0449.

37 king the MOBKL1B-NS5A interaction could not replicate after cells were treated with MOBKL1B siRNA.

38 Human intestinal Caco-2 cells were treated with multiple concentrations of AS II to a

39 Human mast cells were treated with PAF, and the effect on DNA methylatio

40 Primary mouse hepatocytes (PMHs) and Huh7 cells were treated with palmitate or lysophosphatidylcholine

41 Human endothelial and smooth muscle cells were treated with pro-inflammatory cytokines with or wi

42 When cells were treated with SMase, the binding of ALO-D4 to cells

43 When cells were treated with suberoylanilide hydroxamic acid, whic

44 When the cells were treated with the combination C18 + C4 the mature C

45 This interaction was reduced when cells were treated with the ER stress inducer dithiothreitol.

46 and MHC-II significantly increased when CML stem/progenitor cells were treated with the JAK1/2 inhibitor ruxolitinib (RUX

47 Upregulation of TLR expression was not evident when cells were treated with the neurokinin 1 receptor antagonist

48 These cells were treated with trichostatin A or vorinostat (suberoy

49 Human retinal pigment epithelial cells were treated with various combinations of BEV and RES.

50 Studies in which cells were treated with varying concentrations of each isomer