ライフサイエンスコーパス: cellular hypoxia

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1 e as (19)F MRI contrast agents for detecting cellular hypoxia.

2 creased levels of oxygen under conditions of cellular hypoxia.

3 e response to proinflammatory stimuli during cellular hypoxia.

4 in response to combinations of IFN-gamma and cellular hypoxia.

5 ement, drove transcription, and responded to cellular hypoxia.

6 nt that there is a multitude of responses to cellular hypoxia.

7 ollowing trauma-hemorrhage is independent of cellular hypoxia and activation of HIF-1alpha, but it is

8 t a mechanistic link exists between vascular cellular hypoxia and mediators of inflammation associate

9 cs identical to endogenous HIF-1alpha during cellular hypoxia and reoxygenation.

10 unique pattern of development and decline of cellular hypoxia as wound cellularity and proliferation

11 3) are thought to act as proximal sensors of cellular hypoxia by virtue of their mechanism-based depe

12 mitation, alternative carbon metabolism, and cellular hypoxia, conditions that are thought to exist w

13 Fluorescent probes for the direct imaging of cellular hypoxia could be useful tools that complement r

14 the relative contributions of blood flow and cellular hypoxia (dysoxia) to increases in tissue and ve

15 reproducible model for the study of cerebral cellular hypoxia in healthy individuals.

16 complete Hif1alpha null, results in elevated cellular hypoxia in mouse embryos.

17 Cellular hypoxia induced a time-dependent increase in nu

18 Cellular hypoxia is also found in these conditions.

19 Cellular hypoxia is the common final pathway of brain in

20 Cellular hypoxia may be a useful indication of tissue di

21 e tissues such as skeletal muscle, resultant cellular hypoxia necessitates acclimatization to optimiz

22 ulation of macrophage phagocytosis following cellular hypoxia or trauma-hemorrhage.

23 Cellular hypoxia response is regulated at the level of h

24 eudohypoxic drive-the aberrant activation of cellular hypoxia response pathways despite normal oxygen

25 tion of neural tube formation, regulation of cellular hypoxia response, but also Hepatitis C virus re

26 a and p53 proteins is not coupled during the cellular hypoxia response.

27 Prolonged cellular hypoxia results in energy failure and ultimatel

28 been identified as a factor that influences cellular hypoxia sensing, putatively via an action on th

29 oxamine (DFx) and cobalt chloride, mimics of cellular hypoxia, similarly stimulated VEGF mRNA express

30 le to peripheral tissue hypoperfusion and/or cellular hypoxia, simultaneous measurements of tissue pe

31 tumors and is considered to be a marker for cellular hypoxia that it is not produced in most normal

32 a message is maintained during conditions of cellular hypoxia through inhibition of IRP-1-dependent r

33 We set out to determine whether cellular hypoxia, via mitochondrial reactive oxygen spec

34 blot analysis and pimonidazole staining for cellular hypoxia, we demonstrate that hypoxia stabilizes

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