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1 opment of the epidermis, hair follicles, and cellular immune system.
2 ein-Barr virus, is strongly dependent on the cellular immune system.
3 tinction between "self" and "nonself" by the cellular immune system.
4 egulates the development and activity of the cellular immune system.
5 of ligands by T cell receptors (TCRs) of the cellular immune system.
6 ized by numerous pathological changes of the cellular immune system.
7 es and other constituents of the humoral and cellular immune systems.
8 he outcome following interaction between the cellular immune system and cancer-derived exosomes is no
9 elopment of the repertoire of T cells in the cellular immune system and during activation of cytotoxi
11 -2 complexes have substantial effects on the cellular immune system, and this approach is being explo
13 ith laboratory mice, finding that wild mouse cellular immune systems are, comparatively, in a highly
14 petent and polyploid cells by the innate and cellular immune systems did not correlate with the prefe
15 anglia and reactivates upon weakening of the cellular immune system due to various conditions, erupti
16 cluding specific pathways of the humoral and cellular immune systems, have been further described.
17 We propose that PATCs form the basis of a cellular immune system, identifying certain endogenous g
19 hought to involve multiple components of the cellular immune system, including subsets of gammadelta
21 in the understanding of how the humoral and cellular immune system of humans recognizes and destroys
23 o reveal conserved WNV epitopes to an intact cellular immune system that subsequently recognizes infe
24 in vivo presentation of tumor antigen to the cellular immune system with consequent induction of anti
25 response is thought to be activation of the cellular immune system, with T cells, dendritic cells an
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