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1 ell death or proliferation, depending on the cellular microenvironment.
2 and/or phosphorylation, in an ever-changing cellular microenvironment.
3 these loop structures are influenced by the cellular microenvironment.
4 sponds to signals from growth factors in the cellular microenvironment.
5 ssociated with and depend on cues from their cellular microenvironment.
6 simplicity, and minimal interference of the cellular microenvironment.
7 e likely reflect a complex regulation by the cellular microenvironment.
8 ct of complex interactions determined by the cellular microenvironment.
9 t techniques are often incompatible with the cellular microenvironment.
10 nd scaffolds, which collectively make up the cellular microenvironment.
11 etical mechanotransduction events within the cellular microenvironment.
12 exert a coordinated response to their unique cellular microenvironment.
13 in normal cells with possible effects on the cellular microenvironment.
14 teracting with the printed components of the cellular microenvironment.
15 nding to chromatin even in a proinflammatory cellular microenvironment.
16 ange and by other regulatory proteins in the cellular microenvironment.
17 regulatory factors that could be provided by cellular microenvironment.
18 both the transformed cell and the supporting cellular microenvironment.
19 gulator of receptor-mediated activity in the cellular microenvironment.
20 ion but neglected in the absence of specific cellular microenvironments.
21 gies intracellular pathogens employ to alter cellular microenvironments.
22 used as in vitro culture models to mimic 3D cellular microenvironments.
23 ize) into hydrogel templates to construct 3D cellular microenvironments.
24 chemotherapy on cell-intrinsic processes and cellular microenvironments.
25 sidation occurs sequentially and in distinct cellular microenvironments.
26 of native tissues and to construct in vitro cellular microenvironments.
28 ol organ morphogenesis by defining the local cellular microenvironment, a finding that has relevance
29 escribe a technology, referred to as arrayed cellular microenvironments (ACMEs), that allows for a hi
30 d neoplasia includes an increasingly complex cellular microenvironment and dynamic interactions betwe
31 rication tools that serve to both define the cellular microenvironment and enable parallelization of
32 w cells to sense their immediate surrounding cellular microenvironment and make appropriate behaviour
33 uid exchange allowing ease of changes in the cellular microenvironment and precise cell handling.
37 TRAILshort is shed in microvesicles into the cellular microenvironment and therefore confers TRAIL re
38 which regulate gene expression depending on cellular microenvironment and thus modify the physiology
39 signaling molecule capable of modulating the cellular microenvironment and thus, it may be important
40 close proximity can be used to regulate the cellular microenvironment and, as such, are promising ap
41 oteins, thus altering the composition of the cellular "microenvironment" and thereby modulating devel
42 tivate the mTOR pathway, irrespective of the cellular microenvironment, and which play a requisite ro
44 results demonstrate that alterations in the cellular microenvironment are central to the induction o
45 , we characterized the genomic, protein, and cellular microenvironment associated with BCC in compari
46 final destinations, they encounter different cellular microenvironments, but functional responses of
47 ucleus during wound repair, and altering the cellular microenvironment by inducing hypoxia increases
52 ppressive molecules such as adenosine in the cellular microenvironment can reduce macrophage inflamma
55 easing evidence suggests that changes in the cellular microenvironment contribute to tumorigenesis, b
56 question that remains unanswered is how the cellular microenvironment contributes to angiogenesis.
59 eer molecular assemblies spatiotemporally in cellular microenvironment for inhibiting cancer cell gro
60 by which this retrovirus can create an ideal cellular microenvironment for its propagation and mainte
61 erplasia, increase the permissiveness of the cellular microenvironment for neoplastic expression of a
62 oped to engineer the physical aspects of the cellular microenvironment for stem cells, and these tool
63 of substantial amounts of NO and modify the cellular microenvironment (formation of the superoxide r
64 e bisphenol A and oxidizing agent (KBrO3) on cellular microenvironment, gene expression, and chromati
65 al connections between the components of the cellular microenvironment (growth factors, hormones, and
66 this regard since they provide a specialized cellular microenvironment guiding proper morphology and
67 how material and chemical alterations in the cellular microenvironment have wide ranging effects on r
69 timuli and demonstrate the importance of the cellular microenvironment in determining phenotypic beha
75 microscale technologies for controlling the cellular microenvironment in vitro and for performing hi
78 cocultures in regulating various aspects of cellular microenvironment is discussed, as well as the a
81 njury results from severe disruptions in the cellular microenvironment leading to massive loss of neu
84 ons between individual virus strains and the cellular microenvironment of the individual ATM(-) line
85 reciprocal dialogue between cells and their cellular microenvironments often governs the maintenance
87 ied forces and the biophysical nature of the cellular microenvironment play a central role in determi
89 recipients strongly suggests that the local cellular microenvironment plays a prominent role in regu
90 alteration in tumor phenotype indicates that cellular microenvironment plays an important role in def
94 ture of complex, experimentally inaccessible cellular microenvironments, such as the human intestine.
95 r initiation and development by altering the cellular microenvironment that facilitates tumor formati
96 f the immunological synapse is to generate a cellular microenvironment that favors the interactions o
97 in vitro to bind multiple components of the cellular microenvironment that function during wound hea
98 hemical mechanisms and parameters within the cellular microenvironment that initiate the onset of met
99 se of BPA was associated with changes in the cellular microenvironment that may promote survival.
100 dherin adhesion is thus a key element of the cellular microenvironment that provides both mechanical
101 iewed as an experimental manipulation of the cellular microenvironment that purportedly contains the
103 Little is known about the architecture of cellular microenvironments that support stem and precurs
104 In combination with strategies to engineer cellular microenvironments, this type of modeling approa
105 end beyond the synapse but can be limited to cellular microenvironments through uptake by target cell
106 ing blocks provides refined control over the cellular microenvironment, thus enabling functional tiss
107 e increasingly been employed to modulate the cellular microenvironment to promote tissue regeneration
108 ls coordinate spindle orientation with their cellular microenvironment to regulate and direct cell fa
109 ication tools allow precise control over the cellular microenvironment towards stabilizing liver func
110 These events, presumably unsupported by the cellular microenvironment, were followed by gonocyte apo
111 engineering enables precise control over the cellular microenvironment, whereas microfluidics provide
112 technique that is useful for controlling the cellular microenvironment with subcellular resolution.
113 r microarrays, which should better mimic the cellular microenvironment, with sensitive immunofluoresc
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