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1 ntracellular domain of CD44 to the posterior cellular protrusion.
2 nside, and budded from the tip of these thin cellular protrusions.
3 budding sites at the tip or side of the long cellular protrusions.
4 ollicular epithelium and (2) the dynamics of cellular protrusions.
5 mbrane tension in the control of actin-based cellular protrusions.
6 cal adhesions, with concomitant increases in cellular protrusions.
7 auses membrane ruffling and the formation of cellular protrusions.
8 ctural rigidity of most cells or support new cellular protrusions.
9 s well as filopodia, lamellipodia, and other cellular protrusions.
10 gions of dynamic actin remodeling, including cellular protrusions and cell-cell and cell-matrix junct
13 in the cytoskeleton, specifically diminished cellular protrusions and expression of the BLBC-associat
14 al Hh receptor Patched is localized in these cellular protrusions and Hh reception takes place in mem
15 gy characterized by the formation of dynamic cellular protrusions and the assembly of discrete aggreg
16 bundles believed to mediate the formation of cellular protrusions and to provide mechanical support t
17 na/VASP proteins to promote the formation of cellular protrusions and to stimulate invasive migration
18 ion correlated with the dynamic formation of cellular protrusions and was dependent upon cell-to-cell
19 e reduction, a rounded cell shape, decreased cellular protrusions, and a higher nuclear/cytoplasmic r
20 imulated N-cadherin reorganization into thin cellular protrusions, and decreased N-cadherin adhesion.
21 wnian kinetics on flattened membranes and on cellular protrusions, and does not transfer between cell
26 nsion in wild-type embryos reveals elaborate cellular protrusions at ND tips that are not detected in
27 regulates the localization of some mRNAs at cellular protrusions but the underlying mechanisms and f
29 e signaling pathways are thought to generate cellular protrusions by modulating the activity of downs
30 the G protein-coupled receptor at actin-rich cellular protrusions containing VASP, a filopodial marke
32 high levels in leading edge cells and their cellular protrusions during the morphogenetic process of
34 how Arg stimulates actin polymerization and cellular protrusion has not yet been fully elucidated.
36 sh PGC migration depends on the formation of cellular protrusions in form of blebs, a type of protrus
37 colocalizes with both SCRIB and VANGL1 along cellular protrusions in metastatic breast cancer cells,
38 taneously observe Cdc42 and Rac1 activity in cellular protrusions, indicating that Rac1 but not Cdc42
39 shown by loss of villi, tubule dilation, and cellular protrusions into the tubule lumen, was unambigu
40 been linked to the formation of proteolytic cellular protrusions known as invadopodia, undergoes an
41 n the correct formation of other actin-based cellular protrusions (microchaetae and macrochaetae).
43 ected with wild-type ARF6, and resembled the cellular protrusions observed in cells expressing the GT
46 ed for all orientations of stress fibers and cellular protrusions relative to the stretch direction,
47 and randomizes the orientation of polarized cellular protrusions, suggesting that integrin-fibronect
48 This co-movement likely depends on epidermal cellular protrusions that directly contact pObs only whe
50 ocytosis fuels generation of large, invasive cellular protrusions that expand tiny basement membrane
51 ia are highly specialized small antenna-like cellular protrusions that extend from the cell surface o
52 l is traversed by hundreds of extremely long cellular protrusions that maintain long-term contacts be
54 0-mum diameter) vesicles, derived from bulky cellular protrusions, that contain metalloproteinases, R
55 1 enhances the formation of various types of cellular protrusions through directly targeting and down
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