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1 promoter-luciferase constructs into OCCM-30 cementoblasts.
2 the periodontium, including osteoblasts and cementoblasts.
3 clastogenesis-associated molecules in murine cementoblasts.
4 e expression in a line of immortalized mouse cementoblasts.
5 ) carrier + follicle cells, and 3) carrier + cementoblasts.
6 ls, including odontoblasts, osteoblasts, and cementoblasts.
7 ter SV40 transgenic mice were used to obtain cementoblasts.
8 n mRNA was found after PDGF gene delivery to cementoblasts.
9 sgenic mice were used to obtain immortalized cementoblasts.
10 nesis using: 1) OCCM-30 (immortalized murine cementoblasts), 2) RAW 264.7 cells (murine myeloid cells
11 of platelet-derived growth factor stimulates cementoblast activity that is sustained above that of rh
12 n peptide appears to have a direct effect on cementoblast activity that may prove significant during
15 investigation was to evaluate the ability of cementoblasts and dental follicle cells to promote perio
16 ion in expression of BSP mRNA and protein in cementoblasts and surrounding osteoblasts in comparison
17 ve Hertwig's root sheath epithelia, putative cementoblasts, and a morphologically correct enamel orga
18 rs that can give rise to mature osteoblasts, cementoblasts, and fibroblasts within the periodontium.
19 s the balance of OPG and RANKL production by cementoblasts, and further indicate that this effect, in
21 t cell types in vivo and support the role of cementoblasts as a tool to better understand periodontal
22 the apically located cellular cementum, some cementoblasts become embedded in the cementoid matrix an
24 rganic phosphate (ePi) is a key regulator of cementoblast behavior, both in vivo and in vitro, and re
26 results revealed high level transduction of cementoblasts by gene transfer for 7 days as evidenced b
29 PR-deficient progenitors exhibit accelerated cementoblast differentiation with upregulation of nuclea
32 of prior in situ studies, OC is expressed by cementoblasts during root development, but not by cells
34 These findings further our understanding of cementoblast function and suggest that differentially in
36 showed that the mineralized tissue formed by cementoblasts gave strong signals for both BSP and OCN g
43 lture conditions, PDLSCs differentiated into cementoblast-like cells, adipocytes, and collagen-formin
46 contrast, in defects treated with carrier + cementoblasts, mineralized tissues were noted at the hea
47 rs using collagenase/trypsin digestion, only cementoblasts, not PDL cells, are immortalized and thus,
48 on of PDL stem cells committed to osteoblast/cementoblast (O/C) differentiation remains to be elucida
49 ition to expression of genes associated with cementoblasts, OC/CM cells promoted mineral nodule forma
52 , a clonal population of immortalized murine cementoblasts (OCCM-30) was exposed to full-length murin
59 in [OCN], and osteopontin [OPN], markers for cementoblast/osteoblast maturation/mineralization), von
61 SMA9 cells expanded, and differentiated into cementoblasts, osteoblasts, and periodontal ligament fib
64 LRAP, and the proliferation and migration of cementoblast/periodontal ligament cells by LRAP and P172
65 wild-type (WT) osteoclast progenitor and KO cementoblast/periodontal ligament cells displayed more t
66 Proliferation and migration rates of the KO cementoblast/periodontal ligament cells were lower than
69 profile, and biomineralization potential of cementoblasts suggesting that such factors alone or in c
70 hosphate regulates OPN gene transcription in cementoblasts through a pathway that requires a function
74 ortions of the implants in two of the 6-week cementoblast-treated specimens, possibly due in part to
75 ation of SMA9-labeled cells into osteoblasts/cementoblasts, we utilized a Col2.3GFP transgene, while
76 rine primary follicle cells and immortalized cementoblasts were delivered to the defects via biodegra
78 DGF-A may delay mineral formation induced by cementoblasts, while PDGF is clearly required for minera
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