ライフサイエンスコーパス: central action

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1 -induced insulin resistance is driven by its central actions.

2 derstanding the mechanism of its pleiotropic central actions.

3 However, extensive efforts to evaluate the central actions and therapeutic efficacy of IN-OT may be

4 However, in an in vivo test of central action (i.e., stimulus generalization with rats

5 s also a brain neuropeptide that has diverse central actions, including inhibition of food and water

6 In addition to its central actions, leptin exerts biological effects by act

7 nnectivity and its establishment through the central action of Lim-HD governed programs.

8 effect may, in fact, be mediated by a direct central action of nAChR antagonists.

9 Inhibition of cyclooxygenases, the central action of nonsteroidal antiinflammatory drugs (b

10 reinstate cocaine-seeking behavior through a central action of the stress-associated neurohormone cor

11 Although a central action of these agents involving direct perturba

12 the primary mediator of clinically important central actions of alpha2AR agonists and suggest that th

13 n of AT(1) antagonists can effectively block central actions of Ang II, regulating blood pressure and

14 Recent work suggests that the central actions of angiotensin II via the circumventricu

15 To prevent the central actions of angiotensin II, we created anterovent

16 brain c-fos expression may be related to the central actions of angiotensin II.

17 utyric acid (GABA) is a major target for the central actions of cocaine and lidocaine, which can resu

18 Central actions of estrogen (E2) include, among others,

19 t implications with respect to the potential central actions of FGF21.

20 It is suggested that determination of the central actions of glucocorticoids in mediating fast-fee

21 The central actions of insulin, on galanin (GAL) and neurope

22 a provide a foundation for understanding the central actions of kinins and their putative role in med

23 The central actions of leptin are essential for homeostatic

24 us (DMN) may serve as a relay center for the central actions of leptin on thyrotropin-releasing hormo

25 jor contributor, at least in part due to the central actions of leptin.

26 The central actions of NPY and CRF have opposing functions i

27 y used to distinguish between peripheral and central actions of opiates.

28 In vivo, central actions of polyphosphate at the level of the bra

29 Central actions of proinflammatory cytokines, in particu

30 CRH might act as one of the mediators of the central actions of the melanocortin system.

31 e in any species that GAL can antagonize the central actions of VP.

32 s have suggested that glucocorticoids have a central action on cerebral metabolism.

33 levels of plasma fatty acids, negating their central action on hepatic glucose fluxes through (i) inh

34 ctions of nicotine, which precede its direct central actions, serve as a conditioned interoceptive cu

35 Nonetheless, these central actions should be taken into account alongside p

36 vated by leptin and mediate part of leptin's central actions to influence energy balance.

37 nylethylamine itself, is without significant central action when administered at moderate doses, but

38 CoA generation and attempts to integrate its central actions with its peripheral antilipotoxic action

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