ライフサイエンスコーパス: central amygdala

1 ons in the basal ganglia and basolateral and central amygdala.

2 of data paralleled by cFos expression in the central amygdala.

3 bed nucleus of the stria terminalis and the central amygdala.

4 basal amygdala and the medial portion of the central amygdala.

5 ethanol-stimulated GABA release in the mouse central amygdala.

6 eive disproportionally higher input from the central amygdala.

7 modulated BOLD responding to cat fur in the central amygdala.

8 the bed nucleus of the stria terminalis and central amygdala.

9 d increased oxytocin receptor binding in the central amygdala.

10 cohol-induced ERK-phosphorylation in the rat central amygdala, a region involved in regulation of alc

11 wed an increase in Hcrtr2 mRNA levels in the central amygdala, a stress-related brain region, of LgA

12 aventricular nucleus of the hypothalamus and central amygdala, although no effect of the drug on 2-DG

13 cortex (Fctx), dorsal hippocampus (DHPC) and central amygdala (Amyg).

14 stic effects on the expression of CRH in the central amygdala and antagonizes GR-mediated reduction i

15 GAD67 mRNA was increased in the hippocampus, central amygdala and dorsomedial hypothalamus in pups te

16 on of the VTA provoked afterdischarge in the central amygdala and enhanced kindling rate.

17 not only failed to elevate FLI expression in central amygdala and insular cortex, but also failed to

18 This pattern was seen in central amygdala and insular cortex, but not in basolate

19 nce c-Fos-like immunoreactivity (FLI) in the central amygdala and insular cortex.

20 not induce Fos or iNOS in stress-responsive central amygdala and paraventricular hypothalamic neuron

21 in several other limbic sites, including the central amygdala and paraventricular nuclei of the hypot

22 its direct output to the medial part of the central amygdala and the hypothalamic 'aggression area',

23 teral and ventromedial hypothalamus), in the central amygdala and various mid- and hindbrain structur

24 ers of the limbic system (e.g., hippocampus, central amygdala, and both medial and lateral habenula),

25 the well-described action of oxytocin in the central amygdala, and demonstrates that self-defense sup

26 ssion in bed nucleus of stria terminalis and central amygdala, and dopamine, CRF, kappa/dynorphin, an

27 al pons, raphe nuclei, lateral hypothalamus, central amygdala, and insular cortex.

28 lated gamma-aminobutyric acid release in the central amygdala, and reduced ethanol consumption when a

29 ventricular nucleus of the hypothalamus, the central amygdala, and the bed nucleus of the stria termi

30 cingulate cortex, the ventral striatum, the central amygdala, and the bed nucleus of the stria termi

31 nd ERC; moderate strength connections in the central amygdala; and weak connections in the cingulate

32 ections from the parabrachial nucleus to the central amygdala are implicated in pain transmission.

33 Central amygdala (but not prefrontal cortex) infusion of

34 ouble dissociation, AM251 infusions into the central amygdala, but MJN110 infusions into the basolate

35 ion of nicotine-cue-activated Fos neurons in central amygdala, but not orbitofrontal cortex, decrease

36 c-fos expression was also induced in the central amygdala by i.c.v. CRF, unlike the vehicle-injec

37 significantly greater in the BNST, PAG, and central amygdala (Ce) following the microinjection of AV

38 ing Fos immunocytochemistry suggest that the central amygdala (Ce) might be a component of this neura

39 The central amygdala (Ce), particularly its medial sector (C

40 rk stressed the differing involvement of the central amygdala (CeA) and bed nucleus of the stria term

41 In contrast, the central amygdala (CeA) and its rostral extension receive

42 one, alone, increased FLI in VTA, NAC shell, central amygdala (ceA) and laterodorsal bed nucleus of t

43 ed dendritic spine density (DSD) in both the central amygdala (CeA) and medial amygdala (MeA) but not

44 ropeptide Y (NPY) were innately lower in the central amygdala (CeA) and medial amygdala (MeA), but no

45 xy-5-methyl-4-isooxazole receptor (AMPAR) in central amygdala (CeA) and phosphorylation of AMPAR GluA

46 The pathway between central amygdala (CeA) and ventrolateral periaqueductal

47 cent dextran amines identifies the X. laevis central amygdala (CeA) as a target for ascending auditor

48 The central amygdala (CeA) has a key role in learning and ex

49 otropin releasing factor (CRF) system in the central amygdala (CeA) has been implicated in the effect

50 otropin releasing factor (CRF) system in the central amygdala (CeA) has been implicated in the effect

51 The central amygdala (CeA) is a key structure at the limbic-

52 on of the tachykinin 2 (Tac2) pathway in the central amygdala (CeA) is necessary and sufficient for t

53 We hypothesized that CRF-CRFR1 signaling in central amygdala (CeA) mediates stress-induced hyperalge

54 tudies show that the GABAergic system in the central amygdala (CeA) nucleus has a complex role in the

55 The central amygdala (CeA) nucleus, a subcortical structure

56 or conditionally overexpress (OE) CRF in the central amygdala (CeA) of adult mice.

57 cts of local mifepristone infusions into the central amygdala (CeA) on yohimbine-induced reinstatemen

58 The central amygdala (CeA) plays a central role in physiolog

59 xcessive drinking.SIGNIFICANCE STATEMENT The central amygdala (CeA) plays a critical role in the deve

60 The central amygdala (CeA) plays a role in the relationship

61 received ibotenic acid lesions in the MeA or central amygdala (CeA) prior to cat-odor exposure.

62 Pharmacological inactivation of the central amygdala (CeA) severely impaired acquisition and

63 tion potential (AP) firing in neurons of the central amygdala (CeA) that project to the dlBnST, incre

64 Prolonged exposure of the central amygdala (CeA) to elevated corticosteroids (CORT

65 (NAc) shell and core, basolateral (BLA) and central amygdala (CeA), and medial prefrontal cortex (mP

66 A dose of 5 mg DF/kg activated the central amygdala (CeA), bed nucleus of the stria termina

67 In the central amygdala (CeA), ethanol acts via corticotrophin-

68 omedial amygdala (CeM), a subdivision of the central amygdala (CeA), is believed to be the main outpu

69 prague Dawley rats were cannulated targeting central amygdala (CeA), medial amygdala (MeA), or basola

70 d nucleus of the stria terminalis (BNST) and central amygdala (CEA), naltrexone increased FLI in ad l

71 The central amygdala (CeA), once viewed as a passive relay b

72 y in the basolateral amygdala (BLA), but not central amygdala (CeA), reversed deactivation of mPFC py

73 Here we studied the role of central amygdala (CeA), ventral medial prefrontal cortex

74 BLA neurons project to the central amygdala (CeA), which also participates in negat

75 alimbic prefrontal cortex (ilPFC) to inhibit central amygdala (CeA)-mediated Pavlovian reactions.

76 expressed significantly more c-Fos IR in the central amygdala (CeA).

77 n was decreased in the nucleus accumbens and central amygdala (CeA).

78 s in altering excitatory transmission in the central amygdala (CeA).

79 lateral parabrachial nucleus (LPB), and the central amygdala (CeA).

80 n areas that regulate anxiety, including the central amygdala (CEA).

81 in stress system dysfunction, including the central amygdala (CeA).

82 have focused on neuroadaptations within the central amygdala (CeA).

83 taste processing, gustatory cortex (GC) and central amygdala (CeA).

84 tic inhibition in fear output neurons of the central amygdala (CEA).

85 nal groups of rats were given lesions in the central amygdala (CeA).

86 indicate that the lateral subdivision of the central amygdala (CeL) is essential for fear learning.

87 ar processing in the lateral division of the central amygdala (CeL), a structure that orchestrates fe

88 s, located in the lateral subdivision of the central amygdala (CEl), which express protein kinase C-d

89 astrocytes in the medial subdivision of the central amygdala (CeM) determine the synaptic and behavi

90 neurons inhibit output neurons in the medial central amygdala (CEm), and also make reciprocal inhibit

91 he activity of the medial subdivision of the central amygdala (CeM), the canonical amygdala output to

92 septum (LS), lateral amygdala (LatAmyg), and central amygdala (CenAmyg) than less social, naive LD fe

93 ikely form differential connections with the central amygdala (controlling freezing), this process wo

94 In the central amygdala, CRF mRNA levels were increased in both

95 In slices of the central amygdala, DCUK-OEt acted primarily on extrasynap

96 contrast, no sex difference was seen in the central amygdala-dependent acquisition or expression of

97 However, inactivation of the central amygdala disrupted only the expression, but not

98 efflux in the basolateral amygdala, whereas central amygdala efflux remained unchanged.

99 In central amygdala, elevated Tacr1 expression was accompan

100 ndent increases in the responsiveness of the central amygdala ERK pathway to cocaine cues.

101 After 1 d of withdrawal, stimulation of central amygdala ERK phosphorylation increased cocaine s

102 The pain-recipient neurons in the central amygdala expressing CGRP receptors are also crit

103 phenotype and combined MR up-regulation and central amygdala GR deficiency.

104 cortex, bed nucleus of the stria terminalis, central amygdala, hypothalamic paraventricular nucleus (

105 e found an up-regulation of CART mRNA in the central amygdala induced by acute but not chronic stress

106 ay be related to the increase in the NTS and central amygdala inputs leading to inhibition of DMN neu

107 projections from anterior insular cortex to central amygdala is critical to relapse after the cessat

108 rons onto midbrain-projecting neurons in the central amygdala is necessary for context-dependent retr

109 ia nigra pars reticulata, entorhinal cortex, central amygdala, lateral amygdala, arcuate nucleus, and

110 the medial preoptic area, nucleus accumbens, central amygdala, lateral septum, and cortex.

111 The presence of the hormone oxytocin in the central amygdala makes a mother rat willing to put her l

112 Minor projections also go to central amygdala, mediodorsal thalamus, dorsal raphe, an

113 itional effects to decrease accumbens GR and central amygdala MR expression.

114 ap at the level of defined subpopulations of central amygdala neurons and demonstrate that persistent

115 neurons inhibited a subpopulation of medial central amygdala neurons and shunted excitation from the

116 Here we show that PKC-delta-expressing central amygdala neurons are essential for the synaptic

117 use, we identify a specific subpopulation of central amygdala neurons expressing protein kinase C del

118 distinct behavioral modules and suggest that central amygdala neurons instruct predatory hunting acro

119 In rat central amygdala neurons, deletion of Tlr4 altered GABAA

120 n failed to change in CA1 pyramidal neurons, central amygdala neurons, pyramidal neurons of layer II/

121 ergic system in neurons of slices of the rat central amygdala nucleus (CeA), a brain region thought t

122 eus of the hypothalamus, supraoptic nucleus, central amygdala, nucleus tractus solitarius and area po

123 eased neuropeptide Y (NPY) expression in the central amygdala of alcohol-preferring rats, causing hig

124 optogenetic and chemogenetic stimulation of central amygdala of mice elicited predatory-like attacks

125 tor binding was significantly greater in the central amygdala of socially experienced hamsters than i

126 geminal, C1/A1 group, ventrolateral medulla, central amygdala, parabrachial nucleus, cuneate nucleus,

127 follicular compared with midcycle timing in central amygdala, paraventricular and ventromedial hypot

128 However, how the central amygdala participates in such a learning process

129 dministration and that neuronal ensembles in central amygdala play a critical role in this incubation

130 We identified the anterior insular cortex-to-central amygdala projection as a new addiction- and moti

131 ning lethal bites on prey, was mediated by a central amygdala projection to the reticular formation i

132 Given that neurons in the central amygdala responding to DCUK-OEt were recently id

133 FKBP51 in the basolateral amygdala (BLA) or central amygdala resulted in increased anxiety-related b

134 but not vasopressin, administration into the central amygdala reversed the social incompetence of the

135 ypothalamus into areas within the medial and central amygdala, terminating at the medial border of th

136 d in H/S pups, in which fewer neurons in the central amygdala, the bed nucleus of the stria terminali

137 leus of the stria terminalis, the medial and central amygdala, the periaqueductal gray, the dorsal ra

138 TA activation and neural excitability in the central amygdala, the present results are consistent wit

139 at the TRH analog, RX 77368, acts within the central amygdala to vagally stimulate gastric contractil

140 synapses from the lateral subdivision of the central amygdala via A2A receptor activation.

141 Y1 receptor ir was evident in the central amygdala, whereas both Y1- and Y5-immunoreactive

142 ocalized within the parabrachial nucleus and central amygdala, which constitute part of the 'emergenc