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1 have developed a model to disrupt GR in the central nucleus of the amygdala.
2 projects back to the anterior BLA and to the central nucleus of the amygdala.
3 ) electrophysiological responsiveness in the central nucleus of the amygdala.
4 s in several brain sites such as the PVH and central nucleus of the amygdala.
5 rentially activated by administration in the central nucleus of the amygdala.
6 ial border of the lateral subdivision of the central nucleus of the amygdala.
7 R) neurons in the lateral subdivision of the central nucleus of the amygdala.
8 al (CeL) or medial (CeM) subdivisions of the central nucleus of the amygdala.
9 icular nucleus but lower basal levels in the central nucleus of the amygdala.
10 both CRF and glucocorticoid receptors in the central nucleus of the amygdala.
11 leus of the stria terminalis (BNST), and the central nucleus of the amygdala.
12 eus, bed nucleus of the stria terminalis and central nucleus of the amygdala.
13 nistration, reduced CRF immunodensity in the central nucleus of the amygdala.
14 d GR mRNA in the NAc core, ventral BNST, and central nucleus of the amygdala.
15 val-related afferent input was absent in the central nucleus of the amygdala.
16 frontal cortex, basolateral amygdala, or the central nucleus of the amygdala.
17 shows particularly robust expression in the central nucleus of the amygdala.
18 , nucleus accumbens, olfactory tubercle, and central nucleus of the amygdala.
19 striatum, and reduced Dlx5 expression in the central nucleus of the amygdala.
20 icotropin-releasing factor (CRF) mRNA in the central nucleus of the amygdala, a critical node in the
21 delivery and direct administration into the central nucleus of the amygdala, a critical stress-relat
24 esigned to determine the extent to which the central nucleus of the amygdala (ACe) contributes to thi
26 og, 5 microg, and 10 microg) into either the central nucleus of the amygdala (ACe) or nucleus parafas
28 s [that received electrolytic lesions of the central nucleus of the amygdala (ACe)] and control anima
32 , a significant proportion of neurons in the central nucleus of the amygdala and in the hippocampal C
33 , and elevated c-Fos or Zif268 expression in central nucleus of the amygdala and medial paracapsular
34 e, MK-801 increased FLI in cingulate cortex, central nucleus of the amygdala and nucleus accumbens (N
35 , cortical pyramidal neurons, neurons of the central nucleus of the amygdala and parvocellular neuron
38 expression in the parabrachial nucleus, the central nucleus of the amygdala and the basolateral amyg
39 results implicate Barrington's nucleus, the central nucleus of the amygdala and the bed nucleus of t
40 mediated by different brain structures, the central nucleus of the amygdala and the bed nucleus of t
41 everal other regions surveyed, including the central nucleus of the amygdala and the dorsal striatum.
42 ical and functional relationship between the central nucleus of the amygdala and the dorsal vagal com
44 lary and pontine neurons that project to the central nucleus of the amygdala and to the lateral bed n
45 ebrain regions including olfactory tubercle, central nucleus of the amygdala, and bed nucleus of the
46 s of the stria terminalis, ventral pallidum, central nucleus of the amygdala, and cingulate cortex.
47 ary-adrenal axis, CRF mRNA expression in the central nucleus of the amygdala, and CRF(1) mRNA express
48 in the piriform cortex, supraoptic nucleus, central nucleus of the amygdala, and nucleus of the soli
49 ral and paraventricular hypothalamic nuclei, central nucleus of the amygdala, and periaqueductal gray
50 the basolateral nucleus of the amygdala, the central nucleus of the amygdala, and the bed nucleus of
51 tures, especially in the lateral part of the central nucleus of the amygdala, and the CA1 region of t
52 the bed nucleus of the stria terminalis, the central nucleus of the amygdala, and the parvocellular d
53 aventricular nucleus of the hypothalamus and central nucleus of the amygdala, and this was inhibited
54 medial prefrontal cortex, nucleus accumbens, central nucleus of the amygdala, and ventral tegmental a
55 roject to the laterocapsular division of the central nucleus of the amygdala as forming a functionall
56 of the hypothalamus and, importantly, in the central nucleus of the amygdala, as measured by solid ph
57 following KA and in the piriform cortex, and central nucleus of the amygdala at 120 min after KA.
58 hat lacked BDNF mRNA (e.g., medial habenula, central nucleus of the amygdala, bed nucleus of stria te
59 cumbens core, dorsomedial prefrontal cortex, central nucleus of the amygdala, bed nucleus of stria te
60 tems in the extended amygdala, including the central nucleus of the amygdala, bed nucleus of the stri
61 tion of c-fos expression in the hippocampus, central nucleus of the amygdala, bed nucleus of the stri
62 he paraventricular nucleus, arcuate nucleus, central nucleus of the amygdala, bed nucleus of the stri
63 tems in the extended amygdala, including the central nucleus of the amygdala, bed nucleus of the stri
66 the medial zona incerta (MZI) project to the central nucleus of the amygdala (cAMY), horizontal diago
67 locus coeruleus (LC) and an increase in the central nucleus of the amygdala (CAMY), supporting a ton
69 ed tonic oxytocin receptor expression in the central nucleus of the amygdala (cAmyg) through activati
70 optic area (MPO), lateral hypothalamus (LH), central nucleus of the amygdala (Ce) and bed nucleus of
71 reflect dissociable circuits centered on the central nucleus of the amygdala (Ce) and the bed nucleus
72 onents of the central extended amygdala, the central nucleus of the amygdala (Ce) and the lateral bed
74 ies of AT in young monkeys revealed that the central nucleus of the amygdala (Ce) is a key environmen
75 o imaging in young monkeys demonstrated that central nucleus of the amygdala (Ce) metabolism is relat
79 ntricular nucleus of the hypothalamus (PVN), central nucleus of the amygdala (Ce), periaqueductal gra
81 expression, the LA is thought to engage the central nucleus of the amygdala (CE), which serves as th
82 CRH mRNA levels were observed in the PVN and central nucleus of the amygdala (ceA) 4-6 hr after SEB a
83 ticotropin-releasing factor (CRF) within the central nucleus of the amygdala (CeA) and bed nucleus of
85 , and the preoptic area (POA) as well as the central nucleus of the amygdala (CeA) and nucleus of the
87 n-releasing hormone (CRH) are located in the central nucleus of the amygdala (CeA) and paraventricula
88 norepinephrine (NE) transmission within the central nucleus of the amygdala (CeA) and the bed nucleu
89 n (r/h)] and Urocortin I (Ucn I), within the central nucleus of the amygdala (CeA) and the lateral se
91 -aspartate (NMDA) receptor antagonism in the central nucleus of the amygdala (CeA) and the posterior
92 kappa-opioid receptor (KOR) signaling in the central nucleus of the amygdala (CeA) and these neuroada
93 (MCP-1) that colocalize in neurons from the central nucleus of the amygdala (CeA) and ventral tegmen
94 the solitary tract (NTS)/area postrema (AP), central nucleus of the amygdala (CeA) and, to a smaller
95 cognitive function and overactivation of the central nucleus of the amygdala (CeA) are key factors th
96 C), lateral parabrachial nucleus (lPBN), and central nucleus of the amygdala (CeA) as potential sites
97 ype II glucocorticoid receptors (GRs) in the central nucleus of the amygdala (CeA) by delivering lent
98 tral periventricular nucleus (AVPe), and the central nucleus of the amygdala (CeA) contained few LepR
99 no-5-phosphonovalerate (APV) into the BLA or central nucleus of the amygdala (CEA) during fear condit
106 antly increased EGR-1 mRNA expression in the central nucleus of the amygdala (CeA) in a dose-dependen
107 ristics of visceral afferent pathways to the central nucleus of the amygdala (CeA) in brainstem slice
110 amma-aminobutyric acid (GABA) release in the central nucleus of the amygdala (CeA) in Nf1 heterozygou
111 iated with the recruitment of neurons in the central nucleus of the amygdala (CeA) in nondependent ra
112 ctic response to insulin injections into the central nucleus of the amygdala (CeA) in these animals.
114 tion-induced bladder pain, we found that the central nucleus of the amygdala (CeA) is a critical site
116 -9, extracellularly operating enzyme) in the central nucleus of the amygdala (CeA) is crucial for app
117 ases in ethanol intake, CRF signaling in the central nucleus of the amygdala (CeA) is engaged during
122 a2 siRNA vector (pHSVsiLA2) infused into the central nucleus of the amygdala (CeA) of alcohol-preferr
124 C) receptors within the basolateral (BLA) or central nucleus of the amygdala (CeA) on acute fear-like
129 t a newly identified GABA circuit within the central nucleus of the amygdala (CeA) promotes cataplexy
131 ulation of locally projecting neurons in the central nucleus of the amygdala (CeA) that produce the n
132 high glucocorticoid levels may act upon the central nucleus of the amygdala (CeA) to alter normally
133 al recordings of GABAA-mediated IPSPs in the central nucleus of the amygdala (CeA) to explore functio
134 dy, we retrogradely transported dye from the central nucleus of the amygdala (CeA) to identify CeA-pr
136 ar nucleus of the hypothalamus (PVN) and the central nucleus of the amygdala (CeA) was observed durin
137 e CB(1) receptor mRNA and the protein in the central nucleus of the amygdala (CeA) were also investig
138 f [9-41]-alpha-helical CRH into the adjacent central nucleus of the amygdala (CEA) were ineffective,
139 e course of Fox expression in neurons in the central nucleus of the amygdala (CeA) were studied in en
141 iating the anorexia produced by PACAP in the central nucleus of the amygdala (CeA), a limbic structur
143 ABAergic synaptic activity in neurons of the central nucleus of the amygdala (CeA), an important brai
144 hanol drinking reduced NPY and Y1R IR in the central nucleus of the amygdala (CeA), and 24 h of ethan
145 bed nucleus of the stria terminalis (BNST), central nucleus of the amygdala (CeA), and lateral hypot
146 bed nucleus of the stria terminalis (BNST), central nucleus of the amygdala (CeA), and lateral hypot
147 hypothalamic paraventricular nucleus (PVN), central nucleus of the amygdala (CeA), and nucleus of th
148 s Fos-like immunoreactivity (FLI) within the central nucleus of the amygdala (CeA), bed nucleus of th
149 ns containing the lateral hypothalamus (LH), central nucleus of the amygdala (CeA), bed nucleus of th
150 been relatively unexplored compared with the central nucleus of the amygdala (CeA), despite the fact
151 he brain sites within the extended amygdala [central nucleus of the amygdala (CeA), lateral bed nucle
152 o the bed nucleus of stria terminalis (BST), central nucleus of the amygdala (CEA), paraventricular n
153 nspecific antagonists, administered into the central nucleus of the amygdala (CeA), selectively decre
154 gonist R121919 (0, 0.5, 1.5 mug/side) in the central nucleus of the amygdala (CeA), the basolateral n
155 ar nucleus of the hypothalamus (PVN) and the central nucleus of the amygdala (CEA), through complex m
156 n-releasing factor (CRF) is expressed in the central nucleus of the amygdala (CeA), where the CRF rec
158 neurones in the latero-capsular part of the central nucleus of the amygdala (CeA), which is now defi
159 IP3K-A expression is highly enriched in the central nucleus of the amygdala (CeA), which plays a piv
160 basolateral amygdala (BLA) terminals in the central nucleus of the amygdala (CeA)--achieved by viral
177 15%), lateral hypothalamic area (LHA, 25%), central nucleus of the amygdala (CeA, 77%), sublenticula
178 dorsal hippocampus (CA1 pyramidal layer) and central nucleus of the amygdala (CeA; Experiment 3).
179 erally augments nociceptive amygdala (in the central nucleus of the amygdala [CeA]) PACAP immunoreact
182 rons of the lateral/capsular division of the central nucleus of the amygdala (CEAl/c) and oval divisi
183 ticity in the laterocapsular division of the central nucleus of the amygdala (CeLC) generates emotion
184 ticity in the laterocapsular division of the central nucleus of the amygdala (CeLC) in brain slices f
186 C), the medial posteroventral portion of the central nucleus of the amygdala (CeMPV), and the Edinger
187 entral extended amygdala [CEA; including the central nucleus of the amygdala (CN), ventral bed nucleu
188 nd LY34 into the basal amygdala (BA) and the central nucleus of the amygdala (CNA) and recorded sleep
190 ar nucleus of the hypothalamus (PVN) and the central nucleus of the amygdala (CNA) are two forebrain
191 s neurons and tracts, microinjected into the central nucleus of the amygdala (CNA) during the light p
195 e implanted with bilateral cannulas into the central nucleus of the amygdala (CNA) or control area an
196 o in the parabrachial nucleus (PBN), and the central nucleus of the amygdala (CNA), regions thought t
199 ly labeled CRF-immunoreactive neurons in the central nucleus of the amygdala colocalized glucocortico
201 ing the bed nucleus of the stria terminalis, central nucleus of the amygdala, diagonal band of Broca,
202 f these beta-adrenergic antagonists into the central nucleus of the amygdala did not block the dexame
205 nd rats with BLA lesions, and imply that the central nucleus of the amygdala encodes CS-US associatio
206 n in the basolateral region of the amygdala, central nucleus of the amygdala, gustatory portion of th
207 cotropin-releasing factor infusions into the central nucleus of the amygdala had no effect when given
208 mice, we found that GABAergic neurons in the central nucleus of the amygdala heavily innervate neuron
209 in-releasing hormone (Crh) expression in the central nucleus of the amygdala in anhedonic rats, and t
211 The roles of the dorsal hippocampus and the central nucleus of the amygdala in the expression of con
212 labeling in the nucleus accumbens shore and central nucleus of the amygdala, independent of the adre
215 ncreased significantly Fos expression in the central nucleus of the amygdala (lateral part), parabrac
216 in a number of anxiety-related brain areas (central nucleus of the amygdala, locus ceruleus, ventrol
217 ly reported that electrolytic lesions of the central nucleus of the amygdala, made 6 or 30 days after
218 recent studies suggesting that PACAP in the central nucleus of the amygdala may impact the emotional
219 plasticity within other brain regions (i.e., central nucleus of the amygdala) may be crucial for fear
220 circuit from the parabrachial nucleus to the central nucleus of the amygdala mediates appetite suppre
221 noreactivity in the paraventricular nucleus, central nucleus of the amygdala, nucleus of the solitary
223 as corticotropin-releasing factor neurons in central nucleus of the amygdala or bed nucleus of the st
224 the hypothalamic paraventricular nucleus and central nucleus of the amygdala, OX, and another orexige
225 rtices, bed nucleus of the stria terminalis, central nucleus of the amygdala, periaqueductal gray, la
226 istration and that neuronal ensembles in the central nucleus of the amygdala play a critical role in
227 bed nucleus of the stria terminalis, and the central nucleus of the amygdala, prairie voles had highe
228 e tract-tracing techniques revealed that the central nucleus of the amygdala projects to the dorsal v
229 ot control short hairpin RNA, given into the central nucleus of the amygdala reversed CES-induced anh
230 eus, ventrolateral periaqueductal gray area, central nucleus of the amygdala, sublenticular extended
231 ctivation of these neurons projecting to the central nucleus of the amygdala suppresses appetite.
232 The LV system comprises the neurons in the central nucleus of the amygdala that excite DA cells.
233 t underlies early-life anxiety including the central nucleus of the amygdala, the anterior hippocampu
234 the bed nucleus of the stria terminalis, the central nucleus of the amygdala, the anteroventral, ante
235 dorsomedial nuclei of the hypothalamus, the central nucleus of the amygdala, the oval nucleus of the
236 r cell counts of CRH containing cells in the central nucleus of the amygdala to be lower in 24-month-
237 We identify an inhibitory pathway from the central nucleus of the amygdala to the ventrolateral per
238 bed nucleus of the stria terminalis, and the central nucleus of the amygdala, together referred to as
239 play activated Crh-expressing neurons in the central nucleus of the amygdala, typically involved in a
240 of acetylcholine (ACh) were measured in the central nucleus of the amygdala using microdialysis in 2
241 ing hormone (TRH) analog, RX 77368, into the central nucleus of the amygdala was examined in fasted,
242 ntration of [(3)H]DAMGO binding sites in the central nucleus of the amygdala was very low, the concen
243 omplex, retrogradely labelled neurons in the central nucleus of the amygdala were clustered in the ce
244 Significant changes in Fos expression in the central nucleus of the amygdala were not observed at any
246 ala, we combined retrograde tracing from the central nucleus of the amygdala with immunohistochemistr
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