ライフサイエンスコーパス: central obesity

1 , vitamin D and C-reactive protein, and less central obesity).

2 disease (CHD) such as insulin resistance and central obesity.

3 ictive power of overall obesity with that of central obesity.

4 style factors that favour the development of central obesity.

5 of blood pressure than did other measures of central obesity.

6 tion, and distribution, and in excess, cause central obesity.

7 been shown to affect nutrient metabolism and central obesity.

8 excess adiposity is particularly related to central obesity.

9 causes osteoporosis, insulin resistance and central obesity.

10 on of body mass index (BMI) with measures of central obesity.

11 adiposity that combined BMI with measures of central obesity.

12 3.7% were overweight or obese, and 71.4% had central obesity.

13 e tissues in IGT(+) directly associated with central obesity.

14 (4.5-5.0) for obesity, 29.4% (28.9-29.9) for central obesity, 30.5% (30.0-31.0) for prediabetes, 5.1%

15 Central obesity (47.2%) and overweight (38.8%) in women

16 Women with normal-weight central obesity also had a higher mortality risk than th

17 xamine the effects of NPY variant rs16147 on central obesity and abdominal fat distribution in respon

18 he rs16147 single-nucleotide polymorphism on central obesity and abdominal fat distribution were modi

19 However, the pathophysiological link between central obesity and adverse cardiovascular outcomes rema

20 We evaluated the association between central obesity and cardiac mechanics using multivariabl

21 phenotype similar to metabolic syndrome with central obesity and diabetes.

22 we examined the associations of general and central obesity and hypertension among Chinese children.

23 o severe pediatric AD may be associated with central obesity and increased systolic BP.

24 pean whites and are accounted for by greater central obesity and insulin resistance in Indian Asians.

25 Recent studies have suggested that central obesity and insulin resistance may be primary me

26 tors but was eliminated by an adjustment for central obesity and insulin resistance score in Asians.

27 with MS and its components independently of central obesity and insulin resistance.

28 We hypothesized that central obesity and larger WHR are independently associa

29 Insulin resistance, central obesity and lipid abnormalities such as high tri

30 ther ox-LDL mediates the association between central obesity and MS, and whether insulin resistance m

31 suggesting that HAART increases the risk of central obesity and osteoporosis.

32 obesity and WC) and African American women (central obesity and percentage trunk fat) but was invers

33 Our findings highlight the association of central obesity and related cardiometabolic phenotypes a

34 The relationship between central obesity and survival in community-dwelling adult

35 Central obesity and the accumulation of visceral fat are

36 entral adiposity among African American men (central obesity and WC) and African American women (cent

37 otion abnormalities, and ejection fraction), central obesity and WHR remained associated with worse g

38 it is unknown whether AD is associated with central obesity and/or high BP.

39 biological link between obesity (especially central obesity) and increased cancer risk.

40 t can be associated with excess weight gain, central obesity, and dyslipidemia.

41 a, low high-density lipoprotein cholesterol, central obesity, and elevated fasting glucose.

42 t disorders of lipid and glucose metabolism, central obesity, and high blood pressure, with an increa

43 The prevalence of general obesity, central obesity, and hypertension among the children was

44 The prevalence of general obesity, central obesity, and hypertension was high among Chinese

45 onally representative indicators of obesity, central obesity, and MetS among US adults were construct

46 ts and their related nutrients with obesity, central obesity, and MetS, and attempted to explain some

47 overactivity is also known to be present in central obesity, and recent findings demonstrate the con

48 ors include gastroesophageal reflux disease, central obesity, and smoking.

49 g's syndrome, results in insulin resistance, central obesity, and symptoms similar to the metabolic s

50 Weight gain and central obesity are associated with insulin resistance,

51 cording to the results of the current study, central obesity as determined by WC and citrus fruit int

52 Anthropometric measures of overall and central obesity as predictors of NIDDM risk have not bee

53 For women, central obesity, asthma, and arthritis increased the odd

54 02+/-17 cm, WHR was 0.91+/-0.08, and 80% had central obesity based on waist circumference and WHR cri

55 D, including those with normal and high BMI, central obesity but not BMI is directly associated with

56 ing to diabetes, hypertension, osteoporosis, central obesity, cardiovascular morbidity, and increased

57 y associated with PCOS only among women with central obesity (chi(2) = 35.0, p < 0.001) and not for t

58 f MetS occurred (77.3%), and the presence of central obesity conferred the highest risk of developing

59 Normal-weight central obesity defined by WHR is associated with higher

60 Central obesity, defined by increased waist circumferenc

61 le intake, low physical activity, obesity or central obesity, diabetes, hypertension, and dyslipidaem

62 ingly prevalent and strongly associated with central obesity, dyslipidemia, and insulin resistance.

63 The metabolic syndrome is characterized by central obesity, dyslipidemia, elevated blood pressure,

64 0001) and 13.9%, 18.3%, 22.1%, and 24.9% for central obesity (estimated increase 0.78% per year, 0.76

65 Hyperglycemia and central obesity experienced the highest increase.

66 ists of a myriad of abnormalities, including central obesity, glucose intolerance, dyslipidemia, and

67 z score >2, World Health Organization 2006), central obesity (> or = 90th percentile, third National

68 mple, a man with a normal BMI (22 kg/m2) and central obesity had greater total mortality risk than on

69 Persons with normal-weight central obesity had the worst long-term survival.

70 Individuals with normal weight central obesity had the worst long-term survival: a pers

71 in the SA pedigrees were older, had greater central obesity, had higher prevalence of the metabolic

72 tality risk than one with similar BMI but no central obesity (hazard ratio [HR], 1.87 [95% CI, 1.53 t

73 who entered the MetS having a combination of central obesity, high blood pressure, and hyperglycemia

74 hazard ratio [HR], 1.46; 95% CI, 1.02-2.09), central obesity (HR, 1.41; 95% CI, 1.01-1.98), diabetes

75 lity risk than those with similar BMI but no central obesity (HR, 1.48 [CI, 1.35 to 1.62]) and those

76 ifestations of glucocorticoid excess include central obesity, hyperglycaemia, dyslipidaemia, electrol

77 ance of early-onset coronary artery disease, central obesity, hypertension, and diabetes.

78 ors or phenotypes that include dyslipidemia, central obesity, hypertension, and hyperinsulinemia, and

79 nificantly with almost all other measures of central obesity in older and younger men and women.

80 ly affected by increasing body mass index or central obesity in OSA patients and in OSA-free subjects

81 ; and stunting, underweight, overweight, and central obesity in women.

82 and overall obesity, and to a lesser extent central obesity, in normal processes of growth and devel

83 The prevalence of overweight, obesity, and central obesity increased with age (all p<0.0001) and wa

84 Obesity and central obesity indicators were highly intercorrelated a

85 Most correlations between obesity and central obesity indicators were moderate to strong (0.40

86 atients with hypothalamic damage may develop central obesity, insulin resistance, and hyperphagia.

87 CT is associated with sustained increases in central obesity, insulin resistance, dyslipidemia and bl

88 e waist circumferences (WCs) to determine if central obesity is associated with abnormalities that mi

89 Central obesity is associated with intrasphincteric exte

90 In children, central obesity is associated with reduced odds of aller

91 In patients with CAD, normal weight with central obesity is associated with the highest risk of m

92 In contrast,central obesity is directly associated with mortality.

93 ased intraabdominal pressure associated with central obesity is the probable etiology of PTC, a condi

94 Obesity, especially central obesity, is a hereditable trait associated with

95 inding that waist circumference, a marker of central obesity, is associated with greater liver stiffn

96 lack an obvious predisposition to GERD (eg, central obesity, large hiatal hernia).

97 overall obesity in PD pathogenesis; however, central obesity may be associated with higher PD risk am

98 y to omental adipose tissue, suggesting that central obesity may reflect "Cushing's disease of the om

99 phenotype, defined by insulin resistance and central obesity, may play a critical role in LV remodeli

100 Our aim was to compare the effect of central obesity (measured by waist-to-height ratio, WHtR

101 Central obesity measures should be incorporated in child

102 Central obesity most accurately predicts asthma.

103 Central obesity, most commonly approximated by the waist

104 sociated with risk factors of CVD, including central obesity, obesity, type 2 diabetes mellitus, rais

105 In the adjusted model, central obesity (OR = 1.88, 95%CI = 1.18, 3.01) and cons

106 ral obesity (OR = 1.77, 95%CI = 1.11, 2.81), central obesity (OR = 2.09, 95%CI = 1.46,3.01) and consu

107 95% confidence interval [CI]: 3.69-9.55) and central obesity (OR = 3.45, 95% CI: 2.27-5.23) were stro

108 erweight (p<0.0001), obesity (p=0.0008), and central obesity (p<0.0001) were more prevalent in male m

109 y used anthropometric measures to indicate a central obesity pattern and an increased risk of cardiov

110 Although insulin resistance and central obesity play an important role in the pathogenes

111 Despite the recognition that central obesity plays a critical role in chronic disease

112 BACKGROUND & AIMS: Central obesity promotes gastroesophageal reflux, which

113 ible sets from trans-ethnic meta-analysis of central obesity provide more precise localizations of po

114 unclear, but one mechanism proposed is that central obesity raises intra-abdominal pressure, which i

115 Central obesity rather than BMI could be a more importan

116 s and reduce their sedentary time to prevent central obesity-related asthma.

117 is BMI and WHR, as indicators of overall and central obesity respectively, were associated with late

118 ence (WC) were used to determine general and central obesity, respectively.

119 Central obesity results in a cluster of metabolic abnorm

120 More boys had general and central obesity than girls (15.2% vs. 6.9%; 27.4% vs. 11

121 , is not associated with the weight gain and central obesity that is commonly observed in postmenopau

122 is a possible mechanism in the pathway from central obesity to asthma.

123 lative macrocephaly, moderate short stature, central obesity, unprovoked aggressive outbursts, fine i

124 NHLBI definitions of central obesity (waist circumference > or = 88 cm for wo

125 parently healthy individuals and measures of central obesity [waist circumference (WC)] and overall o

126 Central obesity was also associated with higher mortalit

127 Similarly, central obesity was associated with increased odds of no

128 Central obesity was associated with mortality (hazard ra

129 In children, central obesity was associated with reduced odds of alle

130 Central obesity was defined as a waist circumference gre

131 of participants with multiple comorbidities, central obesity was found to be associated with adverse

132 001), although in women, the proportion with central obesity was similar (p=0.50), and in men, the pr

133 A variable called "central obesity" was created on the basis of tertiles of

134 Obesity and central obesity were common, and most of the children ha

135 However, overweight, obesity and central obesity were more prevalent in male migrant work

136 Overweight, obesity, or central obesity were not associated with allergic rhinit

137 mates were consistently lower for those with central obesity when age and BMI were controlled for.

138 ls and high screen time increase the risk of central obesity, which leads to asthma development.

139 ata on the "Asian Indian phenotype" identify central obesity, which occurs at a lower body mass index

140 cle in 22 female and 17 male volunteers with central obesity whose age (mean +/- SD) was 51 +/- 9 yea

141 A fuller understanding of the biology of central obesity will require information regarding the g

142 models examined associations of general and central obesity with hypertension, and between body mass

143 ion, but data addressing the relationship of central obesity with kidney disease in type 1 diabetes a

144 esized that CAD patients with normal BMI but central obesity would have worse survival compared to in

145 es resulting in weight loss and reduction of central obesity would lessen the incidence and costs of