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1 reduced ejection fraction and predominantly central sleep apnea.
2 nts with heart failure to the development of central sleep apnea.
3 esponse to carbon dioxide than those without central sleep apnea (5.1+/-3.1 vs. 2.1+/-1.0 liters per
4 and 20 +/- 27; P = 0.015), the prevalence of central sleep apnea (78% and 39%; P = 0.01), and the mea
9 nea appears to have an adverse effect on SV, central sleep apnea appears to have little or slightly p
10 cs have a high prevalence of obstructive and central sleep apnea associated with Cheyne-Stokes respir
12 for considering obstructive sleep apnea and central sleep apnea associated with Cheyne-Stokes respir
13 reduced ejection fraction and predominantly central sleep apnea, but all-cause and cardiovascular mo
14 that uses a noninvasive ventilator to treat central sleep apnea by delivering servo-controlled inspi
15 We encountered a young woman with severe central sleep apnea caused by a medullary glioma located
16 aseline sleep indices: apnea-hypopnea index, central sleep apnea (central apnea index, >/=5 vs. <5),
17 rpose of this study was to determine whether central sleep apnea (CSA) contributes to mortality in pa
25 on dioxide contributes to the development of central sleep apnea in some patients with heart failure.
26 the SERVE-HF (Adaptive Servo-Ventilation for Central Sleep Apnea in Systolic Heart Failure) trial res
28 sed whether elevations in the obstructive or central sleep apnea index or the presence of Cheyne-Stok
31 of central apnea per hour than those without central sleep apnea (mean [+/-SD], 35+/-24 vs. 0.5+/-1.0
33 ep apnea (central apnea index, >/=5 vs. <5), central sleep apnea or Cheyne-Stokes respiration, obstru
34 .18-5.66) and Cheyne-Stokes respiration with central sleep apnea (OR, 2.27; 95% CI, 1.13-4.56), but n
36 art failure with (n = 9) and without (n = 8) central sleep apnea using transcranial ultrasound during
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