ライフサイエンスコーパス: central venous pressure

1 gement may differ based on patients' initial central venous pressure.

2 outcome when compared to guiding therapy on central venous pressure.

3 by mean aortic pressure, both subtracted by central venous pressure.

4 mean aortic pressure both subtracted by mean central venous pressure.

5 s with congenital heart disease and elevated central venous pressure.

6 decreasing lung compliance without impacting central venous pressure.

7 istress syndrome patients with a low initial central venous pressure.

8 hrough the pulmonary circulation at a normal central venous pressure.

9 86, respectively, compared with 0.55 for the central venous pressure, 0.56 for the global end-diastol

10 iving LVAD support for >/=30 days had higher central venous pressures (11+/-6 versus 8+/-5 mm Hg, P=0

11 h SNP (n = 78) had significantly higher mean central venous pressure (15 vs. 13 mm Hg; p = 0.001), pu

12 ressure 12 mm Hg (median = 8, p = .005); and central venous pressure -2 mm Hg (median = -1, p = .04).

13 +/- 12 mm Hg vs. 33 +/- 8 mm Hg, p = 0.002, central venous pressure: 20 +/- 6 mm Hg vs. 16 +/- 8 mm

14 pulmonary capillary wedge pressure 33%, and central venous pressure 27% while increasing cardiac out

15 2 to 93 +/- 4 beats min(-1)), while reducing central venous pressure (5.5 +/- 07 to 0.2 +/- 0.6 mmHg)

16 ure (9.0 +/- 0.6 to 14.3 +/- 0.8 mm Hg), and central venous pressure (6.6 +/- 0.7 to 10.7 +/- 0.9 mm

17 f exogenous ANP and observations of elevated central venous pressure after a similar volume expansion

18 ty index was not an independent predictor of central venous pressure after adjusting for inferior ven

19 Central venous pressure, airway pressure, pericardial pr

20 We measured heart rate, central venous pressure and arterial pressure during all

21 correlation coefficient between the baseline central venous pressure and change in stroke volume inde

22 relation coefficient, and/or the AUC between central venous pressure and change in stroke volume inde

23 volume occurred in the face of reductions in central venous pressure and circulating blood volume.

24 oth outcomes included greater intraoperative central venous pressure and greater transfusion volumes.

25 en saturation remains low, despite achieving central venous pressure and mean arterial pressure targe

26 F, and systemic and leg oxygen delivery, but central venous pressure and muscle metabolism remained u

27 nificantly with respect to the monitoring of central venous pressure and oxygen and the use of intrav

28 robserver variability in the measurements of central venous pressure and pulmonary artery occlusion p

29 ressure signal (Paw) to pressure tracings of central venous pressure and pulmonary artery occlusion p

30 m pulmonary capillary wedge pressure (PCWP), central venous pressure and SV (via thermodilution) were

31 demonstrated an interaction between initial central venous pressure and the effect of fluid strategy

32 PICCs can be used to measure central venous pressure and to follow trends in a clinic

33 ols(n=9), both toxins decreased arterial and central venous pressures and systemic vascular resistanc

34 reload (pulmonary artery occlusion pressure, central venous pressure) and end-diastolic ventricular v

35 0 g of liver tissue, mean arterial pressure, central venous pressure, and CI were analyzed.

36 pressure variation, stroke volume variation, central venous pressure, and end-expiratory occlusion te

37 index correlated with cool extremities, high central venous pressure, and low 24-hr fluid output; and

38 ng age, elevated serum creatinine, increased central venous pressure, and red blood cell transfusion

39 pressure, cerebral perfusion pressure (CPP), central venous pressure, and urine output before and aft

40 Aortic pressures, central venous pressures, and heart rates were not diffe

41 therapy produced higher survival (P = .008), central venous pressures, and left ventricular ejection

42 ar clamping techniques supplemented with low central venous pressure anesthesia, availability of nove

43 Low central venous pressure anesthetic technique was used in

44 dex (CFIp, calculated as (occlusive pressure-central venous pressure)/(aortic pressure-central venous

45 espiratory distress syndrome patients with a central venous pressure available at enrollment, 609 wit

46 sites in 10 eyes, was associated with higher central venous pressure before treatment (P = 0.03), pro

47 d markers of intravascular volume, including central venous pressure, brain-natriuretic-peptide conce

48 pulmonary artery pressure (sPAP) and higher central venous pressure, but not with other clinical or

49 ation correlated with knee mottling and high central venous pressure, but these correlations were not

50 PICC central venous pressure more than CICC central venous pressure by 1.0 + 3.2 mm Hg (p = 0.02).

51 s no carcinine-induced effect on heart rate, central venous pressure, cardiac index, or stroke index.

52 al-venous equilibrium pressure, arterial and central venous pressure, cardiac output (LiDCOplus; LiDC

53 measured mean arterial pressure, heart rate, central venous pressure, cardiac output, stroke volume v

54 d continuous aortic, pulmonary arterial, and central venous pressures, cardiac output by thermodiluti

55 ring, including intra-arterial catheters and central venous pressure catheters, and more technologica

56 nal tube from previously placed arterial and central venous pressure catheters.

57 n saturation, intra-arterial blood pressure, central venous pressure, chest wall movement, electrocar

58 n and congenital heart disease with elevated central venous pressure complicated by PLE.

59 inical decisions regarding fluid management, central venous pressure continues to be recommended for

60 A low central venous pressure (CVP) (mean threshold <8 mm Hg)

61 Patients who developed WRF had a greater central venous pressure (CVP) on admission (18 +/- 7 mm

62 (CT) to determine the effects of changes in central venous pressure (CVP) on upper airway size.

63 Central venous pressure (CVP) provides information regar

64 Efferent postganglionic muscle SNA, BP, and central venous pressure (CVP) were measured in 14 patien

65 ysiological study, SNA, blood pressure (BP), central venous pressure (CVP), and heart rate were recor

66 rs, intra-arterial blood pressure (BP), ECG, central venous pressure (CVP), and muscle sympathetic ne

67 Arterial blood pressure (BP), central venous pressure (CVP), and peripheral muscle sym

68 Arterial blood pressure (BP), central venous pressure (CVP), and SNA were recorded dur

69 ompare peripheral venous pressure (PVP) with central venous pressure (CVP), as well as other invasive

70 terial blood pressure (BP), heart rate (HR), central venous pressure (CVP), muscle sympathetic nerve

71 ulmonary capillary wedge pressure (PCWP) and central venous pressure (CVP).

72 ent in the emergency department: a) initiate central venous pressure (CVP)/central venous oxygen satu

73 Estimated central venous pressure decreased with LBNP (P<0.05), in

74 tial pulmonary artery occlusion pressure and central venous pressure did not correlate significantly

75 ystolic to diastolic duration), and elevated central venous pressure (expressed as right atrial [RA]

76 2 secs, knee mottling, or cool extremities), central venous pressure, fluid output, and central venou

77 We examined the relationship between initial central venous pressure, fluid strategy, and 60-day mort

78 s in pulmonary artery occlusion pressure and central venous pressure following saline infusion also d

79 Mean central venous pressure from the CICCs was 11 + 7 mm Hg,

80 the receiver operating characteristic of the central venous pressure, global end-diastolic volume ind

81 s without baseline shock, those with initial central venous pressure greater than 8 mm Hg experienced

82 RV dysfunction was defined as central venous pressure >15 mmHg and consistent echocard

83 ean arterial pressure >70 mm Hg; ScvO2 <70%; central venous pressure >8 mm Hg.

84 Pulmonary artery occlusion pressure and central venous pressure have been considered to be relia

85 Additionally, central arterial pressure, central venous pressure, heart rate, arterial blood gas,

86 form the procedure, the inability to monitor central venous pressure in the emergency department, and

87 lure after enalaprilat despite reductions in central venous pressure in this group.

88 re, pulmonary artery occlusion pressure, and central venous pressure, increased and SVR decreased in

89 ules: laparoscopy with pneumoperitoneum, low central venous pressure, intermittent pedicle clamping,

90 The global effect of the fluid challenge on central venous pressure is greater in nonresponders, but

91 erial pressure, pulmonary arterial pressure, central venous pressure, kaolin and celite activated clo

92 With a cut-off value<8 mm Hg for central venous pressure, kappa was 0.33 [-0.03;0.69].

93 ferior vena cava diameter < 2 cm predicted a central venous pressure < 10 mm Hg with a sensitivity of

94 (area under the curve) to discriminate a low central venous pressure (< 10 mm Hg) was 0.91 for inferi

95 n in the remaining 14 patients who all had a central venous pressure<12 mm Hg.

96 ent targeting three physiological variables: central venous pressure, mean arterial pressure, and eit

97 learance group was resuscitated to normalize central venous pressure, mean arterial pressure, and lac

98 he ScvO2 group was resuscitated to normalize central venous pressure, mean arterial pressure, and Scv

99 is-induced hypoperfusion, specific levels of central venous pressure, mean arterial pressure, urine o

100 view of widening the range of cardiac index:central venous pressure measurements and increasing the

101 IRV significantly increased central venous pressure measurements from both catheter

102 of the trauma patient, or FAST, is replacing central venous pressure measurements to detect hemoperic

103 clinical study, three to 12 paired, digital, central venous pressure measurements were recorded from

104 Paired central venous pressure measurements were taken from 19-

105 After TIPS, central venous pressure (median, 11 vs 15 cm H(2)O; P <

106 tage of nursing staff, problems in obtaining central venous pressure monitoring, and challenges in id

107 Analysis by repeated measures showed PICC central venous pressure more than CICC central venous pr

108 Neither central venous pressure nor left ventricular end diastol

109 Neither central venous pressure nor pulmonary artery occlusion p

110 of <60 mm Hg, heart rate of >120 beats/min, central venous pressure of >15 mm Hg, stroke volume inde

111 hange in pericardial pressure, and change in central venous pressure of 1.1 +/- 0.7, 1.1 +/- 0.8, 0.7

112 A central venous pressure of 10 mm Hg was chosen a priori

113 Mean arterial pressure >/=65 mm Hg and central venous pressure of 10 to 15 mm Hg were hemodynam

114 (18% vs 18%; p = 0.928), whereas those with central venous pressure of 8 mm Hg or less experienced l

115 disorders, and disorders involving increased central venous pressure or mesenteric lymphatic obstruct

116 val (p = .05); fluid requirements (p = .05); central venous pressures (p </= .007); indicators of hem

117 related with increases in mean pulmonary and central venous pressures (P<0.05).

118 re-central venous pressure)/(aortic pressure-central venous pressure); pressure values in mm Hg) of t

119 Prior intravenous protamine, central venous pressure prior to protamine, preoperative

120 High baseline central venous pressure, prolonged fluorescein transit t

121 rterial pressure (invasive and noninvasive), central venous pressure, pulmonary arterial pressure, le

122 MAP, heart rate, central venous pressure, pulmonary artery occlusion pres

123 of intracranial pressure, pleural pressure, central venous pressure, pulmonary artery occlusion pres

124 ences in heart rate, mean arterial pressure, central venous pressure, pulmonary artery occlusion pres

125 did not affect ejection fraction or increase central venous pressure, pulmonary pressures, or left at

126 strate a lack of correlation between initial central venous pressure/pulmonary artery occlusion press

127 rterial pressure, pulmonary artery pressure, central venous pressure, pulse oximetry, and end-tidal C

128 ena cava diameter correlated moderately with central venous pressure (R = 0.58), whereas the inferior

129 Central venous pressure recorded via PICCs is slightly h

130 The slope of the multipoint cardiac index:central venous pressure relationship increased (p = .02)

131 pleural pressure, pericardial pressure, and central venous pressure, respectively.

132 In addition, initial central venous pressure, right ventricular end-diastolic

133 sion pressure (CPP), mean arterial pressure, central venous pressure, serum sodium concentrations, se

134 a previous meta-analysis that concluded that central venous pressure should not be used to make clini

135 2 mm Hg between two measurements was 79% for central venous pressure strips without Paw vs. 86% with

136 not obligatory for sustaining venous return, central venous pressure,stroke volume and (.)Q or mainta

137 Whilst lying in the supine posture, central venous pressure (supine, 7 +/- 3 vs. microgravit

138 ith LeTx, as reflected by greater or earlier central venous pressures, systemic vascular resistance,

139 a cava diameter is a more robust estimate of central venous pressure than the inferior vena cava coll

140 haracteristics, patient population, baseline central venous pressure, the correlation coefficient, an

141 At lower initial central venous pressures, the difference between arms wa

142 At higher initial central venous pressures, the difference in treatment be

143 to support the widespread practice of using central venous pressure to guide fluid therapy.

144 y infused prior to LBNP sufficient to return central venous pressure to pre-heat stress values.

145 rial lactate upon clamping as well as in the central venous pressure upon unclamping.

146 The range of central venous pressure values was 1-23 mm Hg with a med

147 f cardiac index (pulse contour analysis) and central venous pressure values.

148 Central venous pressure was kept constant by colloid/cry

149 y that the estimated area under the curve of central venous pressure was smaller in nonresponders was

150 erial blood pressure, electrocardiogram, and central venous pressure were also recorded continuously.

151 Heart rate, blood pressure and central venous pressure were measured.

152 cle sympathetic nerve activity and estimated central venous pressure were recorded during nonhypotens

153 Measurements of central venous pressure were recorded from both sites by

154 During the clinical study, measurements of central venous pressure were recorded from patients who

155 Although blood and central venous pressures were invasively monitored in >

156 We hypothesized that initial central venous pressure would modify the effect of fluid