ライフサイエンスコーパス: central vision

1 closely associated with the deterioration of central vision.

2 retinal pigment epithelium leads to loss of central vision.

3 reported neither for near peripheral nor for central vision.

4 slowly over time, eventually causing loss of central vision.

5 representation of letter feature position in central vision.

6 tive disorder associated with severe loss in central vision.

7 on, common only in normal peripheral but not central vision.

8 of family C who experienced markedly reduced central vision.

9 eneration (AMD), which causes severe loss of central vision.

10 a small region of the retina responsible for central vision.

11 the impact of the scotomata on the patient's central vision.

12 clinical stages are accompanied by impaired central vision.

13 ily rods and secondarily cones, that mediate central vision.

14 The wet form leads to severe loss of central vision.

15 hotophobia, loss of color vision and reduced central vision.

16 ium is associated with a progressive loss of central vision.

17 ive to vertical - disparities that occurs in central vision.

18 scularization, which leads to severe loss of central vision.

19 many positions throughout a wide portion of central vision.

20 tients with diabetic retinopathy and loss of central vision.

21 There was no change in central vision after 6 months of lutein supplementation,

22 There was no change in central vision after 6 months of lutein supplementation.

23 AMD results in loss of central vision and a dependence on low-resolution periph

24 cular dystrophy leads to progressive loss of central vision and shows symptoms similar to age-related

25 milies included progressive deterioration of central vision and subsequently night vision, mild photo

26 was typical CORD with photophobia, decreased central vision, and dyschromatopsia.

27 Such large fields and inclusion of central vision at nearly all sites precluded retinotopic

28 Both maps represent central vision at the posterior end of the border betwee

29 dystrophy and is characterized by decreased central vision, atrophy of the macula and underlying ret

30 nd 20 patients with RP who had retained good central vision (better than 20/32).

31 In central vision, both schemes are capable of producing co

32 Humans exploit the high resolution of central vision by actively moving their eyes three to fo

33 , progressing to severe disease with loss of central vision by the third decade in affected males.

34 lateral, the severe and irreversible loss of central vision experienced by affected persons has been

35 Severe loss of central vision frequently occurs with the exudative (wet

36 Given the prominence of central vision in humans, it has been assumed that visua

37 escence, side vision in young adulthood, and central vision in later life because of progressive loss

38 or (VEGF)-neutralising proteins can preserve central vision in many patients with neovascular age-rel

39 rdinal orientations in the representation of central vision in owl monkey V1 was relatively small and

40 Because of the prominence of central vision in primates, it has generally been assume

41 reasing visual field loss, with concerns for central vision increasing, whereas those for outdoor mob

42 We demonstrate that the loss of central vision induces functional mobilization of motion

43 locus in normally sighted individuals whose central vision is blocked by an artificial scotoma.

44 is well preserved, or late disease, in which central vision is lost.

45 serves as the preferred fixation locus when central vision is lost.

46 On the other hand, unrestricted central vision is not sufficient to ensure normal refrac

47 (AMD), a blinding disorder that compromises central vision, is characterized by the accumulation of

48 study has found that artificial occlusion of central vision leads to rapid emergence, and long-term m

49 hange visual sensory processing, introducing central vision loss (a scotoma).

50 wet, age-related macular degeneration causes central vision loss and represents a major health proble

51 Most patients with AMD-related central vision loss continue to drive, but demonstrate s

52 Our data suggested that age and mild central vision loss did not affect significantly a subje

53 mally-sighted controls and participants with central vision loss due to macular degeneration (MD).

54 's hereditary optic neuropathy (LHON) causes central vision loss from bilateral optic neuropathy.

55 can be used to screen for moderate to severe central vision loss from glaucoma.

56 nderstood about the prevalence and nature of central vision loss in early glaucoma.

57 r degeneration (AMD) is the leading cause of central vision loss in older adults.

58 scope of visual cortex plasticity following central vision loss is essential both for clarifying the

59 ch humans adapt eye movements in response to central vision loss is still not well understood and car

60 These results suggest that central vision loss may give rise to cortical thinning,

61 Therefore we examined the impact of central vision loss on motion perception using random do

62 Here, we explore the possible effects of central vision loss on the optimal saccades during a fac

63 bservers (mean age, 73.8) with long-standing central vision loss practiced an oral sentence-reading t

64 utational model to predict where humans with central vision loss should direct their eye movements in

65 in most patients, although in 1 patient with central vision loss such change was absent.

66 Clinical observations of patients with central vision loss suggest a lengthy adjustment period,

67 e reading speed in people with long-standing central vision loss was evaluated.

68 Age, but not mild central vision loss, significantly affected a subject's

69 f retinal break formation and to prevent the central vision loss.

70 that results in progressive and irreversible central vision loss.

71 ble rehabilitation regimen for patients with central vision loss.

72 2 (LCA2) characterized by the early onset of central vision loss.

73 -onset complex retinal disease that leads to central vision loss.

74 enhancing visual performance for people with central vision loss.

75 up of disorders characterized by progressive central vision loss.

76 s more prominent in parts of MT representing central vision (< or =10 degrees).

77 All patients had myopia, reduced central vision, nystagmus, and electroretinographic evid

78 The effects on MP and central vision of 6 months of lutein supplementation at

79 ritical factor limiting object perception in central vision of individuals with neurodegeneration of

80 characterized by crowding and poor acuity in central vision of the affected eye.

81 e a variety of maculopathies that can reduce central vision permanently.

82 Central vision ranged from normal to reduced in the firs

83 The PRL was located outside the compromised central vision region, typically near the edge of the sc

84 low 24 degrees or 37 degrees of unrestricted central vision, respectively.

85 Central vision typically remains preserved at least unti

86 ERG a- and b-waves, central vision, vestibulomotor function, the spiking pro

87 ients who presented with unexplained loss of central vision, visual field defects, and/or photopsia w

88 ion selectivity, vestibulomotor function and central vision was compared between the D2 and B6 mouse

89 Central vision was unchanged after supplementation.

90 Central vision was unchanged after the period of supplem

91 Changes in MPOD and central vision were determined in a subset of patients r

92 AMD affects central vision which impairs one's ability to drive, rea