ライフサイエンスコーパス: cephalexin

1 All participants received cephalexin.

2 coli elongated by growth in the presence of cephalexin.

3 eplication in the presence of rifampicin and cephalexin.

4 filament, as is found in cells treated with cephalexin.

5 3 orders of magnitude greater than that for cephalexin.

6 Cephalexin, 500 mg 4 times daily, plus trimethoprim-sulf

7 pants were randomly assigned to receive oral cephalexin, 500 mg, and metronidazole, 500 mg (n = 202 p

8 e deacylation-defective wild-type and mutant cephalexin acyl-enzyme intermediates.

9 Specifically, cephalexin acylated PBP3 about 50% faster in a populatio

10 een for growth on the beta-lactam antibiotic cephalexin afforded a unique p-acrylamido-phenylalanine

11 us trimethoprim-sulfamethoxazole compared to cephalexin alone did not result in higher rates of clini

12 l cure rate of uncomplicated cellulitis than cephalexin alone.

13 -lactam antibiotics ampicillin, amoxicillin, cephalexin and cefadroxil, the antineoplastics delta-ami

14 n after cesarean delivery, prophylactic oral cephalexin and metronidazole may be warranted.

15 , double-blind clinical trial comparing oral cephalexin and metronidazole vs placebo for 48 hours fol

16 ng obese women who receive prophylactic oral cephalexin and metronidazole vs placebo for 48 hours fol

17 axis, a postoperative 48-hour course of oral cephalexin and metronidazole, compared with placebo, red

18 using low concentrations of the beta-lactams cephalexin and piperacillin to specifically inhibit FtsI

19 endence of k(cat)/K(m) for benzylpenicillin, cephalexin, and cefoxitin similarly indicated the import

20 nucleoids are present in cells incubated in cephalexin, and this is increased in polA12 mutants.

21 d acylation of PBP3 with three beta-lactams (cephalexin, aztreonam, and piperacillin) in growing cell

22 ntly, when septal PG synthesis is blocked by cephalexin, both EnvC and NlpD are recruited to septal r

23 Acylation of PBP3 with cephalexin, but not aztreonam or piperacillin, appeared

24 Here we find that one such beta-lactam, cephalexin, can, under certain conditions, lead instead

25 minopenicillins and aminocephalosporins (ie, cephalexin, cefaclor, and cefadroxil) of around 20%, as

26 tams, all subjects underwent skin tests with cephalexin, cefaclor, cefadroxil, cefuroxime, ceftriaxon

27 d polymer (MIP) for cephalosporin molecules (cephalexin (CFL) and cephapirin (CFP)), was prepared by

28 rect photolysis (t(1/2) = 0.7, 3.9 h), while cephalexin (CFX) and cephradine (CFD) were mainly transf

29 s, ampicillin (AMP), benzylpenicillin (PEG), cephalexin (CFX), cefazolin (CFL), cefoperazone (CFP), c

30 ural features of dileucine stereoisomers and cephalexin contributing to interaction with the dipeptid

31 , azithromycin, carbamazepine, moxifloxacin, cephalexin, diclofenac, and nitrofurantoin.

32 addition of trimethoprim-sulfamethoxazole to cephalexin did not improve outcomes overall or by subgro

33 ional FtsZ rings assemble in the presence of cephalexin, even after several generations of growth.

34 served from milk components after elution of cephalexin from MIP, indicating selectivity and affinity

35 solid phase extraction (SPE) for recovery of cephalexin from spiked milk samples for further estimati

36 thoxazole group vs 165 (85.5%) of 193 in the cephalexin group (difference, -2.0%; 95% CI, -9.7% to 5.

37 thoxazole group vs 171 (69.0%) of 248 in the cephalexin group (difference, 7.3%; 95% CI, -1.0% to 15.

38 ar > NAAG, delta-ALA, bestatin > cefadroxil, cephalexin > ampicillin, amoxicillin.

39 dical initiator, along with target molecule (cephalexin) in a porogenic material.

40 hydrolysis of cefoxitin, cephaloridine, and cephalexin indicate that an enzyme residue of apparent p

41 GFP localized to potential division sites in cephalexin-induced and ftsI mutant filaments, further su

42 Furthermore, unlike cephalexin, intramolecular aminolysis in the S-analogue

43 aminolysis by a reaction similar to that for cephalexin itself.

44 tion was diagnosed in 13 women (6.4%) in the cephalexin-metronidazole group vs 31 women (15.4%) in th

45 s blocked if cell division is inhibited with cephalexin or by a ftsZts mutation.

46 . tuberculosis to DNA damaging agents, or to cephalexin, or growth of M. tuberculosis in macrophages

47 crogram of oxacillin per ml, 40 microgram of cephalexin per ml, or 2.5 microgram of methicillin per m

48 l agar (CA) with and without 40 microgram of cephalexin per ml.

49 daily, for 7 days (n = 248 participants) or cephalexin plus placebo for 7 days (n = 248 participants

50 ts with uncomplicated cellulitis, the use of cephalexin plus trimethoprim-sulfamethoxazole compared t

51 ed in 189 (76.2%) of 248 participants in the cephalexin plus trimethoprim-sulfamethoxazole group vs 1

52 ed in 182 (83.5%) of 218 participants in the cephalexin plus trimethoprim-sulfamethoxazole group vs 1

53 To determine whether cephalexin plus trimethoprim-sulfamethoxazole yields a h

54 d a clinically important difference favoring cephalexin plus trimethoprim-sulfamethoxazole, further r

55 Etest using BGA and Regan-Lowe agar without cephalexin (RL-C), and disk diffusion using BGA and RL-C

56 assemble in newborn cells in the presence of cephalexin, suggesting that newborn cells contain a site

57 We found that cephalexin, the antibiotic commonly incorporated in both

58 We used cephalexin to generate elongated cells with irregular sh

59 Binding of cephalexin towards prepared MIP was studied in different

60 Gliding movements of the leading poles of cephalexin-treated filamentous cells were observed but n

61 t septation, such as FtsZ overproduction and cephalexin treatment, induced growing cells to swell, bu

62 spitalization, recent skin infection, recent cephalexin use, and household S. aureus or MRSA fomite c

63 contacts were recent skin infection, recent cephalexin use, and USA300 genetic background.

64 tem using isolated membranes, acylation with cephalexin was not impaired by depletion of FtsW or FtsN

65 Molecular imprinted polymer (MIP) against cephalexin was synthesized by co-polymerization of funct

66 be produced by treatment with the antibiotic cephalexin, which blocks cell division but allows cell g

67 The k(cat)/K(m) for cefoxitin and cephalexin with cadmium substituted BcII is dependent on

68 The thioxo derivative of cephalexin, with an amino group in the C7 side chain, un