ライフサイエンスコーパス: ceramide 1-phosphate

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1 ted by lyso-PA, sphingosine 1-phosphate, and ceramide 1-phosphate.

2 lysophosphatidic acid, phosphatidic acid, or ceramide-1-phosphate.

3 The ability of the "caged" ceramide 1-phosphate analogues to release the bioactive

4 ified and characterized interactions between ceramide 1-phosphate and the annexin a2-p11 heterotetram

5 elated sphingolipids, including dihydro-S1P, ceramide 1-phosphates, and sphingosine phosphocholine as

6 exposed phosphate group: phosphatidic acid, ceramide-1-phosphate, and dihydroceramide-1-phosphate.

7 ated by the emerging bioactive sphingolipid, ceramide 1-phosphate (C-1-P) (1).

8 The bioactive sphingolipid, ceramide 1-phosphate (C-1-P), has been implicated as an

9 In this study, ceramide-1-phosphate (C-1-P) was shown to be a specific

10 ssibility that ceramide metabolites, such as ceramide-1-phosphate (C-1-P), may explain these discrepa

11 lipid transfer protein that is specific for ceramide 1-phosphate (C1P) and phyto-C1P.

12 n to this accepted mechanism of two decades, ceramide 1-phosphate (C1P) has been shown to increase th

13 In this study, we demonstrate that ceramide 1-phosphate (C1P) produced by ceramide kinase (

14 osphatidic acid (PA), lysophosphatidic acid, ceramide 1-phosphate (C1P), and sphingosine 1-phosphate

15 ated the potential for another sphingolipid, ceramide 1-phosphate (C1P), to modulate efflux pumps at

16 Phosphorylated sphingolipids ceramide-1-phosphate (C1P) and sphingosine-1-phosphate (

17 Recently, ceramide-1-phosphate (C1P) has been shown to modulate ac

18 expression following ER stress, blockade of ceramide-1-phosphate (C1P) suppressed Tg-induced hBD2/hB

19 Previously, ceramide-1-phosphate (C1P) was demonstrated to be a pote

20 rated that ceramide kinase, and its product, ceramide 1-phosphate (Cer-1-P), were mediators of arachi

21 (N,N-diMe-Sph) were weak inhibitors whereas ceramide-1-phosphate (Cer-1-P) and sphingosine-1-phospha

22 Ceramide-1-phosphate (Cer-1-P), one of the simplest of a

23 r human ceramide kinase (hCERK) activity and ceramide 1-phosphate formation could lead to enhanced ph

24 Ceramide kinase and its product ceramide 1-phosphate have been implicated in cellular pr

25 ritical event in phagocytosis, the effect of ceramide-1-phosphate in promoting the fusion of liposome

26 evels in biological samples by conversion to ceramide-1-phosphate, in a manner analogous to that of t

27 not C(8)-glucosylceramides, sphingosine, or ceramide 1-phosphate, increases ABCA12 mRNA expression i

28 Ceramide-1-phosphate is formed by a calcium-dependent ce

29 In summary, ceramide-1-phosphate is formed during phagocytosis throu

30 Agonist-stimulated formation of ceramide-1-phosphate is now shown to occur in 32PO4-labe

31 d product by thin-layer chromatography since ceramide-1-phosphate is the only radiolabeled lipid in o

32 vector control and simultaneously increased ceramide 1-phosphate levels 2-fold compared with vector

33 Ceramide-1-phosphate may promote phagolysosome formation

34 Neither phosphocholine, ceramide-1-phosphate, nor sphingosine-1-phosphate recapi

35 YSR2 failed to dephosphorylate ceramide 1-phosphate or phosphatidic acid.

36 In vitro, activation of cPLA2 by ceramide-1-phosphate or A23187 induced spinal neuronal d

37 idic acid nor other sphingolipids, ceramide, ceramide-1-phosphate, or sphingosylphosphorylcholine, in

38 ing concentrations of sphingomyelinase D and ceramide-1-phosphate promoted liposomal fusion.

39 lucosylceramide, sulfatide, ganglioside GM1, ceramide 1-phosphate, sphingosine 1-phosphate, dihydroce

40 The C1P binding site in ACD11 and in human ceramide-1-phosphate transfer protein (CPTP) is surround

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