ライフサイエンスコーパス: cerebellar

1 Learning was abolished by cerebellar ablation.

2 atrophy, ventriculomegaly, hydrocephaly, and cerebellar abnormalities.

3 To study cerebellar activity during learning, we made whole-cell

4 Correspondingly, vocal motor and cerebellar activity is greater during active echolocatio

5 Interestingly, disrupting cerebellar activity via transcranial magnetic stimulatio

6 ly, genetic deletion of Parp1 rescued normal cerebellar ADP-ribose levels and reduced the loss of cer

7 Consistent with the preponderance of cerebellar afferents within the pons, we observed a sign

8 Cerebellar aggregation pathology may suggest a prionlike

9 ole of the recently discovered basal ganglia-cerebellar anatomical links.

10 d that the recently discovered basal ganglia-cerebellar anatomical pathway may support the involvemen

11 order diagnosis on different measures of the cerebellar anatomy as well as the effect of possible fac

12 Group differences in cerebellar and parietal neuronal activity occurred durin

13 His examination was consistent with cerebellar and upper motor neuronal dysfunction.

14 right cerebellar vermis and left lobule V of cerebellar anterior lobe were additionally activated for

15 This cerebellar area overlapped previously recognized regions

16 Using fMRI, we identify a cerebellar area that is active when words are predicted

17 Atrophy colocated with cerebellar areas implicated for motor (PSP, MSA) or cogn

18 Intact and pressurized rat superior cerebellar arteries were labelled for confocal immunoflu

19 ells (VSMCs) in the pressurized rat superior cerebellar artery.

20 on, and perceptual organization did not have cerebellar associations.

21 Taken together, our data identify cerebellar astrocytes as key responders to viral infecti

22 nd IFN-induced expression in human and mouse cerebellar astrocytes than did cerebral cortical astrocy

23 failure, levodopa-unresponsive parkinsonism, cerebellar ataxia and pyramidal symptoms.

24 Progressive limb spasticity and cerebellar ataxia are frequently found together in clini

25 cohort of hereditary spastic paraplegia and cerebellar ataxia cases (n = 618) for mutations in POLR3

26 ersus a blinded panel of autosomal recessive cerebellar ataxia experts.

27 and spectrin-associated autosomal recessive cerebellar ataxia Type 1 pathology likely arises from po

28 and spectrin-associated autosomal recessive cerebellar ataxia Type 1, but molecular mechanisms linki

29 ked in two different families to adult-onset cerebellar ataxia, and a de novo truncation mutation res

30 re were defined for each autosomal recessive cerebellar ataxia, and corresponding prediction scores w

31 sing frequency): peripheral neuropathy, 53%; cerebellar ataxia, dysmetria, or dysarthria, 38%; and en

32 ive mutations in WDR81, previously linked to cerebellar ataxia, mental retardation, and disequilibriu

33 ual disability, and many individuals exhibit cerebellar ataxia, subtle facial dysmorphism, strabismus

34 minant parkinsonism and MSA with predominant cerebellar ataxia, which generally correlate with striat

35 poly(ADP-ribose) polymerase/s as a cause of cerebellar ataxia.

36 t cause of hereditary spastic paraplegia and cerebellar ataxia.

37 A RNU12 that was associated with early onset cerebellar ataxia.

38 of the literature on 67 autosomal recessive cerebellar ataxias and personal clinical experience.

39 tive outcome measures for clinical trials on cerebellar ataxias are lacking.

40 Cerebellar ataxias are the result of diverse disease pro

41 Most cerebellar ataxias progress very slowly and quantitative

42 s such as hereditary spastic paraplegias and cerebellar ataxias remain genetically unexplained, impli

43 g molecular diagnoses of autosomal recessive cerebellar ataxias, thereby guiding targeted sequencing

44 We evaluated two scales for rating cerebellar ataxias: the Composite Cerebellar Functional

45 atter fraction, T2 lesion volume, metrics of cerebellar atrophy and cerebellar lesion volume) and mot

46 sive encephalopathy characterized by extreme cerebellar atrophy due to almost total granule neuron lo

47 30s and 40s and show predominant ataxia and cerebellar atrophy features on imaging.

48 eurological features including ataxia and/or cerebellar atrophy on brain MRI.

49 Across 54 studies, clusters of cerebellar atrophy were found for AD, ALS, FTD, MSA, and

50 ntal delay, hypotonia, early-onset seizures, cerebellar atrophy, and osteopenia.

51 her displays later-onset encephalopathy with cerebellar atrophy, ataxia and dystonia.

52 netic resonance imaging revealed symmetrical cerebellar atrophy, which appeared progressive, and a pr

53 severe and progressive brain, brainstem and cerebellar atrophy, with hypomyelination in most.

54 e, ASO7 localized to Purkinje cells, reduced cerebellar ATXN2 expression below 75% for more than 10 w

55 oglial activation, and reduced the levels of cerebellar ATXN2.

56 lls can fire coincident simple spikes during cerebellar behaviours, we varied the proportion (0-20 of

57 Neocortical basal radial glia (bRG) and cerebellar Bergmann glia (BG) are basal progenitors deri

58 This study aims to compare the cerebellar biochemical profiles in preterm (PT) infants

59 important risk factors for impaired preterm cerebellar biochemistry.

60 the granular layer as a major determinant of cerebellar blood-oxygen-level-dependent signals.

61 grafts of fetal dopaminergic cells, control cerebellar cells, or vehicle bilaterally and were studie

62 t of an ataxic phenotype is linked to severe cerebellar cellular pathology, characterized by nuclear

63 e network and enhanced activity in a cortico-cerebellar circuit correlated with the severity of socia

64 may play a central role in control of olivo-cerebellar climbing fibre signals.

65 o previous studies, our findings reveal that cerebellar cognitive areas are reached by the largest pr

66 TC-CPC loop provides a substrate for cerebro-cerebellar communication during cognitive processing.

67 ions about the use of corollary discharge in cerebellar computations, we studied it in mice of both s

68 -mortem humans have established that cerebro-cerebellar connections are contralateral to each other a

69 heavy alcohol consumption, whereas increased cerebellar connectivity in HD may have compensatory effe

70 ks is now well established, yet the specific cerebellar contribution to language processing remains u

71 ll layer (GCL) is fundamental to theories of cerebellar contributions to motor control and learning.

72 Understanding cerebellar contributions to motor coordination requires

73 lar cortex, as required by forward models of cerebellar control.

74 S: Purkinje cells are the sole output of the cerebellar cortex and fire two distinct types of action

75 e and Golgi cells, is the first stage of the cerebellar cortex and processes spatiotemporal informati

76 BPND and SUVR were calculated using the cerebellar cortex as a reference region and were compare

77 eractions among PCs, which suggests that the cerebellar cortex is more functionally diverse than is a

78 We injected these vectors into the cerebellar cortex of rhesus macaques and tested vector e

79 The PiB uptake was quantified as a region to cerebellar cortex ratio.

80 s widely available at the input stage of the cerebellar cortex, as required by forward models of cere

81 g with the pruning of climbing fibres in the cerebellar cortex, implicates the climbing fibre collate

82 inje cells, or for CRF2 in any aspect of the cerebellar cortex.

83 d negative stripes of PCs across most of the cerebellar cortex.

84 ion because they form the sole output of the cerebellar cortex.

85 als to generate climbing fibre inputs to the cerebellar cortex.

86 n at both the input and output layers of the cerebellar cortex.

87 een these two types of impulse is central to cerebellar cortical information processing.

88 ency, which has implications for controlling cerebellar cortical output and motor learning.

89 llo-thalamo-cortical (CTC) and cortico-ponto-cerebellar (CPC) pathways.

90 internal modeling, we hypothesize that right cerebellar Crus I/II supports prediction of upcoming sen

91 between MRI variables of supratentorial and cerebellar damage (grey matter fraction, T2 lesion volum

92 Cerebellar damage has been implicated in information pro

93 , comparing the performance of patients with cerebellar degeneration and matched controls.

94 Mutations in TG6 induce cerebellar degeneration by an unknown mechanism.

95 cognitive and motor dysfunctions arise from cerebellar degeneration.

96 KEY POINTS: A cerebellar dentate nuclei (DN) contribution to volitiona

97 functional magnetic resonance (MR) imaging, cerebellar dentate nuclei (DNs) functional connectivity

98 n-specific knockout of RNF8 or UBC13 impairs cerebellar-dependent learning.

99 sensorimotor adaptation of the upper limb, a cerebellar-dependent process restoring movement accuracy

100 n in Purkinje neurons (PNs) is essential for cerebellar development and for motor learning and altere

101 at Wnt5a signaling is a crucial regulator of cerebellar development and would aid in better understan

102 trating that many key mechanisms controlling cerebellar development are likely conserved between mous

103 rious aspects of development but its role in cerebellar development remains elusive.

104 tern and functional significance of Wnt5a in cerebellar development using Wnt5a(-/-) and Nestin-Cre m

105 neural progenitor cell cycle progression in cerebellar development.

106 erm birth incorporates an increased risk for cerebellar developmental disorders likely contributing t

107 THGr(Ce) below the determined threshold and cerebellar diaschisis in 25 of 26 (96%) cases with THGr(

108 ysed cross-sectional data from 72 cases with cerebellar disease and 36 controls without cerebellar di

109 g the CaV2.1 subunit are associated with the cerebellar disease episodic ataxia type 2 (EA2).

110 ent and would aid in better understanding of cerebellar disease pathogenesis caused due to deregulati

111 h cerebellar disease and 36 controls without cerebellar disease.

112 sed to detect the involvement of DN in other cerebellar disorders.

113 eed-based method was applied to identify the cerebellar DNs resting-state network; first-level and hi

114 .2 vs 1.7+/-1.7, P<0.001), indicating slower cerebellar dysfunction indexes for FRDA than for SCA.

115 Cerebellar dysfunction, as measured with the CCFS and SA

116 tative measurements are required to evaluate cerebellar dysfunction.

117 errations, immunodeficiency, and progressive cerebellar dysfunction.

118 subjects and support the hypotheses that (i) cerebellar efferents target frontal lobe neurons involve

119 t our work using other methodologies showing cerebellar engagement in linguistic prediction and sugge

120 y skill acquisition would be proportional to cerebellar excitability (CBI) changes, whereas later sta

121 We examined whether the cortico-cerebellar executive function network is altered in chil

122 nces, in COMT genetic effects on the cortico-cerebellar executive function network.

123 We evaluated temporal cerebellar expression profiles by RNA sequencing of ATXN

124 l area of the GC of primary neurons from the cerebellar external granule layer of P2 mouse pups of bo

125 r function were associated with increases in cerebellar FCD in NM and thalamus FCD in HD.

126 The cerebellar floccular and parafloccular lobes are housed

127 causing subsequent disruptions in postnatal cerebellar foliation and lamination.

128 background (Atoh1(S193A/lacZ)) exhibit mild cerebellar foliation defects, motor impairments, partial

129 ABSTRACT: Purkinje cells are central to cerebellar function because they form the sole output of

130 support the idea of prediction as a unifying cerebellar function in motor and nonmotor domains.

131 A crucial issue in understanding cerebellar function is the interaction between simple sp

132 Theories of cerebellar function place the inferior olive to cerebell

133 Elucidating cerebellar function will require an understanding of the

134 erse than is assumed by standard theories of cerebellar function.

135 for rating cerebellar ataxias: the Composite Cerebellar Functional Severity (CCFS) Scale and Scale fo

136 nstructed a spatially realistic model of the cerebellar GCL and examined how GCL architecture contrib

137 early and progressive abnormal patterning of cerebellar gene expression.

138 d shelf and enhancer regions of striatal and cerebellar genes.

139 flux and enhanced migration, while in normal cerebellar granule (precursor) cells and MB cells not de

140 Combinatorial expansion by the cerebellar granule cell layer (GCL) is fundamental to th

141 rmal granule cells and the transformed human cerebellar granule cell line DAOY, OGR1 promoted express

142 ons that express high levels of Ptchd1 mRNA: cerebellar granule cell precursors and dentate granule c

143 ere we have shown that deletion of Chd7 from cerebellar granule cell progenitors (GCps) results in re

144 ordings and modeling of synaptic activity at cerebellar granule cell to Purkinje cell synapses of mic

145 By targeting Pten in cerebellar granule cells and activating the AKT1-mTOR pa

146 es to afferent mixing.SIGNIFICANCE STATEMENT Cerebellar granule cells are among the simplest neurons,

147 Cerebellar granule cells, which constitute half the brai

148 ave recently found that the proliferation of cerebellar granule neuron precursors is significantly re

149 required for the regulated proliferation of cerebellar granule neuron progenitors (CGNP) and for the

150 sslinking protein as an entry point into the cerebellar granule neuron system in combination with sup

151 ubiquitin-conjugating enzyme UBC13 in rodent cerebellar granule neurons robustly increases the number

152 bited L1-CAM-dependent neurite elongation in cerebellar granule neurons, a pathway previously shown t

153 scription of a set of long neuronal genes in cerebellar granule neurons.

154 All VOIs were referenced to a subsection of cerebellar gray matter (cere-crus) as well as a parametr

155 d at 50-60 min after injection, using either cerebellar gray matter (SUVRCB) or whole subcortical whi

156 Four RRs (cerebellar gray matter [CGM], whole cerebellum [WCER], p

157 n the white matter integrity of the pons and cerebellar gray matter volume associated with higher 'p

158 In all analyses, cerebellar gray matter was used as the reference region.

159 Using cerebellar gray reference, 18F-T807 data were expressed

160 ent meta-analysis aims to assess patterns of cerebellar grey matter atrophy in seven neurodegenerativ

161 hemisphere (THGr(Ce)) and the contralateral cerebellar hemisphere (THGr(Cb))-by its respective contr

162 ontrast, the role of the lateral cerebellum (cerebellar hemispheres and dentate nuclei, DN) is less w

163 e corroborated by significant improvement of cerebellar hypometabolism (statistical parametric mappin

164 We demonstrate that loss of Wnt5a results in cerebellar hypoplasia and depletion of GABAergic and glu

165 contribute to GCp proliferative defects and cerebellar hypoplasia in GCp-specific Chd7 mouse mutants

166 (GCps) results in reduced GCp proliferation, cerebellar hypoplasia, developmental delay, and motor de

167 mice and humans are usually associated with cerebellar hypoplasia.

168 ant with abnormal cortical lamination and no cerebellar hypoplasia.

169 otypes in the cortex and hippocampus without cerebellar hypoplasia.

170 locomotion, all three patients showed severe cerebellar hypoplasia.

171 s with motor cortex (M1) can be estimated as cerebellar inhibition (CBI): a conditioning pulse of tra

172 Infection, cerebellar injury and supratentorial injury are importan

173 The presence of cerebellar injury was consistently associated with reduc

174 of pattern separation we built models of the cerebellar input layer with spatially correlated input p

175 epending on neuronal activity changes at the cerebellar input stage.

176 ke-timing-dependent plasticity (STDP) at the cerebellar inputs stage.

177 dying this non-cortex-mediated basal ganglia-cerebellar interaction could radically change our perspe

178 motor aspects of language, and suggest that cerebellar internal models of linguistic stimuli support

179 Here, we evaluate the dynamics of cerebellar interpositus nucleus (IpN) neurons over the c

180 Cerebellar involvement in cognition, as well as in senso

181 t semantic prediction.SIGNIFICANCE STATEMENT Cerebellar involvement in language tasks and language ne

182 Investigation of cerebellar involvement provides further insight into the

183 on volume, metrics of cerebellar atrophy and cerebellar lesion volume) and motor/cognitive scores.

184 both motor and cognitive performances, with cerebellar lesion volume, cerebellar Lobules VI, Crus I

185 tate lesions without other focal cerebral or cerebellar lesions were detected at term equivalent age

186 sclerosis (MS), testing the contribution of cerebellar lobular atrophy to both motor and cognitive p

187 matter volume in the occipital lobe and left cerebellar lobule VIIb, which is functionally connected

188 Atrophy of specific cerebellar lobules explains different aspects of motor a

189 iled mapping of clinical dysfunctions to the cerebellar lobules in disease populations is necessary t

190 performances, with cerebellar lesion volume, cerebellar Lobules VI, Crus I and VIIIa atrophy being in

191 signals were widespread throughout multiple cerebellar lobules.

192 are activated by locomotor tasks, termed the cerebellar locomotor region.

193 ts indicate that learning-related changes in cerebellar-M1 connectivity reflect a somatotopy-specific

194 dy-Walker malformation (DWM), a common human cerebellar malformation.

195 Although null Zfp423 mutants develop cerebellar malformations, the underlying mechanism remai

196 oughout postnatal development and in primary cerebellar medulloblastoma tissues.

197 We explore the associations between altered cerebellar metabolite profiles and brain injury topograp

198 Regression analysis showed that cerebellar metrics accounted for extra variance in both

199 motor phenotype onset, and have progressive cerebellar molecular and neurophysiological (Purkinje ce

200 P2 sumoylation is required for regulation of cerebellar motor function and vocal communication, likel

201 Based on a well-supported theory of cerebellar motor function, which ascribes to the cerebel

202 Moreover, these mutant mice exhibited severe cerebellar motor learning deficits.

203 tside the context of learning, modulation of cerebellar-motor cortex connectivity is somatotopically

204 that adaptive learning with the hand affects cerebellar-motor cortex connectivity, not only for the t

205 PT infants had significantly lower cerebellar NAA (p < 0.025) and higher Cho (p < 0.001) at

206 th alcohol intake suggests a crucial role of cerebellar networks in the generation of symptoms in the

207 ores and the degree to which connectivity in cerebellar networks is "idiosyncratic" in an individual

208 ve processing that requires both frontal and cerebellar networks that are disrupted in patients with

209 ansmission in the amygdala, hippocampus, and cerebellar networks.

210 ripts for 3 genes previously associated with cerebellar neurodegeneration.

211 have clinical implications for diagnosis and cerebellar neuroimaging referencing approaches.

212 Further, right posterolateral cerebellar neuromodulation modifies behavior during pred

213 ar ADP-ribose levels and reduced the loss of cerebellar neurons and ataxia in Xrcc1-defective mice, i

214 report that numbers of cerebral cortical and cerebellar neurons are decreased and that cerebral corte

215 Mutant cerebellar neurons have large mitochondria, which exhibi

216 Brainstem and cerebellar neurons implement an internal model to accura

217 associated with a deficit in the ability of cerebellar neurons to form synapses and an increased num

218 of synaptic functions (VGLUT1 and GAD65) in cerebellar neurons.

219 lation of migration of cultured cortical and cerebellar neurons.

220 A dedicated cerebellar nuclear afferent comprised of feedback collat

221 KEY POINTS: Large premotor neurons of the cerebellar nuclei (CbN cells) integrate synaptic inhibit

222 ABSTRACT: Large projection neurons of the cerebellar nuclei (CbN cells), whose activity generates

223 ectly to large premotor neurons of the mouse cerebellar nuclei (CbN cells).

224 sion at synapses from Purkinje cells to deep cerebellar nuclei and at vestibular synapses in mice.

225 gically inhibiting the erratic output of the cerebellar nuclei in the mutant mice improved movement.

226 brain stimulation directed to the interposed cerebellar nuclei reduced dystonia-like postures in thes

227 the output structures of the cerebellum, the cerebellar nuclei, integrate their inputs and influence

228 both Purkinje cells and neurons of the deep cerebellar nuclei.

229 selectively stimulate neurons in the lateral cerebellar nucleus (LCN), a deep cerebellar nucleus that

230 the lateral cerebellar nucleus (LCN), a deep cerebellar nucleus that sends major excitatory output to

231 These findings advance understanding of cerebellar organization in health and sex chromosome ane

232 hods and results clarify the shifts in human cerebellar organization that accompany interwoven variat

233 kdown animals were associated with irregular cerebellar output caused by changes in the intrinsic act

234 nt neurophysiological changes: modulation of cerebellar output mediated in-part by long-term depressi

235 ive properties of subcortical neurons at the cerebellar output stage mediating conditioned behavior.

236 tic excitation from mossy fibres to generate cerebellar output.

237 It cannot be excluded, however, that extra-cerebellar pathology contributed to the present findings

238 e calculated for DN-to-pons and DN-to-middle cerebellar peduncle (MCP) ratios in a region-of-interest

239 obus pallidus (GP) in relation to the middle cerebellar peduncle (MCP), pons, and thalamus after repe

240 cleus (n = 10), colliculi (n = 10), superior cerebellar peduncle (n = 7), caudate nucleus (n = 4), wh

241 ior thalamus, substantia nigra, red nucleus, cerebellar peduncle, colliculi, dentate nucleus, and glo

242 r) field traversed by fibers of the superior cerebellar peduncle.

243 ar vermis hypoplasia with elongated superior cerebellar peduncles (mild "molar tooth sign"), typical

244 mon paediatric brain tumour that arises from cerebellar precursor cells.

245 Thus, the cortico-cerebellar processes tied to the clinical course of ADHD

246 0 suggests that this pathway may function in cerebellar processing into adulthood.

247 medial frontal brain activity by stimulating cerebellar projections.

248 ions of a biophysically realistic model of a cerebellar Purkinje cell in a pattern recognition task s

249 KEY POINTS: Cerebellar Purkinje cells (PCs) generate two types of ac

250 matic representations through the cerebellum.Cerebellar Purkinje cells (PCs) linearly encode whisker

251 nts, including decreased firing frequency of cerebellar Purkinje cells and a decline in motor functio

252 urons that send climbing fibers to innervate cerebellar Purkinje cells for the control of motor learn

253 he limbic forebrain, the locus coeruleus and cerebellar Purkinje cells, or for CRF2 in any aspect of

254 axia, increased cholesterol storage, loss of cerebellar Purkinje neurons and early lethality.

255 In cerebellar Purkinje neurons from postnatal day 30 Snord1

256 uses, and persists until postnatal day 30 in cerebellar Purkinje neurons.

257 fined as 11C-Pittsburgh compound B target-to-cerebellar ratio above 1.5 within a composite cortical v

258 This cerebellar region also responded to prediction error dur

259 duced variability compared with conventional cerebellar regions in amyloid imaging.

260 pital cortex and the peculiar aggregation of cerebellar regions to form a closed core.

261 ted activations in dorsal frontoparietal and cerebellar regions, and task-related deactivations in de

262 frontal and inferior parietal/precuneus) and cerebellar regions.

263 rebrocortical morphology and effect sizes of cerebellar relative to cerebral morphological difference

264 PD patients have limited cerebellar resources that are already utilized for singl

265 he Montreal Cognitive Assessment) and normal cerebellar, sensory, cranial nerve, and autonomic functi

266 In FRDA, 31% of the variance of cerebellar signs with the CCFS and 41% of that with SARA

267 Dynamic clamp studies in cerebellar slices from weanling mice demonstrate that sy

268 n cultured mouse granule neurons and ex vivo cerebellar slices indicate that ZNHIT3 is indispensable

269 vely by regional and voxel-based cortical to cerebellar standard uptake value ratios.

270 ps, indicating that the brainstem and medial cerebellar structures were functionally spared.

271 more valid and accurate resolution of which cerebellar subcomponents are sensitive to sex and sex ch

272 ay represent the structural correlate to the cerebellar symptoms observed in these patients.

273 role for the AMPA receptor subunit GluA3 in cerebellar synaptic plasticity and motor learning in mic

274 ement, and brain size.SIGNIFICANCE STATEMENT Cerebellar systems are implicated in diverse domains of

275 ss the behavioral and neural consequences of cerebellar tDCS during a sentence completion task.

276 ntal cortex and left hippocampus, along with cerebellar, temporal and parietal regions were more subs

277 ine connectivity was correlated with recall, cerebellar-thalamic baseline connectivity was correlated

278 s about the involvement of the basal ganglia-cerebellar-thalamo-cortical system in tic generation; (i

279 d TRPC4 are prominently expressed in healthy cerebellar tissue throughout postnatal development and i

280 d genes, with more pronounced alterations in cerebellar tissue.

281 The strength of cerebellar-to-cerebral pathways for a given muscle may r

282 Cerebellar total volumes of distribution were higher in

283 predominantly parkinsonism [MSA-P], n = 9), cerebellar type MSA (MSA-C, n = 7), PSP (n = 13), and PD

284 other causes being alcohol excess (12%) and cerebellar variant of multiple system atrophy (11%).

285 y delayed and DDR markers are upregulated in cerebellar ventricular zone progenitors.

286 We found that the right cerebellar vermis and left lobule V of cerebellar anteri

287 In addition, the right cerebellar vermis had enhanced connectivity with motor a

288 with Joubert Syndrome, a ciliopathy causing cerebellar vermis hypoplasia and ataxia.

289 The children presented congenital ataxia and cerebellar vermis hypoplasia with elongated superior cer

290 f-generated head movements in the rat caudal cerebellar vermis, an area essential for graviceptive fu

291 sm spectrum disorder diagnosis and increased cerebellar volume (p = .049, uncorrected), but the analy

292 gated scaling relationships between regional cerebellar volume and brain size in humans, which (1) ar

293 rther observed positive correlations between cerebellar volume and cerebral cortical thickness in fro

294 sed candidate endophenotypes, differences in cerebellar volume have been often reported as statistica

295 We then analyzed the cerebellar volume of 328 patients and 353 control subjec

296 , age, sex, and IQ were important sources of cerebellar volume variability, although independent of a

297 Cerebellar volumes were automatically obtained using the

298 To investigate global and lobular cerebellar volumetries in patients with progressive mult

299 tocol describes a novel and rapid method for cerebellar window construction that can be set up in und

300 Here, we describe a cerebellar window that provides access to the mouse cere