ライフサイエンスコーパス: cerebellar cortex

1 ion because they form the sole output of the cerebellar cortex.

2 ral tasks is in part computed locally in the cerebellar cortex.

3 e spontaneous firing rate of GoCs in the rat cerebellar cortex.

4 ate and sustain oscillatory processes in the cerebellar cortex.

5 als to generate climbing fibre inputs to the cerebellar cortex.

6 ons: Brodmann area 19 (occipital cortex) and cerebellar cortex.

7 somatosensory cortices, anterior insula, and cerebellar cortex.

8 e of the most extensively studied regions of cerebellar cortex.

9 ule cells and mapped this convergence across cerebellar cortex.

10 h deficient Ca(2)(+) channel function in the cerebellar cortex.

11 PCs), which represent the sole output of the cerebellar cortex.

12 cy MF signals through the input layer of the cerebellar cortex.

13 evises the inhibitory circuit diagram of the cerebellar cortex.

14 the source for all motor coordination in the cerebellar cortex.

15 transmission through the input layer of the cerebellar cortex.

16 Purkinje cells (PC), the sole output of the cerebellar cortex.

17 types in OPCs from rat optic nerve and mouse cerebellar cortex.

18 between Golgi and granule cells in the mouse cerebellar cortex.

19 paired dendritic claw differentiation in the cerebellar cortex.

20 d protein was significantly increased in the cerebellar cortex.

21 xcessive migration of granule neurons in the cerebellar cortex.

22 s a substantial disynaptic projection to the cerebellar cortex.

23 ant VGLUT in the neocortex, hippocampus, and cerebellar cortex.

24 n granule neuron parallel fiber axons in the cerebellar cortex.

25 n two tangential but orthogonal paths in the cerebellar cortex.

26 oscillations, requiring amplification by the cerebellar cortex.

27 kably selective for the IN compared with the cerebellar cortex.

28 a ubiquitous neurotransmitter throughout the cerebellar cortex.

29 ant VGLUT in the neocortex, hippocampus, and cerebellar cortex.

30 ontact in primary granule neurons of the rat cerebellar cortex.

31 activation of the left prefrontal and right cerebellar cortex.

32 postmitotic mammalian neurons and in the rat cerebellar cortex.

33 ired long-term depression (LTD) induction in cerebellar cortex.

34 n at both the input and output layers of the cerebellar cortex.

35 nt stimulation (tDCS) applied over the right cerebellar cortex.

36 ion of granule neuron dendrite arbors in the cerebellar cortex.

37 ated by plastic changes occurring within the cerebellar cortex.

38 y mossy fibers, thereby gating inputs to the cerebellar cortex.

39 cells (PCs) provide the sole output from the cerebellar cortex.

40 rimary granule neurons of the developing rat cerebellar cortex.

41 anule neurons in dissociated cultures and in cerebellar cortex.

42 naptic granule neuron dendritic claws in the cerebellar cortex.

43 ant VGLUT in the neocortex, hippocampus, and cerebellar cortex.

44 ion of granule neuron dendritic claws in the cerebellar cortex.

45 suggest a novel form of neural coding in the cerebellar cortex.

46 nd external granular layer of the developing cerebellar cortex.

47 naptic granule neuron dendritic claws in the cerebellar cortex.

48 lasticity in target nuclei downstream of the cerebellar cortex.

49 with GABA(A) receptor alpha1 subunits in the cerebellar cortex.

50 ent of granule neuron parallel fibers in the cerebellar cortex.

51 ctures throughout the molecular layer of the cerebellar cortex.

52 th Purkinje cells and granule neurons of the cerebellar cortex.

53 -coded information to the input layer of the cerebellar cortex.

54 ections limited to specific sublayers of the cerebellar cortex.

55 nduction time between the inferior olive and cerebellar cortex.

56 n the C1 zone in the copula pyramidis of the cerebellar cortex.

57 rm cortex, hippocampus, caudate/putamen, and cerebellar cortex.

58 nt not only in the neocortex but also in the cerebellar cortex.

59 pmental control of neuronal migration in the cerebellar cortex.

60 um and their role in the histogenesis of the cerebellar cortex.

61 thway from the cerebellar nuclei back to the cerebellar cortex.

62 als to generate climbing fibre inputs to the cerebellar cortex.

63 to forward signals to climbing fibers in the cerebellar cortex.

64 lele of rs10937625 and reduced expression in cerebellar cortex.

65 stinct areas of the midbrain, brainstem, and cerebellar cortex.

66 laterals and their target neurons within the cerebellar cortex.

67 ssy fibers relaying vestibular inputs to the cerebellar cortex.

68 ion because they form the sole output of the cerebellar cortex.

69 f GABAergic inhibitory synapses in the mouse cerebellar cortex.

70 pment, showing some layer specificity in the cerebellar cortex.

71 erlying communication between modules in the cerebellar cortex.

72 urkinje cells as the major output neurons of cerebellar cortex.

73 y control over the integrated outflow of the cerebellar cortex.

74 branches terminating as mossy fibers in the cerebellar cortex.

75 inje cells, or for CRF2 in any aspect of the cerebellar cortex.

76 sors and subsequent massive apoptosis of the cerebellar cortex.

77 ating the flow of sensory information in the cerebellar cortex.

78 sequently, to the processing of input to the cerebellar cortex.

79 at the terminals of basket cell axons in the cerebellar cortex.

80 lators of presynaptic differentiation in the cerebellar cortex.

81 thin both the deep cerebellar nuclei and the cerebellar cortex.

82 the adult cortex, hippocampus, thalamus, and cerebellar cortex.

83 resent time intervals in a robust way in the cerebellar cortex.

84 d negative stripes of PCs across most of the cerebellar cortex.

85 spiny inhibitory neurons of neostriatum and cerebellar cortex.

86 itory input from Purkinje cells (PCs) of the cerebellar cortex.

87 f instruction, and last in sites outside the cerebellar cortex.

88 ceiving" parts of the C1 and C3 zones in the cerebellar cortex.

89 GBC was found in the right putamen and left cerebellar cortex.

90 rebral cortex and as diffuse deposits in the cerebellar cortex, a similar array of amino-terminally t

91 At the input layer of the cerebellar cortex, a single type of interneuron, the Gol

92 eyelid conditioning, where disconnecting the cerebellar cortex abolishes one component of learning, r

93 In the cerebellar cortex, activation of CB1R inhibits excitator

94 rkinje cells, the sole output neurons of the cerebellar cortex, also directly inhibit granule cells v

95 In the cerebellar cortex, anatomical and functional evidence in

96 The important roles of both cerebellar cortex and AIP nucleus in eyeblink conditioni

97 e GABAergic input from Purkinje cells of the cerebellar cortex and are thought to contribute to the a

98 sensorimotor cortex, thalamus, contralateral cerebellar cortex and deep cerebellar nuclei (FDR q < 0.

99 It contained components simulating cerebellar cortex and deep cerebellar nuclei, and it rec

100 Experiments focusing on the role of cerebellar cortex and deep nuclei in delay versus trace

101 rontal cortex appeared, as well as the right cerebellar cortex and deep nuclei.

102 (UBCs) are glutamatergic interneurons in the cerebellar cortex and dorsal cochlear nucleus.

103 Purkinje cells are the sole output of the cerebellar cortex and fire two distinct types of action

104 S: Purkinje cells are the sole output of the cerebellar cortex and fire two distinct types of action

105 Purkinje neurons are the output cells of the cerebellar cortex and generate spikes in two distinct mo

106 g fiber conveys an all-or-none signal to the cerebellar cortex and help to link learning and timing t

107 , ventral midbrain, frontal cerebral cortex, cerebellar cortex and hippocampus in Ube3a deficient and

108 rning-related activation in the left lateral cerebellar cortex and in the right premotor and inferior

109 pse as an important target of ethanol in the cerebellar cortex and indicate that ethanol significantl

110 current pharmacological disconnection of the cerebellar cortex and intense sensory stimulation in the

111 nal magnetic resonance imaging (fMRI) of the cerebellar cortex and interposed cerebellar nuclei simul

112 egulate numerous circuit elements within the cerebellar cortex and is well suited to contribute to pr

113 tion available about the organization of the cerebellar cortex and its synaptic inputs, relatively li

114 mpact on the flow of information through the cerebellar cortex and may contribute to the mechanism by

115 fMRI signals increased concomitantly in the cerebellar cortex and nuclei during early acquisition of

116 malization methods to assess function of the cerebellar cortex and nuclei during simple hand movement

117 urther investigate the relative roles of the cerebellar cortex and nuclei in eyeblink conditioning, a

118 the Purkinje cells; reduced the size of the cerebellar cortex and nuclei; produced defects in the pa

119 e and Golgi cells, is the first stage of the cerebellar cortex and processes spatiotemporal informati

120 mate the total number of Purkinje neurons in cerebellar cortex and pyramidal neurons in the hippocamp

121 synaptic dendritic claw morphogenesis in the cerebellar cortex and suggest novel functions for SUMO E

122 to control the flow of information into the cerebellar cortex and understanding their responses duri

123 The model, comprising both the cerebellar cortex and vestibular nuclei, reproduces beha

124 tory synapses on Purkinje cells (PCs) in the cerebellar cortex, and long-term potentiation and depres

125 n the cerebral cortex, Purkinje cells in the cerebellar cortex, and motor neurons of the somatic moto

126 e neurons in vitro and in the context of the cerebellar cortex, and overcome the myelin inhibitory si

127 rkinje cells, the only output neurons of the cerebellar cortex, and their postsynaptic target neurons

128 tal, cingulate, insular, temporoparietal and cerebellar cortex, and with a more localized increase in

129 Although the wiring of the cerebellar cortex appears to be uniform, the neurons in

130 Instead, upstream interneurons within the cerebellar cortex are also characterized by similar prop

131 creasingly posterior lobules of the anterior cerebellar cortex are associated with increasingly compl

132 The neuronal circuits of the cerebellar cortex are essential for motor and sensory le

133 ons (MLIs, stellate and basket cells) of the cerebellar cortex are linked together by chemical and el

134 intraparietal sulcus, and posterior superior cerebellar cortex are modulated by atypically high phasi

135 Different patterns of atrophy of the cerebellar cortex are well known.

136 BPND and SUVR were calculated using the cerebellar cortex as a reference region and were compare

137 rdized uptake value ratios (SUVRs) using the cerebellar cortex as a reference region were calculated

138 -SSP values were computed using the pons and cerebellar cortex as reference regions.

139 With the cerebellar cortex as the reference region, the optimal z

140 model estimating DVR (DVR [MRTM]) using the cerebellar cortex as the reference tissue.

141 s widely available at the input stage of the cerebellar cortex, as required by forward models of cere

142 e relevant neural circuits downstream of the cerebellar cortex, as well as the timing requirements of

143 Picrotoxin or muscimol was applied to the cerebellar cortex at the borders of the recording array.

144 modeling with plasma- and reference-region (cerebellar cortex)-based methods was performed.

145 e results suggest that functional input from cerebellar cortex becomes increasingly important for the

146 ing with impaired dendrite patterning in the cerebellar cortex, behavioral analyses reveal that TRPC5

147 type 6, pathology was not restricted to the cerebellar cortex but also involved the cerebellar nucle

148 tribute to the functional recruitment of the cerebellar cortex by decreasing Golgi cell inhibition on

149 stonia via pharmacological excitation of the cerebellar cortex by local application of kainic acid in

150 serve these roles at the input layer of the cerebellar cortex by releasing GABA to inhibit granule c

151 The cerebellar cortex can be used as a reference region for

152 rkinje cells, the sole output neurons of the cerebellar cortex, can also drive motor learning in mice

153 ward inhibition, such as that present in the cerebellar cortex, can contribute to temporal coding.

154 e projections from the inferior olive to the cerebellar cortex, carry sensorimotor error and clock si

155 Neurons in the cerebellar cortex, cerebellar nuclei, and inferior olive

156 ke discharge reflects the main output of the cerebellar cortex, changes in simple spike firing likely

157 Golgi cells are important elements of the cerebellar cortex, controlling the flow of mossy fibre i

158 ys multiple modalities of information to the cerebellar cortex, converging on Purkinje cells (PC), th

159 The cerebellar cortex coordinates movements and maintains ba

160 The development of the cerebellar cortex depends on intrinsic genetic programs

161 There was functional involvement of the cerebellar cortex despite no or little structural change

162 Purkinje cells, the sole output cells of the cerebellar cortex, differs between cerebellar modules an

163 NSCs, when transplanted into the newborn nr cerebellar cortex, do not replace host PNs but contact i

164 show activation of multiple areas within the cerebellar cortex during both perception and motor perfo

165 Recordings from the cerebellar cortex during conditioning have revealed CS-t

166 The siblings presented cerebellar cortex dysplasia characterized by the presenc

167 r architecture is that Purkinje cells in the cerebellar cortex each receive input from a single climb

168 muscle activity, to the hypothesis that the cerebellar cortex embodies complex internal models of li

169 Purkinje cells, the sole output of the cerebellar cortex, encode the timing signals required fo

170 d with the Bergmann glia of the adult murine cerebellar cortex, expresses the stem cell markers Sox2

171 tively destroy Purkinje cells throughout the cerebellar cortex following excitatory conditioning.

172 d by parallel fibre stimulation in slices of cerebellar cortex from P18-25 rats.

173 ns across models were parieto-temporal lobe, cerebellar cortex, frontal lobe, hypothalamus and striat

174 ficits of AS mice do not arise from impaired cerebellar cortex function.

175 Within the cerebellar cortex, functional magnetic resonance imaging

176 es expressed by glutamatergic neurons in the cerebellar cortex (GAP-43, BDNF, and GABA OLE_LINK2>(A)-

177 absence of any overt external stimulus, the cerebellar cortex generates a slow oscillation that is c

178 The well established anatomy of the cerebellar cortex has led to suggestions that cerebellar

179 Purkinje neurons of the cerebellar cortex have been shown to be particularly imp

180 s complete and the major output cells of the cerebellar cortex have been specified.

181 g with the pruning of climbing fibres in the cerebellar cortex, implicates the climbing fibre collate

182 RPC5 regulates dendrite morphogenesis in the cerebellar cortex in a cell-autonomous manner.

183 formation (HF) and, to a lesser degree, the cerebellar cortex in AD cases without diabetes exhibit m

184 bies virus (RV) into selected regions of the cerebellar cortex in cebus monkeys and used retrograde t

185 der Purkinje cells were found in ipsilateral cerebellar cortex in cerebellar lobule HVI and in lobule

186 cating a more fundamental involvement of the cerebellar cortex in CR generation.

187 The role of the cerebellar cortex in eyeblink classical conditioning rem

188 e evidence exists concerning the role of the cerebellar cortex in eyeblink conditioned inhibition.

189 cific role of deep cerebellar nuclei and the cerebellar cortex in eyeblink conditioning are not well

190 This study examined the role of cerebellar cortex in eyeblink conditioning under conditi

191 s (9 mm) suppresses transmission through the cerebellar cortex in low, but not high, alcohol consumin

192 he pathway from the cerebellar nuclei to the cerebellar cortex in mice includes collaterals of cerebe

193 only hypothesized as a major function of the cerebellar cortex in motor control.

194 l support for the necessary participation of cerebellar cortex in normal acquisition of delay eyeblin

195 d raise questions about the role, if any, of cerebellar cortex in trace eyeblink conditioning.

196 n primary neurons and importantly in the rat cerebellar cortex in vivo robustly increases the density

197 ursts can be successfully transmitted to the cerebellar cortex in vivo, having a significant impact o

198 bellar granule neurons, including in the rat cerebellar cortex in vivo, reveals a requirement for the

199 utofluorescence optical imaging in the mouse cerebellar cortex in vivo, this study demonstrates that

200 ging and single-cell recordings in the mouse cerebellar cortex in vivo, this study reexamines the bea

201 alyses in primary rat neurons and in the rat cerebellar cortex in vivo, we report that CaMKIIbeta ope

202 l fiber/Purkinje cell synapses in the rodent cerebellar cortex in vivo.

203 airs migration of granule neurons in the rat cerebellar cortex in vivo.

204 anule neuron parallel fiber axons in the rat cerebellar cortex in vivo.

205 raction-like mechanism of temporal coding in cerebellar cortex in which activity in a subset of granu

206 cells engage mechanisms of plasticity in the cerebellar cortex; in turn, changes in the cerebellum de

207 eatures in the molecular layer of the mutant cerebellar cortex, including the presence of desmosoid p

208 re >/=30% in the deep cerebellar nuclei, the cerebellar cortex, inferior olive, and thalamus relative

209 Large lesions of the midline cerebellar cortex involving the OMV cause saccades to be

210 a new method for stereologic sampling of the cerebellar cortex, involving calculating the volume of t

211 The cerebellar cortex is among the brain's most well-studied

212 cerebellum; activation of this system in the cerebellar cortex is associated with deficits in motor c

213 The cerebellar cortex is centrally involved in motor coordin

214 While research on the cerebellar cortex is crystallizing our understanding of

215 f molecular layer interneurons (MLIs) of the cerebellar cortex is enhanced.

216 At birth, each Purkinje cell (PC) in the cerebellar cortex is innervated by multiple CFs; an acti

217 eractions among PCs, which suggests that the cerebellar cortex is more functionally diverse than is a

218 Regardless of cause, the cerebellar cortex is often a target in ataxia.

219 The molecular layer of the cerebellar cortex is populated by glial progenitors that

220 An unusual feature of the cerebellar cortex is that its output neurons, Purkinje c

221 The climbing fiber input to the cerebellar cortex is thought to provide instructive sign

222 s), suggesting that the main function of the cerebellar cortex is to shape the timing of CRs.

223 Due to the uniform cyto-architecture of the cerebellar cortex, its overall physiological characteris

224 In cerebellar cortex, Kv3.3 expression was found in Purkinj

225 By using 2-photon microscopy in rodent cerebellar cortex labeled with fluorescent indicator dye

226 e, we provide evidence that SLRs unmasked by cerebellar cortex lesions are mediated by an associative

227 m inhibitory interneurons located within the cerebellar cortex limits the extent of neuronal excitati

228 EMENT Purkinje cells, the sole output of the cerebellar cortex, manifest two fundamentally different

229 It has also been suggested that the cerebellar cortex may be important during early stages o

230 Such concerted changes in the circuitry of cerebellar cortex may contribute to certain forms of sen

231 fibres projecting to different parts of the cerebellar cortex may have differences in spike conducti

232 xpression of SLRs depends on both direct and cerebellar cortex-mediated sensory information from the

233 Thus, how a disruption in cerebellar cortex might lead to ataxia is of considerabl

234 ys required for the normal maturation of the cerebellar cortex, notably developmental pathways for gr

235 eta subunit (CTb) into selected areas of the cerebellar cortex of 18 male Wistar rats.

236 osterior cingulate cerebral cortices and the cerebellar cortex of 87 end-of-life patients (64 with AD

237 G methylation in DS and control cerebral and cerebellar cortex of adults and cerebrum of fetuses.

238 Neurophysiological recordings in the cerebellar cortex of awake-behaving animals are revoluti

239 ported by our finding that infusion into the cerebellar cortex of either the Kv1.2 inhibitor tityusto

240 he expression of STK32B was increased in the cerebellar cortex of patients and expression quantitativ

241 tional state of glutamatergic neurons in the cerebellar cortex of patients with schizophrenia.

242 We injected these vectors into the cerebellar cortex of rhesus macaques and tested vector e

243 and immature morphology specifically in the cerebellar cortex of stg mice.

244 number of Purkinje and granule cells in the cerebellar cortex of the three strains.

245 ior olive or reduce the disinhibition of the cerebellar cortex on the deep cerebellar nuclei could tr

246 ions show that reducing the influence of the cerebellar cortex on the oculomotor pathway reduces the

247 Inactivation of either the HVI region of the cerebellar cortex or the cerebellar interposed nuclei (I

248 ate how the inhibitory microcircuitry of the cerebellar cortex orchestrates synaptic integration and

249 ues were significantly over-expressed in the cerebellar cortex (P = 1x10(-)(5)) suggesting possible i

250 In the cerebellar cortex, parallel fiber-to-stellate cell (PF-S

251 has been proposed as a basic function of the cerebellar cortex (particularly the climbing fiber affer

252 In summary, cells in this region of lateral cerebellar cortex perform simple visual functions, such

253 ule cells, which form the input layer of the cerebellar cortex, permit high-resolution patch-clamp re

254 re under afferent control independent of the cerebellar cortex, potentially diversifying its roles in

255 indings suggest that the subdivisions of the cerebellar cortex produced by folding may create functio

256 mpus (CA1), cerebral cortex (layer III), and cerebellar cortex (Purkinje spines), respectively.

257 ciated with deficient CB1R signalling in the cerebellar cortex, putatively linked with compromised Ca

258 The PiB uptake was quantified as a region to cerebellar cortex ratio.

259 dorsal cochlear nucleus (DCN) and vestibular cerebellar cortex receive glutamatergic mossy fiber inpu

260 reater decreases in the occipital cortex and cerebellar cortex, respectively.

261 ns, bidirectional tracer injections into the cerebellar cortex retrogradely labeled somata in the cer

262 Histological analysis of the TR4(-/-) cerebellar cortex reveals reductions in granule cell den

263 inferior olive and multiple areas within the cerebellar cortex showed a robust response to time chang

264 cerebral cortex, hippocampus, striatum, and cerebellar cortex, showed that NHERF-2 was expressed mai

265 ositive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR)

266 y fiber axons, whereas Purkinje cells of the cerebellar cortex strongly expressed Kv3.3 subunits in a

267 was correlated with responses in ipsilateral cerebellar cortex, suggesting a novel computational role

268 tiple ischaemic-like lesions occurred in the cerebellar cortex suggestive of vascular smooth muscle c

269 Stereotyped circuitry within the cerebellar cortex suggests that similar computations are

270 ral morphology of the molecular layer of the cerebellar cortex that are the result of the binge ethan

271 ngs in vivo, and are the only neurons in the cerebellar cortex that express the alpha6delta-containin

272 erate on-beam and off-beam inhibition in the cerebellar cortex that is hypothesized to control the ti

273 ations in the interpositus nucleus (IPN) and cerebellar cortex that were time-locked both to hippocam

274 -the primary source of synaptic input to the cerebellar cortex-that sensory stimulation can produce b

275 In cerebellar cortex the GluR1 AMPA receptor subunit is exp

276 In the cerebellar cortex the processes of Bergmann glia cells e

277 k responses primarily takes place within the cerebellar cortex, the interposed nuclei, or both.

278 In the cerebellar cortex, the labeled fibers were found mostly

279 the sensory mossy fiber input to the IN and cerebellar cortex, then blocking the MCP should abolish

280 their axons are the only projection from the cerebellar cortex to deeper cerebellar structures.

281 The ability of cerebellar cortex to generate population rhythms within

282 dy to demonstrate beam-like responses in the cerebellar cortex to peripheral, MF, and GC stimulation

283 derived from postnatal subependymal zone or cerebellar cortex undergo a phenotypic transformation in

284 eurons regulate plasticity and timing in the cerebellar cortex via the climbing fiber pathway, but di

285 The alpha3-immunoreactivity in the cerebellar cortex was relatively weak, but it was abunda

286 Using slices of cerebellar cortex, we explored how synaptic plasticity a

287 ng fibres mediate synaptic plasticity in the cerebellar cortex, we have examined the effects of conju

288 multiple molecular layer interneurons in the cerebellar cortex, we reveal specific, nonrandom connect

289 to the red nucleus also collateralize to the cerebellar cortex, we used a Cre-dependent viral approac

290 onstrated that the laminations of the TR4-/- cerebellar cortex were changed, including reductions in

291 tory bulb, hippocampus, cerebral cortex, and cerebellar cortex were exceptionally rich in selenoprote

292 which provide the eye-related output of the cerebellar cortex, were found to increase or decrease th

293 ception to this targeting pattern was in the cerebellar cortex, where PMCA2a also concentrates postsy

294 These neurons form the 'input layer' of the cerebellar cortex, where sensorimotor information carrie

295 ellar nuclei (CN) project profusely into the cerebellar cortex, where they make synaptic contacts on

296 are among the first neurons to populate the cerebellar cortex, where they sprout exuberant axon coll

297 rebellar nuclear connections that can bypass cerebellar cortex, whereas a cerebellar cortical long-te

298 placeable binding was largely reduced in the cerebellar cortex, which in mice was spatially indisting

299 ve identified a pathway linking the vlPAG to cerebellar cortex, which terminates as climbing fibres i

300 s pharmacologically disrupted input from the cerebellar cortex while training with an interstimulus i