1 h cerebellar disease and 36 controls without cerebellar disease.

2 ad range of cognitive and motor phenomena in cerebellar disease.

3 nd offer a possible model of immune-mediated cerebellar disease.

4 tric morbidity of patients with degenerative cerebellar diseases.

5           Despite the absence of symptomatic cerebellar disease, a decrease in the number of Purkinje

6 ysed cross-sectional data from 72 cases with cerebellar disease and 36 controls without cerebellar di

7 probe the integrity of specific functions in cerebellar disease, and new imaging methods to quantitat

8 ion of VZ progenitors in the pathogenesis of cerebellar diseases associated with deregulated Shh sign

9 ntention tremor plus other clinical signs of cerebellar disease (cerebellar tremor).

10 ed a series of 31 patients with degenerative cerebellar diseases, compared with 21 patients with Hunt

11 g the CaV2.1 subunit are associated with the cerebellar disease episodic ataxia type 2 (EA2).

12  all mood disorders in both the degenerative cerebellar diseases group (68%) and the Huntington's dis

13 se group (43%); the rate in the degenerative cerebellar diseases group was significantly higher than

14 any, if not most, patients with degenerative cerebellar diseases may benefit from psychiatric interve

15 rs was 77% in the patients with degenerative cerebellar diseases, nearly identical to that in the pat

16 ent and would aid in better understanding of cerebellar disease pathogenesis caused due to deregulati

17 hange was present in 26% of the degenerative cerebellar diseases patients, 48% of the Huntington's di

18           A total of 19% of the degenerative cerebellar diseases subjects and 71% of the Huntington's

19  disorders in the patients with degenerative cerebellar diseases suggests that many, if not most, pat