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1 tative measurements are required to evaluate cerebellar dysfunction.
2 e display behavioral defects consistent with cerebellar dysfunction.
3 errations, immunodeficiency, and progressive cerebellar dysfunction.
4 cts had ataxia or other detected evidence of cerebellar dysfunction.
5 g of the behavioral deficits associated with cerebellar dysfunction.
6 type attributable to motor hyperactivity and cerebellar dysfunction.
7 ponses in the IN were suppressed, suggesting cerebellar dysfunction.
8 emor in limbs when immobile, consistent with cerebellar dysfunction.
9 , 9 of 16 positive control patients also had cerebellar dysfunction.
10 paired balance, which may be associated with cerebellar dysfunction.
11 companied by an intention tremor, typical of cerebellar dysfunction.
12 bit deficits in motor learning indicative of cerebellar dysfunction.
13 ese difficulties may be attributable to mild cerebellar dysfunction.
14 d between cerebellar K1 and the intensity of cerebellar dysfunction.
15 ss, to more pronounced signs and symptoms of cerebellar dysfunction.
16 yeblink conditioning was used as a marker of cerebellar dysfunction.
17 levels, completely 'normalizing' the fronto-cerebellar dysfunctions.
18 sorders characterized by clinically variable cerebellar dysfunction and accompanied by involvement of
20 ta-III spectrin function appears to underpin cerebellar dysfunction and degeneration in both diseases
21 ercent], P=0.007), even after adjustment for cerebellar dysfunction and for stratification variables
22 Here, we investigated PS1-E280A-associated cerebellar dysfunction and found that it occurs early in
23 taxia type 1 presents with brief episodes of cerebellar dysfunction and persistent neuromyotonia and
24 disorder characterised by brief paroxysms of cerebellar dysfunction and persistent neuromyotonia.
25 anges in DISC1 and unc5H3 may be relevant to cerebellar dysfunction and schizophrenia respectively, i
27 n, cognitive impairment, pyramidal tract and cerebellar dysfunction, and white matter loss in the cen
29 Acquired forms are often associated with cerebellar dysfunction arising due to instability of the
30 ant potential implications for patients with cerebellar dysfunction as well as to motor control studi
32 mature pattern resembles that of people with cerebellar dysfunction, because we know that this adapta
42 ic exercise ameliorates, at least partially, cerebellar dysfunction in the sHW rat, an excellent mode
43 ies have investigated the pathophysiology of cerebellar dysfunction in this neuroinflammatory disorde
44 .2 vs 1.7+/-1.7, P<0.001), indicating slower cerebellar dysfunction indexes for FRDA than for SCA.
45 arked psychomotor retardation, and prominent cerebellar dysfunction manifested by nystagmus, intentio
48 res (MSA-P), 8 MSA patients with principally cerebellar dysfunction (MSA-C), and 6 sporadic olivopont
49 taxia score (SARA, a standardized measure of cerebellar dysfunction on clinical examination, scores r
50 nts (26%) reported episodes of brainstem and cerebellar dysfunction or persistent vertigo several mon
51 Posterior fossa syndrome is characterized by cerebellar dysfunction, oromotor/oculomotor apraxia, emo
52 Although, in EA2 the emphasis has been on cerebellar dysfunction, patients also exhibit episodic,
53 tion, and their absence leads to PC loss and cerebellar dysfunction similar to that observed in AT pa
54 a clinical syndrome dominated by progressive cerebellar dysfunction that might elude standard diagnos
55 Consistent with this behavioral evidence of cerebellar dysfunction, there is increased death of gran
56 rological mutants, detected initially due to cerebellar dysfunction, was identified as models for epi
57 ice expressing mutant beta-III spectrin have cerebellar dysfunction with altered mGluR1alpha localiza
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