ライフサイエンスコーパス: cerebellar granule cell

1 s of [Ca(2+)](i) than other cells, including cerebellar granule cells.

2 inst Tat-induced apoptosis in PC12 cells and cerebellar granule cells.

3 ay an inductive role in the embryogenesis of cerebellar granule cells.

4 , alpha6beta3delta, expressed exclusively in cerebellar granule cells.

5 for the slower decay of spontaneous IPSCs in cerebellar granule cells.

6 nd mature electrophysiological properties of cerebellar granule cells.

7 pmental speeding of miniature EPSCs in mouse cerebellar granule cells.

8 equired for proper migration and survival of cerebellar granule cells.

9 ABAergic synaptic activity in cultured mouse cerebellar granule cells.

10 This hypothesis was also tested in cultured cerebellar granule cells.

11 d stage-specific effects on the migration of cerebellar granule cells.

12 edgehog promotes proliferation of developing cerebellar granule cells.

13 -CAM have overlapping expression patterns in cerebellar granule cells.

14 f tetrodotoxin on ATX-induced Ca2+ influx in cerebellar granule cells.

15 nce of function of adult sensory neurons and cerebellar granule cells.

16 osely the time course of the native I(SA) in cerebellar granule cells.

17 5-methyl-4-isoxazolepropionate) receptors on cerebellar granule cells.

18 overexpress wild-type and mutant SNAP-25 in cerebellar granule cells.

19 tion and glutamate release from cultured rat cerebellar granule cells.

20 sion proteins in PC12 cells and cultured rat cerebellar granule cells.

21 resting intracellular Ca2+ levels in weaver cerebellar granule cells.

22 o control the development of early postnatal cerebellar granule cells.

23 ate excitotoxicity and lithium protection in cerebellar granule cells.

24 tamate-induced excitotoxicity in cultures of cerebellar granule cells.

25 on results in the extensive death of midline cerebellar granule cells.

26 ad, and viability of primary cultures of rat cerebellar granule cells.

27 not polyadenylated, and highly expressed in cerebellar granule cells.

28 the high voltage-activated R-type current in cerebellar granule cells.

29 hat NF-66, but not NF-L is, expressed in the cerebellar granule cells.

30 notropic glutamate receptors, using cultured cerebellar granule cells.

31 xogenous GABA were made from cultured murine cerebellar granule cells.

32 s and endocytosis in synaptic boutons of rat cerebellar granule cells.

33 nduced increased [Ca2+]i was measured in rat cerebellar granule cells.

34 dependent death pathway downstream of BAX in cerebellar granule cells.

35 to a shift in viral tropism from the glia to cerebellar granule cells.

36 st currents through extrasynaptic GABAARs in cerebellar granule cells.

37 ays a critical role in promoting survival of cerebellar granule cells.

38 h Atoh1 and NeuroD1, a marker of postmitotic cerebellar granule cells.

39 d increase neurite outgrowth from developing cerebellar granule cells.

40 l thalamus, dentate gyrus granule cells, and cerebellar granule cells.

41 can participate in integration and firing of cerebellar granule cells.

42 -1 in primary cortical neurons and postnatal cerebellar granule cells.

43 the cerebellum and increases the survival of cerebellar granule cells.

44 thereby regulating excitability in cultured cerebellar granule cells.

45 of cerebellar Purkinje cells and atrophy of cerebellar granule cells.

46 of excitatory AMPA receptors in adult murine cerebellar granule cells.

47 lium-derived factor (PEDF) protects immature cerebellar granule cells (1-3 days in vitro) against ind

48 the inhibition of cAMP formation in cultured cerebellar granule cells; 2) protection against excitoto

49 elease can activate GABAA receptors in adult cerebellar granule cells: action potential-evoked exocyt

50 y, we suggest that BDNF and FGF2 may protect cerebellar granule cells against excitotoxicity by alter

51 selective ablation of Abl family kinases in cerebellar granule cells alone does not cause any abnorm

52 In cerebellar granule cells, although the frequency of inhi

53 synaptic vesicles in a subset of boutons on cerebellar granule cells, an effect that was reversed by

54 By targeting Pten in cerebellar granule cells and activating the AKT1-mTOR pa

55 ly impaired, leading to massive apoptosis of cerebellar granule cells and degeneration of Purkinje ce

56 ypes and in phenotypic maintenance of mature cerebellar granule cells and dorsal root ganglia.

57 ads to a dramatic reduction in the number of cerebellar granule cells and ectopic location of many of

58 with neuronostatin promoted the migration of cerebellar granule cells and elicited direct depolarizin

59 hIP1 and Kv4.2 are concentrated in somata of cerebellar granule cells and in synaptic glomeruli that

60 BSN colocalized with one another in cultured cerebellar granule cells and in synaptic junction membra

61 Here we show that in both cerebellar granule cells and in Xenopus laevis oocytes e

62 antly up-regulated during differentiation of cerebellar granule cells and is absent from astrocytes i

63 inant contributors to the A-type currents in cerebellar granule cells and Ito in rat ventricle.

64 s system prevented apoptosis of cultured rat cerebellar granule cells and macrophages, and the preven

65 nt roles of Smarc proteins and argue against cerebellar granule cells and other progeny of hGFAP-posi

66 hough the NR2C subunit is highly enriched in cerebellar granule cells and plays a unique role in cere

67 ential role in the migration and survival of cerebellar granule cells and precerebellar neurons and f

68 Its expression is later restricted to cerebellar granule cells and precerebellar neurons exten

69 cations within the hindbrain, giving rise to cerebellar granule cells and precerebellar nuclei.

70 HGF is neuroprotective for cerebellar granule cells and promotes growth of human me

71 reeminence of Ptch1 as a tumor suppressor in cerebellar granule cells and reveal other genomic events

72 ch KCl withdrawal induces apoptotic death of cerebellar granule cells and suggest that additional ele

73 Synapses between cerebellar granule cells and their postsynaptic targets,

74 cerebellar granule cells; they produce both cerebellar granule cells and ventral derivatives, some o

75 th SRSF2, hnRNP H1/F, ALYREF and hnRNP A1 in cerebellar granule cells and with SRSF2, hnRNP H1/F and

76 articipates in the AraC-induced apoptosis of cerebellar granule cells and, if so, the relationship be

77 r theory predicts that the dimensions of the cerebellar granule-cell and Drosophila Kenyon-cell repre

78 ATX is neurotoxic in primary cultures of rat cerebellar granule cells, and this neuronal death is pre

79 pontaneously develop ataxia, degeneration of cerebellar granule cells, and vacuolation of white matte

80 T-20 cells, pituitary gonadotropes, cultured cerebellar granule cells, and yeast with high concentrat

81 es to afferent mixing.SIGNIFICANCE STATEMENT Cerebellar granule cells are among the simplest neurons,

82 Cerebellar granule cells are an attractive model system

83 e, we show that multiple synapses of waggler cerebellar granule cells are arrested at an immature sta

84 Cerebellar granule cells are inhibited phasically by GAB

85 aptic alpha6-containing GABA(A) receptors in cerebellar granule cells are selectively regulated by AM

86 Mutant cerebellar granule cells are susceptible to exogenous an

87 Cerebellar granule cells are the most abundant neurons i

88 Cerebellar granule cells are the most abundant neurons i

89 Cerebellar granule cells are the most abundant type of n

90 RRL clones comprised the EGL and cerebellar granule cells, as expected.

91 ternalization of GABAA receptors in cultured cerebellar granule cells, as reflected by diminished imm

92 In vitro and in vivo cerebellar granule cell assays show a decrease in neuron

93 nsients in individual presynaptic boutons of cerebellar granule cells at near-physiological temperatu

94 port that activation of GABA(A) receptors on cerebellar granule cell axons modulates both transmitter

95 e have shown that exogenous GABA depolarizes cerebellar granule cell axons through local activation o

96 The growth of cerebellar granule cell axons was examined by placing fo

97 t of clinical illness and slows apoptosis of cerebellar granule cells but has no effect on white matt

98 rtant roles in the apoptotic death of weaver cerebellar granule cells, but the downstream events asso

99 n the regulation of kainate neurotoxicity in cerebellar granule cells by calcium entry through voltag

100 it is possible that inhibition of IK(SO) in cerebellar granule cells by synaptically released zinc m

101 VPA treatment of rat cerebellar granule cells caused a robust dose- and time-

102 Whereas human cerebellar granule cells (CGC) express only MT1 receptor

103 otoxins produce neuronal injury and death in cerebellar granule cells (CGC) following acute exposure.

104 rahydrofolate (5-FTHF) into primary cultured cerebellar granule cells (CGC) was studied.

105 on primary neuronal cultures, consisting of cerebellar granule cells (CGC).

106 and zeta are present in rat primary cultured cerebellar granule cells (CGCs) 6-14 days in vitro (DIV)

107 y expressed in mouse cerebellum and cultured cerebellar granule cells (CGCs) and modulates P/Q-type c

108 ncreasing the expression of NMDA subunits in cerebellar granule cells (CGCs) by transfection was stud

109 ICE)-like cysteine proteases-in apoptosis of cerebellar granule cells (CGCs) caused by withdrawal of

110 excitatory synaptic currents in cultures of cerebellar granule cells (CGCs) from NR2A knockout (NR2A

111 relatively simple and compact morphology of cerebellar granule cells (CGCs) has led to the view that

112 One of these is a model of cerebellar granule cells (CGCs) in which apoptosis is in

113 Cerebellar granule cells (CGCs) need NMDAR stimulation t

114 ncrease of tonic current was observed in the cerebellar granule cells (CGCs) of alpha1-/- compared wi

115 -tagged PSD-95 (PSD-95gfp) into cultured rat cerebellar granule cells (CGCs) to investigate the role

116 A miniature IPSCs (mIPSCs) from cultured rat cerebellar granule cells (CGCs) while varying the extrac

117 To further examine Srg1 expression in cerebellar granule cells (CGCs), we used an in vitro cel

118 apoptotic and necrotic death of EtOH-exposed cerebellar granule cells (CGCs).

119 P endonuclease, were significantly higher in cerebellar granule cells compared with astrocytes.

120 Cerebellar granule cells constitute the majority of neur

121 weaver cerebellar granule cells could be rescued from cell deat

122 ssive mouse mutation that causes progressive cerebellar granule cell death and peripheral motor axon

123 erization of patterns of germ cell death and cerebellar granule cell death in homozygous weavers with

124 t in a well characterized in vitro paradigm, cerebellar granule cell death induced by withdrawal of d

125 In cerebellar granule cells, delta subunit-containing GABA(

126 Comparatively, mitochondria isolated from cerebellar granule cells demonstrated a higher Ca2+ upta

127 Cerebellar granule cells demonstrated high TUNEL stainin

128 role for a CaMKK2/CaMKIV cascade involved in cerebellar granule cell development and show specificall

129 , Ig-NRG, plays a crucial role in aspects of cerebellar granule cell development in vitro.

130 s indicate that Nr-CAM and L1 play a role in cerebellar granule cell development, and suggest that cl

131 g developmental apoptosis in early postnatal cerebellar granule cells, dorsal root ganglia, embryonic

132 excitatory postsynaptic currents (EPSCs) in cerebellar granule cells during a time-varying, quantifi

133 A expression was rapidly induced in cultured cerebellar granule cells during apoptosis induced by a l

134 e examined how MeHg affects the migration of cerebellar granule cells during early postnatal developm

135 FGF22 is expressed by cerebellar granule cells during the period when they rec

136 Cerebellar granule cells exhibit distinct modes of migra

137 Primary XRCC1 heterozygous mouse cerebellar granule cells exhibit increased strand break

138 xin-sensitive stimulation of 22Na+ influx in cerebellar granule cells exposed to ATX.

139 torage; and viability of primary cultures of cerebellar granule cells exposed to NMDA receptor agonis

140 Apparently, the PM of cerebellar granule cells exposed to NMDA was more depola

141 Unlike mammals, only few cerebellar granule cells expressed AptCB1R transcripts a

142 Nr-CAM-deficient cerebellar granule cells failed to extend neurites in vi

143 Cerebellar granule cells fire in bursts, and their paral

144 ced life span, ataxic gait with apoptosis of cerebellar granule cells followed by Purkinje cell deple

145 Mossy fiber afferents to cerebellar granule cells form the primary synaptic relay

146 In cultured rat cerebellar granule cells, FRET between CFP-SNAP-25 and Y

147 Immunohistochemistry of primary cerebellar granule cells from ataxia3 mice demonstrated

148 sistant phosphomimetic MEF2D mutant protects cerebellar granule cells from cell death after DNA damag

149 and extrasynaptic GABA-mediated currents in cerebellar granule cells from Gabra6(100R/100R) and Gabr

150 rescued excitatory synaptic transmission in cerebellar granule cells from stargazer mice, the decay

151 Cerebellar granule cell GABAA receptor responses to alco

152 e we demonstrate that genetic differences in cerebellar granule cell GABAA receptor responses to recr

153 vel and implications of such interactions at cerebellar granule cell GABAA receptors are discussed.

154 Here, we show that strengthening of mouse cerebellar granule cell GABAergic synapses occurs by a d

155 Dissociated cultures of cerebellar granule cells (GCs) demonstrate improved long

156 ple reactomes of epigenetic pathway genes in cerebellar granule cells (GCs) during circuit formation.

157 not KChIP2) were expressed at high levels in cerebellar granule cells (GCs).

158 esicle priming that is strongly expressed in cerebellar granule cells (GCs).

159 Ethanol enhanced function of cerebellar granule cell GIRKs coupled to GABAB receptors

160 at GABAergic neuronal progenitors as well as cerebellar granule cells give rise to MBG3 with their di

161 In cultured cerebellar granule cells, GlnT is expressed only on neur

162 Golgi cells (GoCs) of the cerebellum inhibit cerebellar granule cells (GrCs) and are driven both by f

163 ted neurite outgrowth of rat postnatal day 6 cerebellar granule cells grown on a substratum of NgCAM,

164 HEK293 cells, and native GABAA receptors of cerebellar granule cells, hippocampal neurons, and thala

165 This resulted in early cerebellar granule cell hyper-proliferation and a 48% in

166 )-alpha6 subunit mRNA, which is expressed in cerebellar granule cells in an activity-independent mann

167 We show here that Cbln1 is secreted from cerebellar granule cells in complex with a related prote

168 Cerebellar granule cells in culture are vulnerable to NM

169 We used rat cerebellar granule cells in culture because these neuron

170 The results demonstrate that cerebellar granule cells in culture express functional k

171 ([Ca2+]i) in rat cortical neurons and mouse cerebellar granule cells in culture.

172 these observations, presynaptic terminals of cerebellar granule cells in dilute-lethal mice showed in

173 Patch-clamp recordings were made from rat cerebellar granule cells in primary culture.

174 ion of 5-bromo-2'-deoxyuridine-labeled mouse cerebellar granule cells in slice cultures, underscoring

175 or CaMKIV disrupts the ability of developing cerebellar granule cells in the external granule cell la

176 programs regulate the autonomous turning of cerebellar granule cells in vitro.

177 its role in the cell cycle-related death of cerebellar granule cells in vivo.

178 dent, p53-independent cell death response in cerebellar granule cells in vivo.

179 sults suggest that AraC-induced apoptosis of cerebellar granule cells involves the expression of both

180 The cerebellar granule cell is the most numerous neuron in t

181 t growth factor (FGF) receptor activation in cerebellar granule cells is associated with increased GA

182 gions to MeHg exposure, and profound loss of cerebellar granule cells is detected in the brains of pa

183 between proliferation and differentiation in cerebellar granule cells is poorly understood.

184 he molecular correlate of R-type currents in cerebellar granule cells is the alpha1E subunit, which y

185 this phenomenon are beginning to emerge: in cerebellar granule cells, it alters the gain of transmis

186 with this idea, we detected UPR induction in cerebellar granule cells lacking stargazin.

187 Combinatorial expansion by the cerebellar granule cell layer (GCL) is fundamental to th

188 ic and nontransgenic mice was highest in the cerebellar granule cell layer and hippocampal neuronal l

189 idence suggests that local activation of the cerebellar granule cell layer produces a much more restr

190 The localization of apoptosis to the cerebellar granule cell layer, identified these cells as

191 bution of TWIK-1, with highest levels in the cerebellar granule cell layer, thalamic reticular nucleu

192 c loss of GABA(A) receptor expression in the cerebellar granule cell layer.

193 mGlu1b receptor mRNA levels increased in the cerebellar granule cell layer.

194 ule cell layer and throughout the cortex and cerebellar granule cell layer.

195 on of TREK-2 was primarily restricted to the cerebellar granule cell layer.

196 ughout all forebrain areas as well as in the cerebellar granule cell layer.

197 rmal granule cells and the transformed human cerebellar granule cell line DAOY, OGR1 promoted express

198 en receptors (ERs) expressed in the immature cerebellar granule cell line E(t)C.1 by transfecting suc

199 Dissociated cerebellar granule cells maintained in medium containing

200 fects on integration of excitatory inputs by cerebellar granule cells might result from different mod

201 ein (GRIP) and the physiologic regulation of cerebellar granule cell migration by SR.

202 ental abnormalities in the cerebellum due to cerebellar granule cell migration defects.

203 ed on our studies, we propose that defective cerebellar granule cell migration secondary to disorgani

204 Ca(2+) elevations triggers the completion of cerebellar granule cell migration.

205 ally inhibited PC12 cell differentiation and cerebellar granule cell migration.

206 o mediate SDF-1alpha/CXCR4 signal in guiding cerebellar granule cell migration.

207 3 gene product, shown previously to regulate cerebellar granule cell migrations, also controls the gu

208 the studies reported here, using an in vitro cerebellar granule cell model, suggest that levels of GA

209 levation of nuclear CaM also was observed in cerebellar granule cells, neocortical neurons, and denta

210 activation is required for proliferation of cerebellar granule cell neuron precursors during develop

211 ical, and histological findings of a case of cerebellar granule cell neuronopathy (GCN), a JCV-associ

212 e have demonstrated that JC virus can infect cerebellar granule cell neurons and cortical pyramidal n

213 ary cell culture revealed that Ifi27l2a(-/-) cerebellar granule cell neurons and macrophages but not

214 grated into the cerebellum, and WNV-infected cerebellar granule cell neurons expressed higher levels

215 d macrophages, dendritic cells, fibroblasts, cerebellar granule cell neurons, and cortical neurons re

216 crophages, dendritic cells, fibroblasts, and cerebellar granule cell neurons, but not cortical neuron

217 ies were investigated in primary cultures of cerebellar granule cell neurons.

218 which is required for normal development of cerebellar granule cell neurons.

219 erived neurotrophic factor (BDNF) protein in cerebellar granule cell neurons.

220 CNS, we demonstrated a dramatic increase in cerebellar granule cell number that appeared to be attri

221 have examined the effects of somatostatin in cerebellar granule cells of early postnatal mice, becaus

222 els of BDNF and higher rates of apoptosis in cerebellar granule cells of neuroD2(-/-) mice indicate t

223 , a finding also observed in apoptotic mouse cerebellar granule cells on postnatal day 5.

224 lices rich in OECs enhanced axonal growth of cerebellar granule cells or hippocampal neurons; axons g

225 a has been attributed to loss of AMPARs from cerebellar granule cells, other cerebellar neurons have

226 Here we show that migration of cerebellar granule cells out of their proliferative zone

227 rmalities, we examined the ultrastructure of cerebellar granule cell output synapses and measured the

228 ors and A(1) receptors are both localized on cerebellar granule cell parallel fiber terminals and bas

229 At the synapse between cerebellar granule cell parallel fibers (PFs) and Purkin

230 pendent manner by comparing synapses made by cerebellar granule cell parallel fibers onto Golgi cells

231 xpressed by developing inner ear hair cells, cerebellar granule cells, precerebellar neurons, and col

232 Here we show that cerebellar granule cell precursors (GCPs) migrate along

233 Hedgehog signaling controls proliferation of cerebellar granule cell precursors (GCPs), and its aberr

234 ons that express high levels of Ptchd1 mRNA: cerebellar granule cell precursors and dentate granule c

235 These HSPG co-receptors are expressed by cerebellar granule cell precursors and promote Shh bindi

236 facial branchiomotor nucleus, the spread of cerebellar granule cell precursors to form the external

237 nuclei, while later derivatives comprise of cerebellar granule cell precursors, a unique proliferati

238 d, Sonic hedgehog, as a powerful mitogen for cerebellar granule cell precursors.

239 specifically deleted Smarca4 and Smarcb1 in cerebellar granule cell precursors.

240 Cerebellar granule cells prepared from p53 knock-out mic

241 Cerebellar granule cell progenitors (GCP) proliferate ex

242 tic cerebellum in which the proliferation of cerebellar granule cell progenitors (GCPs) in response t

243 ere we have shown that deletion of Chd7 from cerebellar granule cell progenitors (GCps) results in re

244 illustrate the utility of MADM, we show that cerebellar granule cell progenitors are fated at an earl

245 Cerebellar granule cell progenitors proliferate postnata

246 og (Shh) signaling, which normally regulates cerebellar granule cell proliferation.

247 tering mice have been shown to have abnormal cerebellar granule cell-Purkinje cell synapses and leane

248 Cerebellar granule cells receive five orders of magnitud

249 Cerebellar granule cells receive mossy fiber inputs that

250 After their final mitosis, cerebellar granule cells remain in the external granular

251 -isoxazolepropionic acid (AMPA) receptors in cerebellar granule cells requires stargazin, a member of

252 s [delayed Ca(2+) deregulation (DCD)] in rat cerebellar granule cells resulting from chronic activati

253 e imaging of Venus-tagged ASTN1 in migrating cerebellar granule cells reveals the intracellular traff

254 cipated passive and active properties of the cerebellar granule cell's unmyelinated axon.

255 is concentrated along the plasma membrane of cerebellar granule cell somata and dendrites.

256 nal Ca(2+) channel gamma subunit, exhibits a cerebellar granule cell-specific brain-derived neurotrop

257 on of GABA(A) receptor alpha6, delta (mature cerebellar granule cell-specific proteins), and beta3 su

258 t experiments on hypoglossal motoneurons and cerebellar granule cells suggest that the resting curren

259 vels of genes encoding proteins that support cerebellar granule cell survival, including brain-derive

260 wly, K(+) was retained in the cytoplasm, and cerebellar granule cells survived the challenge; in the

261 ng.In conclusion, this study demonstrates in cerebellar granule cells that external pH can either red

262 w that Cbln1 is a glycoprotein secreted from cerebellar granule cells that is essential for three pro

263 extracellular matrix protein synthesized in cerebellar granule cells that plays an important role in

264 TARP expression in gamma-2-lacking stargazer cerebellar granule cells--the classic model of TARP defi

265 r cells are not restricted to producing only cerebellar granule cells; they produce both cerebellar g

266 ting the derivation of medulloblastomas from cerebellar granule cells through activation of the Sonic

267 ge in intrinsic membrane excitability of rat cerebellar granule cells through modification of Kv4 A-t

268 Cerebellar granule cell to Purkinje cell synapses have b

269 ordings and modeling of synaptic activity at cerebellar granule cell to Purkinje cell synapses of mic

270 For cerebellar granule cell to stellate cell (grc-->SC) syna

271 iating the chemotactic response of migrating cerebellar granule cells to BDNF through its regulation

272 We have used primary cultures of rat cerebellar granule cells to examine mechanisms whereby g

273 Exposure of cerebellar granule cells to glutamate induces a rapid in

274 Furthermore, stimulation of fused knockout cerebellar granule cells to proliferate with Sonic Hh re

275 Likewise, in cerebellar granule cells transfected with vGlut1-pHluori

276 anule cell-Purkinje cell synapses and leaner cerebellar granule cells undergo abnormal apoptosis duri

277 uced currents by external H+ was examined in cerebellar granule cells using whole-cell and single-cha

278 AraC-induced apoptosis of cerebellar granule cells was preceded by an increase in

279 Neurite outgrowth of cerebellar granule cells was stimulated on astrocytes ex

280 Using cultured rat cerebellar granule cells, we investigated whether BDNF a

281 A-type currents in rat cerebellar granule cells were decreased two days after A

282 he modulation of GAP-43 expression, cultured cerebellar granule cells were exposed to these transmitt

283 ine kinetics and consequences to morphology, cerebellar granule cells were treated in vitro with d-th

284 Primary cerebellar granule cells were used in this study to dete

285 Cultured cerebellar granule cells were used to examine the protec

286 ate (NMDA) receptors are highly expressed in cerebellar granule cells where they mediate the majority

287 eport here that erbB4 is expressed in mature cerebellar granule cells, where it appears to be concent

288 rongly for glucocorticoid receptors, such as cerebellar granule cells, where PP5 is either absent or

289 ons, such as hippocampal pyramidal cells and cerebellar granule cells, where we have previously also

290 mRNA for ClC-2 in SCG neurons but not in rat cerebellar granule cells which do not possess a hyperpol

291 Cerebellar granule cells, which constitute half the brai

292 ncipal cytopathology appears to be a loss of cerebellar granule cells, which frequently display conde

293 uman brain contains approximately 60 billion cerebellar granule cells, which outnumber all other brai

294 f the TrkC ligand neurotrophin-3 (NT-3) from cerebellar granule cells, which provide major afferent i

295 polarized manner facing the leading pole of cerebellar granule cells with a migratory morphology.

296 Pretreatment of cerebellar granule cells with IL-10 (1-50 ng/ml) elicite

297 g term, but not acute, treatment of cultured cerebellar granule cells with LiCl induces a concentrati

298 Pretreatment of aging cerebellar granule cells with lithium or VPA alone provi

299 ceptor, or BDNF, act as chemoattractants for cerebellar granule cells, with SDF-1 action being select

300 ptors increased kainate receptor activity in cerebellar granule cells without changing NMDA receptors