ライフサイエンスコーパス: cerebellar vermis

1 frontal and the paracentral motor cortex and cerebellar vermis.

2 the prefrontal cortex, corpus callosum, and cerebellar vermis.

3 Two of these are ongoing within the cerebellar vermis.

4 Controls had greater activation in the cerebellar vermis.

5 rietal sulcus, insula, frontal operculum and cerebellar vermis.

6 leus, and lobules I, IV, V, IX, and X of the cerebellar vermis.

7 ly, frontal operculum bilaterally and in the cerebellar vermis.

8 ecrease of vascular resistance in the dorsal cerebellar vermis.

9 oys in the posterior-inferior lobules of the cerebellar vermis.

10 te nucleus, globus pallidus, cerebellum, and cerebellar vermis.

11 kinases (ERKs) and p38 MAPK in the anterior cerebellar vermis.

12 total and frontal brain, basal ganglia, and cerebellar vermis.

13 recursor pool of cells that develop into the cerebellar vermis.

14 sels particularly in the cerebral cortex and cerebellar vermis.

15 ateral thalamus and premotor cortex, and the cerebellar vermis.

16 after transection of the posterior inferior cerebellar vermis.

17 the dorsal striatum, olfactory tubercle, and cerebellar vermis.

18 l cortex, the left prefrontal cortex and the cerebellar vermis.

19 of the frontal lobes but not in the superior cerebellar vermis.

20 taining GABA-A/BDZ receptors in the superior cerebellar vermis.

21 ns of K1, and DV bilaterally in the superior cerebellar vermis.

22 f-generated head movements in the rat caudal cerebellar vermis, an area essential for graviceptive fu

23 me is a congenital brain malformation of the cerebellar vermis and brainstem with abnormalities of ax

24 characterized by hypoplasia/dysplasia of the cerebellar vermis and by ataxia, hypotonia, oculomotor a

25 tter reduction in anterior subregions of the cerebellar vermis and hemisphere in the asymptomatic pre

26 fect in GCP proliferation was similar in the cerebellar vermis and hemispheres in all patients with c

27 uronal loss/dysfunction was indicated in the cerebellar vermis and hemispheres in both diseases by lo

28 We found that the right cerebellar vermis and left lobule V of cerebellar anteri

29 amus, dentate nucleus, cerebellar peduncles, cerebellar vermis and lobules V and VI, and corpus callo

30 e ataxic, attributed to the reduction of the cerebellar vermis and some regions of the hemispheres.

31 l striatum and ventral striatum, and midline cerebellar vermis and subgenual cingulate cortex.

32 dial, cortico-nuclear zone consisting of the cerebellar vermis and the fastigial nucleus.

33 A similar increase in the cerebellar vermis and the left thalamus likewise suggest

34 input and motor output (anterior and dorsal cerebellar vermis) and the maintenance of equilibrium (v

35 l cortices as well as the caudate, thalamus, cerebellar vermis, and cerebrum in 20 first-episode psyc

36 ulate cortex, lingual gyrus, striate cortex, cerebellar vermis, and left thalamus.

37 infancy, atrophy of the cerebral cortex and cerebellar vermis, and mild atrophy of the cerebellar he

38 the thalamus, globus pallidus, hippocampus, cerebellar vermis, and very low expression was detected

39 same distribution, and also in the superior cerebellar vermis; and the non-ACD group had significant

40 d precursor cell proliferation in the medial cerebellar (vermis) anlage after E11.5.

41 (Senior-Loken syndrome), liver fibrosis, and cerebellar vermis aplasia (Joubert syndrome) in approxim

42 nherited ciliopathy giving rise to NPHP with cerebellar vermis aplasia and retinal degeneration.

43 ts association with retinal degeneration and cerebellar vermis aplasia in Joubert syndrome are poorly

44 Decreased volume of the superior cerebellar vermis appears to represent an important subs

45 men/globus pallidus, sensorimotor cortex and cerebellar vermis, as well as increases in the precuneus

46 recessive disorder marked by agenesis of the cerebellar vermis, ataxia, hypotonia, oculomotor apraxia

47 trophy, progressive gait ataxia with tremor, cerebellar vermis atrophy, and optic-nerve thinning.

48 failure in early infancy and are affected by cerebellar vermis atrophy, ataxia, and peripheral neurop

49 ctivations (increased rCBF) were observed in cerebellar vermis, brainstem and right anterior cingulat

50 Using whole-cell recording from slices of cerebellar vermis derived from juvenile (P18-25) or adul

51 ion, activation patterns were present in the cerebellar vermis during bimanual coordination tasks, wi

52 In addition, the right cerebellar vermis had enhanced connectivity with motor a

53 Unlike the cerebellar vermis, however, MSTd neurons also carry a sp

54 and posterior fossa malformations including cerebellar vermis hypoplasia (CVH), mega-cisterna magna

55 with Joubert Syndrome, a ciliopathy causing cerebellar vermis hypoplasia and ataxia.

56 Finally, we report cerebellar vermis hypoplasia in 35% of CHARGE syndrome p

57 syndrome and link reduced FGF signalling to cerebellar vermis hypoplasia in a human syndrome.

58 The children presented congenital ataxia and cerebellar vermis hypoplasia with elongated superior cer

59 cessive multisystem disease characterized by cerebellar vermis hypoplasia with prominent superior cer

60 The ciliopathy Joubert syndrome is marked by cerebellar vermis hypoplasia, a phenotype for which the

61 evelopmental brain disorder characterized by cerebellar vermis hypoplasia, abnormal eye movement, ata

62 atrophy, dysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facial dysmorphism.

63 ooth sign" on axial brain MRI, together with cerebellar vermis hypoplasia, ataxia, and psychomotor de

64 and abnormal signal), thin corpus callosum, cerebellar vermis hypoplasia, optic nerve hypoplasia and

65 urological disorder featuring absence of the cerebellar vermis (i.e. midline).

66 ic resonance image analysis, we measured the cerebellar vermis in 125 normal individuals with a broad

67 us, and fusiform gyri and reduced GMV in the cerebellar vermis in FXS at both timepoints, suggesting

68 Injury to the cerebellar vermis in patients with mTBI and anxiety may

69 Cerebellar fusion and absence of cerebellar vermis is often associated with supratentoria

70 daloid, and hippocampal volumes, and smaller cerebellar vermis lobules VI and VII, in comparison with

71 ly smaller volumes in the posterior-inferior cerebellar vermis (lobules VIII-X; effect size, 0.54; P

72 ized developmental links between frontal and cerebellar vermis neural abnormalities were supported, i

73 nsors but similar to lobules 9 and 10 of the cerebellar vermis (nodulus and uvula), MSTd neurons resp

74 nuclear fractions of postmortem samples from cerebellar vermis of 10 patients with schizophrenia and

75 recorded by two-photon Ca(2+) imaging in the cerebellar vermis of awake behaving mice.

76 jection of low doses of kainic acid into the cerebellar vermis of mice elicited reliable and reproduc

77 bitrary densitometric units [P=.003]) in the cerebellar vermis of schizophrenic subjects.

78 and linear density of Purkinje cells in the cerebellar vermis of subjects with and without schizophr

79 The influence of the cerebellar vermis on prefrontal and striatal circuitry s

80 colliculus resulting from dysfunction in the cerebellar vermis or the basal ganglia.

81 metabolism in right amygdala/hippocampus and cerebellar vermis (P < 0.001), relative to global brain.

82 his tumor, which arises predominantly in the cerebellar vermis, preferentially affects children betwe

83 We found that caudal cerebellar vermis Purkinje cells and cerebellar nuclei n

84 thies characterized by severe defects of the cerebellar vermis, ranging from hypoplasia to aplasia.

85 cts, including mediolateral expansion of the cerebellar vermis, reduced thickness of the granule cell

86 e and mental stress tasks, increased rCBF in cerebellar vermis, right anterior cingulate and right in

87 loss of volume was observed in the superior cerebellar vermis; the volume loss persisted regardless

88 The cerebellar vermis was strongly activated by capsaicin, w

89 al cortex, hippocampus, corpus callosum, and cerebellar vermis were compared between mother-reared (n

90 Blocks of alcohol-fixed cerebellar vermis were dissected at autopsy from the bra

91 of abnormal levels of the MAP kinases in the cerebellar vermis were linked to additional downstream t

92 brain malformation characterized by missing cerebellar vermis with apparent fusion of the cerebellar

93 tify a group of Purkinje cells in the caudal cerebellar vermis with responses that reflect an estimat