ライフサイエンスコーパス: cerebellar white matter

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1 in mice was spatially indistinguishable from cerebellar white matter.

2 with numerous lesions in the hippocampus and cerebellar white matter.

3 , including cells in the corpus callosum and cerebellar white matter.

4 s and basal ganglia, and to a lesser extent, cerebellar white matter.

5 nje cells and spongiform degeneration of the cerebellar white matter.

6 the corpus callosum and fimbria/fornix, and cerebellar white matter.

7 ntified extensive nanoparticle signal in the cerebellar white matter and circumscribed cortical gray

8 on of proliferative clusters of cells in the cerebellar white matter and dorsal brainstem of postnata

9 Brain T2 lesion load, cerebellar white matter and gray matter volumes, and tra

10 Men had higher fractional anisotropy (FA) in cerebellar white matter and in the left superior longitu

11 lower volume in the basilar (ventral) pons, cerebellar white matter and visual cortex.

12 sis pontis, middle cerebellar peduncles, and cerebellar white matter, and elevated succinate on magne

13 the middle cerebellar peduncles and adjacent cerebellar white matter are thought to be highly sensiti

14 ense white matter voxels was performed using cerebellar white matter as the intensity reference.

15 ltinuclear globoid cells in the cerebral and cerebellar white matter associated with severe gliosis.

16 connections as well as projection fibers and cerebellar white matter being among the affected tracts.

17 acteristics are (i) significant cerebral and cerebellar white matter disease, (ii) associated astrocy

18 e increases in anisotropy within the midline cerebellar white matter in the second study correlated w

19 also optic radiation, brainstem tracts, and cerebellar white matter, in combination with delayed mye

20 of Purkinje cells, Bergmann gliosis and deep cerebellar white matter inflammatory infiltrates.

21 ion tensor MRI abnormalities consistent with cerebellar white matter injury in 3 of 4 subjects.

22 nje cells and spongiform degeneration of the cerebellar white matter may be important factors in the

23 Thus, dividing progenitors in the cerebellar white matter migrate through the white matter

24 We investigated GABAergic signaling to cerebellar white matter NG2 cells in a mouse model of DW

25 ot NAA, evokes an inward membrane current in cerebellar white matter oligodendrocytes, which is reduc

26 The three receptors were also detected in cerebellar white matter, primarily during myelination.

27 h less severe Purkinje cell loss also showed cerebellar white matter thinning and demyelination.

28 formed there, be readily transported to the cerebellar white matter where PrPSc is found.

29 ar streams: a dorsal pathway into developing cerebellar white matter, where the migrating cells dispe

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