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1 rior temporal, putamen, lingual, cuneus, and cerebellum).
2 files to both competencies (e.g., R.MFG with cerebellum).
3 3 were inverse in the frontal cortex and the cerebellum.
4  cleavage, as does Astn2 isolated from mouse cerebellum.
5 ons and thalamus, while HD had higher FCD in cerebellum.
6 ies in total gray matter, basal ganglia, and cerebellum.
7 erior cingulate cortex (PCC), precuneus, and cerebellum.
8 ed receptor that is most highly expressed in cerebellum.
9 ased GBCr in the PFC and reduced GBCr in the cerebellum.
10 ior cingulate, precuneus, lingual gyrus, and cerebellum.
11 brain and low in the non-affected area, i.e. cerebellum.
12 set, characterized by hypoplasia of pons and cerebellum.
13 14) tDCS applied to the right posterolateral cerebellum.
14 tion of cortical folding of the cerebrum and cerebellum.
15 p2 expression approximately 40% in the mouse cerebellum.
16 of the glutamatergic transmission in the EAE cerebellum.
17 ods to quantitatively map atrophy across the cerebellum.
18 induced by aberrant afferent inputs from the cerebellum.
19 ary somatosensory cortex, frontal areas, and cerebellum.
20 curacy achieved by CTAC, particularly in the cerebellum.
21 s are highlighted: the preoptic area and the cerebellum.
22 brain regions, including the hippocampus and cerebellum.
23 ce, and strongly supports rate coding in the cerebellum.
24 implications for cognitive processing in the cerebellum.
25 ficant cholestanol increase in the brain and cerebellum.
26 man adult visual cortex, frontal cortex, and cerebellum.
27  studies to send feedback collaterals to the cerebellum.
28 preferential cholestanol accumulation in the cerebellum.
29 filtering of sensorimotor information in the cerebellum.
30 including the hypothalamus, hippocampus, and cerebellum.
31 llate cell-Purkinje GABAergic synapse in the cerebellum.
32 onfirmed the loss of CN neurons from the Sey cerebellum.
33 ability and preferential degeneration of the cerebellum.
34 s by stripping antibodies and re-probing the cerebellum.
35 tiation and migration in the embryonic mouse cerebellum.
36 campal formation, and little CARTp-ir in the cerebellum.
37 hite matter volume throughout the cortex and cerebellum.
38  originate from cells located outside of the cerebellum.
39 ges occur in association with atrophy of the cerebellum.
40 or these receptors in human, monkey, and rat cerebellum.
41 sumoylation of FOXP2 in the developing mouse cerebellum.
42 directionally connects the neocortex and the cerebellum.
43  variety of cells in the mouse neocortex and cerebellum.
44 gulated in the CSF of MS patients and in EAE cerebellum.
45 d, less evidence exists on the impact on the cerebellum.
46 tion of dendrites such as the hippocampus or cerebellum.
47 g +/- 0.079 and 0.061 mug/g +/- 0.012 in the cerebellum.
48 rocessed in circuitry similar to that of the cerebellum.
49 of the dorsal root ganglia, spinal cord, and cerebellum.
50 , habenula, preoptic area, hypothalamus, and cerebellum.
51  arborization of Purkinje cells in the mouse cerebellum.
52 enerated in other sensory structures and the cerebellum.
53  role in ASD, such as the frontal cortex and cerebellum.
54 nnected to a focal area in the dorsal medial cerebellum.
55  detrimental effects on the integrity of the cerebellum.
56 creased activation in right Crus I/II of the cerebellum.
57 major supsraspinal motor control centre, the cerebellum.
58 temporal lobes (8.69%), basal ganglia (22%), cerebellum(17.39%), brainstem(9%) and thalamus(4%).
59 y the neocortex (76), hypothalamus (63), and cerebellum (26).
60 bellar motor function, which ascribes to the cerebellum a role in short-term prediction through inter
61                                           In cerebellum, a rapid increase in gene expression occurred
62                It has been proposed that the cerebellum acquires internal models of mental processes
63                       It is thought that the cerebellum acquires internal models of mental processes
64  the cannabinoid CB1 receptor (CB1 R) in the cerebellum; activation of this system in the cerebellar
65    In addition to reduced volume of pons and cerebellum, affected individuals had microcephaly, psych
66 MLIs are an important circuit element in the cerebellum allowing for accurate motor control.
67 ugh, very little was known about STDP in the cerebellum, although it is thought to play a critical ro
68                                          The cerebellum, although traditionally considered a motor st
69  fiber-to-unipolar brush cell synapse in the cerebellum, AMPAR-mediated EPSCs last for hundreds of mi
70  this new functional interaction between the cerebellum and basal ganglia, and put forward a hypothes
71  may be an involvement of the basal ganglia, cerebellum and brainstem, with or without hemorrhage and
72 ingly supports a more articulated view where cerebellum and cortex, working closely in concert with b
73 ave not been studied in detail regarding the cerebellum and depression.
74  The induction of synaptic plasticity in the cerebellum and elsewhere usually involves intracellular
75 larity was specific for promoter regions and cerebellum and frontal cortex expression, suggesting a m
76 chnique validation by imaging taurine in the cerebellum and hippocampus regions of the rat brain.
77 rmed by climbing fibers on Purkinje cells in cerebellum and inhibitory synapses formed by parvalbumin
78 thelial cells, the intestinal tract, and the cerebellum and is activated by alterations of its membra
79 lly abundant metabolites were evident in the cerebellum and liver.
80  inhibited HH signaling-driven growth of the cerebellum and medulloblastoma.
81 esions were most commonly found in the right cerebellum and most apparent in patients with primary pr
82 NIFICANCE STATEMENT Connectivity between the cerebellum and motor cortex is a critical pathway for th
83 orm of PCH with small, normally proportioned cerebellum and should be screened in individuals with sy
84 lying robustness of circuit formation in the cerebellum and speculate that adaptive reprogramming of
85  transcriptomic alterations occurring in the cerebellum and spinal cord and monitored immune cell sur
86 alterations in the transcriptome of both the cerebellum and spinal cord that was consistent with glia
87 ying disease processes jointly affecting the cerebellum and the cerebrum.
88 ge analysis pipelines optimized for both the cerebellum and the cerebrum.
89                                          The cerebellum and the motor cortex appear to be critical fo
90 mplex in the development and function of the cerebellum and the skeletal system.
91 lly tight disynaptic connections between the cerebellum and the striatum.
92                        The occipital cortex, cerebellum and whole brain were first evaluated as candi
93              ChIP-seq was performed in adult cerebellum and Wiz peaks were found at promoters and tra
94 ts revealed PCH, small normally proportioned cerebellum, and corpus callosum anomalies.
95 ipital lobes, temporal lobes, limbic system, cerebellum, and frontoparietal cortices, as shown by ana
96 found pathology both in the striatum and the cerebellum, and functional disorganisation was reported
97                                    Midbrain, cerebellum, and hippocampus were evaluated as the refere
98 y in the right anterior insula and bilateral cerebellum, and hypoactivity in the dorsal medial prefro
99 lumetric abnormalities in the basal ganglia, cerebellum, and prefrontal cortices.
100 drome predominantly affecting the brainstem, cerebellum, and spinal cord.
101 doreference regions (i.e., occipital cortex, cerebellum, and whole brain) to obtain SUVRoccip, SUVRce
102                        Purkinje cells of the cerebellum appear to be the most sensitive to impaired I
103 ntext, the regulatory role of rs113288603 in cerebellum appears to be consistent with the known invol
104 ur work establishes that MLIs in the lateral cerebellum are broadly activated during movement with ca
105 standing of how sex differences in the human cerebellum are distributed and determined.
106 thophysiological and atrophic changes in the cerebellum are documented in Parkinson's disease.
107  consequences of inflammation locally in the cerebellum are prevented by peripheral treatment with th
108 n apoptosis, whereas the cerebral cortex and cerebellum are unaffected.
109  that control local blood flow supply in the cerebellum are unclear.
110  that control local blood flow supply in the cerebellum are unclear.
111  preferential increase in cholestanol in the cerebellum arising from CYP27A1 deficiency.
112 together, these novel findings establish the cerebellum as a key node in the distributed brain networ
113  (DVR) and static SUV ratio (SUVR) using the cerebellum as a reference tissue.
114 eration as well as the practice of using the cerebellum as reference region for ligand neuroimaging s
115                                    Using the cerebellum as reference region, we applied the simplifie
116                      These data identify the cerebellum as the main site of dysfunction in DYT1, and
117         Striatal SRTM BPND using midbrain or cerebellum as the reference region was significantly low
118                                     With the cerebellum as the reference region, regional binding pot
119  was the binding potential (BPND), using the cerebellum as the reference region.
120 e simplified reference tissue model with the cerebellum as the reference tissue for nonspecific bindi
121 postnatal stages to adult stages, and in the cerebellum at adulthood.
122 cid metabolism and epigenetic changes in the cerebellum at presymptomatic stages of NPC disease repre
123                                           In cerebellum, basket cells (BCs) innervate the soma and ax
124                      We show that within the cerebellum, BDNF-expressing cells are restricted to the
125 ranial magnetic stimulation delivered to the cerebellum before a test pulse over motor cortex.
126 y and functional connectivity pattern in the cerebellum between PD patients and healthy controls.
127 ent with a role for the right posterolateral cerebellum beyond motor aspects of language, and suggest
128                                       In the cerebellum, both compensatory and detrimental contributi
129 d regional cerebral blood flow (in inner and cerebellum brainstem regions) remaining higher in the bo
130 s of deep brain structures (the striatum and cerebellum), but not the cerebral cortex.
131                   This primarily affects the cerebellum, but also results in extracerebellar symptoms
132                     TorsinA knockdown in the cerebellum, but not in the basal ganglia, was sufficient
133 garded as a 'network' disorder including the cerebellum, but the exact pathogenesis being unknown.
134 e viral tracing, we show that innervation of cerebellum by rubrospinal neuron collaterals is remarkab
135 lves distinct physiological processes in the cerebellum (CB) and primary motor cortex (M1).
136         In contrast, the role of the lateral cerebellum (cerebellar hemispheres and dentate nuclei, D
137 reference regions were also evaluated: whole cerebellum, cerebellar gray matter, atlas-based white ma
138 ion of kinematic representations through the cerebellum.Cerebellar Purkinje cells (PCs) linearly enco
139  when comparing astrocytes from spinal cord, cerebellum, cerebral cortex, and hippocampus.
140      Granule cells at the input layer of the cerebellum comprise over half the neurons in the human b
141 ebellar function place the inferior olive to cerebellum connection at the centre of motor behaviour.
142 he metabolite urea in the OVT73 striatum and cerebellum, consistent with our recently published obser
143   ABSTRACT: It is well known that the medial cerebellum controls saccadic speed and accuracy.
144 pothesized, activity in right posterolateral cerebellum correlated with the predictability of the upc
145  track neural activity over multiple days of cerebellum-dependent eyeblink conditioning in mice, that
146 ys for a given muscle may reflect aspects of cerebellum-dependent motor adaptation.
147 ein adducts, whereas the cerebral cortex and cerebellum did not respond at any time.
148 pulations in the hippocampus, neocortex, and cerebellum during development.
149 s that preterm birth impedes function of the cerebellum even in the absence of focal cerebellar lesio
150 gly, such Sox2(+) S100(-) cells of the adult cerebellum expanded after adequate physiological stimuli
151 pport amygdala modulation of CS input to the cerebellum (Experiment 3).
152 n rodents, torsinA knockdown in the immature cerebellum failed to produce dystonia.
153                         Baseline parietal to cerebellum FDG metabolism ratios predicted MMSE (beta=0.
154  evidence for a functional topography of the cerebellum first defined in task-based functional magnet
155 t manner by sex (relative expansion of total cerebellum, flocculus, and Crus II-lobule VIIIB volumes
156 lar window that provides access to the mouse cerebellum for intravital imaging, thereby allowing for
157              These results indicate that the cerebellum forms a crucial part of the feedforward contr
158                Loss of Zdhhc13 in cortex and cerebellum from 3- and 24 m old hetero- and homozygous m
159  regulates BBB permeability and protects the cerebellum from infection and immunopathology.
160 iated with higher DNMT3B expression in adult cerebellum from the Genotype-Tissue Expression project (
161 prefrontal cortex, striatum, hippocampus and cerebellum) from 41 schizophrenia patients and 47 contro
162 rafish embryos, as well as in vitro cultured cerebellum granule neuron progenitors (CGNPs) and SmoM2-
163  ratios were calculated (t = 80-100 minutes, cerebellum gray matter reference).
164 yelination with atrophy of basal ganglia and cerebellum (H-ABC).
165 glut1 is prominent in granular layers of the cerebellum, habenula, preglomerular nuclei, and several
166                                          The cerebellum has been hypothesized to form a crucial part
167                                    While the cerebellum has been shown to be part of the speech motor
168                  Associative learning in the cerebellum has previously focused on single movements.
169                            INTRODUCTION: The cerebellum has strong cortical and subcortical connectiv
170            Here we found that the developing cerebellum has unappreciated progenitor plasticity, sinc
171 tionally connecting the hypothalamus and the cerebellum, has been detected, being specifically involv
172 rhythmic behavior.SIGNIFICANCE STATEMENT The cerebellum helps fine-tune coordinated motor actions via
173                                  The lateral cerebellum (hemispheres and DN) also participates in mod
174 l area (VTA), habenula, periaqueductal gray, cerebellum, hypothalamus, and hippocampal CA3.
175 e causal evidence for the involvement of the cerebellum in adapting to delayed action effects, and th
176 h previous studies suggesting a role for the cerebellum in an internal feedback mechanism.
177    Although evidence suggests a role for the cerebellum in cognition, granule cells are known to enco
178  medial frontal networks and the role of the cerebellum in cognitive processing.
179 ters were equal for cortical regions and the cerebellum in control subjects but different in the puta
180 eased in the thalamus, entorhinal cortex and cerebellum in FFI.
181 e to CNS haemangioblastoma in the cortex and cerebellum in mice that present with highly vascular tum
182                  One of the functions of the cerebellum in motor learning is to predict and account f
183 ncluded the cingulate cortices, thalami, and cerebellum in patients with bvFTD, with the addition of
184 unctional MRI to investigate the role of the cerebellum in performing a dual motor and cognitive task
185 hese data are consistent with a role for the cerebellum in semantic processing and semantic predictio
186             To date, the precise role of the cerebellum in speech motor control remains unclear, as i
187                 Current models implicate the cerebellum in the prediction of the sensory consequences
188 nsorimotor behavior, but the function of the cerebellum in the rodent whisker system is unknown.
189     There was reduced activation in the left cerebellum in the transcranial direct current stimulatio
190 l pathway may support the involvement of the cerebellum in tic production; (iii) furnishes prediction
191 2 expression levels in Purkinje cells of the cerebellum in vivo, we reduced Foxp2 expression approxim
192 C (0.6% +/- 2.7%, P < 0.01) was found in the cerebellum, in comparison with ZTACUC (8.1% +/- 3.5%, P
193 ctional connections with a region in ventral cerebellum, in the vicinity of lobules VII/VIII.
194 infusion into the striatum, but not into the cerebellum, indicating that defects in striatal neurons
195      These data provide insight into how the cerebellum influences medial frontal networks and the ro
196 ow that a Hebbian form of STDP occurs at the cerebellum input stage, providing the substrate for phas
197                                          The cerebellum is a large hindbrain structure that is increa
198                                          The cerebellum is a prominent part of the vertebrate hindbra
199                            Assuming that the cerebellum is a valid reference region, simplified metho
200         In addition to single movements, the cerebellum is also believed to be important for learning
201                                 However, the cerebellum is also consistently implicated in nonmotor f
202                                          The cerebellum is critical for learning the appropriate timi
203     Together, these results suggest that the cerebellum is crucial for maintaining accurate feedforwa
204               It has been suggested that the cerebellum is important in dual-tasking.
205 to alpha1-adrenoceptors, suggesting that the cerebellum is of limited usefulness as a reference tissu
206               First, the skull overlying the cerebellum is removed, and then the window is applied to
207 neuromodulation to provide evidence that the cerebellum is specifically involved in semantic predicti
208             Medulloblastoma arising from the cerebellum is the most common pediatric brain malignancy
209 and comprehension.SIGNIFICANCE STATEMENT The cerebellum is traditionally seen as a motor structure th
210 lar deposits, in (18)F-florbetaben SUVR when cerebellum is used as the reference.
211 Medulloblastoma, an aggressive cancer of the cerebellum, is among the most common pediatric brain tum
212 tex only in foetal life stages, while in the cerebellum it was also expressed postnatally.
213  than the Purkinje cells they innervate, and cerebellum-like circuits, including the insect mushroom
214 iltering has been established previously for cerebellum-like sensory structures in fish, suggesting a
215 motor area, rostral cingulate motor area and cerebellum likely contributes to progressive micrographi
216                                              Cerebellum lobules were segmented using SUIT V.3.0 to es
217 ingulate/paracingulate gyri (ACG/ApCG), left cerebellum (lobules IV/V and VIII), bilateral superior f
218                                       In the cerebellum, NEEP21/Nsg1 expression becomes largely restr
219 ion of FOXP2 in the brain, in particular the cerebellum, nor the effects of any posttranslational mod
220 zed contributions, we identified EZH2 in the cerebellum, NR3C1 in the cerebral cortex and SRF in the
221 f three autosomes in the cerebral cortex and cerebellum of adult and developing brain of Bub1b(H/H) m
222  observed throughout the cerebral cortex and cerebellum of an affected proband, expanding our underst
223 while those with GABAB2 were enhanced in the cerebellum of Cacna1aCtmKO/CtmKO mice.
224 l production; (ii) spatial separation in the cerebellum of cholesterol/cholestanol-metabolizing P450s
225 n and ubiquitin-aggregated structures in the cerebellum of Mecp2 knockout mouse model (Mecp2 (-/y) )
226 xpression of cytokines and chemokines in the cerebellum of MIA offspring, including increase in the n
227 ation of FOXP2 at K674 (K673 in mice) in the cerebellum of neonates.
228 esponsible for abnormal morphogenesis of the cerebellum of Npc1-deficient mice and show, for the firs
229  demonstrated diaschisis in the cerebrum and cerebellum of patients with glioma.
230 in the sensory motor cortex, hippocampus and cerebellum of post-mortem brains from HD individuals, pa
231 pression analyses in the cerebral cortex and cerebellum of these mice identified a network of schizop
232 as 16 intraparenchymal deposits (four in the cerebellum) of a recombinant adenoassociated viral vecto
233  it has been reported that the Pax6-null Sey cerebellum only has minor defects involving granule cell
234 ntal problems and ataxia, and atrophy of the cerebellum or even the whole brain in about half of the
235 0-min dynamic PET scan and the use of either cerebellum or midbrain as the reference region.
236 s studied (hippocampus, ventral pallidum and cerebellum), or of the effects of chronic ketamine admin
237 (p<0.01), left crus I, right VIIb and entire cerebellum (p<0.05 for each comparison) and between PwMS
238 terior corona radiata, external capsule, and cerebellum (P<0.05, family-wise error-corrected).
239                                              Cerebellum plasticity may explain the lack of statistica
240                Purkinje cells of the primate cerebellum play critical but poorly understood roles in
241                          Additionally, whole cerebellum, pons, centrum semiovale, and a composite reg
242 nd thalami and increased connectivity in the cerebellum, pons, left amygdala, and orbitofrontal corti
243 ghly understudied, despite the fact that the cerebellum possesses many more neurons than the cerebral
244 iferation of a progenitor niche in postnatal cerebellum predisposed to oncogenic induction of medullo
245 tructures of the isocortex, hippocampus, and cerebellum, presumably reflecting the trafficking of rec
246 nhance the degenerative phenotype of the NPC cerebellum provides strong support for the notion that l
247 e cells form the primary synaptic relay into cerebellum, providing an ideal site to process signal in
248 inhibitory neurons at the input stage of the cerebellum, providing feedforward and feedback inhibitio
249 tively associated with activity in the right cerebellum (PTSD vs. NTC), and illness severity was nega
250                        Purkinje cells of the cerebellum receive approximately 180,000 parallel fibre
251 alogy, 25% of (11)C-CUMI-101 uptake in human cerebellum reflects binding to alpha1-adrenoceptors, sug
252 inate fasciculus, ventral frontal, and right cerebellum regions; and amygdala functional connectivity
253 ortical (putamen, thalamus, globus pallidus, cerebellum) regions.
254       Sumoylation of FOXP2 in neonatal mouse cerebellum regulates Purkinje cell development and motor
255 he expansion and folding of the cerebrum and cerebellum, respectively.
256  in tendons and brain (preferentially in the cerebellum) rich in cholesterol and cholestanol, the 5al
257 ndicates that posterolateral portions of the cerebellum (right Crus I/II) contribute to language proc
258  an independent set of prefrontal cortex and cerebellum samples.
259 to provide motor efferent information to the cerebellum, satisfying predictions about the use of coro
260                                       In the cerebellum, sensory information is conveyed to Purkinje
261 ement across a lobule-specific region of the cerebellum showing high temporal correspondence within t
262 s ubiquitously expressed with high levels in cerebellum, skeletal muscle, thymus and kidney.
263  midbrain, and in the dentate nucleus of the cerebellum (t's > 2.7, P's < 0.02).
264  neural activity in the mTBI patients in the cerebellum-thalamo-cortical and the fronto-basal-ganglia
265 e THs, RNA-seq analysis was conducted in the cerebellum, thalamus-pituitary and liver of tilapia trea
266 re TH-responsive (FDR < 0.05) in the tilapia cerebellum, thalamus-pituitary and liver, respectively.
267 ice, with significantly higher levels in the cerebellum than in the cerebrum.
268 ior olive conveys instructive signals to the cerebellum that drive sensorimotor learning.
269 rmal perivascular proliferative niche in the cerebellum that persisted in adult animals but did not p
270 d their normal period, resulting in a larger cerebellum that persists into adulthood, with consequent
271                                       In the cerebellum, the amount of intact gadodiamide accounts fo
272 nsight into how the output structures of the cerebellum, the cerebellar nuclei, integrate their input
273                                       In the cerebellum, the molecular layer is regarded as the main
274 Tbr1 and Tbr2 expression is found in the Sey cerebellum, these are cell-specific markers of cerebella
275 s are essential for motor learning, with the cerebellum thought to be required only for the error-bas
276 s with high levels of binding to that in the cerebellum, thus demonstrating the binding specificity a
277 through the analysis of sub regions of a rat cerebellum tissue section.
278 se hypothalamus, hippocampus, neocortex, and cerebellum to determine estrous cycle-specific changes i
279 reinforces the sensitivity of the developing cerebellum to microtubule defects.
280 r temporal gyri (MFG and ITG) and resistant (cerebellum) to classical AD pathology.
281 olive sends instructive motor signals to the cerebellum via the climbing fibre projection, which send
282 ficant decrease in hippocampus, midbrain, or cerebellum VT Baseline striatal SRTM BPND did not differ
283  executive function-related Crus I/II in the cerebellum was analysed.
284                               Interestingly, cerebellum was the area most affected, with greater myel
285                                              Cerebellum was used as a reference region in all imaging
286 ference regions: cerebellar gray (CG), whole cerebellum (WC), WC with brainstem (WC + B), pons, and w
287 our RRs (cerebellar gray matter [CGM], whole cerebellum [WCER], pons, and subcortical white matter [S
288 ventral striatum, substantia nigra (SN), and cerebellum were manually drawn on coregistered MR images
289 tients as nodes within the basal ganglia and cerebellum were more strongly connected to one another t
290 tructural covariance between the ACC and the cerebellum were observed in the MDD group.
291 es; (ii) there is greater involvement of the cerebellum when immediate recall tasks involve more comp
292 acterized by impaired growth of the pons and cerebellum, which frequently follows a degenerative cour
293 ts in a preferential reduction of PCs in the cerebellum, which is likely mediated by decreased neuron
294 arely Pax2(+) interneurons in the developing cerebellum, which opposes the "temporal identity transit
295 milarity between the cerebral cortex and the cerebellum, which points to potential brain cell dediffe
296 n the orbitofrontal cortex, hippocampus, and cerebellum; white matter integrity in the uncinate fasci
297 g2 expression strikingly disappears from the cerebellum with age.
298 erent channels, which separately connect the cerebellum with the motor areas and nonmotor areas of th
299  = 4.31, P < .001), and the crus I/II of the cerebellum (z score = 3.77, P < .001), a region connecte
300 ly corresponding to the motor regions of the cerebellum (z score = 3.96 and 3.42 in right and left si

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