ライフサイエンスコーパス: cerebral artery occlusion

1 rtery occlusion and 360 with isolated middle cerebral artery occlusion).

2 l carotid, basilar, and M1 segment of middle cerebral artery occlusions).

3 neonatal and adult rats by transient middle cerebral artery occlusion.

4 t cavity for 7 d, beginning 7 d after middle cerebral artery occlusion.

5 d into rat brains 6 h after transient middle cerebral artery occlusion.

6 nal infarcts such as those induced by middle cerebral artery occlusion.

7 arge peri-infarct cortex region after middle cerebral artery occlusion.

8 emic brain damage caused by permanent middle cerebral artery occlusion.

9 ously or intra-arterially after right middle cerebral artery occlusion.

10 ct cortex before and after unilateral middle cerebral artery occlusion.

11 me 11 for collateral remodeling after middle cerebral artery occlusion.

12 larly starting 3 days after 30 min of middle cerebral artery occlusion.

13 me in aged rats following a transient middle cerebral artery occlusion.

14 e and oxidative stress in response to middle cerebral artery occlusion.

15 t rats were subjected to a 90-minutes middle cerebral artery occlusion.

16 to stroke rats from days 6 to 9 after middle cerebral artery occlusion.

17 when initiated both before and after middle cerebral artery occlusion.

18 nuated in CD36-null mice subjected to middle cerebral artery occlusion.

19 ype animals after a 90 min reversible middle cerebral artery occlusion.

20 lesion volume induced by a transient middle cerebral artery occlusion.

21 ring and subjected to transient (2 h) middle cerebral artery occlusion.

22 to affect brain damage from transient middle cerebral artery occlusion.

23 in the ischemic penumbra region after middle cerebral artery occlusion.

24 in vivo rat stroke model of transient middle cerebral artery occlusion.

25 We used the stroke model of temporary middle cerebral artery occlusion.

26 ic cortex of rats 7 days after distal middle cerebral artery occlusion.

27 pidly upregulate STC2 after transient middle cerebral artery occlusion.

28 Rats were subjected to embolic middle cerebral artery occlusion.

29 infarction volume was observed after middle cerebral artery occlusion.

30 ischemia was induced by 30 minutes of middle cerebral artery occlusion.

31 Cerebral ischemia was induced by middle cerebral artery occlusion.

32 in the brains of 2D2 mice 14 d after middle cerebral artery occlusion.

33 tes AhR activation in the brain after middle cerebral artery occlusion.

34 ittermates received sham or transient middle cerebral artery occlusion.

35 weeks into the treatment by transient middle cerebral artery occlusion.

36 bra using a mouse model of reversible middle cerebral artery occlusion.

37 r bundles from degeneration following middle cerebral artery occlusion.

38 brains 6 h or 7 days after transient middle cerebral artery occlusion.

39 85% reduction of infarct volume after middle cerebral artery occlusion; 54% rescue of low skeletal mu

40 termates were treated with reversible middle cerebral artery occlusion (90 min; 22 hr reperfusion).

41 inflammatory cytokine profile in the middle cerebral artery occlusion-affected right brain hemispher

42 H have improved outcomes after 60-min middle cerebral artery occlusion and 24-h reperfusion.

43 We used rodent models of middle cerebral artery occlusion and cell-culture models of neu

44 ) of Niaspan, starting 24 hours after middle cerebral artery occlusion and daily for 14 days.

45 ere subjected to 60 min of reversible middle cerebral artery occlusion and evaluated for infarct volu

46 n status, is increased following both middle cerebral artery occlusion and oxygen-glucose deprivation

47 e model of focal cerebral ischemia by middle cerebral artery occlusion and reperfusion (I/R) in male

48 In vivo, transient middle cerebral artery occlusion and reperfusion in kinase-dead

49 ion in brain infarct size after acute middle cerebral artery occlusion and reperfusion, strongly supp

50 icient Rag1(-/-) mice after 60 min of middle cerebral artery occlusion and reperfusion.

51 ts were subjected to right hemisphere middle-cerebral artery occlusion and reperfusion.

52 l or energy-restricted diets prior to middle cerebral artery occlusion and reperfusion.

53 ficit, and infarct size at 24 h after middle cerebral artery occlusion and reperfusion.

54 were subjected to 90 min of transient middle cerebral artery occlusion and treated with either a cont

55 ed significantly in rats subjected to middle cerebral artery occlusion and treated with no intravenou

56 Adult male rats were subjected to middle cerebral artery occlusion and were treated with or witho

57 ed in mice (by permanent or transient middle cerebral artery occlusion) and rats (by 3-vessel occlusi

58 trophic lateral sclerosis (SOD1G93A), middle cerebral artery occlusion, and multiple mini-strokes.

59 after (late post-ischemia) 60 min of middle cerebral artery occlusion, and were killed 10 days after

60 patients who had ischaemic stroke and major cerebral artery occlusion beyond 3 h of symptom onset.

61 In rats with middle cerebral artery occlusion, both the recovery of motor sk

62 ly monitored rats received 2 hours of middle cerebral artery occlusion by intraluminal suture, result

63 ri-infarct zone from 24 to 72 h after middle cerebral artery occlusion by means of immunohistochemica

64 its and poststroke inflammation after middle cerebral artery occlusion by preventing microglia polari

65 were subjected to 2 hrs of transient middle cerebral artery occlusion by the intraluminal occlusion

66 .5%) rats were subjected to permanent middle cerebral artery occlusion by the intraluminal occlusion

67 Following transient middle cerebral artery occlusion, ck2beta(-/-) mice displayed s

68 e scores at those times, and proximal middle cerebral artery occlusion demonstrated prior to treatmen

69 calculated in patients with proximal middle cerebral artery occlusion (derivation cohort) with known

70 of either sex subjected to transient middle cerebral artery occlusion developed dramatically smaller

71 either sex challenged with transient middle cerebral artery occlusion developed significantly smalle

72 male mice underwent unilateral distal middle cerebral artery occlusion (dMCA) occlusion and were imag

73 , 5 and 7 days after permanent distal middle cerebral artery occlusion (dMCAO) in mice compared to ve

74 Ischaemia was induced by distal middle cerebral artery occlusion (dMCAO) in normoxic (30% inhal

75 e rats received a 90-min right distal middle cerebral artery occlusion (dMCAo).

76 initiated at 48 h after mouse distal middle cerebral artery occlusion (dMCAO).

77 volume of mice subjected to transient middle cerebral artery occlusion even up to 3 to 5 hours after

78 rct volume in a standardized model of middle cerebral artery occlusion, even when the agent is admini

79 and wild-type (WT) mice to 1 hour of middle cerebral artery occlusion followed by 23 hours of reperf

80 ivo focal ischemic model, 2 h of left middle cerebral artery occlusion followed by 24 h of reperfusio

81 ed to 90-min ischemia induced by left middle cerebral artery occlusion followed by 72-h reperfusion.

82 nously 10 minutes after initiation of middle cerebral artery occlusion followed by reperfusion.

83 were subjected to 30 min of transient middle cerebral artery occlusion, followed by 0, 0.5, 3, and 6

84 and hyperlipidemic mice to transient middle cerebral artery occlusion, followed by measurement of st

85 Male Swiss Webster mice underwent middle cerebral artery occlusion for 1 h followed by reperfusio

86 We performed right middle cerebral artery occlusion for 3 hours, administered reco

87 Mice were subjected to middle cerebral artery occlusion for 40 min, followed by reperf

88 ebral ischemia was induced in mice by middle cerebral artery occlusion for 60 minutes and s-NSCs were

89 othelin receptors following permanent middle cerebral artery occlusion for 7 days.

90 Sprague-Dawley rats were subjected to middle cerebral artery occlusion for 70 min followed by reperfu

91 bral ischemia was produced in rats by middle cerebral artery occlusion for 90 min.

92 Animals were subjected to transient middle cerebral artery occlusion for 90 mins.

93 e produced focal cerebral ischemia by middle cerebral artery occlusion for 90 minutes in the adult ra

94 A 60 minute middle cerebral artery occlusion in C57 mice resulted in over 5

95 We induced permanent middle cerebral artery occlusion in female mice that previously

96 ne the role of AhR in stroke, we used middle cerebral artery occlusion in mice and oxygen-glucose dep

97 the severity of stroke in a model of middle cerebral artery occlusion in mice.

98 spontaneous functional recovery after middle cerebral artery occlusion in mice.

99 y, and cerebral edema formation after middle cerebral artery occlusion in mice.

100 owing acute focal ischemia induced by middle cerebral artery occlusion in normotensive rats.

101 apsulated clodronate before transient middle cerebral artery occlusion in postnatal day 7 rats.

102 nvestigated the effects of VEGF after middle cerebral artery occlusion in rats using a series of beha

103 e ET(B) receptors following permanent middle cerebral artery occlusion in rats.

104 e, and clinical outcome in a model of middle cerebral artery occlusion in rats.

105 he ischemic brain following transient middle cerebral artery occlusion in rats.

106 and 14 (n = 7) after 2 hr unilateral middle cerebral artery occlusion in rats.

107 mbra when administered six hours post middle cerebral artery occlusion in rats.

108 and until completion of 15 min distal middle cerebral artery occlusion in spontaneously hypertensive

109 retinal ischemia induced by temporary middle cerebral artery occlusion in the adult rat.

110 Middle cerebral artery occlusion in the rat brain resulted in a

111 After transient middle cerebral artery occlusion in the rat brain, elevated upt

112 al ischaemia was induced by transient middle cerebral artery occlusion in the rat using the filament

113 ilized two model systems of ischemia, middle cerebral artery occlusion in vivo and oxygen-glucose dep

114 umbilical cord blood cells 48 h after middle cerebral artery occlusion increased Akt phosphorylation

115 ologically induced excitotoxicity and middle cerebral artery occlusion-induced brain damage.

116 ainst NMDA-induced excitotoxicity and middle cerebral artery occlusion-induced stroke in mice.

117 Infarct volume following middle cerebral artery occlusion is dramatically diminished in

118 ggression; (3) larger infarcts in the middle cerebral artery occlusion ischemic stroke model; and (4)

119 erebral ischemia induced by transient middle cerebral artery occlusion it selectively dilates arterio

120 in the ischemic brain after transient middle cerebral artery occlusion leading to increased intracran

121 ld-type mice were subjected to 1 h of middle cerebral artery occlusion (MCAO) and 24-72 h of reperfus

122 nhibitor, in a rat model of transient middle cerebral artery occlusion (MCAO) and an in vitro model o

123 s consist of rats subjected to 2 h of middle cerebral artery occlusion (MCAo) and at 24 h after MCAo

124 severe ischemic injury, as induced by middle cerebral artery occlusion (MCAo) and if this protection

125 ts (n=57) were subjected to permanent middle cerebral artery occlusion (MCAo) and injected intravenou

126 barrier (BBB) permeability following middle cerebral artery occlusion (MCAo) and intrastriatal trans

127 unction and tissue injury response to middle cerebral artery occlusion (MCAO) and reperfusion.

128 on are attenuated following transient middle cerebral artery occlusion (MCAO) and reperfusion.

129 he animals were subjected to a 2-hour middle cerebral artery occlusion (MCAO) and sacrificed at 24 ho

130 odel encompasses a combination of the middle cerebral artery occlusion (MCAO) and spatial restraint s

131 acute brain injury: stroke caused by middle cerebral artery occlusion (MCAO) and traumatic brain inj

132 reventing brain damage from transient middle cerebral artery occlusion (MCAO) as was estrone.

133 tar rats underwent unilateral embolic middle cerebral artery occlusion (MCAo) by a single fibrin rich

134 (reproductive senescence) shows that middle cerebral artery occlusion (MCAo) causes a larger cortica

135 f age, rat pups underwent a permanent middle cerebral artery occlusion (MCAo) coupled with a temporar

136 l of permanent and transient (45 min) middle cerebral artery occlusion (MCAO) during the hyperacute,

137 to the ischemic site after transient middle cerebral artery occlusion (MCAO) followed by reperfusion

138 -Tg) littermates underwent reversible middle cerebral artery occlusion (MCAO) for 1 hour followed by

139 riments, rats were subjected to right middle cerebral artery occlusion (MCAO) for 2 h under ketamine/

140 study, the model of reversible right middle cerebral artery occlusion (MCAO) for 2 h was used.

141 t male C57BL/6 mice were subjected to middle cerebral artery occlusion (MCAO) for stroke induction.

142 for the 30-min, 60-min and permanent middle cerebral artery occlusion (MCAO) group, respectively.

143 We also tested H(2) in middle cerebral artery occlusion (MCAO) in adult rats (MCAO n=9

144 Following transient middle cerebral artery occlusion (MCAO) in adult rats, expressi

145 o the cerebral cortex before a 60-min middle cerebral artery occlusion (MCAo) in adult rats.

146 emia and 2) infarct volumes 24h after Middle Cerebral Artery Occlusion (MCAO) in all 3 types of mice.

147 d preconditioning following 90 min of middle cerebral artery occlusion (MCAO) in male Wistar rats.

148 dismutase mimetic, against 90 min of middle cerebral artery occlusion (MCAO) in male Wistar rats.

149 Since we previously demonstrated that middle cerebral artery occlusion (MCAO) in mice induces sheddin

150 evaluated various outcomes following middle cerebral artery occlusion (MCAO) in ovariectomised femal

151 addition, the functional recovery to middle cerebral artery occlusion (MCAO) in rats and hMCT2 trans

152 ) reduces cerebral infarct size after middle cerebral artery occlusion (MCAO) in rats through an unkn

153 roved behavioral outcome after distal middle cerebral artery occlusion (MCAO) in rats.

154 peutic effect in a model of permanent middle cerebral artery occlusion (MCAo) in rats.

155 ischemic brain damage after permanent middle cerebral artery occlusion (MCAO) in SHR-SP rats and whet

156 ression increased following 30 min of middle cerebral artery occlusion (MCAO) in the mouse brain in b

157 followed by continuous decline after middle cerebral artery occlusion (MCAO) in the mouse brain.

158 Ischemia induced either by embolic middle cerebral artery occlusion (MCAO) in vivo or by oxygen an

159 Experimental middle cerebral artery occlusion (MCAO) increases tPA activity

160 ane and subjected to 2 h of temporary middle cerebral artery occlusion (MCAo) induced by means of a p

161 We found that middle cerebral artery occlusion (MCAO) induces microglial acti

162 Middle cerebral artery occlusion (MCAO) is a popular model in e

163 ue-Dawley rats using the intraluminal middle cerebral artery occlusion (MCAO) method.

164 prove sensorimotor functions in a rat middle cerebral artery occlusion (MCAO) model after a single in

165 tion were investigated in a permanent middle cerebral artery occlusion (MCAO) model in the adult mous

166 We used transient rat middle cerebral artery occlusion (MCAO) model of brain ischemia

167 cle-treated groups in a 12h permanent middle cerebral artery occlusion (MCAO) model of focal ischemia

168 e intravenous (IV) injection in a rat middle cerebral artery occlusion (MCAO) model of ischemia/reper

169 AE) model of multiple sclerosis and a middle cerebral artery occlusion (MCAO) model of stroke, LSR wa

170 Using an in vivo middle cerebral artery occlusion (MCAO) model only the 57kDa fr

171 lized a murine transient endovascular middle cerebral artery occlusion (MCAO) model to examine the in

172 focal cerebral ischemia in the mouse middle cerebral artery occlusion (MCAO) model.

173 mpact of gemfibrozil in two permanent middle cerebral artery occlusion (MCAO) models in young adult m

174 n injury in mouse photothrombotic and middle cerebral artery occlusion (MCAo) models.

175 on ischemic stroke was studied in the middle cerebral artery occlusion (MCAo) mouse model.

176 hemia was induced by permanent distal middle cerebral artery occlusion (MCAO) on day 14 of vehicle or

177 A reversible intraluminar suture middle cerebral artery occlusion (MCAO) procedure was used to p

178 ale rats were subjected to a two-hour middle cerebral artery occlusion (MCAO) procedure.

179 vely) that are neuroprotective in the middle cerebral artery occlusion (MCAO) rat stroke model.

180 ague-Dawley rats undergoing permanent middle cerebral artery occlusion (MCAO) received three intraven

181 late as 5 h after 60 min of transient middle cerebral artery occlusion (MCAO) reduced infarct volume

182 tudy, rats were sacrificed 24 h after middle cerebral artery occlusion (MCAO) stroke and gene transcr

183 immune cell populations in mice after middle cerebral artery occlusion (MCAO) strongly implicates a m

184 itioning stimulus in a mouse model of middle cerebral artery occlusion (MCAO) to examine whether impr

185 35-55 peptide to C57BL/6 mice before middle cerebral artery occlusion (MCAO) to induce an anti-infla

186 dified nylon suture in a rat model of middle cerebral artery occlusion (MCAO) under two anesthesia re

187 hr after both transient and permanent middle cerebral artery occlusion (MCAo) via continuous DC recor

188 Two weeks later, permanent middle cerebral artery occlusion (MCAO) was induced by intralum

189 Infarct sizes 72 h after 60 min middle cerebral artery occlusion (MCAo) were on average 30 +/-

190 greater in diabetic animals following middle cerebral artery occlusion (MCAO) when compared to non-di

191 GRN(+/-) and PGRN(-/-) mice underwent middle cerebral artery occlusion (MCAO) with monitoring of cere

192 Studies were conducted following middle cerebral artery occlusion (MCAO) with or without reperfu

193 pite smaller infarcts after transient middle cerebral artery occlusion (MCAO) with the suture model.

194 This study utilized middle cerebral artery occlusion (MCAO) with tissue plasminogen

195 replacement were subjected to 2 h of middle cerebral artery occlusion (MCAO), and phosphorylated STA

196 One week later the animals received a middle cerebral artery occlusion (MCAO), and T(2)-weighted MRI

197 of stroke, via transient intraluminal middle cerebral artery occlusion (MCAO), or SHAM surgery.

198 In a mouse model of middle cerebral artery occlusion (MCAO), p38 MAPK activation wa

199 ion in stroke volume in rats with the middle cerebral artery occlusion (MCAO), provided the BDNF is c

200 ia induced by permanent and transient middle cerebral artery occlusion (MCAO), we observed an initial

201 levels of estradiol protects against middle cerebral artery occlusion (MCAO)-induced brain injury du

202 estradiol protects the cortex against middle cerebral artery occlusion (MCAO)-induced cell death.

203 eraction between tPA and LRP plays on middle cerebral artery occlusion (MCAO)-induced NF-kappaB-media

204 ed to permanent, 60-min and 30-min of middle cerebral artery occlusion (MCAO).

205 a focal stroke caused by a transient middle cerebral artery occlusion (MCAO).

206 schemia produced in rats by permanent middle cerebral artery occlusion (MCAO).

207 l of focal ischemic stroke induced by middle cerebral artery occlusion (MCAO).

208 ats infused with HUCB cells 48h after middle cerebral artery occlusion (MCAO).

209 with ATX or vehicle prior to a 60-min middle cerebral artery occlusion (MCAo).

210 outcome were assessed after permanent middle-cerebral artery occlusion (MCAO).

211 es) was induced by intraluminal right middle cerebral artery occlusion (MCAO).

212 injury (CCI) and another to transient middle cerebral artery occlusion (MCAO).

213 ly with HNG before being subjected to middle cerebral artery occlusion (MCAO).

214 N in the intraluminal suture model of middle cerebral artery occlusion (MCAo).

215 ats were subjected to permanent right middle cerebral artery occlusion (MCAo).

216 BL/6 mice were subjected to transient middle cerebral artery occlusion (MCAO).

217 reperfusion injury in a rat model of middle cerebral artery occlusion (MCAO).

218 of the integrity of the NVU following middle cerebral artery occlusion (MCAO).

219 d to behavioral tests after 90 min of middle cerebral artery occlusion (MCAO).

220 wo groups for permanent and temporary middle cerebral artery occlusion (MCAO).

221 sly injected into rats 24 hours after middle cerebral artery occlusion (MCAo).

222 Rats underwent 2 hr of middle cerebral artery occlusion (MCAO).

223 Rats were subjected to embolic middle cerebral artery occlusion (MCAO).

224 Rats in the model groups underwent middle cerebral artery occlusion (MCAO).

225 a was induced by a transient (90 min) middle cerebral artery occlusion (MCAO).

226 ts with experimental stroke caused by middle cerebral artery occlusion (MCAO).

227 proteins, following a 1-h reversible middle cerebral artery occlusion (MCAO).

228 T) mice were subjected to a transient middle cerebral artery occlusion (MCAO).

229 ittermate were subjected to 45 min of middle cerebral artery occlusion (MCAO).

230 129/SV mice were subjected to 30-min middle cerebral artery occlusion (MCAo)/reperfusion and serial

231 four time points following permanent middle cerebral artery occlusion (MCAO): 1, 3, 7, and 14 days.

232 In the present study, transient middle cerebral artery occlusions (MCAO) were induced for vario

233 in were investigated with a permanent middle cerebral artery occlusion model in the rat.

234 eases infarct volume in the transient middle cerebral artery occlusion model of stroke.

235 al model of hypoxia/ischemia and in a middle cerebral artery occlusion model of transient focal ische

236 ave improved cerebral blood flow in a middle cerebral artery occlusion model than mice expressing an

237 A transient middle cerebral artery occlusion model was used to investigate

238 In the middle cerebral artery occlusion model, the volume and fraction

239 ase in stroke volume in the permanent middle cerebral artery occlusion model.

240 cerebrovascular protection in a mouse middle cerebral artery occlusion model.

241 e C57BL/6 mice using the intraluminal middle cerebral artery occlusion model.

242 en used the murine suture and embolic middle cerebral artery occlusion models of stroke to investigat

243 groups: 30-min, 60-min, and permanent middle cerebral-artery occlusion (n=12 rats for each group).

244 hemic stroke and in mice subjected to middle cerebral artery occlusion, natural killer (NK) cells dis

245 n females, we evaluated the effect of middle cerebral artery occlusion on infarct size and peripheral

246 y rats (12 months old) with permanent middle cerebral artery occlusion or sham operations on multiple

247 thrombotic cortical injury, transient middle cerebral artery occlusion, or neonatal hypoxic-ischemic

248 Animals were subjected to permanent middle cerebral artery occlusion (pMCAo) or a sham surgical pro

249 es in transient (tMCAO) and permanent middle cerebral artery occlusion (pMCAO) stroke models using Ex

250 1 and in most cases 2 h of permanent middle cerebral artery occlusion (pMCAO), mild sensory stimulat

251 Following transient middle cerebral artery occlusion, progesterone was administered

252 At 6 hrs following middle cerebral artery occlusion, rats were treated in a blinde

253 At 6 hrs following middle cerebral artery occlusion, rats were treated in a blinde

254 against cerebral ischemia induced by middle cerebral artery occlusion, reducing percent hemispheric

255 In the middle cerebral artery occlusion-reperfusion model of transient

256 deficit in a hyperglycemic rat right middle cerebral artery occlusion/reperfusion (MCAO/R) model.

257 gical impairment in mice subjected to middle cerebral artery occlusion/reperfusion.

258 istration of PAN-811 i.c.v. 1 h after middle cerebral artery occlusion resulted in a 59% reduction in

259 e analyses of rat brains subjected to middle cerebral artery occlusion revealed marked increases in e

260 rcted tissue at 72 h after reversible middle cerebral artery occlusion (rMCAo) in adult rats.

261 xcitatory neurotoxicity in reversible middle cerebral artery occlusion (rMCAO) model in vivo.

262 Following right middle cerebral artery occlusion (rMCAo) using an intraluminal

263 infarction using a stroke model with middle cerebral artery occlusion (see figure).

264 4-Fc decoy receptor immediately after middle cerebral artery occlusion significantly reduced infarct

265 TAT-C1aB in mice following transient middle cerebral artery occlusion significantly reduced ischemic

266 In the model of transient middle cerebral artery occlusion stroke Gna(i2)(fl/fl)/PF4-Cre

267 ved functional outcome in a transient middle cerebral artery occlusion stroke model.

268 ment of blood flow anomaly in a mouse middle cerebral artery occlusion stroke model.

269 s implanted with CTX-DP 4 weeks after middle cerebral artery occlusion stroke prompted investigation

270 r evaluated in vivo using a transient middle cerebral artery occlusion (t-MCAO) model of stroke.

271 We investigated in a murine model of middle cerebral artery occlusion the effect of blocking SIDS by

272 In a mouse model of thrombin-induced middle cerebral artery occlusion, the efficacy of the diabody w

273 ed neuronal death following transient middle cerebral artery occlusion (tMCAO) by up to 90% with an e

274 Furthermore, by transient middle cerebral artery occlusion (tMCAO) in rats, the transcrip

275 Transient middle cerebral artery occlusion (tMCAO) in spontaneously hyper

276 A transient middle cerebral artery occlusion (tMCAO) model was used to esta

277 ronic diaschisis by using a transient middle cerebral artery occlusion (tMCAO) rat model.

278 Transient middle cerebral artery occlusion (tMCAO) significantly increase

279 rial thrombosis models: the transient middle cerebral artery occlusion (tMCAO) stroke model and tail

280 injury in a mouse model of transient middle cerebral artery occlusion (tMCAO).

281 anxiety) in rats following transient middle cerebral artery occlusion (tMCAO).

282 corresponding controls, to transient middle cerebral artery occlusion (tMCAO).

283 uced in adult rats by 90-minute right middle cerebral artery occlusion (tMCAO).

284 .) 15 min before undergoing transient middle cerebral artery occlusion (tMCAO; 2 h occlusion) with re

285 Following middle cerebral artery occlusion to induce stroke in mice, immu

286 nt with an ischemic lesion (permanent middle cerebral artery occlusion) to determine the effect of No

287 ients with internal carotid artery or middle cerebral artery occlusions transferred over an 11-month

288 l vascular endothelial cell death and middle cerebral artery occlusion-triggered cerebrovascular dama

289 The infarct produced by middle cerebral artery occlusion was 49% smaller in CD36-null m

290 ere reperfusion after photothrombolic middle cerebral artery occlusion was increased in Klkb1(-/-) mi

291 Middle cerebral artery occlusion was induced for 1 hour (follow

292 (BM) chimeras subjected to transient middle cerebral artery occlusion, we found that CD36(-/-) mice

293 Following middle cerebral artery occlusion, we observed a rapid increase

294 ver, using a mouse model of transient middle cerebral artery occlusion, we observed that cerebral inf

295 Using two distinct models of acute middle cerebral artery occlusion, we show by next-generation se

296 lumes following a 60-minute transient middle cerebral artery occlusion were determined in adult male

297 Rats subjected to 2h of transient middle cerebral artery occlusion were studied temporally over 1

298 Rats subjected to embolic middle cerebral artery occlusion were treated with atorvastatin

299 in infarct volume was observed after middle cerebral artery occlusion with 4 days of reperfusion in

300 ollowing experimental stroke, using a middle cerebral artery occlusion with reperfusion model.