ライフサイエンスコーパス: cerebral blood volume

1 rectly correlated to regional differences in cerebral blood volume.

2 e values are inversely proportional to local cerebral blood volume.

3 were maps of apparent diffusion and relative cerebral blood volume.

4 , 11-40] vs 14 [range, 6-22] mL/100 mL/min), cerebral blood volume (2.4 [range, 1.6- 4.2] vs 3.9 [ran

5 ative cerebral blood flow (16-53% decrease), cerebral blood volume (6-33% decrease), and tissue mean

6 showed an average 60% decrease in normalized cerebral blood volume (adults P < 0.05; children P < 0.0

7 First, we used MRI to map cerebral blood volume, an established correlate of basal

8 and radiologic (Gaussian normalized relative cerebral blood volume and apparent diffusion coefficient

9 hemodynamic properties of gliomas including cerebral blood volume and blood flow, vascular permeabil

10 ating increased Gaussian-normalized relative cerebral blood volume and Gaussian-normalized relative c

11 flow, prolonged mean transit time, elevated cerebral blood volume and high mean transit time/cerebra

12 ilate the cerebral vasculature, and increase cerebral blood volume and intracranial pressure while de

13 istically significant difference in relative cerebral blood volume and metabolic ratios between high-

14 maging technique that independently measures cerebral blood volume and oxygenation, continuously, in

15 ntributed to the IL-1beta-induced changes in cerebral blood volume and the ADC of brain water.

16 lution, layer-specific measurements of BOLD, cerebral blood volume, and cerebral blood flow in region

17 cal parameter such as cerebral blood flow or cerebral blood volume, and direct mapping of neural acti

18 longer duration, less focal increase in CBF, cerebral blood volume, and hyperoxygenation, the duratio

19 diffusion coefficients, cerebral blood flow, cerebral blood volume, and intratumoral susceptibility s

20 5%, 5.0%, and 1.9%, for cerebral blood flow, cerebral blood volume, and mean transit time, respective

21 eate images of relative blood flow, relative cerebral blood volume, and mean transit time.

22 hy had an average 80% decrease in normalized cerebral blood volume at the core of the lesion (P < 0.0

23 sing optical imaging of light scattering and cerebral blood volume, autofluorescence flavoprotein ima

24 total hemoglobin concentration, i.e. in the cerebral blood volume, by -7+/-3 microM, as opposed to a

25 xygenation level-dependent (BOLD) signal and cerebral blood volume (CBV) and blood flow (CBF), which

26 vity during voluntary behaviors by measuring cerebral blood volume (CBV) and neural activity in the s

27 d 570 and 610 nm optical signals to estimate cerebral blood volume (CBV) and oxygenation, respectivel

28 Simultaneously, we measured changes in cerebral blood volume (CBV) as a proxy of drug effects o

29 rmed at 570 nm to provide functional maps of cerebral blood volume (CBV) changes and at 610 nm to est

30 tudies with blood oxygen level-dependent and cerebral blood volume (CBV) contrasts at 9.4 tesla, as w

31 blood oxygenation level-dependent (BOLD) and cerebral blood volume (CBV) fMRI signal.

32 ndent (BOLD), cerebral blood flow (CBF), and cerebral blood volume (CBV) HRF to ultrashort forelimb s

33 ation of (18)F-FDG uptake, permeability, and cerebral blood volume (CBV) in children with pediatric b

34 sly measuring neural activity and changes in cerebral blood volume (CBV) in the somatosensory cortex

35 collected prospectively, with guidance from cerebral blood volume (CBV) MR imaging measurements.

36 genesis and test whether MRI measurements of cerebral blood volume (CBV) provide an imaging correlate

37 Images of regional cerebral blood volume (CBV) were generated from echo-pla

38 Absolute cerebral blood volume (CBV), absolute cerebral blood flo

39 enerating maps of cerebral blood flow (CBF), cerebral blood volume (CBV), and mean transit time (MTT)

40 Comparisons of mean CBF, cerebral blood volume (CBV), and mean transit time (MTT)

41 Cerebral blood volume (CBV), cerebral blood flow (CBF),

42 15)O-H(2)O to generate parametric images for cerebral blood volume (CBV), cerebral blood flow (CBF),

43 the dynamic spatiotemporal relationships of cerebral blood volume (CBV), deoxygenated hemoglobin (Hb

44 m measurements of cerebral blood flow (CBF), cerebral blood volume (CBV), oxygen extraction fraction

45 s a significant, acute reduction (15-30%) in cerebral blood volume (CBV), which is dependent on TNF-a

46 Here we show that cerebral blood volume (CBV)-weighted fMRI with a blood p

47 l based thresholds for mean transit time and cerebral blood volume (CBV).

48 th mean tissue cerebral blood flow (CBF) and cerebral blood volume (CBV); venous and arterial peak en

49 nges in receptor occupancy and hemodynamics [cerebral blood volume (CBV)] in the domains of space, ti

50 for blood-oxygen-level-dependent (BOLD) and cerebral-blood-volume (CBV)-based laminar fMRI and used

51 umor-to-blood [T/B] ratio), vascularization (cerebral blood volume [CBV]), and vascular permeability

52 Cerebral blood flow, cerebral blood volume, cerebral oxygen metabolism (CMRO2

53 gh mean transit time/cerebral blood flow and cerebral blood volume/cerebral blood flow ratios.

54 n tomography imaging of cerebral blood flow, cerebral blood volume, CMRO2, and oxygen extraction frac

55 anges in cerebral blood flow (DeltaCBF/CBF), cerebral blood volume (DeltaCBV/CBV), and transverse rel

56 Temporoparietal cerebral blood volume, expressed as a percentage of the

57 d to marijuana-induced alteration in resting cerebral blood volume/flow or downregulation of cannabin

58 which were obtained in contrast agent-aided cerebral blood volume fMRI and total hemoglobin-based op

59 Here, we use optical techniques to measure cerebral blood volume, hemoglobin oxygenation (S(t)O(2))

60 e range in Pa(CO2), cortical blood flow, and cerebral blood volume in animals studied using vertical

61 t shows predictive timing, with increases of cerebral blood volume in anticipation of trial onsets ev

62 sm, transient decreases in cell swelling and cerebral blood volume in the surround are consistent wit

63 e child with arrested progression normalized cerebral blood volume in this region.

64 The measured relative cerebral blood volumes in the peritumoral region in high

65 Relative cerebral blood volumes in these regions were calculated

66 VLCBV was defined as cerebral blood volume<2.5th percentile of brain contrala

67 The clinical applications of cerebral blood volume maps obtained with perfusion MR im

68 bility contrast magnetic resonance perfusion cerebral blood volume maps were co-registered, segmented

69 raction between Gaussian-normalized relative cerebral blood volume (nrCBV) and Gaussian-normalized re

70 23) were associated with increasing relative cerebral blood volume of NER (rCBVNER), which was higher

71 Decreased cerebral blood volume on PCT was the most accurate predi

72 e properties including chemical composition, cerebral blood volume, perfusion, vascular permeability,

73 Relative cerebral blood volume (rCBV) (maximum rCBV [rCBV(max)] a

74 The hypothesis was that a change in relative cerebral blood volume (rCBV) 1 month after RT-TMZ is pre

75 Relative cerebral blood volume (rCBV) and blood flow (rCBF) maps

76 Relative cerebral blood volume (rCBV) and flow (rCBF) maps were a

77 xin injection caused an increase in regional cerebral blood volume (rCBV) around the lesion site afte

78 ied to MR imaging-based whole-tumor relative cerebral blood volume (rCBV) histograms.

79 i.v.) in control monkeys increased relative cerebral blood volume (rCBV) in a number of brain region

80 cant differences were noted in age, relative cerebral blood volume (rCBV) in contrast-enhanced region

81 Relative cerebral blood volume (rCBV) maps were created from anal

82 Relative cerebral blood volume (rCBV) ratio is one of the best no

83 ffusion coefficient (ADC) with high relative cerebral blood volume (rCBV) represented elevated cholin

84 Regional cerebral blood volume (rCBV) was determined by using dyn

85 Tumor relative cerebral blood volume (rCBV) was estimated from each DSC

86 R imaging (PWI), especially maps of regional cerebral blood volume (rCBV), may provide similar diagno

87 ntrast-enhanced perfusion-weighted (relative cerebral blood volume [rCBV]) imaging were evaluated in

88 Significantly higher fMRI signals [relative cerebral blood volumes (rCBVs)] and atrophy were observe

89 etic stimulation of TH(VTA) neurons enhanced cerebral blood volume signals in striatal target regions

90 tic theory, yields quantitative estimates of cerebral blood volume that reflect the underlying microv

91 cerebral lesion was present, and normalized cerebral blood volume values were analysed using a Food

92 brain incorporation rate of 11C-AA (K*) and cerebral blood volume (Vb), as well as CBF, were generat

93 Very low cerebral blood volume (VLCBV), diffusion, and hypoperfus

94 njection of IL-1beta (1 ng in 1 microliter), cerebral blood volume was significantly increased, the b

95 ng global hemodynamic changes using BOLD and cerebral blood volume-weighted (CBVw) fMRI.

96 Cerebral blood flow and cerebral blood volume were determined with intravenous H

97 an 8-month-old infant, mean transit time and cerebral blood volume were low relative to cerebral bloo

98 al flow was normal but mean transit time and cerebral blood volume were low, consistent with perfusio