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1 ertebral and basilar arteries (the posterior cerebral circulation).
2 remained unaffected in the celiac artery and cerebral circulation.
3 n MS, ET-1 is released from the brain to the cerebral circulation.
4 shRNA lentiviral particles injected into the cerebral circulation.
5 endothelial function in the BH(4)-deficient cerebral circulation.
6 usly expressed in the vasculature, including cerebral circulation.
7 or proximal vessel occlusion in the anterior cerebral circulation.
8 t soluble Abeta can significantly impair the cerebral circulation.
9 sanoid that produces vasoconstriction in the cerebral circulation.
10 embolism to the arterial tree, including the cerebral circulation.
11 channels in terminal arterioles from rabbit cerebral circulation.
12 n venous and arterial input functions in the cerebral circulation.
13 he carotid bed and 37 +/- 6% at 20/s for the cerebral circulation.
14 be related to vasospastic phenomena in human cerebral circulation.
15 Alzheimer disease (AD) suffer from impaired cerebral circulation.
17 vascular smooth muscle cells (cVSMCs) of the cerebral circulation, although the identity of their mol
18 imum reduction was 37 +/- 6% at 20/s for the cerebral circulation and 11 +/- 3% at 2/s for bulk carot
19 vel dilatory signaling pathway for CO in the cerebral circulation and appears to be the first demonst
20 ongenital vascular variants of the posterior cerebral circulation and cerebral hypoperfusion could pa
22 eposition and fibrin accumulation within the cerebral circulation and would be expected to contribute
23 ment a dissociation between the systemic and cerebral circulations and potentially explain earlier cl
25 t the parasympathetic nerves innervating the cerebral circulation contain NOS and furthermore, since
27 ted by ET-1, which is likely released in the cerebral circulation from reactive astrocytes in plaques
28 nd stent placement in the carotid artery and cerebral circulation has given rise to concerns about th
29 azard with proven deleterious effects on the cerebral circulation, including a decrease in cerebral b
30 ndow of thrombolysis and prolong the time of cerebral circulation intervention for neurosurgical proc
33 eculate that autonomic neural control of the cerebral circulation is tonically active and likely play
34 ts, and that, when measured in the posterior cerebral circulation, it is the higher rather than lower
35 rate hypoxia with lack of expected change in cerebral circulation, leading to hypotension, bradycardi
38 wn influences of autonomic activation on the cerebral circulation played a smaller but significant ro
39 proximal arterial occlusion in the anterior cerebral circulation that was confirmed on vessel imagin
40 in II (Ang II) exerts detrimental effects on cerebral circulation, the mechanisms of which have not b
41 is to discuss arterial anomalies of anterior cerebral circulation, their prevalence and demonstrate r
42 lium produces endothelial dysfunction in the cerebral circulation through a mechanism involving oxida
44 Significantly higher normalized BFVs in the cerebral circulation were measured in children with both
45 synthase (eNOS) contributes to regulation of cerebral circulation, whereas that produced by neuronal
46 on of the parasympathetic innervation of the cerebral circulation with emphasis on the vertebral and
47 nt reduction in resistance that involved the cerebral circulation with little effect on bulk carotid
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