ライフサイエンスコーパス: cerebral cortex

1 esembles the layered structure of the brain (cerebral cortex).

2 with the notion of lateralized functions in cerebral cortex.

3 hrough which retinal information reaches the cerebral cortex.

4 n other brain regions including amygdala and cerebral cortex.

5 egionally and temporally with folding of the cerebral cortex.

6 erns of brain regions distributed across the cerebral cortex.

7 ht to influence the laminar formation of the cerebral cortex.

8 represents the folding characteristic of the cerebral cortex.

9 mpaired neuronal migration in the developing cerebral cortex.

10 was not observed in astrocytes in slices of cerebral cortex.

11 ebellum possesses many more neurons than the cerebral cortex.

12 al pathway of mice lacking active Met in the cerebral cortex.

13 to the unparalleled performance of the human cerebral cortex.

14 a nigra pars compacta (SNpc) and, later, the cerebral cortex.

15 hat may have contributed to the expansion of cerebral cortex.

16 jection of oAbeta extracted directly from AD cerebral cortex.

17 l subtypes in both the mature and developing cerebral cortex.

18 listic microvascular networks from the mouse cerebral cortex.

19 neuron data from six distinct regions of the cerebral cortex.

20 e complex and heterogeneous structure of the cerebral cortex.

21 osite directions between the hippocampus and cerebral cortex.

22 expression of anti-oxidative enzymes in the cerebral cortex.

23 ied and well-understood parts of the primate cerebral cortex.

24 -related increase in aneuploidy in the mouse cerebral cortex.

25 t and distributed patterns of atrophy in the cerebral cortex.

26 nt amounts or types of Abeta deposits in the cerebral cortex.

27 nformation exchange between the thalamus and cerebral cortex.

28 ion in many brain areas, most notably in the cerebral cortex.

29 spine turnover becomes limited in the adult cerebral cortex.

30 itotic spindle orientation in the developing cerebral cortex.

31 nnectivity with the atrophied regions in the cerebral cortex.

32 s, and therefore absent in animals lacking a cerebral cortex.

33 age-dependent neurodegeneration in the aging cerebral cortex.

34 en excitatory and inhibitory synapses in the cerebral cortex.

35 anges in the state of population dynamics in cerebral cortex.

36 s (the striatum and cerebellum), but not the cerebral cortex.

37 for proteases expressed in the somatosensory cerebral cortex.

38 perior parietal lobes, and in the paramedian cerebral cortex.

39 odic, nonmotoric abnormalities involving the cerebral cortex.

40 al swollen morphology in the hippocampus and cerebral cortex.

41 pallidus and thalamus, and thickness of the cerebral cortex.

42 is limited to surface structures such as the cerebral cortex.

43 aling a polyclonal architecture of the human cerebral cortex.

44 fates is fundamental for the function of the cerebral cortex.

45 parietal, superior temporal, and paramidline cerebral cortex.

46 ivo photo-control of visual responses in the cerebral cortex.

47 Memories of experiences are stored in the cerebral cortex.

48 neuronal circuits during development of the cerebral cortex.

49 hension is unlikely to exist anywhere in the cerebral cortex.

50 onary expansion and elaboration of the human cerebral cortex.

51 derived interneurons to the striatum and the cerebral cortex.

52 but had no overt neuron degeneration in the cerebral cortex.

53 or cells, the transit amplifying cell of the cerebral cortex.

54 scriptional target of Ascl1 in the embryonic cerebral cortex.

55 imuli and behavior have been observed in the cerebral cortex.

56 pses, about 1 per microm(3) in the mammalian cerebral cortex.

57 the hippocampus and 22.1 +/- 4.9 muM in the cerebral cortex.

58 cture that sits between the striatum and the cerebral cortex.

59 ogenitors and the resultant formation of the cerebral cortex.

60 iate into functional neurons in the neonatal cerebral cortex.

61 ions in the basal ganglia, nucleus gyrus and cerebral cortex.

62 brillary lesions and neuritic plaques in the cerebral cortex.

63 ndritic spine density in the hippocampus and cerebral cortex.

64 ny developmental features with the mammalian cerebral cortex.

65 f P450arom, but also 17beta-estradiol in the cerebral cortex.

66 metric spatial neurovascular coupling in the cerebral cortex.

67 n against ischemia/reperfusion injury of the cerebral cortex.

68 detailing circuit mapping strategies in the cerebral cortex.

69 y generated, "immature" neurons of the adult cerebral cortex.

70 studies have shown widespread changes in the cerebral cortex.

71 in time at early developmental stages of the cerebral cortex.

72 2SH3- or CB2SH3+ in neurons of the adult rat cerebral cortex.

73 The claustrum is connected with the cerebral cortex.

74 is thought to depend on connections with the cerebral cortex.

75 city-related gene expression profiles in the cerebral cortex.

76 n of oxygenated hemoglobin ([oxy-Hb]) in the cerebral cortex.

77 er regions within and between right and left cerebral cortex.

78 tory lineages emerging in rostral and caudal cerebral cortex.

79 e coordination of neural networks throughout cerebral cortex.

80 ciousness in the Front or in the Back of the Cerebral Cortex?

81 ker immunotagging of nuclei from adult human cerebral cortex; 2) fluorescence-activated nuclei sortin

82 the basal extracellular concentration in rat cerebral cortex (4.3+/-1.5 microM).

83 paraventricular thalamic nucleus (64%), and cerebral cortex (63%).

84 ing an ectopic neuronal layer that resembles cerebral cortex abnormalities in humans.

85 a rare condition characterized by a reduced cerebral cortex accompanied with intellectual disability

86 ation directions between the hippocampus and cerebral cortex across wake and SWS.

87 h level of mmu-miR-455-3p (P = 0.004) in the cerebral cortex (AD-affected) region of brain and low in

88 ion induced by Abeta applied directly to the cerebral cortex, administered intravascularly, or overpr

89 nd to inhibit AC activity in mouse lungs and cerebral cortex after systemic administration.

90 h we previously showed to be elevated in the cerebral cortex Alzheimer's disease.

91 l the distinctive radial organization of the cerebral cortex and allow for the study of neuronal migr

92 brains from control animals, i.e. <1% in the cerebral cortex and approximately 0.1% in the cerebellum

93 Brain anomalies, including atrophy of the cerebral cortex and brainstem, and cerebellar aplasia we

94 sive connections with various regions of the cerebral cortex and by hypotheses surrounding its possib

95 ied with increases in apoptosis, whereas the cerebral cortex and cerebellum are unaffected.

96 tyrosine (3-NT) protein adducts, whereas the cerebral cortex and cerebellum did not respond at any ti

97 ency of aneuploidy of three autosomes in the cerebral cortex and cerebellum of adult and developing b

98 cleated neurons were observed throughout the cerebral cortex and cerebellum of an affected proband, e

99 differential gene expression analyses in the cerebral cortex and cerebellum of these mice identified

100 ebral hemispheres as a whole (right and left cerebral cortex and cerebral nuclei together) and their

101 essed in various brain regions including the cerebral cortex and hippocampus.

102 forms were also selectively decreased in the cerebral cortex and in the synaptic compartment of patie

103 uctural and functional organization of human cerebral cortex and its variation across individuals and

104 s, which may receive synaptic input from the cerebral cortex and other brain regions beyond the core

105 the cerebellum and abnormal processes in the cerebral cortex and pons.

106 ed by slow waves and spindles throughout the cerebral cortex and REM sleep by an "activated," low-vol

107 antibody-stained paraffin sections of mouse cerebral cortex and spinal cord, human postmortem brain,

108 ntified EZH2 in the cerebellum, NR3C1 in the cerebral cortex and SRF in the basal forebrain.

109 s from tissue to the synaptic compartment of cerebral cortex and striatum strongly supports our appro

110 Additionally, propagation between cerebral cortex and subcortical structures reverses dire

111 ma model of fast rhythmic oscillation in the cerebral cortex and suggest that spike synchrony and pha

112 diffusion MRI measurements of the developing cerebral cortex and support the possibility that, in gyr

113 d MT5-MMP are broadly expressed in the mouse cerebral cortex and that MT3-MMP loss-of-function interf

114 regions important for memory formation, the cerebral cortex and the CA1-hippocampal field.

115 rrent with increasing similarity between the cerebral cortex and the cerebellum, which points to pote

116 endogenous classes, including cells from the cerebral cortex and the retina.

117 ccompanied by diminished glial activation in cerebral cortex and white matter of insulin-treated FIV(

118 The cerebral cortex and white matter showed mild signal inte

119 ing demonstrated restricted diffusion in the cerebral cortex and white matter with corresponding low

120 had minimal Abeta plaque accumulation in the cerebral cortex and, thus, may show limited or no benefi

121 mAbs were measured by ELISA in fetal plasma, cerebral cortex, and cerebrospinal fluid, blood-brain ba

122 ing astrocytes from spinal cord, cerebellum, cerebral cortex, and hippocampus.

123 DYRK1A is strongly expressed in the cerebral cortex, and its overexpression leads to defecti

124 omical volumes, including total gray matter, cerebral cortex, and putamen.

125 The OE, the olfactory bulb (OB), the cerebral cortex, and the cerebellum were analyzed for ox

126 lood-brain barrier (BBB) function in the rat cerebral cortex, and, by using the discovered mechanism,

127 actors to predict connections of the primate cerebral cortex: architectonic similarity (structural mo

128 Ipsilateral motor areas of cerebral cortex are active during arm movements and even

129 Neurons in the cerebral cortex are constantly integrating different typ

130 xpansion and complexification of the primate cerebral cortex are largely linked to the emergence of t

131 Neurons in the cerebral cortex are of two major types: excitatory and i

132 STATEMENT: The functional complexity of the cerebral cortex arises from an array of distinct neurona

133 Using the mouse cerebral cortex as a model, Sox11 and Sox4 were examined

134 propagation of a wave of activity across the cerebral cortex as saccade planning and remapping procee

135 e information being processed throughout the cerebral cortex as well as maintain the modular structur

136 In addition to innervating the cerebral cortex, basal forebrain cholinergic (BFc) neuro

137 troduce a three-dimensional atlas of the cat cerebral cortex based on established cytoarchitectonic a

138 -/- embryonic fibroblasts and neurons in the cerebral cortex both show lipid droplet abnormalities.

139 rized from sequencing of mRNA and miRNA from cerebral cortex, brain stem, midbrain and cerebellum of

140 hus likely to reflect damage not only to the cerebral cortex but also to underlying axonal pathways,

141 art of the stem cell niche in the developing cerebral cortex, but their in vivo role in controlling t

142 rs are essential for early patterning of the cerebral cortex, but whether EMX1 mediates interhemisphe

143 xity and heterogeneity of neuromodulation of cerebral cortex by cholinergic stimulation, an area of a

144 o plasmid electroporation and into the mouse cerebral cortex by in vivo AAV injection.

145 ort that relief of hypoxia in the developing cerebral cortex by ingrowth of blood vessels temporo-spa

146 icient NSC differentiation in the developing cerebral cortex by providing oxygen and possibly regulat

147 f SVZ neural precursor cells in the prenatal cerebral cortex by testing for the presence and distribu

148 The (18)F(-)/(18)F-FDG uptake in the cerebral cortex, cerebellum, parotid grand, myocardium,

149 e genes that function downstream of MeCP2 in cerebral cortex circuitry, and identify upregulation of

150 of language is represented in regions of the cerebral cortex collectively known as the 'semantic syst

151 Circuits in the cerebral cortex consist of thousands of neurons connecte

152 The cerebral cortex contains "immature" neurons, which are g

153 The mature cerebral cortex contains a wide diversity of neuron phen

154 The cerebral cortex contains layers of neurons sequentially

155 The cerebral cortex continuously undergoes changes in its st

156 that, indeed, the extracellular impedance in cerebral cortex could be high and non-resistive, and we

157 , which share extensive connections with the cerebral cortex, could be selectively targeted by major

158 The human cerebral cortex depends for its normal development and s

159 s with Huntington disease whose striatum and cerebral cortex develop inclusions associated with exten

160 system development (P-value = 1.26E-3), and cerebral cortex development (P-value = 1.31E-2).

161 the m6A RNA mark in regulating the timing of cerebral cortex development in mouse and human.

162 ovel functional contributor to the mammalian cerebral cortex development, and that the pathological m

163 hat EURL is expressed during human and mouse cerebral cortex development, and we report that alterati

164 The human cerebral cortex develops through an elaborate succession

165 erized by ID and/or DD, malformations of the cerebral cortex, epilepsy, vision problems, musculoskele

166 expected, light activation of opsins in the cerebral cortex evoked robust, short-latency increases i

167 Significance statement: The cerebral cortex exerts higher brain functions including

168 cent evidence, stimulus-tuned neurons in the cerebral cortex exhibit reduced variability in firing ra

169 the basal ganglia, to frontal regions of the cerebral cortex (FC).

170 nvestigators have paid much attention to the cerebral cortex, few studies have detailed the basal gan

171 Why the cerebral cortex folds in some mammals but not in others

172 (NL3) or neuroligin 2 (NL2) in the adult rat cerebral cortex following in utero electroporation (IUEP

173 D), a wave of neuronal depolarization in the cerebral cortex following traumatic brain injury or cere

174 i and to compute the distance to the nearest cerebral cortex for 9626 contacts.

175 ecific posterior cortical areas in the human cerebral cortex for the selective retrieval of object an

176 stributed by the systemic circulation to the cerebral cortex (for amnesia and loss of consciousness)

177 As the cerebral cortex forms, specialized molecular cascades di

178 regarding the structural development of the cerebral cortex from childhood to adulthood, and provide

179 Here, we use RNA-seq in cerebral cortex from rats subjected to CR and LA-supplem

180 low and oxygenation disturbances that impair cerebral cortex growth and cause life-long cognitive and

181 , we showed that mouse Kif20b is involved in cerebral cortex growth and midbody organization of neura

182 bclasses of GABAergic cells (up to 15 in the cerebral cortex) has furthered the understanding of GABA

183 e demonstrate that two broad networks in the cerebral cortex have access to the adrenal medulla.

184 acroscopic anatomical characteristics of the cerebral cortex have been identified in individuals affe

185 lly connected with widespread regions of the cerebral cortex, hippocampus and amygdala.

186 WDR73 protein is expressed in human cerebral cortex, hippocampus, and cultured embryonic kid

187 Extracts from the whole brain and from the cerebral cortex, hippocampus, and olfactory bulbs exhibi

188 We measured the area of the cerebral cortex in a longitudinal sample of 974 individu

189 activity, in multielectrode recordings from cerebral cortex in anesthetized marmoset monkeys.

190 ventral surface of the medulla oblongata or cerebral cortex in brain slices of MeCP2-knockout and wi

191 an important role in the development of the cerebral cortex in humans and mice.

192 choline in mice regulates development of the cerebral cortex in the offspring.

193 l type-specific neuronal connectivity in the cerebral cortex in vivo has long been a challenge for ne

194 a-Pcdh-A1) or down (by gamma-Pcdh-C3) in the cerebral cortex in vivo, using conditional transgenic al

195 s that reflect those found in the developing cerebral cortex in vivo.

196 , i.e. the distance of each contact from the cerebral cortex, in order to discriminate between white

197 mation transfer across multiple nodes in the cerebral cortex, including visual, posterior parietal, a

198 We show that presynaptic activity in the cerebral cortex increases the persistence of glutamate i

199 The oxidation in the cerebral cortex induced by pentylenetetrazole was signif

200 Sensory areas of the cerebral cortex integrate the sensory inputs with the on

201 The mammalian cerebral cortex is a dense network composed of local, su

202 The ventricular zone (VZ) of the developing cerebral cortex is a pseudostratified epithelium that co

203 of reproducible folding in the gyrencephalic cerebral cortex is a topic of great interest to neurosci

204 Mammalian cerebral cortex is accepted as being critical for volunt

205 The cerebral cortex is built during embryonic neurogenesis,

206 The anatomy of cerebral cortex is characterized by two genetically inde

207 Development of the cerebral cortex is influenced by sensory experience duri

208 usion anisotropy within the developing fetal cerebral cortex is longitudinally characterized in the r

209 contrast, the number of inputs per neuron in cerebral cortex is more uniform and large.

210 The cerebral cortex is organized into specialized sensory ar

211 The cerebral cortex is plastic and represents the world acco

212 The mammalian cerebral cortex is responsible for the highest levels of

213 ether new neurons are added in the postnatal cerebral cortex is still debated.

214 The cerebral cortex is subdivided into six layers based on m

215 The cerebral cortex is the source of our most complex cognit

216 ent reorganisation of functional maps in the cerebral cortex is well described in the primary sensory

217 m, which receives dense innervation from the cerebral cortex, is believed to set the stage for the be

218 us system development, including that of the cerebral cortex, is the formation of higher-order neural

219 neurons in some brain regions, including the cerebral cortex, it is constitutively expressed.

220 rigins of the TDP-43 pathology reside in the cerebral cortex itself, secondarily in corticofugal fibr

221 o those of the fetal tissue to organize into cerebral cortex-like regions.

222 culture approach for generating a laminated cerebral cortex-like structure, named human cortical sph

223 During development of the cerebral cortex, local GABAergic interneurons recognize

224 suggests that the functional architecture of cerebral cortex may impose fundamental restrictions on t

225 Malformations of the cerebral cortex (MCCs) are devastating developmental dis

226 Neurons and networks in the cerebral cortex must operate reliably despite multiple s

227 t human pluripotent stem cell (hPSC)-derived cerebral cortex neurons form large-scale networks that r

228 Substantial expansion in the number of cerebral cortex neurons is thought to underlie cognitive

229 , neither the cellular organization of their cerebral cortex nor indices of structural neuronal plast

230 a lasting impact on the organization of the cerebral cortex observable decades later.

231 architecturally defined regions in the human cerebral cortex, occupying the most anterior part of the

232 ffuse and neuritic plaques were found in the cerebral cortex of 26-44 cases.

233 -weighted signal intensities, we studied the cerebral cortex of a large cohort of patients in early s

234 levels of aneuploidy during adulthood in the cerebral cortex of Bub1b(H/H) mice.

235 ased morphometry (VBM) to assess whether the cerebral cortex of captive chimpanzees that learned to v

236 BACE1 inhibition on dendritic spines in the cerebral cortex of constitutive and conditional Sez6 kno

237 ed during early postnatal development in the cerebral cortex of germ-line heterozygous Pten mutant mi

238 gene 1 (Wig1) is markedly upregulated in the cerebral cortex of HD patients.

239 The cerebral cortex of humans and macaques has specialized r

240 ical abnormalities in layer formation of the cerebral cortex of L1-deficient mice partially overlap w

241 roinflammatory factors was also found in the cerebral cortex of Malat1 KO mice after ischemic stroke

242 Heparan sulfate from the cerebral cortex of MPS IIIA mice showed enhanced ability

243 ed to identify molecular profiles across the cerebral cortex of postmortem human brains (n = 6).

244 T5A serotonin receptor were increased in the cerebral cortex of PrP(-/-), as compared with WT mice.

245 enin signaling is elevated in the developing cerebral cortex of Pten haploinsufficient mice, and a he

246 Here we use the developing cerebral cortex of Pten(+/-) mice to investigate the tra

247 ns within the networks of interest where the cerebral cortex of superagers was thicker than that of t

248 The cerebral cortex of the mouse has become one of the most

249 ingle-cell sequencing of 36 neurons from the cerebral cortex of three normal individuals.

250 frequency oscillations (LFOs) throughout the cerebral cortex of tottering (tg/tg) mice, a widely used

251 teristics similar to a phenotype seen in the cerebral cortex of Vldlr(null) mice.

252 s with the neuronal radial migration, in the cerebral cortex, of the neurons overexpressing NL3.

253 The Dp140 isoform was expressed in the cerebral cortex only in foetal life stages, while in the

254 ncrease in the area and neuron number of the cerebral cortex over evolutionary time systematically ch

255 nd cerebellar neurons are decreased and that cerebral cortex progressively thins in Aspa(Nur7/Nur7) m

256 In the cerebral cortex, prostaglandins are major contributors t

257 These include the pyramidal neurons in the cerebral cortex, Purkinje cells in the cerebellar cortex

258 In the developing cerebral cortex, radial glia progenitors (RGPs) generate

259 EEG delta activity through the activation of cerebral cortex, rather than to induce behavioral wakefu

260 nuclei relative to wild-type controls in the cerebral cortex, reaching a frequency as high as 40.3% f

261 nriched in supragranular layers of the human cerebral cortex relative to mouse distinguish major cort

262 velopment and structural organization of the cerebral cortex remain unknown.

263 llective population dynamics and function of cerebral cortex remains unknown.

264 strate that the high-power LFOs in the tg/tg cerebral cortex represent a highly abnormal excitability

265 Understanding the amazingly complex human cerebral cortex requires a map (or parcellation) of its

266 Development of the cerebral cortex requires regulation of proliferation and

267 Neurons in the cerebral cortex respond inconsistently to a repeated sen

268 re is a characteristic change in the way the cerebral cortex responds to perturbations, as can be ass

269 The cerebral cortex retains its fundamental organization, la

270 Electron microscopic analysis of the cerebral cortex revealed that oxytocin receptors were ma

271 evels were decreased in the putamen, but not cerebral cortex samples, of post-mortem Huntington's dis

272 In the developing cerebral cortex, sequential transcriptional programs tak

273 Variation in cerebral cortex size and complexity is thought to contri

274 nomously in culture, suggesting that primate cerebral cortex size is regulated at least in part at th

275 ensation as a key process ensuring mammalian cerebral cortex size.

276 In the developing cerebral cortex, spindle orientation defects result in s

277 s virus to identify the areas of the primate cerebral cortex that communicate through multisynaptic c

278 Of eleven isoforms in SCN and cerebral cortex that exhibit exon variation across two p

279 a structural brain abnormality involving the cerebral cortex that results from impaired neuronal migr

280 1) retrotransposition in the hippocampus and cerebral cortex that would have major implications for n

281 o increased vascular permeability in the rat cerebral cortex, through activation of NMDA receptors.

282 vides i) a preclinical basis for engineering cerebral cortex tissue autografts and ii) a biofidelic 3

283 connectivity projection map from the entire cerebral cortex to the dorsal striatum or caudoputamen (

284 r the mind, conceptually associated with the cerebral cortex, to influence autonomic and endocrine sy

285 data on the effects of diet and aging on the cerebral cortex transcriptome, and also emphasises the i

286 n Level Dependent (BOLD) fMRI signals in the cerebral cortex under normal physiological conditions ma

287 s relay sensory and motor information to the cerebral cortex using both single spikes and high-freque

288 unction and impairs lactate transport in the cerebral cortex using rat models of HE (bile duct ligati

289 Demyelination in the cerebral cortex was related to inflammatory infiltrates

290 To investigate its basis in the primate cerebral cortex, we measured single neuron responses to

291 contrast to the prenatal development of the cerebral cortex, when cell production, migration, and la

292 Notably, gene sets pertaining to the cerebral cortex (which did not differ in volume between

293 us has extensive connections with the entire cerebral cortex, which can also serve to integrate infor

294 c and hypoxia-induced lactate release in the cerebral cortex, which was normalized by OP treatment.

295 , it is suggested that a special area in the cerebral cortex with a relatively faster growth speed co

296 ion of the volume of activated tissue in the cerebral cortex with concurrent three-dimensional mappin

297 right time and in proper numbers to build a cerebral cortex with the appropriate size and structure?

298 owed closely the genetic organization of the cerebral cortex, with change rates varying as a function

299 hnology, the extreme complexity of the human cerebral cortex, with its approximately 10(14) synapses,

300 SCs were topically applied to the surface of cerebral cortex within 1 hour of experimental TBI.