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1 l death cascade in adult, but not embryonic, cerebral cortical neurons.
2 g transcriptional activation in cultured rat cerebral cortical neurons.
3 alpha-syn was also induced by VPA in rat cerebral cortical neurons.
4 2.3-Xq23 associated with arrest of migrating cerebral cortical neurons.
5 r, with systematically abnormal migration of cerebral cortical neurons.
6 tissue-type plasminogen activator (tPA) from cerebral cortical neurons.
7 port systems for glutamine are also found in cerebral cortical neurons, a predominantly GABAergic ner
9 oxygenase-2 (COX-2) is expressed in selected cerebral cortical neurons and is involved in synaptic si
10 her, Slit2, expressed in COS cells, repelled cerebral cortical neurons and olfactory interneuron prec
11 ay revealed that the choroid plexus repelled cerebral cortical neurons and olfactory interneuron prec
12 ha1 subunit-containing receptors in cultured cerebral cortical neurons and the role of protein kinase
13 lls of the testis, theca cells of the ovary, cerebral cortical neurons, and cerebellar Purkinje cells
14 on studies suggest that the laminar fates of cerebral cortical neurons are determined by environmenta
16 ol inhibited NMDA-gated currents in cultured cerebral cortical neurons at concentrations well below t
17 cycle, producing more early-born, deep-layer cerebral cortical neurons but depleting the cortical pro
18 (RGs) and serve as the direct precursors for cerebral cortical neurons, but factors that control thei
19 us system pathology appeared largely normal, cerebral cortical neurons cultured from neonatal Spg20-/
20 1 and A2a adenosine receptor agonists on rat cerebral cortical neurons discharges was examined using
21 granule cell neurons and macrophages but not cerebral cortical neurons, embryonic fibroblasts, or den
22 l data, primary cultures of TIMP-2 knock-out cerebral cortical neurons exhibit significantly reduced
23 ethod to direct viral vector transduction to cerebral cortical neurons expressing the neuregulin (NRG
24 VEGF also protected primary cultures of rat cerebral cortical neurons from hypoxia and glucose depri
25 R(+)-WIN 55212-2 also protected cultured cerebral cortical neurons from in vitro hypoxia and gluc
27 e properly can lead to an abnormal arrest of cerebral cortical neurons in proliferative zones near th
29 sible nuclear oxidative DNA damage occurs in cerebral cortical neurons in response to transient gluta
30 -dependent maintenance of GSH homeostasis in cerebral cortical neurons in the defense against oxidati
31 approximately half of these transgenic mice, cerebral cortical neurons in various cortical regions (p
33 osis of PC12 cells, sympathetic neurons, and cerebral cortical neurons is suppressed by the G1/S bloc
35 well as at axonal branch points in cultured cerebral cortical neurons, prefiguring a functional role
36 th recombinant tPA promotes cell survival in cerebral cortical neurons previously exposed to hypoxic
37 vely active in retinal neurons compared with cerebral cortical neurons suggesting that it is not a pa
41 eins and localizes to the ER in cultured rat cerebral cortical neurons, where it colocalizes with spa
42 3 and 4 and IGF1 are coexpressed by primate cerebral cortical neurons, where their expression is enh
43 impairment on the proteasome, we treated rat cerebral cortical neurons with oligomycin, antimycin, or
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