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1 din-1, claudin-5 or ZO-1 expression in ovine cerebral cortices.
2 ding through temporal, parietal, and frontal cerebral cortices.
3 to enable generation of larger, convoluted, cerebral cortices.
4 direct communication between left and right cerebral cortices and so, in this subject, a purely cort
5 anterior cingulate, and posterior cingulate cerebral cortices and the cerebellar cortex of 87 end-of
9 decreased neuronal death was observed in the cerebral cortices, brain stems, and cerebella of caspase
10 ses of lpa(1)((-/-)) lpa(2)((-/-)) embryonic cerebral cortices did not reveal obvious differences in
11 ly in the amygdala, entorhinal, and auditory cerebral cortices during the first REM sleep episodes af
12 nscriptome analysis of human and mouse fetal cerebral cortices exposed to ethanol in vitro and in viv
15 carried out electron microscopic analysis of cerebral cortices from fluoxetine-treated guinea pigs.
16 ositive astrocytes acutely isolated from the cerebral cortices of 4- to 12-day old rats were examined
17 gnificantly increased levels of VDAC1 in the cerebral cortices of 6-, 12- and 24-month-old APP transg
18 properties of Glu receptors (GluRs) from the cerebral cortices of AD and non-AD brains and found that
20 Connexin-32 abundance was higher (P<0.05) in cerebral cortices of fetuses at 60% of gestation (3.0+/-
21 re connexin-32 and connexin-43 expression in cerebral cortices of fetuses at 60%, 80%, and 90% of ges
22 connexin-43 abundance was higher (P<0.05) in cerebral cortices of fetuses at 90% of gestation (0.44+/
24 icant brain volume shrinkage occurred in the cerebral cortices of monkeys drinking >/= 3 g/kg ethanol
26 rimary astrocyte cultures were prepared from cerebral cortices of one-day-old Sprague-Dawley rats.
29 f IL6, CD11c, IL1beta, CD40 and CD11b in the cerebral cortices of the Tg2576 mice compared with their
30 upratentorial hypoperfusion of the bilateral cerebral cortices on the left side and severe left tempo
31 tor cells (NPCs) and neurons from developing cerebral cortices, revealing hundreds of differentially
32 ers of quantifiable GAD cells in the rostral cerebral cortices were different between groups, both ip
33 aly with partially collapsed skull; (2) thin cerebral cortices with subcortical calcifications; (3) m
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