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1 ts, compared with only 6.0 for those without cerebral emboli.
2 tients, compared with 17.9 for those without cerebral emboli.
3 s been suggested as a cause for asymptomatic cerebral emboli.
4 ata and possible late relapses mainly due to cerebral emboli.
5 ts without skin manifestations, particularly cerebral emboli (32.8% vs 18.4%, P = .01), without incre
6 ht to evaluate the incidence of asymptomatic cerebral emboli (ACE) during ablation of atrial fibrilla
8 al fibrillation is associated with a risk of cerebral emboli attributed to cardioversions and numerou
11 rgoing LV ablation experienced a total of 16 cerebral emboli, in comparison with zero patients underg
16 on in cognitive functioning in patients with cerebral emboli over 2 years, with a mean increase in sc
20 ns on damaged endothelial surface, and small cerebral emboli were detected in vivo by PET imaging.
21 so indicated faster decline in patients with cerebral emboli, with a mean increase in score of 12.0 i
22 ealed more rapid deterioration in those with cerebral emboli, with a mean increase in score of 59.0 i
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