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1 induced by controlled cortical impact on one cerebral hemisphere.
2 ctions of PV are with S2 and PV of the other cerebral hemisphere.
3 mm min(-1), respectively; n = 5) across the cerebral hemisphere.
4 f the laminar architecture of the developing cerebral hemisphere.
5 zed by epilepsy and degeneration of a single cerebral hemisphere.
6 at extend over nearly the full length of the cerebral hemisphere.
7 gion near the temporal pole of the mammalian cerebral hemisphere.
8 stinguish it from the medial boundary of the cerebral hemisphere.
9 activity of the right (language nondominant) cerebral hemisphere.
10 ning across the lateral surface of the right cerebral hemisphere.
11 zations can spread widely across the injured cerebral hemisphere.
12 y movable microelectrodes spanning an entire cerebral hemisphere.
13 died in a patient who was born with only one cerebral hemisphere.
14 r degeneration primarily affecting the right cerebral hemisphere.
15 r patients with lesions situated in the left cerebral hemisphere.
16 losum is the major commissure connecting the cerebral hemispheres.
17 ions between the ectoderm and the developing cerebral hemispheres.
18 in and incorporate into the neocortex of the cerebral hemispheres.
19 onal transfer of information between the two cerebral hemispheres.
20 nvolved receive their motor output from both cerebral hemispheres.
21 in, and biological features of, normal human cerebral hemispheres.
22 fiber tract that connects the left and right cerebral hemispheres.
23 and astrogliosis in dorsal striatum in both cerebral hemispheres.
24 predominantly in the posterior region of the cerebral hemispheres.
25 n the mammalian brain, linking the bilateral cerebral hemispheres.
26 ural and functional connectivity between the cerebral hemispheres.
27 rved postmortem chimpanzee (Pan troglodytes) cerebral hemispheres.
28 rized by overgrowth of either one of the two cerebral hemispheres.
29 of the leptomeninges over large areas of the cerebral hemispheres.
30 olves incomplete or failed separation of the cerebral hemispheres.
31 acterized by an incomplete separation of the cerebral hemispheres.
32 n of axonal projections that connect the two cerebral hemispheres.
33 cerebral arteries serving the left and right cerebral hemispheres.
34 non-motor cortical regions within and across cerebral hemispheres.
35 needed for the expansion and bifurcation of cerebral hemispheres.
36 hic affecting the spine, cranial nerves, and cerebral hemispheres.
37 ined as the incomplete separation of the two cerebral hemispheres.
38 e neuroendocrine system from any part of the cerebral hemispheres.
39 oss after seizures and (b) with a more focal cerebral hemisphere abnormality usually due to vascular
43 most of the external morphology of the right cerebral hemisphere and a fragment of fossilized face th
44 bral nuclei form the ventral division of the cerebral hemisphere and are thought to play an important
46 cessing was strongly lateralized to the left cerebral hemisphere and involved a network of regions in
47 ade to demonstrate the medial surface of the cerebral hemisphere and the medial wall of the lateral v
48 omotopic and other locations in the opposite cerebral hemisphere and to a surrounding array of eight
49 g on the neurons and neuroglial cells in the cerebral hemispheres and associated fiber tracts of the
51 fferent and afferent connections between the cerebral hemispheres and between the cortex and thalamus
52 ificant age-related differences in volume of cerebral hemispheres and caudate nuclei (controls, but n
53 ray of cerebral cortical layering, fusion of cerebral hemispheres and cerebellar folia, and aberrant
56 s; in mice lacking Foxg1, development of the cerebral hemispheres and olfactory epithelium (OE) is se
57 ereby limits neuronal trafficking across the cerebral hemispheres and reduces input to cortical inhib
58 is a reduction in white matter volume in the cerebral hemispheres and structural abnormalities in mye
59 rectomy (i.e. surgical removal of one entire cerebral hemisphere) and 10 age- and sex-matched control
60 l cells (sensory ganglia and outer aspect of cerebral hemispheres) and glial cells (radial glia, sate
61 izures, progressive degeneration of a single cerebral hemisphere, and autoimmunity directed against g
62 ch-motor regions of the non-dominant (right) cerebral hemisphere, and extend this theory to include t
63 t of marginal zone components, fusion of the cerebral hemispheres, and scalloping of the dentate gyru
64 fibre tract in the brain, connecting the two cerebral hemispheres, and thereby facilitating the integ
66 s originating from the contralesional intact cerebral hemisphere are capable of sprouting into the de
68 the rest of the brain (and, conversely, the cerebral hemispheres are smaller with respect to the cer
71 amining the connectional organization of the cerebral hemispheres as a whole (right and left cerebral
72 n the perisylvian area of the left and right cerebral hemispheres, as well as in the frontal and occi
73 developed marked infarcts in the ipsilateral cerebral hemisphere at 24 hr and showed significant loss
75 ight ratios of ipsilateral and contralateral cerebral hemispheres at 12 hrs, 24 hrs, or 2, 3, or 7 da
77 re consistent with the known predominance of cerebral hemisphere axonal injury in cardiac arrest and
78 usion, fibrin accumulates in the ipsilateral cerebral hemisphere, based upon immunoblotting, and loca
79 hyperintensities in the white matter of the cerebral hemispheres, brainstem, cerebellum, and cervica
80 ntral nervous system (CNS) that were high in cerebral hemispheres but progressively decreased toward
81 ive of edema in the posterior regions of the cerebral hemispheres, but the changes often involved oth
83 sed by about 50% the weight loss of the left cerebral hemisphere caused by hypoxia-ischemia in neonat
84 Brain structures investigated included the cerebral hemispheres, cerebral cortex, diencephalon, cau
86 Nearly every tissue section from bilateral cerebral hemispheres contained either macroscopic or mic
89 of 50 fetuses, and the medial surface of the cerebral hemisphere could be traced posteriorly around t
91 the corpus callosum, cortical axons from one cerebral hemisphere cross the midline to reach their tar
92 that resulted in holoprosencephaly (a single cerebral hemisphere), cyclopia (a single midline eye), a
96 coarse coding hypothesis postulates that the cerebral hemispheres differ in their breadth of semantic
98 omatic mutations that affect each developing cerebral hemisphere differently with more neurons than e
99 onous activation of cortical loci in the two cerebral hemispheres during development leads to the sta
100 ient and symmetrically in the cortex of both cerebral hemispheres except for precentral gyri in the s
102 human forebrain, and in its severe form, the cerebral hemispheres fail to completely separate into tw
103 of the human forebrain and midface where the cerebral hemispheres fail to separate into distinct left
104 itudinal changes in network activity in each cerebral hemisphere, focusing specifically on the "presy
105 ls of N434A, but not H435A, decreased in the cerebral hemispheres following bilateral injection into
106 h with defects in neurulation, fusion of the cerebral hemispheres, formation of the great forebrain c
108 A recent study of a child born with one cerebral hemisphere has revealed an extreme developmenta
110 demonstrated along the medial surface of the cerebral hemisphere in 36 (72%) of 50 fetuses, and the m
114 Understanding the role of the unaffected cerebral hemisphere in the motor recovery process has be
117 In addition, defects in development of the cerebral hemispheres in Foxg1(-/-) mice are not rescued
119 us callosum (CC) connects the left and right cerebral hemispheres in mammals and its development requ
120 size and anatomical connectivity across the cerebral hemispheres in persons with Tourette's disorder
121 genetic determination of the left and right cerebral hemispheres in the nonright-handed twin pairs i
122 nferior frontal and temporal regions of both cerebral hemispheres in the titled and untitled conditio
129 either the unmyelinated axon of the immature cerebral hemispheres is relatively resistant to traumati
130 ted with progressive destruction of a single cerebral hemisphere, is an autoimmune disease in which o
133 ends in part on multiple regions of the left cerebral hemisphere, located outside the classic languag
134 within the distributed motor system in both cerebral hemispheres, more so in patients with greater i
135 Tumor locations were diencephalon (n = 58), cerebral hemisphere (n = 3), and cerebellum (n = 17).
139 r, were transplanted into the spinal cord or cerebral hemisphere of immunosuppressed normal and myeli
141 surface area and volume of V1 in one or both cerebral hemispheres of 15 neurologically normal human b
144 metry of venous drainage in the pathological cerebral hemisphere on CT and MRI dynamic angiography.
145 te a differential role of the left and right cerebral hemispheres on immune functions in humans.
146 gliomas, we injected hMSCs directly into the cerebral hemisphere opposite an established human glioma
147 ted changes were greater in magnitude in the cerebral hemisphere opposite the more clinically affecte
148 iencephalon, but also in the olfactory bulbs/cerebral hemispheres, optic tectum/tegmentum, retina, an
150 occipital-temporal scalp areas of the right cerebral hemisphere regardless of the visual field of st
151 rtals and Cro-Magnon 1, who have the largest cerebral hemispheres relative to cerebellum volume of an
152 e hypothesis that asymmetric routing between cerebral hemispheres represents an important mechanism f
154 ephalon failed to bulge or separate into two cerebral hemispheres, resulting in holoprosencephaly.
155 typically bilaterally distributed across the cerebral hemispheres, show similarity to known white mat
156 ured cerebral volume on four contiguous 5 mm cerebral hemisphere slices at each time point, using an
157 o cortical sulci over the convexities of the cerebral hemispheres, sparing the brainstem, cerebellum
158 elencephalon, the embryonic precursor of the cerebral hemispheres, specialized cell types form a midl
161 ducted a cytoarchitectonic analysis by using cerebral hemispheres that were cut at oblique angles and
162 The microprobes were inserted through the cerebral hemisphere, the superior colliculus and the mid
163 The larger units of analysis, including the cerebral hemispheres, the major grey and white matter st
164 aschitic hemisphere-that is, the ipsilateral cerebral hemisphere (THGr(Ce)) and the contralateral cer
165 notypic features such as failure of anterior cerebral hemisphere to divide, hydrocephalus and cleft p
166 ntly better for patients with cerebellar and cerebral hemisphere tumors (n = 75) than those with tumo
167 ction (GTR) was attempted for cerebellar and cerebral hemisphere tumors, with biopsy or less aggressi
168 rder characterized by an enlarged, malformed cerebral hemisphere, typically causing epilepsy that req
169 ose of this study was to examine how the two cerebral hemispheres use perceptual information about wo
171 ns that connect homotopic regions of the two cerebral hemispheres via the corpus callosum and that ar
172 allosal projection neurons (CPN) connect the cerebral hemispheres via the corpus callosum, integratin
174 eter measured with the medial surface of the cerebral hemisphere was 10.7 mm, compared with the true
175 male cats anaesthetized with chloralose, one cerebral hemisphere was exposed and the middle cerebral
176 rotid ligation, perfusion of the ipsilateral cerebral hemisphere was markedly diminished, suggesting
177 r each case, the entire hippocampus from one cerebral hemisphere was sliced into 5mm slabs (5-7 slabs
178 During mammalian evolution, expansion of the cerebral hemispheres was accompanied by expansion of the
179 metries of input from the two eyes into each cerebral hemisphere, we measured ocular dominance column
181 nes and early members of the genus Homo, the cerebral hemispheres were large in proportion to the cer
183 nce between blood flow in the right and left cerebral hemispheres when autoregulation was impaired in
184 f the BF-1 gene results in hypoplasia of the cerebral hemispheres, which is more severe in structures
185 wavefronts selectively over the ipsilateral cerebral hemispheres with a velocity of 3.8 +/- 0.70 mm
186 zation are present in the left vs. the right cerebral hemisphere, with the left hemisphere showing a
187 n brain is composed of two broadly symmetric cerebral hemispheres, with an abundance of reciprocal an
188 was hypothesized that disconnection between cerebral hemispheres would disrupt mental simulation of
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