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1 induced by controlled cortical impact on one cerebral hemisphere.
2 ctions of PV are with S2 and PV of the other cerebral hemisphere.
3  mm min(-1), respectively; n = 5) across the cerebral hemisphere.
4 f the laminar architecture of the developing cerebral hemisphere.
5 zed by epilepsy and degeneration of a single cerebral hemisphere.
6 at extend over nearly the full length of the cerebral hemisphere.
7 gion near the temporal pole of the mammalian cerebral hemisphere.
8 stinguish it from the medial boundary of the cerebral hemisphere.
9 activity of the right (language nondominant) cerebral hemisphere.
10 ning across the lateral surface of the right cerebral hemisphere.
11 zations can spread widely across the injured cerebral hemisphere.
12 y movable microelectrodes spanning an entire cerebral hemisphere.
13 died in a patient who was born with only one cerebral hemisphere.
14 r degeneration primarily affecting the right cerebral hemisphere.
15 r patients with lesions situated in the left cerebral hemisphere.
16 losum is the major commissure connecting the cerebral hemispheres.
17 ions between the ectoderm and the developing cerebral hemispheres.
18 in and incorporate into the neocortex of the cerebral hemispheres.
19 onal transfer of information between the two cerebral hemispheres.
20 nvolved receive their motor output from both cerebral hemispheres.
21 in, and biological features of, normal human cerebral hemispheres.
22 fiber tract that connects the left and right cerebral hemispheres.
23  and astrogliosis in dorsal striatum in both cerebral hemispheres.
24 predominantly in the posterior region of the cerebral hemispheres.
25 n the mammalian brain, linking the bilateral cerebral hemispheres.
26 ural and functional connectivity between the cerebral hemispheres.
27 rved postmortem chimpanzee (Pan troglodytes) cerebral hemispheres.
28 rized by overgrowth of either one of the two cerebral hemispheres.
29 of the leptomeninges over large areas of the cerebral hemispheres.
30 olves incomplete or failed separation of the cerebral hemispheres.
31 acterized by an incomplete separation of the cerebral hemispheres.
32 n of axonal projections that connect the two cerebral hemispheres.
33 cerebral arteries serving the left and right cerebral hemispheres.
34 non-motor cortical regions within and across cerebral hemispheres.
35  needed for the expansion and bifurcation of cerebral hemispheres.
36 hic affecting the spine, cranial nerves, and cerebral hemispheres.
37 ined as the incomplete separation of the two cerebral hemispheres.
38 e neuroendocrine system from any part of the cerebral hemispheres.
39 oss after seizures and (b) with a more focal cerebral hemisphere abnormality usually due to vascular
40                   Does complete removal of a cerebral hemisphere affect either of these processes?
41  matter, in terms of tissue volume, from the cerebral hemispheres after traumatic brain injury.
42 trol was found in language areas of the left cerebral hemisphere along the Sylvian fissure.
43 most of the external morphology of the right cerebral hemisphere and a fragment of fossilized face th
44 bral nuclei form the ventral division of the cerebral hemisphere and are thought to play an important
45                    Innervated regions in the cerebral hemisphere and brainstem fall into nine general
46 cessing was strongly lateralized to the left cerebral hemisphere and involved a network of regions in
47 ade to demonstrate the medial surface of the cerebral hemisphere and the medial wall of the lateral v
48 omotopic and other locations in the opposite cerebral hemisphere and to a surrounding array of eight
49 g on the neurons and neuroglial cells in the cerebral hemispheres and associated fiber tracts of the
50 imals exhibit a reduction in the size of the cerebral hemispheres and associated structures.
51 fferent and afferent connections between the cerebral hemispheres and between the cortex and thalamus
52 ificant age-related differences in volume of cerebral hemispheres and caudate nuclei (controls, but n
53 ray of cerebral cortical layering, fusion of cerebral hemispheres and cerebellar folia, and aberrant
54  sensory-motor functions, and suprasegmental cerebral hemispheres and cerebellum.
55                 The corpus callosum connects cerebral hemispheres and is the largest axon tract in th
56 s; in mice lacking Foxg1, development of the cerebral hemispheres and olfactory epithelium (OE) is se
57 ereby limits neuronal trafficking across the cerebral hemispheres and reduces input to cortical inhib
58 is a reduction in white matter volume in the cerebral hemispheres and structural abnormalities in mye
59 rectomy (i.e. surgical removal of one entire cerebral hemisphere) and 10 age- and sex-matched control
60 l cells (sensory ganglia and outer aspect of cerebral hemispheres) and glial cells (radial glia, sate
61 izures, progressive degeneration of a single cerebral hemisphere, and autoimmunity directed against g
62 ch-motor regions of the non-dominant (right) cerebral hemisphere, and extend this theory to include t
63 t of marginal zone components, fusion of the cerebral hemispheres, and scalloping of the dentate gyru
64 fibre tract in the brain, connecting the two cerebral hemispheres, and thereby facilitating the integ
65                           The cerebellum and cerebral hemispheres appear to have evolved reciprocally
66 s originating from the contralesional intact cerebral hemisphere are capable of sprouting into the de
67                     The human left and right cerebral hemispheres are anatomically and functionally a
68  the rest of the brain (and, conversely, the cerebral hemispheres are smaller with respect to the cer
69                                      The two cerebral hemispheres are specialised for different cogni
70            Diffuse astrocytomas of the adult cerebral hemispheres are unique among tumours in human b
71 amining the connectional organization of the cerebral hemispheres as a whole (right and left cerebral
72 n the perisylvian area of the left and right cerebral hemispheres, as well as in the frontal and occi
73 developed marked infarcts in the ipsilateral cerebral hemisphere at 24 hr and showed significant loss
74 by sleeping with only one eye closed and one cerebral hemisphere at a time.
75 ight ratios of ipsilateral and contralateral cerebral hemispheres at 12 hrs, 24 hrs, or 2, 3, or 7 da
76 ight ratios of ipsilateral and contralateral cerebral hemispheres at 48 hrs postischemia.
77 re consistent with the known predominance of cerebral hemisphere axonal injury in cardiac arrest and
78 usion, fibrin accumulates in the ipsilateral cerebral hemisphere, based upon immunoblotting, and loca
79  hyperintensities in the white matter of the cerebral hemispheres, brainstem, cerebellum, and cervica
80 ntral nervous system (CNS) that were high in cerebral hemispheres but progressively decreased toward
81 ive of edema in the posterior regions of the cerebral hemispheres, but the changes often involved oth
82                      These data suggest both cerebral hemispheres can benefit from supportive sentenc
83 sed by about 50% the weight loss of the left cerebral hemisphere caused by hypoxia-ischemia in neonat
84   Brain structures investigated included the cerebral hemispheres, cerebral cortex, diencephalon, cau
85                                 Maps in each cerebral hemisphere code information from the opposite s
86   Nearly every tissue section from bilateral cerebral hemispheres contained either macroscopic or mic
87 e stimulus and that may be controlled by the cerebral hemisphere contralateral to the limb.
88                   Virtually all parts of the cerebral hemispheres contribute to a triple descending p
89 of 50 fetuses, and the medial surface of the cerebral hemisphere could be traced posteriorly around t
90            The surgical disconnection of the cerebral hemispheres creates an extraordinary opportunit
91 the corpus callosum, cortical axons from one cerebral hemisphere cross the midline to reach their tar
92 that resulted in holoprosencephaly (a single cerebral hemisphere), cyclopia (a single midline eye), a
93        In ECMO animals, cerebral blood flow (cerebral hemispheres) decreased from a baseline measurem
94 brain, or it can alternate over time between cerebral hemispheres, depending on the species.
95            Although the left and right human cerebral hemispheres differ both functionally and anatom
96 coarse coding hypothesis postulates that the cerebral hemispheres differ in their breadth of semantic
97            Schizophrenia may involve the two cerebral hemispheres differentially.
98 omatic mutations that affect each developing cerebral hemisphere differently with more neurons than e
99 onous activation of cortical loci in the two cerebral hemispheres during development leads to the sta
100 ient and symmetrically in the cortex of both cerebral hemispheres except for precentral gyri in the s
101                                       As the cerebral hemispheres expand, they extend caudally, passi
102 human forebrain, and in its severe form, the cerebral hemispheres fail to completely separate into tw
103 of the human forebrain and midface where the cerebral hemispheres fail to separate into distinct left
104 itudinal changes in network activity in each cerebral hemisphere, focusing specifically on the "presy
105 ls of N434A, but not H435A, decreased in the cerebral hemispheres following bilateral injection into
106 h with defects in neurulation, fusion of the cerebral hemispheres, formation of the great forebrain c
107                    And yet, even though each cerebral hemisphere has its own set of capacities, with
108      A recent study of a child born with one cerebral hemisphere has revealed an extreme developmenta
109                                   Within the cerebral hemispheres, hsp110 is present in neurons in al
110 demonstrated along the medial surface of the cerebral hemisphere in 36 (72%) of 50 fetuses, and the m
111 processing in the left compared to the right cerebral hemisphere in nondeficit schizophrenia.
112 ning across the lateral surface of the right cerebral hemisphere in persons at high risk.
113 n arises from the dominant role of the right cerebral hemisphere in regulating attention.
114     Understanding the role of the unaffected cerebral hemisphere in the motor recovery process has be
115                Imaging of the surface of the cerebral hemispheres in both explant cultures and brains
116 ions ultimately disrupt the formation of the cerebral hemispheres in dorsal regions.
117   In addition, defects in development of the cerebral hemispheres in Foxg1(-/-) mice are not rescued
118 cle that discussed the separate roles of the cerebral hemispheres in humans.
119 us callosum (CC) connects the left and right cerebral hemispheres in mammals and its development requ
120  size and anatomical connectivity across the cerebral hemispheres in persons with Tourette's disorder
121  genetic determination of the left and right cerebral hemispheres in the nonright-handed twin pairs i
122 nferior frontal and temporal regions of both cerebral hemispheres in the titled and untitled conditio
123                                         Each cerebral hemisphere initially processes one half of the
124 d to provide a model for the organization of cerebral hemisphere inputs to this network.
125                 Since the medial wall of the cerebral hemisphere is commonly spared following stroke,
126                                     The left cerebral hemisphere is dominant for language, and many a
127             For instance, in humans the left cerebral hemisphere is largely responsible for language
128               Because the medial wall of the cerebral hemisphere is often spared following brain inju
129 either the unmyelinated axon of the immature cerebral hemispheres is relatively resistant to traumati
130 ted with progressive destruction of a single cerebral hemisphere, is an autoimmune disease in which o
131                                              Cerebral hemisphere lambda1 was lower in cardiac arrest
132          Following intra-nasal delivery, low cerebral hemisphere levels of variants were obtained at
133 ends in part on multiple regions of the left cerebral hemisphere, located outside the classic languag
134  within the distributed motor system in both cerebral hemispheres, more so in patients with greater i
135  Tumor locations were diencephalon (n = 58), cerebral hemisphere (n = 3), and cerebellum (n = 17).
136              Understanding the principles of cerebral hemisphere neural network organization is essen
137 s, SD 14.7) with 156 paired watershed vessel/cerebral hemisphere observations.
138 re stereotactically implanted into the right cerebral hemisphere of Fischer rats.
139 r, were transplanted into the spinal cord or cerebral hemisphere of immunosuppressed normal and myeli
140 ulated in ischemic region rather than normal cerebral hemisphere of MCAO rats.
141 surface area and volume of V1 in one or both cerebral hemispheres of 15 neurologically normal human b
142 a pleiotropic role in the development of the cerebral hemispheres of the brain.
143                                              Cerebral hemispheres of the offspring were examined at t
144 metry of venous drainage in the pathological cerebral hemisphere on CT and MRI dynamic angiography.
145 te a differential role of the left and right cerebral hemispheres on immune functions in humans.
146 gliomas, we injected hMSCs directly into the cerebral hemisphere opposite an established human glioma
147 ted changes were greater in magnitude in the cerebral hemisphere opposite the more clinically affecte
148 iencephalon, but also in the olfactory bulbs/cerebral hemispheres, optic tectum/tegmentum, retina, an
149                         Virtually all of the cerebral hemisphere projects on the upper brainstem beha
150  occipital-temporal scalp areas of the right cerebral hemisphere regardless of the visual field of st
151 rtals and Cro-Magnon 1, who have the largest cerebral hemispheres relative to cerebellum volume of an
152 e hypothesis that asymmetric routing between cerebral hemispheres represents an important mechanism f
153             VAF and caudal SRAF in the right cerebral hemisphere responded maximally to midline horiz
154 ephalon failed to bulge or separate into two cerebral hemispheres, resulting in holoprosencephaly.
155 typically bilaterally distributed across the cerebral hemispheres, show similarity to known white mat
156 ured cerebral volume on four contiguous 5 mm cerebral hemisphere slices at each time point, using an
157 o cortical sulci over the convexities of the cerebral hemispheres, sparing the brainstem, cerebellum
158 elencephalon, the embryonic precursor of the cerebral hemispheres, specialized cell types form a midl
159 tion, the development of the patient's right cerebral hemisphere terminated.
160         They also learn better with the left cerebral hemisphere than with the right hemisphere.
161 ducted a cytoarchitectonic analysis by using cerebral hemispheres that were cut at oblique angles and
162    The microprobes were inserted through the cerebral hemisphere, the superior colliculus and the mid
163  The larger units of analysis, including the cerebral hemispheres, the major grey and white matter st
164 aschitic hemisphere-that is, the ipsilateral cerebral hemisphere (THGr(Ce)) and the contralateral cer
165 notypic features such as failure of anterior cerebral hemisphere to divide, hydrocephalus and cleft p
166 ntly better for patients with cerebellar and cerebral hemisphere tumors (n = 75) than those with tumo
167 ction (GTR) was attempted for cerebellar and cerebral hemisphere tumors, with biopsy or less aggressi
168 rder characterized by an enlarged, malformed cerebral hemisphere, typically causing epilepsy that req
169 ose of this study was to examine how the two cerebral hemispheres use perceptual information about wo
170 n neurons (CPN), whose axons connect the two cerebral hemispheres via the corpus callosum (CC).
171 ns that connect homotopic regions of the two cerebral hemispheres via the corpus callosum and that ar
172 allosal projection neurons (CPN) connect the cerebral hemispheres via the corpus callosum, integratin
173       MRI measured affected and non-affected cerebral hemisphere volumes for HME and non-HME cases, i
174 eter measured with the medial surface of the cerebral hemisphere was 10.7 mm, compared with the true
175 male cats anaesthetized with chloralose, one cerebral hemisphere was exposed and the middle cerebral
176 rotid ligation, perfusion of the ipsilateral cerebral hemisphere was markedly diminished, suggesting
177 r each case, the entire hippocampus from one cerebral hemisphere was sliced into 5mm slabs (5-7 slabs
178 During mammalian evolution, expansion of the cerebral hemispheres was accompanied by expansion of the
179 metries of input from the two eyes into each cerebral hemisphere, we measured ocular dominance column
180                                         Both cerebral hemispheres were actively involved in speech pr
181 nes and early members of the genus Homo, the cerebral hemispheres were large in proportion to the cer
182                                          The cerebral hemispheres were segmented semi-automatically,
183 nce between blood flow in the right and left cerebral hemispheres when autoregulation was impaired in
184 f the BF-1 gene results in hypoplasia of the cerebral hemispheres, which is more severe in structures
185  wavefronts selectively over the ipsilateral cerebral hemispheres with a velocity of 3.8 +/- 0.70 mm
186 zation are present in the left vs. the right cerebral hemisphere, with the left hemisphere showing a
187 n brain is composed of two broadly symmetric cerebral hemispheres, with an abundance of reciprocal an
188  was hypothesized that disconnection between cerebral hemispheres would disrupt mental simulation of

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