ライフサイエンスコーパス: cerebral hypoxia

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1 s beneficial in other conditions with global cerebral hypoxia.

2 Cerebral hypoxia and ischemia may stimulate neurogenesis

3 Cerebral hypoxia and ischemia trigger endogenous protect

4 ted lactate/glucose ratio were observed with cerebral hypoxia and ischemia.

5 mportance lies in the likely exacerbation of cerebral hypoxia and the contribution of such seizure ac

6 dicates ongoing cell injury following severe cerebral hypoxia, and that recovery of oxidative phospho

7 zed rabbits were first subjected to 8 min of cerebral hypoxia by breathing 3% oxygen and then to 8 mi

8 The mechanism of cerebral hypoxia-induced myoclonic jerks is not known.

9 f memantine for 7 days significantly reduced cerebral hypoxia-induced neurodegeneration in the CA1 of

10 Cerebral hypoxia is a major component of immediate and s

11 xpressed in hypoxic brain regions of a mouse cerebral hypoxia-ischaemia (H/I) model.

12 ls of induced hypothermia as treatment after cerebral hypoxia-ischaemia in term newborn infants.

13 ic db/db mice suffered less damage following cerebral hypoxia-ischemia (H/I) than male db/db mice.

14 tex displays impaired growth after transient cerebral hypoxia-ischemia (HI) at preterm gestation that

15 hether whole body hypothermia after neonatal cerebral hypoxia-ischemia (HI) could broaden the therape

16 ive was to determine the effects of neonatal cerebral hypoxia-ischemia (HI) on ODP.

17 We have previously shown that cerebral Hypoxia-ischemia (HI) results in activation of

18 cerebrovascular diseases, including neonatal cerebral hypoxia-ischemia (HI).

19 rmal lung function, after induction of acute cerebral hypoxia-ischemia by occlusion of the brachiocep

20 Cerebral hypoxia-ischemia caused death of O4+ late OPCs

21 Cerebral hypoxia-ischemia causes encephalopathy and neur

22 s study tested the hypotheses that (1) acute cerebral hypoxia-ischemia changes laryngeal adductor, la

23 Our understanding of events associated with cerebral hypoxia-ischemia during cardiopulmonary bypass

24 Moreover, cerebral hypoxia-ischemia impaired neurobehavioral perfo

25 with therapeutic hypothermia after transient cerebral hypoxia-ischemia in a piglet model of perinatal

26 nd improved neurological functions following cerebral hypoxia-ischemia in the neonatal rat.

27 campal neurons of neonatal rats subjected to cerebral hypoxia-ischemia in vivo.

28 Bromodeoxyuridine uptake assay showed that cerebral hypoxia-ischemia inhibited proliferation of ste

29 dendrocyte progenitor cells (OPCs) following cerebral hypoxia-ischemia, a previously developed neonat

30 In various animal models of cerebral hypoxia-ischemia, it is not clear whether neuro

31 ther preterm birth asphyxia or induced acute cerebral hypoxia-ischemia, minute ventilation initially

32 neration by XO as a major cause of damage in cerebral hypoxia-ischemia.

33 Cs and improved neurological functions after cerebral hypoxia-ischemia.

34 intracerebroventricular injection 3 h after cerebral hypoxia-ischemia.

35 n damage in those animals subjected to prior cerebral hypoxia-ischemia.

36 t size in 7-day-old rat pups with an in vivo cerebral hypoxia/ischemia model.

37 During cerebral hypoxia or ischaemia, mitochondrial dysfunction

38 Cerebral hypoxia was induced by lowering the FiO2 (0.05-

39 ns such as amyotrophic lateral sclerosis and cerebral hypoxia, which are associated with neuronal GLT

40 ular syncope, with abnormal movements due to cerebral hypoxia, which may be difficult to differentiat

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