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1 Cs and improved neurological functions after cerebral hypoxia-ischemia.
2 intracerebroventricular injection 3 h after cerebral hypoxia-ischemia.
3 n damage in those animals subjected to prior cerebral hypoxia-ischemia.
4 neration by XO as a major cause of damage in cerebral hypoxia-ischemia.
5 dendrocyte progenitor cells (OPCs) following cerebral hypoxia-ischemia, a previously developed neonat
6 rmal lung function, after induction of acute cerebral hypoxia-ischemia by occlusion of the brachiocep
9 s study tested the hypotheses that (1) acute cerebral hypoxia-ischemia changes laryngeal adductor, la
10 Our understanding of events associated with cerebral hypoxia-ischemia during cardiopulmonary bypass
11 ic db/db mice suffered less damage following cerebral hypoxia-ischemia (H/I) than male db/db mice.
12 tex displays impaired growth after transient cerebral hypoxia-ischemia (HI) at preterm gestation that
13 hether whole body hypothermia after neonatal cerebral hypoxia-ischemia (HI) could broaden the therape
18 with therapeutic hypothermia after transient cerebral hypoxia-ischemia in a piglet model of perinatal
21 Bromodeoxyuridine uptake assay showed that cerebral hypoxia-ischemia inhibited proliferation of ste
23 ther preterm birth asphyxia or induced acute cerebral hypoxia-ischemia, minute ventilation initially
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